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1
Identity, structure, and function of the mitochondrial permeability transition pore: controversies, consensus, recent advances, and future directions
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Identity, structure, and function of the mitochondrial permeability transition pore: controversies, consensus, recent advances, and future directions

Cell death and differentiation, 2023-08, Vol.30 (8), p.1869-1885 [Peer Reviewed Journal]

2023. The Author(s). ;The Author(s) 2023. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2023 ;ISSN: 1350-9047 ;EISSN: 1476-5403 ;DOI: 10.1038/s41418-023-01187-0 ;PMID: 37460667

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2
The RISK pathway leading to mitochondria and cardioprotection: how everything started
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The RISK pathway leading to mitochondria and cardioprotection: how everything started

Basic research in cardiology, 2023-05, Vol.118 (1), p.22-22, Article 22 [Peer Reviewed Journal]

The Author(s) 2023 ;2023. The Author(s). ;The Author(s) 2023. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;ISSN: 1435-1803 ;ISSN: 0300-8428 ;EISSN: 1435-1803 ;DOI: 10.1007/s00395-023-00992-5 ;PMID: 37233787

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3
Recent progress in the use of mitochondrial membrane permeability transition pore in mitochondrial dysfunction-related disease therapies
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Recent progress in the use of mitochondrial membrane permeability transition pore in mitochondrial dysfunction-related disease therapies

Molecular and cellular biochemistry, 2021-01, Vol.476 (1), p.493-506 [Peer Reviewed Journal]

Springer Science+Business Media, LLC, part of Springer Nature 2020 ;COPYRIGHT 2020 Springer ;Springer Science+Business Media, LLC, part of Springer Nature 2020. ;ISSN: 0300-8177 ;EISSN: 1573-4919 ;DOI: 10.1007/s11010-020-03926-0 ;PMID: 33000352

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4
RIPK3 Activates MLKL-mediated Necroptosis and Inflammasome Signaling during Streptococcus Infection
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RIPK3 Activates MLKL-mediated Necroptosis and Inflammasome Signaling during Streptococcus Infection

American journal of respiratory cell and molecular biology, 2021-05, Vol.64 (5), p.579-591 [Peer Reviewed Journal]

Copyright American Thoracic Society May 2021 ;Copyright © 2021 by the American Thoracic Society 2021 ;ISSN: 1044-1549 ;EISSN: 1535-4989 ;DOI: 10.1165/rcmb.2020-0312OC ;PMID: 33625952

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5
Molecular mechanisms of cell death: recommendations of the Nomenclature Committee on Cell Death 2018
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Molecular mechanisms of cell death: recommendations of the Nomenclature Committee on Cell Death 2018

Cell death and differentiation, 2018-03, Vol.25 (3), p.486-541 [Peer Reviewed Journal]

2018. This work is published under http://creativecommons.org/licenses/by-nc-sa/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;Attribution - NonCommercial - ShareAlike ;ADMC Associazione Differenziamento e Morte Cellulare 2018 ;ISSN: 1350-9047 ;ISSN: 1476-5403 ;EISSN: 1476-5403 ;DOI: 10.1038/s41418-017-0012-4 ;PMID: 29362479

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6
Pathogenesis of cardiac ischemia reperfusion injury is associated with CK2α-disturbed mitochondrial homeostasis via suppression of FUNDC1-related mitophagy
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Pathogenesis of cardiac ischemia reperfusion injury is associated with CK2α-disturbed mitochondrial homeostasis via suppression of FUNDC1-related mitophagy

Cell death and differentiation, 2018-06, Vol.25 (6), p.1080-1093 [Peer Reviewed Journal]

2018. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;ADMC Associazione Differenziamento e Morte Cellulare 2018 ;ISSN: 1350-9047 ;EISSN: 1476-5403 ;DOI: 10.1038/s41418-018-0086-7 ;PMID: 29540794

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7
Opening of mitochondrial permeability transition pore in cardiomyocytes: is ferutinin a suitable tool for its assessment?
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Opening of mitochondrial permeability transition pore in cardiomyocytes: is ferutinin a suitable tool for its assessment?

