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1
CuII(atsm) inhibits ferroptosis: Implications for treatment of neurodegenerative disease
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CuII(atsm) inhibits ferroptosis: Implications for treatment of neurodegenerative disease

British journal of pharmacology, 2020-02, Vol.177 (3), p.656-667 [Peer Reviewed Journal]

2019 The British Pharmacological Society ;2020 The British Pharmacological Society ;ISSN: 0007-1188 ;EISSN: 1476-5381 ;DOI: 10.1111/bph.14881 ;PMID: 31655003

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2
ALS-associated TDP-43 induces endoplasmic reticulum stress, which drives cytoplasmic TDP-43 accumulation and stress granule formation
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ALS-associated TDP-43 induces endoplasmic reticulum stress, which drives cytoplasmic TDP-43 accumulation and stress granule formation

PloS one, 2013-11, Vol.8 (11), p.e81170-e81170 [Peer Reviewed Journal]

COPYRIGHT 2013 Public Library of Science ;2013 Walker et al. This is an open-access article distributed under the terms of the Creative Commons Attribution License: http://creativecommons.org/licenses/by/3.0/ (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2013 Walker et al 2013 Walker et al ;ISSN: 1932-6203 ;EISSN: 1932-6203 ;DOI: 10.1371/journal.pone.0081170 ;PMID: 24312274

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3
Copper delivery to the CNS by CuATSM effectively treats motor neuron disease in SODG93A mice co-expressing the Copper-Chaperone-for-SOD
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Copper delivery to the CNS by CuATSM effectively treats motor neuron disease in SODG93A mice co-expressing the Copper-Chaperone-for-SOD

Neurobiology of disease, 2016-05, Vol.89, p.1-9 [Peer Reviewed Journal]

EISSN: 1095-953X ;DOI: 10.1016/j.nbd.2016.01.020

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4
Comment: Cu-ATSM to treat and image amyotrophic lateral sclerosis
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Comment: Cu-ATSM to treat and image amyotrophic lateral sclerosis

Neurology, 2015-05, Vol.84 (20), p.2038-2038 [Peer Reviewed Journal]

2015 American Academy of Neurology ;ISSN: 0028-3878 ;EISSN: 1526-632X ;DOI: 10.1212/WNL.0000000000001600 ;PMID: 25904689

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5
CuII(atsm) improves the neurological phenotype and survival of SOD1G93A mice and selectively increases enzymatically active SOD1 in the spinal cord
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CuII(atsm) improves the neurological phenotype and survival of SOD1G93A mice and selectively increases enzymatically active SOD1 in the spinal cord

Scientific reports, 2017-02, Vol.7 (1), p.42292-42292, Article 42292 [Peer Reviewed Journal]

Copyright Nature Publishing Group Feb 2017 ;Copyright © 2017, The Author(s) 2017 The Author(s) ;ISSN: 2045-2322 ;EISSN: 2045-2322 ;DOI: 10.1038/srep42292 ;PMID: 28205575

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6
Prion protein “gamma-cleavage”: characterizing a novel endoproteolytic processing event
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Prion protein “gamma-cleavage”: characterizing a novel endoproteolytic processing event

Cellular and molecular life sciences : CMLS, 2016-02, Vol.73 (3), p.667-683 [Peer Reviewed Journal]

Springer Basel 2015 ;Springer International Publishing 2016 ;ISSN: 1420-682X ;EISSN: 1420-9071 ;DOI: 10.1007/s00018-015-2022-z ;PMID: 26298290

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7
Increasing Cu bioavailability inhibits Aβ oligomers and tau phosphorylation
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Increasing Cu bioavailability inhibits Aβ oligomers and tau phosphorylation

Proceedings of the National Academy of Sciences - PNAS, 2009-01, Vol.106 (2), p.381-386 [Peer Reviewed Journal]

2009 by The National Academy of Sciences of the USA ;ISSN: 0027-8424 ;EISSN: 1091-6490 ;DOI: 10.1073/pnas.0809057106 ;PMID: 19122148

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8
Metal ionophore treatment restores dendritic spine density and synaptic protein levels in a mouse model of Alzheimer's disease
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Metal ionophore treatment restores dendritic spine density and synaptic protein levels in a mouse model of Alzheimer's disease

