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1
0-2-Pyridyl thionophosphate and thionophosphonate ester(s) - prepd. by phosphorylation of 2-hydroxy-pyridine, useful as insecticides and acaricides
0-2-Pyridyl thionophosphate and thionophosphonate ester(s) - prepd. by phosphorylation of 2-hydroxy-pyridine, useful as insecticides and acaricides
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0-2-Pyridyl thionophosphate and thionophosphonate ester(s) - prepd. by phosphorylation of 2-hydroxy-pyridine, useful as insecticides and acaricides

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2
0,0'-Dicyanoethyl-phosphoramidites, a process for their preparation and their use in phosphorylation reactions
0,0'-Dicyanoethyl-phosphoramidites, a process for their preparation and their use in phosphorylation reactions
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0,0'-Dicyanoethyl-phosphoramidites, a process for their preparation and their use in phosphorylation reactions

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3
0,0'-Dicyanoethyl-phosphoramidites, a process for their preparation and their use in phosphorylation reactions
0,0'-Dicyanoethyl-phosphoramidites, a process for their preparation and their use in phosphorylation reactions
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0,0'-Dicyanoethyl-phosphoramidites, a process for their preparation and their use in phosphorylation reactions

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4
0004 Salidroside Ameliorates Chronic Intermittent Hypoxia-induced Endothelial Insulin Resistance via Suppression of ERK1/2 Activation
0004 Salidroside Ameliorates Chronic Intermittent Hypoxia-induced Endothelial Insulin Resistance via Suppression of ERK1/2 Activation
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0004 Salidroside Ameliorates Chronic Intermittent Hypoxia-induced Endothelial Insulin Resistance via Suppression of ERK1/2 Activation

Sleep (New York, N.Y.), 2018-04, Vol.41 (suppl_1), p.A1-A2 [Peer Reviewed Journal]

Sleep Research Society 2018. Published by Oxford University Press [on behalf of the Sleep Research Society]. All rights reserved. For permissions, please email: journals.permissions@oup.com 2018 ;Copyright © 2018 Sleep Research Society ;ISSN: 0161-8105 ;EISSN: 1550-9109 ;DOI: 10.1093/sleep/zsy061.003

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5
0005 Efficacy of SM-1 in a Transient Insomnia Model
0005 Efficacy of SM-1 in a Transient Insomnia Model
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0005 Efficacy of SM-1 in a Transient Insomnia Model

Sleep (New York, N.Y.), 2018-04, Vol.41 (suppl_1), p.A2-A2 [Peer Reviewed Journal]

Sleep Research Society 2018. Published by Oxford University Press [on behalf of the Sleep Research Society]. All rights reserved. For permissions, please email: journals.permissions@oup.com 2018 ;Copyright © 2018 Sleep Research Society ;ISSN: 0161-8105 ;EISSN: 1550-9109 ;DOI: 10.1093/sleep/zsy061.004

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6
0006 The Pharmacokinetics and Pharmacodynamics of SM-1
0006 The Pharmacokinetics and Pharmacodynamics of SM-1
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0006 The Pharmacokinetics and Pharmacodynamics of SM-1

Sleep (New York, N.Y.), 2018-04, Vol.41 (suppl_1), p.A2-A3 [Peer Reviewed Journal]

Sleep Research Society 2018. Published by Oxford University Press [on behalf of the Sleep Research Society]. All rights reserved. For permissions, please email: journals.permissions@oup.com 2018 ;Copyright © 2018 Sleep Research Society ;ISSN: 0161-8105 ;EISSN: 1550-9109 ;DOI: 10.1093/sleep/zsy061.005

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7
0035. Mitochondrial uncoupling contributes to fever in sepsis
0035. Mitochondrial uncoupling contributes to fever in sepsis
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0035. Mitochondrial uncoupling contributes to fever in sepsis

Intensive care medicine experimental, 2014-09, Vol.2 (Suppl 1), Article O8 [Peer Reviewed Journal]

Greco et al; licensee Springer. 2014. This article is published under license to BioMed Central Ltd. This is an Open Access article distributed under the terms of the Creative Commons Attribution License ( ), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited. ;Greco et al; licensee Springer. 2014 ;ISSN: 2197-425X ;EISSN: 2197-425X ;DOI: 10.1186/2197-425X-2-S1-O8

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8
005 Nox2-deficient bone marrow-derived macrophages exhibit defects in cell spreading and migration
005 Nox2-deficient bone marrow-derived macrophages exhibit defects in cell spreading and migration
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005 Nox2-deficient bone marrow-derived macrophages exhibit defects in cell spreading and migration

Heart (British Cardiac Society), 2010-02, Vol.96 (4), p.e2-e2 [Peer Reviewed Journal]

2009, Published by the BMJ Publishing Group Limited For permission to use, (where not already granted under a licence) please go to http://group.bmj.com/group/rights-licensing/permissions. ;Copyright: 2009 (c) 2009, Published by the BMJ Publishing Group Limited For permission to use, (where not already granted under a licence) please go to http://group.bmj.com/group/rights-licensing/permissions. ;ISSN: 1355-6037 ;EISSN: 1468-201X ;DOI: 10.1136/hrt.2009.191049e