Fundamental & clinical pharmacology, 2023-08 [Peer Reviewed Journal]

Distributed under a Creative Commons Attribution 4.0 International License ;ISSN: 0767-3981 ;EISSN: 1472-8206 ;DOI: 10.1111/fcp.12879 ;PMID: 36797226

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8
The mitochondrial chaperone TRAP1 regulates F-ATP synthase channel formation
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The mitochondrial chaperone TRAP1 regulates F-ATP synthase channel formation

Cell death and differentiation, 2022-12, Vol.29 (12), p.2335-2346 [Peer Reviewed Journal]

2022. The Author(s). ;The Author(s) 2022. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2022 ;ISSN: 1350-9047 ;EISSN: 1476-5403 ;DOI: 10.1038/s41418-022-01020-0 ;PMID: 35614131

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9
Oxidized DNA fragments exit mitochondria via mPTP- and VDAC-dependent channels to activate NLRP3 inflammasome and interferon signaling
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Oxidized DNA fragments exit mitochondria via mPTP- and VDAC-dependent channels to activate NLRP3 inflammasome and interferon signaling

Immunity (Cambridge, Mass.), 2022-08, Vol.55 (8), p.1370-1385.e8 [Peer Reviewed Journal]

2022 Elsevier Inc. ;Copyright © 2022 Elsevier Inc. All rights reserved. ;ISSN: 1074-7613 ;EISSN: 1097-4180 ;DOI: 10.1016/j.immuni.2022.06.007 ;PMID: 35835107

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10
Molecular mechanisms and consequences of mitochondrial permeability transition
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Article
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Molecular mechanisms and consequences of mitochondrial permeability transition

Nature reviews. Molecular cell biology, 2022-04, Vol.23 (4), p.266-285 [Peer Reviewed Journal]

2021. Springer Nature Limited. ;Springer Nature Limited 2021. ;ISSN: 1471-0072 ;EISSN: 1471-0080 ;DOI: 10.1038/s41580-021-00433-y ;PMID: 34880425

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11
Cell death pathways: intricate connections and disease implications
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Cell death pathways: intricate connections and disease implications

The EMBO journal, 2021-03, Vol.40 (5), p.e106700-n/a [Peer Reviewed Journal]

2021 The Authors ;2021 The Authors. ;2021 EMBO ;ISSN: 0261-4189 ;EISSN: 1460-2075 ;DOI: 10.15252/embj.2020106700 ;PMID: 33439509

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12
Targeting the Mitochondrial Permeability Transition Pore to Prevent Age-Associated Cell Damage and Neurodegeneration
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Targeting the Mitochondrial Permeability Transition Pore to Prevent Age-Associated Cell Damage and Neurodegeneration

Oxidative medicine and cellular longevity, 2021, Vol.2021, p.6626484-15 [Peer Reviewed Journal]

Copyright © 2021 Andrew C. Kent et al. ;Copyright © 2021 Andrew C. Kent et al. This is an open access article distributed under the Creative Commons Attribution License (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. https://creativecommons.org/licenses/by/4.0 ;Copyright © 2021 Andrew C. Kent et al. 2021 ;ISSN: 1942-0900 ;EISSN: 1942-0994 ;DOI: 10.1155/2021/6626484 ;PMID: 33574977

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13
Heart failure–induced activation of phospholipase iPLA2γ generates hydroxyeicosatetraenoic acids opening the mitochondrial permeability transition pore
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Article
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Heart failure–induced activation of phospholipase iPLA2γ generates hydroxyeicosatetraenoic acids opening the mitochondrial permeability transition pore

The Journal of biological chemistry, 2018-01, Vol.293 (1), p.115-129 [Peer Reviewed Journal]

2018 © 2018 by The American Society for Biochemistry and Molecular Biology, Inc. ;2018 by The American Society for Biochemistry and Molecular Biology, Inc. 2018 The American Society for Biochemistry and Molecular Biology, Inc. ;ISSN: 0021-9258 ;EISSN: 1083-351X ;DOI: 10.1074/jbc.RA117.000405 ;PMID: 29158256

