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1
Active MLKL triggers the NLRP3 inflammasome in a cell-intrinsic manner
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Active MLKL triggers the NLRP3 inflammasome in a cell-intrinsic manner

Proceedings of the National Academy of Sciences - PNAS, 2017-02, Vol.114 (6), p.E961-E969 [Peer Reviewed Journal]

Volumes 1–89 and 106–114, copyright as a collective work only; author(s) retains copyright to individual articles ;Copyright National Academy of Sciences Feb 7, 2017 ;ISSN: 0027-8424 ;EISSN: 1091-6490 ;DOI: 10.1073/pnas.1613305114 ;PMID: 28096356

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2
NLRP1 Inflammasome Activation Induces Pyroptosis of Hematopoietic Progenitor Cells
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NLRP1 Inflammasome Activation Induces Pyroptosis of Hematopoietic Progenitor Cells

Immunity (Cambridge, Mass.), 2012-12, Vol.37 (6), p.1009-1023 [Peer Reviewed Journal]

2012 Elsevier Inc. ;Copyright © 2012 Elsevier Inc. All rights reserved. ;Copyright Elsevier Limited Dec 14, 2012 ;2012 Elsevier Inc. All rights reserved. 2012 ;ISSN: 1074-7613 ;EISSN: 1097-4180 ;DOI: 10.1016/j.immuni.2012.08.027 ;PMID: 23219391

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3
A small-molecule inhibitor of the NLRP3 inflammasome for the treatment of inflammatory diseases
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A small-molecule inhibitor of the NLRP3 inflammasome for the treatment of inflammatory diseases

Nature medicine, 2015-03, Vol.21 (3), p.248-255 [Peer Reviewed Journal]

COPYRIGHT 2015 Nature Publishing Group ;COPYRIGHT 2015 Nature Publishing Group ;Copyright Nature Publishing Group Mar 2015 ;ISSN: 1078-8956 ;EISSN: 1546-170X ;DOI: 10.1038/nm.3806 ;PMID: 25686105

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4
Myeloid-derived miR-223 regulates intestinal inflammation via repression of the NLRP3 inflammasome
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Myeloid-derived miR-223 regulates intestinal inflammation via repression of the NLRP3 inflammasome

The Journal of experimental medicine, 2017-06, Vol.214 (6), p.1737-1752 [Peer Reviewed Journal]

2017 Neudecker et al. ;Copyright Rockefeller University Press Jun 5, 2017 ;2017 Neudecker et al. 2017 ;ISSN: 0022-1007 ;EISSN: 1540-9538 ;DOI: 10.1084/jem.20160462 ;PMID: 28487310

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5
Human DPP9 represses NLRP1 inflammasome and protects against autoinflammatory diseases via both peptidase activity and FIIND domain binding
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Human DPP9 represses NLRP1 inflammasome and protects against autoinflammatory diseases via both peptidase activity and FIIND domain binding

The Journal of biological chemistry, 2018-12, Vol.293 (49), p.18864-18878 [Peer Reviewed Journal]

2018 © 2018 Zhong et al. ;2018 Zhong et al. ;2018 Zhong et al. 2018 Zhong et al. ;ISSN: 0021-9258 ;EISSN: 1083-351X ;DOI: 10.1074/jbc.RA118.004350 ;PMID: 30291141

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6
TBK1 and IKKε Act Redundantly to Mediate STING-Induced NF-κB Responses in Myeloid Cells
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TBK1 and IKKε Act Redundantly to Mediate STING-Induced NF-κB Responses in Myeloid Cells

Cell reports (Cambridge), 2020-04, Vol.31 (1), p.107492, Article 107492 [Peer Reviewed Journal]

2020 The Author(s) ;Copyright © 2020 The Author(s). Published by Elsevier Inc. All rights reserved. ;ISSN: 2211-1247 ;EISSN: 2211-1247 ;DOI: 10.1016/j.celrep.2020.03.056 ;PMID: 32268090

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7
Germline NLRP1 Mutations Cause Skin Inflammatory and Cancer Susceptibility Syndromes via Inflammasome Activation
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Germline NLRP1 Mutations Cause Skin Inflammatory and Cancer Susceptibility Syndromes via Inflammasome Activation

Cell, 2016-09, Vol.167 (1), p.187-202.e17 [Peer Reviewed Journal]