PloS one, 2011-03, Vol.6 (3), p.e17669 [Peer Reviewed Journal]

COPYRIGHT 2011 Public Library of Science ;COPYRIGHT 2011 Public Library of Science ;2011 Adlard et al. This is an open-access article distributed under the terms of the Creative Commons Attribution License: https://creativecommons.org/licenses/by/4.0/ (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;Adlard et al. 2011 ;ISSN: 1932-6203 ;EISSN: 1932-6203 ;DOI: 10.1371/journal.pone.0017669 ;PMID: 21412423

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9
Regular Physical Exercise Modulates Iron Homeostasis in the 5xFAD Mouse Model of Alzheimer’s Disease
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Regular Physical Exercise Modulates Iron Homeostasis in the 5xFAD Mouse Model of Alzheimer’s Disease

International journal of molecular sciences, 2021-08, Vol.22 (16), p.8715 [Peer Reviewed Journal]

2021 by the authors. Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (https://creativecommons.org/licenses/by/4.0/). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2021 by the authors. 2021 ;ISSN: 1422-0067 ;ISSN: 1661-6596 ;EISSN: 1422-0067 ;DOI: 10.3390/ijms22168715 ;PMID: 34445419

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10
The hypoxia imaging agent CuII(atsm) is neuroprotective and improves motor and cognitive functions in multiple animal models of Parkinson's disease
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The hypoxia imaging agent CuII(atsm) is neuroprotective and improves motor and cognitive functions in multiple animal models of Parkinson's disease

The Journal of experimental medicine, 2012-04, Vol.209 (4), p.837-854 [Peer Reviewed Journal]

2012 Hung et al. 2012 ;ISSN: 0022-1007 ;EISSN: 1540-9538 ;DOI: 10.1084/jem.20112285 ;PMID: 22473957

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11
impaired mitochondrial electron transport chain increases retention of the hypoxia imaging agent diacetylbis(4-methylthiosemicarbazonato)copperII
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impaired mitochondrial electron transport chain increases retention of the hypoxia imaging agent diacetylbis(4-methylthiosemicarbazonato)copperII

Proceedings of the National Academy of Sciences - PNAS, 2012-01, Vol.109 (1), p.47-52 [Peer Reviewed Journal]

copyright © 1993—2008 the National Academy of Sciences of the United States of America ;Copyright National Academy of Sciences Jan 3, 2012 ;ISSN: 0027-8424 ;EISSN: 1091-6490 ;DOI: 10.1073/pnas.1116227108 ;PMID: 22173633

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12
Copper-ATSM as a Treatment for ALS: Support from Mutant SOD1 Models and Beyond
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Copper-ATSM as a Treatment for ALS: Support from Mutant SOD1 Models and Beyond

Life (Basel, Switzerland), 2020-11, Vol.10 (11), p.271 [Peer Reviewed Journal]

2020 by the authors. 2020 ;ISSN: 2075-1729 ;EISSN: 2075-1729 ;DOI: 10.3390/life10110271 ;PMID: 33158182

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13
Diacetylbis(N(4)-methylthiosemicarbazonato) Copper(II) (CuII(atsm)) Protects against Peroxynitrite-induced Nitrosative Damage and Prolongs Survival in Amyotrophic Lateral Sclerosis Mouse Model
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Diacetylbis(N(4)-methylthiosemicarbazonato) Copper(II) (CuII(atsm)) Protects against Peroxynitrite-induced Nitrosative Damage and Prolongs Survival in Amyotrophic Lateral Sclerosis Mouse Model

The Journal of biological chemistry, 2011-12, Vol.286 (51), p.44035-44044 [Peer Reviewed Journal]

2011 © 2011 ASBMB. Currently published by Elsevier Inc; originally published by American Society for Biochemistry and Molecular Biology. ;2011 by The American Society for Biochemistry and Molecular Biology, Inc. 2011 ;ISSN: 0021-9258 ;EISSN: 1083-351X ;DOI: 10.1074/jbc.M111.274407 ;PMID: 22033929

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14
Iron accumulation in skeletal muscles of old mice is associated with impaired regeneration after ischaemia–reperfusion damage
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Iron accumulation in skeletal muscles of old mice is associated with impaired regeneration after ischaemia–reperfusion damage

