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1
Trends in diagnosis, referral, red flag onset, patient profiles and natural outcome of de novo cardiac amyloidosis and their multidisciplinary implications
Trends in diagnosis, referral, red flag onset, patient profiles and natural outcome of de novo cardiac amyloidosis and their multidisciplinary implications
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Trends in diagnosis, referral, red flag onset, patient profiles and natural outcome of de novo cardiac amyloidosis and their multidisciplinary implications

embargo 640030 ;ISSN: 1784-973X ;ISSN: 0001-5385 ;EISSN: 1784-973X ;DOI: 10.1080/00015385.2021.1976450

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2
Exploration and validation of signature genes and immune associations in septic cardiomyopathy
Exploration and validation of signature genes and immune associations in septic cardiomyopathy
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Exploration and validation of signature genes and immune associations in septic cardiomyopathy

Clinical and experimental hypertension (1993), 2024-12, Vol.46 (1), p.2302570-2302570 [Peer Reviewed Journal]

ISSN: 1064-1963 ;EISSN: 1525-6006 ;DOI: 10.1080/10641963.2024.2302570 ;PMID: 38281072

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3
Catheter Ablation for Ventricular Tachycardia in Patients With Desmoplakin Cardiomyopathy
Catheter Ablation for Ventricular Tachycardia in Patients With Desmoplakin Cardiomyopathy
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Catheter Ablation for Ventricular Tachycardia in Patients With Desmoplakin Cardiomyopathy

ISSN: 2405-500X ;EISSN: 2405-5018 ;DOI: 10.1016/j.jacep.2023.11.017 ;DOI: 10.5167/uzh-257657

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4
Current advances on the therapeutic potential of scutellarin: an updated review
Current advances on the therapeutic potential of scutellarin: an updated review
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Current advances on the therapeutic potential of scutellarin: an updated review

Natural products and bioprospecting, 2024-12, Vol.14 (1), p.20-20 [Peer Reviewed Journal]

The Author(s) 2024 ;2024. The Author(s). ;The Author(s) 2024. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;ISSN: 2192-2195 ;EISSN: 2192-2209 ;DOI: 10.1007/s13659-024-00441-3 ;PMID: 38436812

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5
Post translational modifications of connexin 43 in ventricular arrhythmias after myocardial infarction
Post translational modifications of connexin 43 in ventricular arrhythmias after myocardial infarction
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Post translational modifications of connexin 43 in ventricular arrhythmias after myocardial infarction

Molecular biology reports, 2024-12, Vol.51 (1), p.329-329 [Peer Reviewed Journal]

The Author(s), under exclusive licence to Springer Nature B.V. 2024. Springer Nature or its licensor (e.g. a society or other partner) holds exclusive rights to this article under a publishing agreement with the author(s) or other rightsholder(s); author self-archiving of the accepted manuscript version of this article is solely governed by the terms of such publishing agreement and applicable law. ;2024. The Author(s), under exclusive licence to Springer Nature B.V. ;ISSN: 0301-4851 ;EISSN: 1573-4978 ;DOI: 10.1007/s11033-024-09290-2 ;PMID: 38393658

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6
Clinicopathological alterations in wild mammals from the reservoir system of Trypanosoma cruzi: a scoping review
Clinicopathological alterations in wild mammals from the reservoir system of Trypanosoma cruzi: a scoping review
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Clinicopathological alterations in wild mammals from the reservoir system of Trypanosoma cruzi: a scoping review

Animal diseases, 2024-12, Vol.4 (1), p.6-12 [Peer Reviewed Journal]

The Author(s) 2024 ;The Author(s) 2024. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;EISSN: 2731-0442 ;DOI: 10.1186/s44149-024-00111-4

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7
Trisomy 21-driven metabolite alterations are linked to cellular injuries in Down syndrome
Trisomy 21-driven metabolite alterations are linked to cellular injuries in Down syndrome
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Trisomy 21-driven metabolite alterations are linked to cellular injuries in Down syndrome

Cellular and molecular life sciences : CMLS, 2024-12, Vol.81 (1), p.112-112 [Peer Reviewed Journal]

The Author(s) 2024 ;2024. The Author(s). ;The Author(s) 2024. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;ISSN: 1420-682X ;EISSN: 1420-9071 ;DOI: 10.1007/s00018-024-05127-0 ;PMID: 38433139

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8
STAT protein family and cardiovascular diseases: overview of pathological mechanisms and therapeutic implications
STAT protein family and cardiovascular diseases: overview of pathological mechanisms and therapeutic implications
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STAT protein family and cardiovascular diseases: overview of pathological mechanisms and therapeutic implications

Molecular biology reports, 2024-12, Vol.51 (1), p.440-440 [Peer Reviewed Journal]

