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0020. Microcirculatory perfusion and vascular reactivity are altered in post cardiac arrest patients, irrespective of target temperature management to 33° vs 36° (substudy TTM)
0020. Microcirculatory perfusion and vascular reactivity are altered in post cardiac arrest patients, irrespective of target temperature management to 33° vs 36° (substudy TTM)
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0020. Microcirculatory perfusion and vascular reactivity are altered in post cardiac arrest patients, irrespective of target temperature management to 33° vs 36° (substudy TTM)

Intensive care medicine experimental, 2014-09, Vol.2 (Suppl 1), Article O1 [Peer Reviewed Journal]

Koopmans et al; licensee Springer. 2014. This article is published under license to BioMed Central Ltd. This is an Open Access article distributed under the terms of the Creative Commons Attribution License ( ), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited. ;Koopmans et al; licensee Springer. 2014 ;ISSN: 2197-425X ;EISSN: 2197-425X ;DOI: 10.1186/2197-425X-2-S1-O1 ;PMID: 27484293

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0026. Voluntary activation of the sympathetic nervous system and attenuation of the innate immune response in humans
0026. Voluntary activation of the sympathetic nervous system and attenuation of the innate immune response in humans
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0026. Voluntary activation of the sympathetic nervous system and attenuation of the innate immune response in humans

Intensive care medicine experimental, 2014-09, Vol.2 (Suppl 1), p.O2 [Peer Reviewed Journal]

Kox et al; licensee Springer. 2014. This article is published under license to BioMed Central Ltd. This is an Open Access article distributed under the terms of the Creative Commons Attribution License ( ), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited. ;Kox et al; licensee Springer. 2014 ;ISSN: 2197-425X ;EISSN: 2197-425X ;DOI: 10.1186/2197-425X-2-S1-O2

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3
0027. Kinetic of muscle mass regulation during experimental sepsis
0027. Kinetic of muscle mass regulation during experimental sepsis
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0027. Kinetic of muscle mass regulation during experimental sepsis

Intensive care medicine experimental, 2014-09, Vol.2 (Suppl 1), p.O3 [Peer Reviewed Journal]

Palao et al; licensee Springer. 2014. This article is published under license to BioMed Central Ltd. This is an Open Access article distributed under the terms of the Creative Commons Attribution License ( ), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited. ;Palao et al; licensee Springer. 2014 ;ISSN: 2197-425X ;EISSN: 2197-425X ;DOI: 10.1186/2197-425X-2-S1-O3

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4
0030. Effect of exercise training on muscle function in a recovery model of critical illness
0030. Effect of exercise training on muscle function in a recovery model of critical illness
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0030. Effect of exercise training on muscle function in a recovery model of critical illness

Intensive care medicine experimental, 2014-09, Vol.2 (Suppl 1), Article O4 [Peer Reviewed Journal]

Sigurta et al; licensee Springer. 2014. This article is published under license to BioMed Central Ltd. This is an Open Access article distributed under the terms of the Creative Commons Attribution License ( ), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited. ;Sigurta et al; licensee Springer. 2014 ;ISSN: 2197-425X ;EISSN: 2197-425X ;DOI: 10.1186/2197-425X-2-S1-O4

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5
0032. Relationship between microcirculatory alterations and venous-to-arterial carbon dioxide differences in patients with septic shock
0032. Relationship between microcirculatory alterations and venous-to-arterial carbon dioxide differences in patients with septic shock
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0032. Relationship between microcirculatory alterations and venous-to-arterial carbon dioxide differences in patients with septic shock

Intensive care medicine experimental, 2014-09, Vol.2 (Suppl 1), Article O5 [Peer Reviewed Journal]

Ospina-Tascón et al; licensee Springer. 2014. This article is published under license to BioMed Central Ltd. This is an Open Access article distributed under the terms of the Creative Commons Attribution License ( ), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited. ;Ospina-Tascón et al; licensee Springer. 2014 ;ISSN: 2197-425X ;EISSN: 2197-425X ;DOI: 10.1186/2197-425X-2-S1-O5