2016 Elsevier Inc. ;Copyright © 2016 Elsevier Inc. All rights reserved. ;Distributed under a Creative Commons Attribution 4.0 International License ;ISSN: 0092-8674 ;EISSN: 1097-4172 ;DOI: 10.1016/j.cell.2016.09.001 ;PMID: 27662089

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8
RIPK3 promotes cell death and NLRP3 inflammasome activation in the absence of MLKL
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RIPK3 promotes cell death and NLRP3 inflammasome activation in the absence of MLKL

Nature communications, 2015-02, Vol.6 (1), p.6282-6282, Article 6282 [Peer Reviewed Journal]

Copyright Nature Publishing Group Feb 2015 ;Copyright © 2015, Nature Publishing Group, a division of Macmillan Publishers Limited. All Rights Reserved. 2015 Nature Publishing Group, a division of Macmillan Publishers Limited. All Rights Reserved. ;ISSN: 2041-1723 ;EISSN: 2041-1723 ;DOI: 10.1038/ncomms7282 ;PMID: 25693118

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9
Adipose Tissue Macrophages Promote Myelopoiesis and Monocytosis in Obesity
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Adipose Tissue Macrophages Promote Myelopoiesis and Monocytosis in Obesity

Cell metabolism, 2014-05, Vol.19 (5), p.821-835 [Peer Reviewed Journal]

2014 Elsevier Inc. ;Copyright © 2014 Elsevier Inc. All rights reserved. ;2014 Elsevier Inc. All rights reserved. 2014 ;ISSN: 1550-4131 ;EISSN: 1932-7420 ;DOI: 10.1016/j.cmet.2014.03.029 ;PMID: 24807222

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10
IL-18 Production from the NLRP1 Inflammasome Prevents Obesity and Metabolic Syndrome
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IL-18 Production from the NLRP1 Inflammasome Prevents Obesity and Metabolic Syndrome

Cell metabolism, 2016-01, Vol.23 (1), p.155-164 [Peer Reviewed Journal]

2016 Elsevier Inc. ;Copyright © 2016 Elsevier Inc. All rights reserved. ;ISSN: 1550-4131 ;EISSN: 1932-7420 ;DOI: 10.1016/j.cmet.2015.09.024 ;PMID: 26603191

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11
RIPK1 Regulates RIPK3-MLKL-Driven Systemic Inflammation and Emergency Hematopoiesis
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RIPK1 Regulates RIPK3-MLKL-Driven Systemic Inflammation and Emergency Hematopoiesis

Cell, 2014-05, Vol.157 (5), p.1175-1188 [Peer Reviewed Journal]

2014 Elsevier Inc. ;Copyright © 2014 Elsevier Inc. All rights reserved. ;ISSN: 0092-8674 ;EISSN: 1097-4172 ;DOI: 10.1016/j.cell.2014.04.019 ;PMID: 24813849

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12
A Toxoplasma gondii Gluconeogenic Enzyme Contributes to Robust Central Carbon Metabolism and Is Essential for Replication and Virulence
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A Toxoplasma gondii Gluconeogenic Enzyme Contributes to Robust Central Carbon Metabolism and Is Essential for Replication and Virulence

Cell host & microbe, 2015-08, Vol.18 (2), p.210-220 [Peer Reviewed Journal]

2015 Elsevier Inc. ;Copyright © 2015 Elsevier Inc. All rights reserved. ;ISSN: 1931-3128 ;EISSN: 1934-6069 ;DOI: 10.1016/j.chom.2015.07.008 ;PMID: 26269956

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13
The pathogen Candida albicans hijacks pyroptosis for escape from macrophages
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The pathogen Candida albicans hijacks pyroptosis for escape from macrophages

mBio, 2014-03, Vol.5 (2), p.e00003-e00014 [Peer Reviewed Journal]

Copyright © 2014 Uwamahoro et al. 2014 Uwamahoro et al. ;ISSN: 2161-2129 ;EISSN: 2150-7511 ;DOI: 10.1128/mbio.00003-14 ;PMID: 24667705

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14
Deficiency in coatomer complex I causes aberrant activation of STING signalling
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Deficiency in coatomer complex I causes aberrant activation of STING signalling