The Author(s), under exclusive licence to Springer Nature B.V. 2024. Springer Nature or its licensor (e.g. a society or other partner) holds exclusive rights to this article under a publishing agreement with the author(s) or other rightsholder(s); author self-archiving of the accepted manuscript version of this article is solely governed by the terms of such publishing agreement and applicable law. ;2024. The Author(s), under exclusive licence to Springer Nature B.V. ;ISSN: 0301-4851 ;EISSN: 1573-4978 ;DOI: 10.1007/s11033-024-09371-2 ;PMID: 38520542

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9
ALKBH1 promotes HIF-1α-mediated glycolysis by inhibiting N-glycosylation of LAMP2A
ALKBH1 promotes HIF-1α-mediated glycolysis by inhibiting N-glycosylation of LAMP2A
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ALKBH1 promotes HIF-1α-mediated glycolysis by inhibiting N-glycosylation of LAMP2A

Cellular and molecular life sciences : CMLS, 2024-12, Vol.81 (1), p.130-130 [Peer Reviewed Journal]

The Author(s) 2024 ;2024. The Author(s). ;The Author(s) 2024. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;ISSN: 1420-682X ;EISSN: 1420-9071 ;DOI: 10.1007/s00018-024-05152-z ;PMID: 38472355

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10
SIRT3/6: an amazing challenge and opportunity in the fight against fibrosis and aging
SIRT3/6: an amazing challenge and opportunity in the fight against fibrosis and aging
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SIRT3/6: an amazing challenge and opportunity in the fight against fibrosis and aging

Cellular and molecular life sciences : CMLS, 2024-12, Vol.81 (1), p.69-69, Article 69 [Peer Reviewed Journal]

The Author(s) 2024 ;2024. The Author(s). ;The Author(s) 2024. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;ISSN: 1420-682X ;EISSN: 1420-9071 ;DOI: 10.1007/s00018-023-05093-z ;PMID: 38294557

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11
Neutrophil extracellular traps mediate cardiomyocyte ferroptosis via the Hippo–Yap pathway to exacerbate doxorubicin-induced cardiotoxicity
Neutrophil extracellular traps mediate cardiomyocyte ferroptosis via the Hippo–Yap pathway to exacerbate doxorubicin-induced cardiotoxicity
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Neutrophil extracellular traps mediate cardiomyocyte ferroptosis via the Hippo–Yap pathway to exacerbate doxorubicin-induced cardiotoxicity

Cellular and molecular life sciences : CMLS, 2024-12, Vol.81 (1), p.122-122 [Peer Reviewed Journal]

The Author(s) 2024 ;2024. The Author(s). ;The Author(s) 2024. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;ISSN: 1420-682X ;EISSN: 1420-9071 ;DOI: 10.1007/s00018-024-05169-4 ;PMID: 38456997

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12
Protective effects of arbutin against doxorubicin-induced cardiac damage
Protective effects of arbutin against doxorubicin-induced cardiac damage
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Protective effects of arbutin against doxorubicin-induced cardiac damage

Molecular biology reports, 2024-12, Vol.51 (1), p.532 [Peer Reviewed Journal]

The Author(s), under exclusive licence to Springer Nature B.V. 2024. Springer Nature or its licensor (e.g. a society or other partner) holds exclusive rights to this article under a publishing agreement with the author(s) or other rightsholder(s); author self-archiving of the accepted manuscript version of this article is solely governed by the terms of such publishing agreement and applicable law. ;2024. The Author(s), under exclusive licence to Springer Nature B.V. ;ISSN: 0301-4851 ;EISSN: 1573-4978 ;DOI: 10.1007/s11033-024-09488-4 ;PMID: 38637360

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13
An update of new/potential cardiovascular markers: a narrative review
An update of new/potential cardiovascular markers: a narrative review
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An update of new/potential cardiovascular markers: a narrative review

Molecular biology reports, 2024-12, Vol.51 (1), p.179-179, Article 179 [Peer Reviewed Journal]

The Author(s), under exclusive licence to Springer Nature B.V. 2024. Springer Nature or its licensor (e.g. a society or other partner) holds exclusive rights to this article under a publishing agreement with the author(s) or other rightsholder(s); author self-archiving of the accepted manuscript version of this article is solely governed by the terms of such publishing agreement and applicable law. ;2024. The Author(s), under exclusive licence to Springer Nature B.V. ;ISSN: 0301-4851 ;EISSN: 1573-4978 ;DOI: 10.1007/s11033-023-08978-1 ;PMID: 38252393

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14
Association of SLC30A8 rs13266634 gene polymorphism with type 2 diabetes mellitus (T2DM) in a population of Noakhali, Bangladesh: a case–control study
Association of SLC30A8 rs13266634 gene polymorphism with type 2 diabetes mellitus (T2DM) in a population of Noakhali, Bangladesh: a case–control study
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Association of SLC30A8 rs13266634 gene polymorphism with type 2 diabetes mellitus (T2DM) in a population of Noakhali, Bangladesh: a case–control study

Egyptian Journal of Medical Human Genetics, 2024-12, Vol.25 (1), p.22-11 [Peer Reviewed Journal]

