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1
Imeglimin attenuates NLRP3 inflammasome activation by restoring mitochondrial functions in macrophages
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Imeglimin attenuates NLRP3 inflammasome activation by restoring mitochondrial functions in macrophages

Journal of pharmacological sciences, 2024-06, Vol.155 (2), p.35-43 [Peer Reviewed Journal]

2024 The Authors ;Copyright © 2024 The Authors. Production and hosting by Elsevier B.V. All rights reserved. ;ISSN: 1347-8613 ;EISSN: 1347-8648 ;DOI: 10.1016/j.jphs.2024.03.004 ;PMID: 38677784

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2
EFHD1 promotes osteosarcoma proliferation and drug resistance by inhibiting the opening of the mitochondrial membrane permeability transition pore (mPTP) by binding to ANT3
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EFHD1 promotes osteosarcoma proliferation and drug resistance by inhibiting the opening of the mitochondrial membrane permeability transition pore (mPTP) by binding to ANT3

Cellular and molecular life sciences : CMLS, 2024-05, Vol.81 (1) [Peer Reviewed Journal]

The Author(s) 2024 ;ISSN: 1420-682X ;EISSN: 1420-9071 ;DOI: 10.1007/s00018-024-05254-8

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3
The Enigma of Norbormide, a Rattus -Selective Toxicant
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The Enigma of Norbormide, a Rattus -Selective Toxicant

Cells (Basel, Switzerland), 2024-05, Vol.13 (9), p.788 [Peer Reviewed Journal]

2024 by the authors. Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (https://creativecommons.org/licenses/by/4.0/). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;EISSN: 2073-4409 ;DOI: 10.3390/cells13090788 ;PMID: 38727324

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4
Peptides Targeting the IF1-ATP Synthase Complex Modulate the Permeability Transition Pore in Cancer HeLa Cells
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Peptides Targeting the IF1-ATP Synthase Complex Modulate the Permeability Transition Pore in Cancer HeLa Cells

International journal of molecular sciences, 2024-05, Vol.25 (9), p.4655 [Peer Reviewed Journal]

COPYRIGHT 2024 MDPI AG ;2024 by the authors. Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (https://creativecommons.org/licenses/by/4.0/). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;ISSN: 1422-0067 ;ISSN: 1661-6596 ;EISSN: 1422-0067 ;DOI: 10.3390/ijms25094655 ;PMID: 38731874

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5
Tissue-specific differences in Ca 2+ sensitivity of the mitochondrial permeability transition pore (PTP). Experiments in male rat liver and heart
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Tissue-specific differences in Ca 2+ sensitivity of the mitochondrial permeability transition pore (PTP). Experiments in male rat liver and heart

Physiological reports, 2024-05, Vol.12 (10), p.e16056 [Peer Reviewed Journal]

2024 The Author(s). Physiological Reports published by Wiley Periodicals LLC on behalf of The Physiological Society and the American Physiological Society. ;EISSN: 2051-817X ;PMID: 38777811

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6
The mitochondrial ATP synthase is a negative regulator of the mitochondrial permeability transition pore
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The mitochondrial ATP synthase is a negative regulator of the mitochondrial permeability transition pore

Proceedings of the National Academy of Sciences - PNAS, 2023-12, Vol.120 (51), p.e2303713120-e2303713120 [Peer Reviewed Journal]

Copyright National Academy of Sciences Dec 19, 2023 ;ISSN: 0027-8424 ;EISSN: 1091-6490 ;DOI: 10.1073/pnas.2303713120 ;PMID: 38091291

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7
Mitochondrial permeability transition regulator, cyclophilin D, is transcriptionally activated by C/EBP during adipogenesis
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Mitochondrial permeability transition regulator, cyclophilin D, is transcriptionally activated by C/EBP during adipogenesis

The Journal of biological chemistry, 2023-12, Vol.299 (12), p.105458-105458, Article 105458 [Peer Reviewed Journal]

2023 The Authors ;Copyright © 2023 The Authors. Published by Elsevier Inc. All rights reserved. ;2023 The Authors 2023 ;ISSN: 0021-9258 ;EISSN: 1083-351X ;DOI: 10.1016/j.jbc.2023.105458 ;PMID: 37949231