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6
0033. Hypothermia protects brain mitochondrial function from hypoxia in sepsis
0033. Hypothermia protects brain mitochondrial function from hypoxia in sepsis
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0033. Hypothermia protects brain mitochondrial function from hypoxia in sepsis

Intensive care medicine experimental, 2014-09, Vol.2 (Suppl 1), Article O6 [Peer Reviewed Journal]

Chisholm et al; licensee Springer. 2014. This article is published under license to BioMed Central Ltd. This is an Open Access article distributed under the terms of the Creative Commons Attribution License ( ), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited. ;Chisholm et al; licensee Springer. 2014 ;ISSN: 2197-425X ;EISSN: 2197-425X ;DOI: 10.1186/2197-425X-2-S1-O6

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7
0034. Changes of TLR2, TLR4, MYD88 MRNA expressions on peripheral blood mononuclear cell in severe sepsis patients during treatment with thymosin α1
0034. Changes of TLR2, TLR4, MYD88 MRNA expressions on peripheral blood mononuclear cell in severe sepsis patients during treatment with thymosin α1
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0034. Changes of TLR2, TLR4, MYD88 MRNA expressions on peripheral blood mononuclear cell in severe sepsis patients during treatment with thymosin α1

Intensive care medicine experimental, 2014-09, Vol.2 (Suppl 1), Article O7 [Peer Reviewed Journal]

Tang et al; licensee Springer. 2014. This article is published under license to BioMed Central Ltd. This is an Open Access article distributed under the terms of the Creative Commons Attribution License ( ), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited. ;Tang et al; licensee Springer. 2014 ;ISSN: 2197-425X ;EISSN: 2197-425X ;DOI: 10.1186/2197-425X-2-S1-O7

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8
0035. Mitochondrial uncoupling contributes to fever in sepsis
0035. Mitochondrial uncoupling contributes to fever in sepsis
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0035. Mitochondrial uncoupling contributes to fever in sepsis

Intensive care medicine experimental, 2014-09, Vol.2 (Suppl 1), Article O8 [Peer Reviewed Journal]

Greco et al; licensee Springer. 2014. This article is published under license to BioMed Central Ltd. This is an Open Access article distributed under the terms of the Creative Commons Attribution License ( ), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited. ;Greco et al; licensee Springer. 2014 ;ISSN: 2197-425X ;EISSN: 2197-425X ;DOI: 10.1186/2197-425X-2-S1-O8

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9
0036. Confocal imaging of impaired mitochondrial function in the cerebral cortex of rats during haemorrhagic shock in vivo
0036. Confocal imaging of impaired mitochondrial function in the cerebral cortex of rats during haemorrhagic shock in vivo
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0036. Confocal imaging of impaired mitochondrial function in the cerebral cortex of rats during haemorrhagic shock in vivo

Intensive care medicine experimental, 2014-09, Vol.2 (Suppl 1), Article O9 [Peer Reviewed Journal]

Ida et al; licensee Springer. 2014. This article is published under license to BioMed Central Ltd. This is an Open Access article distributed under the terms of the Creative Commons Attribution License ( ), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited. ;Ida et al; licensee Springer. 2014 ;ISSN: 2197-425X ;EISSN: 2197-425X ;DOI: 10.1186/2197-425X-2-S1-O9

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10
0041. The effect of low-dose hydrocortisone after severe burn injury: a microarray longitudinal study
0041. The effect of low-dose hydrocortisone after severe burn injury: a microarray longitudinal study
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0041. The effect of low-dose hydrocortisone after severe burn injury: a microarray longitudinal study

Intensive care medicine experimental, 2014-09, Vol.2 (Suppl 1), Article O10 [Peer Reviewed Journal]

Textoris et al; licensee Springer. 2014. This article is published under license to BioMed Central Ltd. This is an Open Access article distributed under the terms of the Creative Commons Attribution License ( ), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited. ;Textoris et al; licensee Springer. 2014 ;ISSN: 2197-425X ;EISSN: 2197-425X ;DOI: 10.1186/2197-425X-2-S1-O10