The Author(s) 2024 ;COPYRIGHT 2024 Springer ;The Author(s) 2024. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;ISSN: 1110-8630 ;EISSN: 2090-2441 ;DOI: 10.1186/s43042-024-00484-8

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15
Assessment of left atrial function by two-dimensional speckle tracking echocardiography in patients with metabolic-associated fatty liver disease
Assessment of left atrial function by two-dimensional speckle tracking echocardiography in patients with metabolic-associated fatty liver disease
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Assessment of left atrial function by two-dimensional speckle tracking echocardiography in patients with metabolic-associated fatty liver disease

The Egyptian heart journal, 2024-12, Vol.76 (1), p.42-42 [Peer Reviewed Journal]

The Author(s) 2024 ;2024. The Author(s). ;COPYRIGHT 2024 Springer ;The Author(s) 2024. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;ISSN: 1110-2608 ;EISSN: 2090-911X ;DOI: 10.1186/s43044-024-00462-w ;PMID: 38553577

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16
Does epigenetic markers of HLA gene show association with coronary artery disease in Indian subjects?
Does epigenetic markers of HLA gene show association with coronary artery disease in Indian subjects?
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Does epigenetic markers of HLA gene show association with coronary artery disease in Indian subjects?

Molecular biology reports, 2024-12, Vol.51 (1), p.173-173, Article 173 [Peer Reviewed Journal]

The Author(s), under exclusive licence to Springer Nature B.V. 2024. Springer Nature or its licensor (e.g. a society or other partner) holds exclusive rights to this article under a publishing agreement with the author(s) or other rightsholder(s); author self-archiving of the accepted manuscript version of this article is solely governed by the terms of such publishing agreement and applicable law. ;2024. The Author(s), under exclusive licence to Springer Nature B.V. ;ISSN: 0301-4851 ;EISSN: 1573-4978 ;DOI: 10.1007/s11033-023-08974-5 ;PMID: 38252175

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17
Over-expression of mir-181a-3p in serum of breast cancer patients as diagnostic biomarker
Over-expression of mir-181a-3p in serum of breast cancer patients as diagnostic biomarker
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Over-expression of mir-181a-3p in serum of breast cancer patients as diagnostic biomarker

Molecular biology reports, 2024-12, Vol.51 (1), p.372-372 [Peer Reviewed Journal]

The Author(s), under exclusive licence to Springer Nature B.V. 2024. Springer Nature or its licensor (e.g. a society or other partner) holds exclusive rights to this article under a publishing agreement with the author(s) or other rightsholder(s); author self-archiving of the accepted manuscript version of this article is solely governed by the terms of such publishing agreement and applicable law. ;2024. The Author(s), under exclusive licence to Springer Nature B.V. ;ISSN: 0301-4851 ;EISSN: 1573-4978 ;DOI: 10.1007/s11033-024-09272-4 ;PMID: 38411870

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18
Appropriate timing of veno-arterial extracorporeal membrane oxygenation initiation after cardiac surgery
Appropriate timing of veno-arterial extracorporeal membrane oxygenation initiation after cardiac surgery
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Appropriate timing of veno-arterial extracorporeal membrane oxygenation initiation after cardiac surgery

Cardiothoracic Surgeon, 2024-12, Vol.32 (1), p.2-9, Article 2 [Peer Reviewed Journal]

The Author(s) 2024 ;COPYRIGHT 2024 Springer ;The Author(s) 2024. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;ISSN: 2662-2203 ;ISSN: 2636-333X ;EISSN: 2662-2203 ;DOI: 10.1186/s43057-023-00120-y

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19
Nitro-fatty acids: mechanisms of action, roles in metabolic diseases, and therapeutics
Nitro-fatty acids: mechanisms of action, roles in metabolic diseases, and therapeutics
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Article 		Add to My Research

Nitro-fatty acids: mechanisms of action, roles in metabolic diseases, and therapeutics

Current medicine (Cham, Switzerland), 2024-12, Vol.3 (1), p.3 [Peer Reviewed Journal]

The Author(s) 2024 ;The Author(s) 2024. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;EISSN: 2731-0868 ;DOI: 10.1007/s44194-024-00030-z

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20
Epicardial fat thickness among neonates of diabetic mothers attending the neonatal intensive care unit at Fayoum University Hospital: a case control study
Epicardial fat thickness among neonates of diabetic mothers attending the neonatal intensive care unit at Fayoum University Hospital: a case control study
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Article 		Add to My Research

Epicardial fat thickness among neonates of diabetic mothers attending the neonatal intensive care unit at Fayoum University Hospital: a case control study

The Gazette of the Egyptian Paediatric Association, 2024-12, Vol.72 (1), p.13-10 [Peer Reviewed Journal]

The Author(s) 2024 ;COPYRIGHT 2024 Springer ;The Author(s) 2024. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;ISSN: 1110-6638 ;EISSN: 2090-9942 ;DOI: 10.1186/s43054-024-00254-6

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