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1
Function and mechanisms of enteroendocrine cells and gut hormones in metabolism
Function and mechanisms of enteroendocrine cells and gut hormones in metabolism
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Function and mechanisms of enteroendocrine cells and gut hormones in metabolism

Nature reviews. Endocrinology, 2019-04, Vol.15 (4), p.226-237 [Peer Reviewed Journal]

COPYRIGHT 2019 Nature Publishing Group ;2019© Springer Nature Limited 2019 ;ISSN: 1759-5029 ;EISSN: 1759-5037 ;DOI: 10.1038/s41574-019-0168-8 ;PMID: 30760847

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2
Bacterial Metabolite Indole Modulates Incretin Secretion from Intestinal Enteroendocrine L Cells
Bacterial Metabolite Indole Modulates Incretin Secretion from Intestinal Enteroendocrine L Cells
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Bacterial Metabolite Indole Modulates Incretin Secretion from Intestinal Enteroendocrine L Cells

Cell reports (Cambridge), 2014-11, Vol.9 (4), p.1202-1208 [Peer Reviewed Journal]

2014 The Authors ;Copyright © 2014 The Authors. Published by Elsevier Inc. All rights reserved. ;2014 The Authors 2014 ;ISSN: 2211-1247 ;EISSN: 2211-1247 ;DOI: 10.1016/j.celrep.2014.10.032 ;PMID: 25456122

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3
Short-Chain Fatty Acids Stimulate Glucagon-Like Peptide-1 Secretion via the G-Protein―Coupled Receptor FFAR2
Short-Chain Fatty Acids Stimulate Glucagon-Like Peptide-1 Secretion via the G-Protein―Coupled Receptor FFAR2
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Short-Chain Fatty Acids Stimulate Glucagon-Like Peptide-1 Secretion via the G-Protein―Coupled Receptor FFAR2

Diabetes (New York, N.Y.), 2012-02, Vol.61 (2), p.364-371 [Peer Reviewed Journal]

2015 INIST-CNRS ;COPYRIGHT 2012 American Diabetes Association ;COPYRIGHT 2012 American Diabetes Association ;Copyright American Diabetes Association Feb 2012 ;2012 by the American Diabetes Association. 2012 ;ISSN: 0012-1797 ;EISSN: 1939-327X ;DOI: 10.2337/db11-1019 ;PMID: 22190648 ;CODEN: DIAEAZ

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4
G protein-coupled receptors as new therapeutic targets for type 2 diabetes
G protein-coupled receptors as new therapeutic targets for type 2 diabetes
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G protein-coupled receptors as new therapeutic targets for type 2 diabetes

Diabetologia, 2016-02, Vol.59 (2), p.229-233 [Peer Reviewed Journal]

The Author(s) 2015 ;Springer-Verlag Berlin Heidelberg 2016 ;ISSN: 0012-186X ;EISSN: 1432-0428 ;DOI: 10.1007/s00125-015-3825-z ;PMID: 26661410

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5
Bile Acids Trigger GLP-1 Release Predominantly by Accessing Basolaterally Located G Protein–Coupled Bile Acid Receptors
Bile Acids Trigger GLP-1 Release Predominantly by Accessing Basolaterally Located G Protein–Coupled Bile Acid Receptors
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Bile Acids Trigger GLP-1 Release Predominantly by Accessing Basolaterally Located G Protein–Coupled Bile Acid Receptors

Endocrinology (Philadelphia), 2015-11, Vol.156 (11), p.3961-3970 [Peer Reviewed Journal]

ISSN: 0013-7227 ;EISSN: 1945-7170 ;DOI: 10.1210/en.2015-1321 ;PMID: 26280129

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6
Identification and Characterization of GLP-1 Receptor-Expressing Cells Using a New Transgenic Mouse Model
Identification and Characterization of GLP-1 Receptor-Expressing Cells Using a New Transgenic Mouse Model
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Identification and Characterization of GLP-1 Receptor-Expressing Cells Using a New Transgenic Mouse Model

Diabetes (New York, N.Y.), 2014-04, Vol.63 (4), p.1224-1233 [Peer Reviewed Journal]

2015 INIST-CNRS ;COPYRIGHT 2014 American Diabetes Association ;COPYRIGHT 2014 American Diabetes Association ;Copyright American Diabetes Association Apr 2014 ;ISSN: 0012-1797 ;EISSN: 1939-327X ;DOI: 10.2337/db13-1440 ;PMID: 24296712 ;CODEN: DIAEAZ