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11
0042. Burn injury stabilises extracellular atp and induces microvesicle production in skin
0042. Burn injury stabilises extracellular atp and induces microvesicle production in skin
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0042. Burn injury stabilises extracellular atp and induces microvesicle production in skin

Intensive care medicine experimental, 2014-09, Vol.2 (Suppl 1), Article O11 [Peer Reviewed Journal]

Katbeh et al; licensee Springer. 2014. This article is published under license to BioMed Central Ltd. This is an Open Access article distributed under the terms of the Creative Commons Attribution License ( ), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited. ;Katbeh et al; licensee Springer. 2014 ;ISSN: 2197-425X ;EISSN: 2197-425X ;DOI: 10.1186/2197-425X-2-S1-O11

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12
0078. Establishing a detailed short-term rat model of partial ischaemia/reperfusion injury
0078. Establishing a detailed short-term rat model of partial ischaemia/reperfusion injury
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0078. Establishing a detailed short-term rat model of partial ischaemia/reperfusion injury

Intensive care medicine experimental, 2014-09, Vol.2 (Suppl 1), Article P1 [Peer Reviewed Journal]

Sabbatini et al; licensee Springer. 2014. This article is published under license to BioMed Central Ltd. This is an Open Access article distributed under the terms of the Creative Commons Attribution License ( ), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited. ;Sabbatini et al; licensee Springer. 2014 ;ISSN: 2197-425X ;EISSN: 2197-425X ;DOI: 10.1186/2197-425X-2-S1-P1

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13
0082. Early circulating lipid and cytokine profiles prognosticate in a rat model of faecal peritonitis
0082. Early circulating lipid and cytokine profiles prognosticate in a rat model of faecal peritonitis
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0082. Early circulating lipid and cytokine profiles prognosticate in a rat model of faecal peritonitis

Intensive care medicine experimental, 2014-09, Vol.2 (Suppl 1), Article P2 [Peer Reviewed Journal]

Khaliq and Singer; licensee Springer. 2014. This article is published under license to BioMed Central Ltd. This is an Open Access article distributed under the terms of the Creative Commons Attribution License ( ), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited. ;Khaliq and Singer; licensee Springer. 2014 ;ISSN: 2197-425X ;EISSN: 2197-425X ;DOI: 10.1186/2197-425X-2-S1-P2

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14
0083. Hepatoprotective effects of hydrogen sulphide against acute liver failure
0083. Hepatoprotective effects of hydrogen sulphide against acute liver failure
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0083. Hepatoprotective effects of hydrogen sulphide against acute liver failure

Intensive care medicine experimental, 2014-09, Vol.2 (Suppl 1), Article P3 [Peer Reviewed Journal]

Tokuda and Ichinose; licensee Springer. 2014. This article is published under license to BioMed Central Ltd. This is an Open Access article distributed under the terms of the Creative Commons Attribution License ( ), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited. ;Tokuda and Ichinose; licensee Springer. 2014 ;ISSN: 2197-425X ;EISSN: 2197-425X ;DOI: 10.1186/2197-425X-2-S1-P3

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15
0092. Ischemic pre/post-conditioning protects the microcirculation in experimental sepsis
0092. Ischemic pre/post-conditioning protects the microcirculation in experimental sepsis
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0092. Ischemic pre/post-conditioning protects the microcirculation in experimental sepsis

Intensive care medicine experimental, 2014-09, Vol.2 (Suppl 1), Article P4 [Peer Reviewed Journal]

Cortes et al; licensee Springer. 2014. This article is published under license to BioMed Central Ltd. This is an Open Access article distributed under the terms of the Creative Commons Attribution License ( ), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited. ;Cortes et al; licensee Springer. 2014 ;ISSN: 2197-425X ;EISSN: 2197-425X ;DOI: 10.1186/2197-425X-2-S1-P4