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8
Identity, structure, and function of the mitochondrial permeability transition pore: controversies, consensus, recent advances, and future directions
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Identity, structure, and function of the mitochondrial permeability transition pore: controversies, consensus, recent advances, and future directions

Cell death and differentiation, 2023-08, Vol.30 (8), p.1869-1885 [Peer Reviewed Journal]

2023. The Author(s). ;The Author(s) 2023. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2023 ;ISSN: 1350-9047 ;EISSN: 1476-5403 ;DOI: 10.1038/s41418-023-01187-0 ;PMID: 37460667

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9
Opening of mitochondrial permeability transition pore in cardiomyocytes: is ferutinin a suitable tool for its assessment?
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Opening of mitochondrial permeability transition pore in cardiomyocytes: is ferutinin a suitable tool for its assessment?

Fundamental & clinical pharmacology, 2023-08 [Peer Reviewed Journal]

Distributed under a Creative Commons Attribution 4.0 International License ;ISSN: 0767-3981 ;EISSN: 1472-8206 ;DOI: 10.1111/fcp.12879 ;PMID: 36797226

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10
Mechanistic correlation between mitochondrial permeability transition pores and mitochondrial ATP dependent potassium channels in ischemia reperfusion
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Mechanistic correlation between mitochondrial permeability transition pores and mitochondrial ATP dependent potassium channels in ischemia reperfusion

Biomedicine & pharmacotherapy, 2023-06, Vol.162, p.114599-114599, Article 114599 [Peer Reviewed Journal]

2023 ;Copyright © 2023. Published by Elsevier Masson SAS. ;ISSN: 0753-3322 ;EISSN: 1950-6007 ;DOI: 10.1016/j.biopha.2023.114599 ;PMID: 37004326

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11
The RISK pathway leading to mitochondria and cardioprotection: how everything started
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The RISK pathway leading to mitochondria and cardioprotection: how everything started

Basic research in cardiology, 2023-05, Vol.118 (1), p.22-22, Article 22 [Peer Reviewed Journal]

The Author(s) 2023 ;2023. The Author(s). ;The Author(s) 2023. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;ISSN: 1435-1803 ;ISSN: 0300-8428 ;EISSN: 1435-1803 ;DOI: 10.1007/s00395-023-00992-5 ;PMID: 37233787

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12
The Haves and Have-Nots: The Mitochondrial Permeability Transition Pore across Species
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The Haves and Have-Nots: The Mitochondrial Permeability Transition Pore across Species

Cells (Basel, Switzerland), 2023-05, Vol.12 (10), p.1409 [Peer Reviewed Journal]

COPYRIGHT 2023 MDPI AG ;2023 by the authors. Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (https://creativecommons.org/licenses/by/4.0/). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2023 by the authors. 2023 ;ISSN: 2073-4409 ;EISSN: 2073-4409 ;DOI: 10.3390/cells12101409 ;PMID: 37408243

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13
The Mitochondrial Permeability Transition Pore-Current Knowledge of Its Structure, Function, and Regulation, and Optimized Methods for Evaluating Its Functional State
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The Mitochondrial Permeability Transition Pore-Current Knowledge of Its Structure, Function, and Regulation, and Optimized Methods for Evaluating Its Functional State

Cells (Basel, Switzerland), 2023-04, Vol.12 (9), p.1273 [Peer Reviewed Journal]

COPYRIGHT 2023 MDPI AG ;2023 by the authors. Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (https://creativecommons.org/licenses/by/4.0/). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2023 by the authors. 2023 ;ISSN: 2073-4409 ;EISSN: 2073-4409 ;DOI: 10.3390/cells12091273 ;PMID: 37174672

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14
Diplacone Isolated from Paulownia tomentosa Mature Fruit Induces Ferroptosis-Mediated Cell Death through Mitochondrial Ca 2+ Influx and Mitochondrial Permeability Transition
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Diplacone Isolated from Paulownia tomentosa Mature Fruit Induces Ferroptosis-Mediated Cell Death through Mitochondrial Ca 2+ Influx and Mitochondrial Permeability Transition