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7
Preproglucagon Neurons in the Nucleus of the Solitary Tract Are the Main Source of Brain GLP-1, Mediate Stress-Induced Hypophagia, and Limit Unusually Large Intakes of Food
Preproglucagon Neurons in the Nucleus of the Solitary Tract Are the Main Source of Brain GLP-1, Mediate Stress-Induced Hypophagia, and Limit Unusually Large Intakes of Food
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Preproglucagon Neurons in the Nucleus of the Solitary Tract Are the Main Source of Brain GLP-1, Mediate Stress-Induced Hypophagia, and Limit Unusually Large Intakes of Food

Diabetes (New York, N.Y.), 2019-01, Vol.68 (1), p.21-33 [Peer Reviewed Journal]

2018 by the American Diabetes Association. ;Copyright American Diabetes Association Jan 1, 2019 ;2018 by the American Diabetes Association. 2018 ;ISSN: 0012-1797 ;EISSN: 1939-327X ;DOI: 10.2337/db18-0729 ;PMID: 30279161

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8
The G Protein-coupled Receptor Family C Group 6 Subtype A (GPRC6A) Receptor Is Involved in Amino Acid-induced Glucagon-like Peptide-1 Secretion from GLUTag Cells
The G Protein-coupled Receptor Family C Group 6 Subtype A (GPRC6A) Receptor Is Involved in Amino Acid-induced Glucagon-like Peptide-1 Secretion from GLUTag Cells
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The G Protein-coupled Receptor Family C Group 6 Subtype A (GPRC6A) Receptor Is Involved in Amino Acid-induced Glucagon-like Peptide-1 Secretion from GLUTag Cells

The Journal of biological chemistry, 2013-02, Vol.288 (7), p.4513-4521 [Peer Reviewed Journal]

2013 © 2013 ASBMB. Currently published by Elsevier Inc; originally published by American Society for Biochemistry and Molecular Biology. ;2013 by The American Society for Biochemistry and Molecular Biology, Inc. 2013 ;ISSN: 0021-9258 ;EISSN: 1083-351X ;DOI: 10.1074/jbc.M112.402677 ;PMID: 23269670

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9
Free Fatty Acid Receptors in Enteroendocrine Cells
Free Fatty Acid Receptors in Enteroendocrine Cells
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Free Fatty Acid Receptors in Enteroendocrine Cells

Endocrinology (Philadelphia), 2018-07, Vol.159 (7), p.2826-2835 [Peer Reviewed Journal]

COPYRIGHT 2018 Endocrine Society ;ISSN: 1945-7170 ;ISSN: 0013-7227 ;EISSN: 1945-7170 ;DOI: 10.1210/en.2018-00261 ;PMID: 29688303

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10
Transcriptomic profiling of pancreatic alpha, beta and delta cell populations identifies delta cells as a principal target for ghrelin in mouse islets
Transcriptomic profiling of pancreatic alpha, beta and delta cell populations identifies delta cells as a principal target for ghrelin in mouse islets
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Transcriptomic profiling of pancreatic alpha, beta and delta cell populations identifies delta cells as a principal target for ghrelin in mouse islets

Diabetologia, 2016-10, Vol.59 (10), p.2156-2165 [Peer Reviewed Journal]

The Author(s) 2016 ;Springer-Verlag Berlin Heidelberg 2016 ;ISSN: 0012-186X ;EISSN: 1432-0428 ;DOI: 10.1007/s00125-016-4033-1 ;PMID: 27390011

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11
Nutrient-Induced Cellular Mechanisms of Gut Hormone Secretion
Nutrient-Induced Cellular Mechanisms of Gut Hormone Secretion
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Nutrient-Induced Cellular Mechanisms of Gut Hormone Secretion

Nutrients, 2021-03, Vol.13 (3), p.883 [Peer Reviewed Journal]

2021 by the authors. Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (http://creativecommons.org/licenses/by/4.0/). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2021 by the authors. 2021 ;ISSN: 2072-6643 ;EISSN: 2072-6643 ;DOI: 10.3390/nu13030883 ;PMID: 33803183

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12
Nutritional regulation of glucagon-like peptide-1 secretion
Nutritional regulation of glucagon-like peptide-1 secretion
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Nutritional regulation of glucagon-like peptide-1 secretion

The Journal of physiology, 2009-01, Vol.587 (1), p.27-32 [Peer Reviewed Journal]

2009 The Authors. Journal compilation © 2009 The Physiological Society ;2009 The Authors. Journal compilation © 2009 The Physiological Society 2009 ;ISSN: 0022-3751 ;EISSN: 1469-7793 ;DOI: 10.1113/jphysiol.2008.164012 ;PMID: 19001044

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13
A cross-platform approach identifies genetic regulators of human metabolism and health
A cross-platform approach identifies genetic regulators of human metabolism and health
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A cross-platform approach identifies genetic regulators of human metabolism and health

Nature genetics, 2021-01, Vol.53 (1), p.54-64 [Peer Reviewed Journal]