Nature communications, 2022-04, Vol.13 (1), p.2321-2321, Article 2321 [Peer Reviewed Journal]

2022. The Author(s). ;The Author(s) 2022. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2022 ;ISSN: 2041-1723 ;EISSN: 2041-1723 ;DOI: 10.1038/s41467-022-29946-6 ;PMID: 35484149

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15
NLRP1 restricts butyrate producing commensals to exacerbate inflammatory bowel disease
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NLRP1 restricts butyrate producing commensals to exacerbate inflammatory bowel disease

Nature communications, 2018-09, Vol.9 (1), p.3728-11, Article 3728 [Peer Reviewed Journal]

2018. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2018 ;ISSN: 2041-1723 ;EISSN: 2041-1723 ;DOI: 10.1038/s41467-018-06125-0 ;PMID: 30214011

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16
ATF3 Is a Key Regulator of Macrophage IFN Responses
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ATF3 Is a Key Regulator of Macrophage IFN Responses

The Journal of immunology (1950), 2015-11, Vol.195 (9), p.4446-4455 [Peer Reviewed Journal]

Copyright © 2015 by The American Association of Immunologists, Inc. ;ISSN: 0022-1767 ;EISSN: 1550-6606 ;DOI: 10.4049/jimmunol.1500204 ;PMID: 26416280

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17
The RNA-binding protein Tristetraprolin (TTP) is a critical negative regulator of the NLRP3 inflammasome
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The RNA-binding protein Tristetraprolin (TTP) is a critical negative regulator of the NLRP3 inflammasome

The Journal of biological chemistry, 2017-04, Vol.292 (17), p.6869-6881 [Peer Reviewed Journal]

2017 © 2017 ASBMB. Currently published by Elsevier Inc; originally published by American Society for Biochemistry and Molecular Biology. ;2017 by The American Society for Biochemistry and Molecular Biology, Inc. ;2017 by The American Society for Biochemistry and Molecular Biology, Inc. 2017 The American Society for Biochemistry and Molecular Biology, Inc. ;ISSN: 0021-9258 ;EISSN: 1083-351X ;DOI: 10.1074/jbc.M116.772947 ;PMID: 28302726

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18
The B30.2 Domain of Pyrin, the Familial Mediterranean Fever Protein, Interacts Directly with Caspase-1 to Modulate IL-1β Production
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The B30.2 Domain of Pyrin, the Familial Mediterranean Fever Protein, Interacts Directly with Caspase-1 to Modulate IL-1β Production

Proceedings of the National Academy of Sciences - PNAS, 2006-06, Vol.103 (26), p.9982-9987 [Peer Reviewed Journal]

Copyright 2006 National Academy of Sciences of the United States of America ;2006 by The National Academy of Sciences of the USA 2006 ;ISSN: 0027-8424 ;EISSN: 1091-6490 ;DOI: 10.1073/pnas.0602081103 ;PMID: 16785446

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19
Differential recognition of HIV-stimulated IL-1β and IL-18 secretion through NLR and NAIP signalling in monocyte-derived macrophages
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Differential recognition of HIV-stimulated IL-1β and IL-18 secretion through NLR and NAIP signalling in monocyte-derived macrophages

PLoS pathogens, 2021-04, Vol.17 (4), p.e1009417-e1009417 [Peer Reviewed Journal]

2021 Triantafilou et al. This is an open access article distributed under the terms of the Creative Commons Attribution License: http://creativecommons.org/licenses/by/4.0/ (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2021 Triantafilou et al 2021 Triantafilou et al ;ISSN: 1553-7374 ;ISSN: 1553-7366 ;EISSN: 1553-7374 ;DOI: 10.1371/journal.ppat.1009417 ;PMID: 33861800

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20
Homeostasis-altering molecular processes as mechanisms of inflammasome activation
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Homeostasis-altering molecular processes as mechanisms of inflammasome activation

Nature reviews. Immunology, 2017-03, Vol.17 (3), p.208-214 [Peer Reviewed Journal]

COPYRIGHT 2017 Nature Publishing Group ;COPYRIGHT 2017 Nature Publishing Group ;Copyright Nature Publishing Group Mar 2017 ;ISSN: 1474-1733 ;EISSN: 1474-1741 ;DOI: 10.1038/nri.2016.151 ;PMID: 28163301

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