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16
0093. Mitochondrial function of immune cells in severe sepsis and septic shock - a prospective observational cohort study
0093. Mitochondrial function of immune cells in severe sepsis and septic shock - a prospective observational cohort study
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0093. Mitochondrial function of immune cells in severe sepsis and septic shock - a prospective observational cohort study

Intensive care medicine experimental, 2014-09, Vol.2 (Suppl 1), Article P5 [Peer Reviewed Journal]

Merz et al; licensee Springer. 2014. This article is published under license to BioMed Central Ltd. This is an Open Access article distributed under the terms of the Creative Commons Attribution License ( ), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited. ;Merz et al; licensee Springer. 2014 ;ISSN: 2197-425X ;EISSN: 2197-425X ;DOI: 10.1186/2197-425X-2-S1-P5

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17
0094. Monocyte tace activity profile during sepsis and systemic inflammatory response syndrome
0094. Monocyte tace activity profile during sepsis and systemic inflammatory response syndrome
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0094. Monocyte tace activity profile during sepsis and systemic inflammatory response syndrome

Intensive care medicine experimental, 2014-09, Vol.2 (Suppl 1), Article P6 [Peer Reviewed Journal]

O'Callaghan et al; licensee Springer. 2014. This article is published under license to BioMed Central Ltd. This is an Open Access article distributed under the terms of the Creative Commons Attribution License ( ), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited. ;O'Callaghan et al; licensee Springer. 2014 ;ISSN: 2197-425X ;EISSN: 2197-425X ;DOI: 10.1186/2197-425X-2-S1-P6

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18
0095. Transvenous vagus nerve stimulation does not modulate the innate immune response in humans in vivo during experimental endotoxemia
0095. Transvenous vagus nerve stimulation does not modulate the innate immune response in humans in vivo during experimental endotoxemia
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0095. Transvenous vagus nerve stimulation does not modulate the innate immune response in humans in vivo during experimental endotoxemia

Intensive care medicine experimental, 2014-09, Vol.2 (Suppl 1), Article P7 [Peer Reviewed Journal]

Kox et al; licensee Springer. 2014. This article is published under license to BioMed Central Ltd. This is an Open Access article distributed under the terms of the Creative Commons Attribution License ( ), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited. ;Kox et al; licensee Springer. 2014 ;ISSN: 2197-425X ;EISSN: 2197-425X ;DOI: 10.1186/2197-425X-2-S1-P7

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19
0096. Evaluation by videomicroscopy (SDF) of the renal cortex microcirculation and convoluted tubules in acute renal failure during severe sepsis. Experimental study
0096. Evaluation by videomicroscopy (SDF) of the renal cortex microcirculation and convoluted tubules in acute renal failure during severe sepsis. Experimental study
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0096. Evaluation by videomicroscopy (SDF) of the renal cortex microcirculation and convoluted tubules in acute renal failure during severe sepsis. Experimental study

Intensive care medicine experimental, 2014-09, Vol.2 (Suppl 1), Article P8 [Peer Reviewed Journal]

Liberatore et al; licensee Springer. 2014. This article is published under license to BioMed Central Ltd. This is an Open Access article distributed under the terms of the Creative Commons Attribution License ( ), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited. ;Liberatore et al; licensee Springer. 2014 ;ISSN: 2197-425X ;EISSN: 2197-425X ;DOI: 10.1186/2197-425X-2-S1-P8

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20
0097. Myeloid-derived suppressor cells attenuate inflammation and improve the survival of microbial sepsis
0097. Myeloid-derived suppressor cells attenuate inflammation and improve the survival of microbial sepsis
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0097. Myeloid-derived suppressor cells attenuate inflammation and improve the survival of microbial sepsis

Intensive care medicine experimental, 2014-09, Vol.2 (Suppl 1), Article P9 [Peer Reviewed Journal]

Tong et al; licensee Springer. 2014. This article is published under license to BioMed Central Ltd. This is an Open Access article distributed under the terms of the Creative Commons Attribution License ( ), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited. ;Tong et al; licensee Springer. 2014 ;ISSN: 2197-425X ;EISSN: 2197-425X ;DOI: 10.1186/2197-425X-2-S1-P9

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