Journal of cachexia, sarcopenia and muscle, 2021-04, Vol.12 (2), p.476-492 [Peer Reviewed Journal]

2021 The Authors. Journal of Cachexia, Sarcopenia and Muscle published by John Wiley & Sons Ltd on behalf of Society on Sarcopenia, Cachexia and Wasting Disorders ;2021 The Authors. Journal of Cachexia, Sarcopenia and Muscle published by John Wiley & Sons Ltd on behalf of Society on Sarcopenia, Cachexia and Wasting Disorders. ;2021. This work is published under http://creativecommons.org/licenses/by-nc-nd/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;ISSN: 2190-5991 ;EISSN: 2190-6009 ;DOI: 10.1002/jcsm.12685 ;PMID: 33665974

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15
Enhancing survival motor neuron expression extends lifespan and attenuates neurodegeneration in mutant TDP-43 mice
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Enhancing survival motor neuron expression extends lifespan and attenuates neurodegeneration in mutant TDP-43 mice

Human molecular genetics, 2016-09, Vol.25 (18), p.4080-4093 [Peer Reviewed Journal]

The Author 2016. Published by Oxford University Press. All rights reserved. For Permissions, please email: journals.permissions@oup.com. ;ISSN: 0964-6906 ;EISSN: 1460-2083 ;DOI: 10.1093/hmg/ddw247 ;PMID: 27466204

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16
Proteome-wide systems genetics identifies UFMylation as a regulator of skeletal muscle function
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Proteome-wide systems genetics identifies UFMylation as a regulator of skeletal muscle function

eLife, 2022-12, Vol.11 [Peer Reviewed Journal]

2022, Molendijk et al. ;2022, Molendijk et al 2022 Molendijk et al ;ISSN: 2050-084X ;EISSN: 2050-084X ;DOI: 10.7554/ELIFE.82951 ;PMID: 36472367

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17
Microglial ferroptotic stress causes non-cell autonomous neuronal death
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Microglial ferroptotic stress causes non-cell autonomous neuronal death

Molecular neurodegeneration, 2024-02, Vol.19 (1), p.14-14, Article 14 [Peer Reviewed Journal]

2024. The Author(s). ;COPYRIGHT 2024 BioMed Central Ltd. ;The Author(s) 2024 ;ISSN: 1750-1326 ;EISSN: 1750-1326 ;DOI: 10.1186/s13024-023-00691-8 ;PMID: 38317225

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18
Phosphorylation of hnRNP K by cyclin-dependent kinase 2 controls cytosolic accumulation of TDP-43
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Phosphorylation of hnRNP K by cyclin-dependent kinase 2 controls cytosolic accumulation of TDP-43

Human molecular genetics, 2015-03, Vol.24 (6), p.1655-1669 [Peer Reviewed Journal]

The Author 2014. Published by Oxford University Press. All rights reserved. For Permissions, please email: journals.permissions@oup.com. ;ISSN: 0964-6906 ;EISSN: 1460-2083 ;DOI: 10.1093/hmg/ddu578 ;PMID: 25410660

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19
Neuroprotective copper bis(thiosemicarbazonato) complexes promote neurite elongation
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Neuroprotective copper bis(thiosemicarbazonato) complexes promote neurite elongation

PloS one, 2014-02, Vol.9 (2), p.e90070-e90070 [Peer Reviewed Journal]

COPYRIGHT 2014 Public Library of Science ;COPYRIGHT 2014 Public Library of Science ;2014 Bica et al. This is an open-access article distributed under the terms of the Creative Commons Attribution License: http://creativecommons.org/licenses/by/4.0/ (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2014 Bica et al 2014 Bica et al ;ISSN: 1932-6203 ;EISSN: 1932-6203 ;DOI: 10.1371/journal.pone.0090070 ;PMID: 24587210

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20
ZnII(atsm) is protective in amyotrophic lateral sclerosis model mice via a copper delivery mechanism
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ZnII(atsm) is protective in amyotrophic lateral sclerosis model mice via a copper delivery mechanism

Neurobiology of disease, 2015-09, Vol.81, p.20-24 [Peer Reviewed Journal]

EISSN: 1095-953X ;DOI: 10.1016/j.nbd.2015.02.023

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