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14
Mitochondrial Drp1 recognizes and induces excessive mPTP opening after hypoxia through BAX-PiC and LRRK2-HK2
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Article
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Mitochondrial Drp1 recognizes and induces excessive mPTP opening after hypoxia through BAX-PiC and LRRK2-HK2

Cell death & disease, 2021-11, Vol.12 (11), p.1050-1050, Article 1050 [Peer Reviewed Journal]

2021. The Author(s). ;The Author(s) 2021. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2021 ;ISSN: 2041-4889 ;EISSN: 2041-4889 ;DOI: 10.1038/s41419-021-04343-x ;PMID: 34741026

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15
The mitochondrial ATP synthase is a negative regulator of the mitochondrial permeability transition pore
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The mitochondrial ATP synthase is a negative regulator of the mitochondrial permeability transition pore

Proceedings of the National Academy of Sciences - PNAS, 2023-12, Vol.120 (51), p.e2303713120-e2303713120 [Peer Reviewed Journal]

Copyright National Academy of Sciences Dec 19, 2023 ;ISSN: 0027-8424 ;EISSN: 1091-6490 ;DOI: 10.1073/pnas.2303713120 ;PMID: 38091291

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16
New insights into acute pancreatitis
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Article
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New insights into acute pancreatitis

Nature reviews. Gastroenterology & hepatology, 2019-08, Vol.16 (8), p.479-496 [Peer Reviewed Journal]

COPYRIGHT 2019 Nature Publishing Group ;US Government 2019. ;ISSN: 1759-5045 ;EISSN: 1759-5053 ;DOI: 10.1038/s41575-019-0158-2 ;PMID: 31138897

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17
Hibernator-Derived Cells Show Superior Protection and Survival in Hypothermia Compared to Non-Hibernator Cells
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Article
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Hibernator-Derived Cells Show Superior Protection and Survival in Hypothermia Compared to Non-Hibernator Cells

International journal of molecular sciences, 2020-03, Vol.21 (5), p.1864 [Peer Reviewed Journal]

2020. This work is licensed under http://creativecommons.org/licenses/by/3.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2020 by the authors. 2020 ;ISSN: 1422-0067 ;ISSN: 1661-6596 ;EISSN: 1422-0067 ;DOI: 10.3390/ijms21051864 ;PMID: 32182837

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18
Reduction of elevated proton leak rejuvenates mitochondria in the aged cardiomyocyte
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Reduction of elevated proton leak rejuvenates mitochondria in the aged cardiomyocyte

eLife, 2020-12, Vol.9 [Peer Reviewed Journal]

2020, Zhang et al. ;COPYRIGHT 2020 eLife Science Publications, Ltd. ;2020, Zhang et al 2020 Zhang et al ;ISSN: 2050-084X ;EISSN: 2050-084X ;DOI: 10.7554/eLife.60827 ;PMID: 33319746

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19
The Mitochondrial Permeability Transition Pore: Channel Formation by F-ATP Synthase, Integration in Signal Transduction, and Role in Pathophysiology
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The Mitochondrial Permeability Transition Pore: Channel Formation by F-ATP Synthase, Integration in Signal Transduction, and Role in Pathophysiology

Physiological reviews, 2015-10, Vol.95 (4), p.1111-1155 [Peer Reviewed Journal]

Copyright © 2015 the American Physiological Society. ;Copyright © 2015 the American Physiological Society 2015 American Physiological Society ;ISSN: 0031-9333 ;EISSN: 1522-1210 ;DOI: 10.1152/physrev.00001.2015 ;PMID: 26269524 ;CODEN: PHREA7

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20
Mitochondrial calcium exchange in physiology and disease
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Article
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Mitochondrial calcium exchange in physiology and disease

Physiological reviews, 2022-04, Vol.102 (2), p.893-992 [Peer Reviewed Journal]

Copyright American Physiological Society Apr 2022 ;Copyright © 2022 the American Physiological Society. 2022 American Physiological Society ;ISSN: 0031-9333 ;EISSN: 1522-1210 ;DOI: 10.1152/physrev.00041.2020 ;PMID: 34698550

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