International journal of molecular sciences, 2023-04, Vol.24 (8) [Peer Reviewed Journal]

EISSN: 1422-0067 ;PMID: 37108220

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15
1,3,8-Triazaspiro[4.5]decane Derivatives Inhibit Permeability Transition Pores through a F O -ATP Synthase c Subunit Glu 119 -Independent Mechanism That Prevents Oligomycin A-Related Side Effects
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1,3,8-Triazaspiro[4.5]decane Derivatives Inhibit Permeability Transition Pores through a F O -ATP Synthase c Subunit Glu 119 -Independent Mechanism That Prevents Oligomycin A-Related Side Effects

International journal of molecular sciences, 2023-03, Vol.24 (7) [Peer Reviewed Journal]

EISSN: 1422-0067 ;PMID: 37047160

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16
Promising Strategy of mPTP Modulation in Cancer Therapy: An Emerging Progress and Future Insight
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Article
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Promising Strategy of mPTP Modulation in Cancer Therapy: An Emerging Progress and Future Insight

International journal of molecular sciences, 2023-03, Vol.24 (6), p.5564 [Peer Reviewed Journal]

COPYRIGHT 2023 MDPI AG ;2023 by the authors. Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (https://creativecommons.org/licenses/by/4.0/). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2023 by the authors. 2023 ;ISSN: 1422-0067 ;ISSN: 1661-6596 ;EISSN: 1422-0067 ;DOI: 10.3390/ijms24065564 ;PMID: 36982637

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17
A Comparative Study on the Effects of the Lysine Reagent Pyridoxal 5-Phosphate and Some Thiol Reagents in Opening the Tl + -Induced Mitochondrial Permeability Transition Pore
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A Comparative Study on the Effects of the Lysine Reagent Pyridoxal 5-Phosphate and Some Thiol Reagents in Opening the Tl + -Induced Mitochondrial Permeability Transition Pore

International journal of molecular sciences, 2023-01, Vol.24 (3), p.2460 [Peer Reviewed Journal]

2023 by the authors. Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (https://creativecommons.org/licenses/by/4.0/). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2023 by the authors. 2023 ;ISSN: 1422-0067 ;ISSN: 1661-6596 ;EISSN: 1422-0067 ;DOI: 10.3390/ijms24032460 ;PMID: 36768782

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18
Genetic Inhibition of Mitochondrial Permeability Transition Pore Exacerbates Ryanodine Receptor 2 Dysfunction in Arrhythmic Disease
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Genetic Inhibition of Mitochondrial Permeability Transition Pore Exacerbates Ryanodine Receptor 2 Dysfunction in Arrhythmic Disease

Cells (Basel, Switzerland), 2023-01, Vol.12 (2), p.204 [Peer Reviewed Journal]

2023 by the authors. Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (https://creativecommons.org/licenses/by/4.0/). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2023 by the authors. 2023 ;ISSN: 2073-4409 ;EISSN: 2073-4409 ;DOI: 10.3390/cells12020204 ;PMID: 36672139

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19
Calcium signaling induced by 15-deoxy-prostamide-J2 promotes cell death by activating PERK, IP3R, and the mitochondrial permeability transition pore
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Calcium signaling induced by 15-deoxy-prostamide-J2 promotes cell death by activating PERK, IP3R, and the mitochondrial permeability transition pore

Oncotarget, 2022-12, Vol.13 (1), p.1380-1396

Copyright: © 2022 Ladin et al. ;ISSN: 1949-2553 ;EISSN: 1949-2553 ;DOI: 10.18632/oncotarget.28334 ;PMID: 36580536

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20
The mitochondrial chaperone TRAP1 regulates F-ATP synthase channel formation
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The mitochondrial chaperone TRAP1 regulates F-ATP synthase channel formation

Cell death and differentiation, 2022-12, Vol.29 (12), p.2335-2346 [Peer Reviewed Journal]

2022. The Author(s). ;The Author(s) 2022. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2022 ;ISSN: 1350-9047 ;EISSN: 1476-5403 ;DOI: 10.1038/s41418-022-01020-0 ;PMID: 35614131

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