COPYRIGHT 2021 Nature Publishing Group ;Copyright Nature Publishing Group Jan 2021 ;ISSN: 1061-4036 ;EISSN: 1546-1718 ;DOI: 10.1038/s41588-020-00751-5 ;PMID: 33414548

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14
Na+-D-glucose Cotransporter SGLT1 is Pivotal for Intestinal Glucose Absorption and Glucose-Dependent Incretin Secretion
Na+-D-glucose Cotransporter SGLT1 is Pivotal for Intestinal Glucose Absorption and Glucose-Dependent Incretin Secretion
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Na+-D-glucose Cotransporter SGLT1 is Pivotal for Intestinal Glucose Absorption and Glucose-Dependent Incretin Secretion

Diabetes (New York, N.Y.), 2012-01, Vol.61 (1), p.187-196 [Peer Reviewed Journal]

2015 INIST-CNRS ;Copyright American Diabetes Association Jan 2012 ;2012 by the American Diabetes Association. 2012 ;ISSN: 0012-1797 ;EISSN: 1939-327X ;DOI: 10.2337/db11-1029 ;PMID: 22124465 ;CODEN: DIAEAZ

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15
Distribution and characterisation of Glucagon-like peptide-1 receptor expressing cells in the mouse brain
Distribution and characterisation of Glucagon-like peptide-1 receptor expressing cells in the mouse brain
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Distribution and characterisation of Glucagon-like peptide-1 receptor expressing cells in the mouse brain

Molecular metabolism (Germany), 2015-10, Vol.4 (10), p.718-731 [Peer Reviewed Journal]

The Authors ;2015 The Authors ;2015 The Authors 2015 ;ISSN: 2212-8778 ;EISSN: 2212-8778 ;DOI: 10.1016/j.molmet.2015.07.008 ;PMID: 26500843

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16
A brainstem circuit for nausea suppression
A brainstem circuit for nausea suppression
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A brainstem circuit for nausea suppression

Cell reports (Cambridge), 2022-06, Vol.39 (11), p.110953-110953, Article 110953 [Peer Reviewed Journal]

2022 The Author(s) ;Copyright © 2022 The Author(s). Published by Elsevier Inc. All rights reserved. ;ISSN: 2211-1247 ;EISSN: 2211-1247 ;DOI: 10.1016/j.celrep.2022.110953 ;PMID: 35705049

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17
Interleukin-6 enhances insulin secretion by increasing glucagon-like peptide-1 secretion from L cells and alpha cells
Interleukin-6 enhances insulin secretion by increasing glucagon-like peptide-1 secretion from L cells and alpha cells
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Interleukin-6 enhances insulin secretion by increasing glucagon-like peptide-1 secretion from L cells and alpha cells

Nature medicine, 2011-11, Vol.17 (11), p.1481-1489 [Peer Reviewed Journal]

COPYRIGHT 2011 Nature Publishing Group ;COPYRIGHT 2011 Nature Publishing Group ;Copyright Nature Publishing Group Nov 2011 ;ISSN: 1078-8956 ;EISSN: 1546-170X ;DOI: 10.1038/nm.2513 ;PMID: 22037645

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18
Insulin inhibits glucagon release by SGLT2-induced stimulation of somatostatin secretion
Insulin inhibits glucagon release by SGLT2-induced stimulation of somatostatin secretion
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Insulin inhibits glucagon release by SGLT2-induced stimulation of somatostatin secretion

Nature communications, 2019-01, Vol.10 (1), p.139-139, Article 139 [Peer Reviewed Journal]

This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2019 ;ISSN: 2041-1723 ;EISSN: 2041-1723 ;DOI: 10.1038/s41467-018-08193-8 ;PMID: 30635569

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19
Chylomicrons stimulate incretin secretion in mouse and human cells
Chylomicrons stimulate incretin secretion in mouse and human cells
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Chylomicrons stimulate incretin secretion in mouse and human cells

Diabetologia, 2017-12, Vol.60 (12), p.2475-2485 [Peer Reviewed Journal]

The Author(s) 2017 ;Diabetologia is a copyright of Springer, 2017. ;Distributed under a Creative Commons Attribution 4.0 International License ;ISSN: 0012-186X ;EISSN: 1432-0428 ;DOI: 10.1007/s00125-017-4420-2 ;PMID: 28866808

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20
Models and Tools for Studying Enteroendocrine Cells
Models and Tools for Studying Enteroendocrine Cells
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Models and Tools for Studying Enteroendocrine Cells

Endocrinology (Philadelphia), 2018-12, Vol.159 (12), p.3874-3884 [Peer Reviewed Journal]

Copyright © 2018 Endocrine Society 2018 ;Copyright © 2018 Endocrine Society ;ISSN: 1945-7170 ;ISSN: 0013-7227 ;EISSN: 1945-7170 ;DOI: 10.1210/en.2018-00672 ;PMID: 30239642

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