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9
007 Roles of P47phox S303/S304 phosphorylation in TNF -induced endothelial reactive oxygen species production and mitogen-activated protein kinase activation
007 Roles of P47phox S303/S304 phosphorylation in TNF -induced endothelial reactive oxygen species production and mitogen-activated protein kinase activation
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007 Roles of P47phox S303/S304 phosphorylation in TNF -induced endothelial reactive oxygen species production and mitogen-activated protein kinase activation

Heart (British Cardiac Society), 2010-02, Vol.96 (4), p.e2-e3 [Peer Reviewed Journal]

Copyright: 2009 (c) 2009, Published by the BMJ Publishing Group Limited For permission to use, (where not already granted under a licence) please go to http://group.bmj.com/group/rights-licensing/permissions. ;ISSN: 1355-6037 ;EISSN: 1468-201X ;DOI: 10.1136/hrt.2009.191049g

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10
007 Roles of P47phox S303/S304 phosphorylation in TNFα-induced endothelial reactive oxygen species production and mitogen-activated protein kinase activation
007 Roles of P47phox S303/S304 phosphorylation in TNFα-induced endothelial reactive oxygen species production and mitogen-activated protein kinase activation
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007 Roles of P47phox S303/S304 phosphorylation in TNFα-induced endothelial reactive oxygen species production and mitogen-activated protein kinase activation

Heart (British Cardiac Society), 2010-02, Vol.96 (4), p.e2 [Peer Reviewed Journal]

2009, Published by the BMJ Publishing Group Limited For permission to use, (where not already granted under a licence) please go to http://group.bmj.com/group/rights-licensing/permissions. ;ISSN: 1355-6037 ;EISSN: 1468-201X ;DOI: 10.1136/hrt.2009.191049g

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11
0083. Hepatoprotective effects of hydrogen sulphide against acute liver failure
0083. Hepatoprotective effects of hydrogen sulphide against acute liver failure
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0083. Hepatoprotective effects of hydrogen sulphide against acute liver failure

Intensive care medicine experimental, 2014-09, Vol.2 (Suppl 1), Article P3 [Peer Reviewed Journal]

Tokuda and Ichinose; licensee Springer. 2014. This article is published under license to BioMed Central Ltd. This is an Open Access article distributed under the terms of the Creative Commons Attribution License ( ), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited. ;Tokuda and Ichinose; licensee Springer. 2014 ;ISSN: 2197-425X ;EISSN: 2197-425X ;DOI: 10.1186/2197-425X-2-S1-P3

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12
009The effect of cardiac magnetic resonance on human circulating leukocytes
009The effect of cardiac magnetic resonance on human circulating leukocytes
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009The effect of cardiac magnetic resonance on human circulating leukocytes

Heart (British Cardiac Society), 2017-04, Vol.103 (Suppl 1), p.A7-A8 [Peer Reviewed Journal]

ISSN: 1355-6037 ;EISSN: 1468-201X ;DOI: 10.1136/heartjnl-2017-311399.9

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13
009 The effect of cardiac magnetic resonance on human circulating leukocytes
009 The effect of cardiac magnetic resonance on human circulating leukocytes
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009 The effect of cardiac magnetic resonance on human circulating leukocytes

Heart (British Cardiac Society), 2017-04, Vol.103 (Suppl 1), p.A7-A8 [Peer Reviewed Journal]

2017, Published by the BMJ Publishing Group Limited. For permission to use (where not already granted under a licence) please go to http://group.bmj.com/group/rights-licensing/permissions ;ISSN: 1355-6037 ;EISSN: 1468-201X ;DOI: 10.1136/heartjnl-2017-311399.9

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14
01. Pathogenesis
01. Pathogenesis
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01. Pathogenesis

Journal of scleroderma and related disorders, 2018-02, Vol.3 (1_suppl), p.32-68 [Peer Reviewed Journal]

The Author(s) 2017 ;The Author(s) 2017 2017 SAGE Publications ;ISSN: 2397-1983 ;EISSN: 2397-1991 ;DOI: 10.1177/2397198317753309 ;PMID: 35381987

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15
01.13Igf1R signalling contributes to T cell dependent inflammation in rheumatoid arthritis
01.13Igf1R signalling contributes to T cell dependent inflammation in rheumatoid arthritis
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01.13Igf1R signalling contributes to T cell dependent inflammation in rheumatoid arthritis

Annals of the rheumatic diseases, 2017-03, Vol.76 (Suppl 1), p.A6-A6 [Peer Reviewed Journal]

ISSN: 0003-4967 ;EISSN: 1468-2060 ;DOI: 10.1136/annrheumdis-2016-211048.13

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16
01.13 Igf1R signalling contributes to T cell dependent inflammation in rheumatoid arthritis
01.13 Igf1R signalling contributes to T cell dependent inflammation in rheumatoid arthritis
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01.13 Igf1R signalling contributes to T cell dependent inflammation in rheumatoid arthritis

Annals of the rheumatic diseases, 2017-03, Vol.76 (Suppl 1), p.A6 [Peer Reviewed Journal]

2017, Published by the BMJ Publishing Group Limited. For permission to use (where not already granted under a licence) please go to http://group.bmj.com/group/rights-licensing/permissions ;Copyright: 2017 (c) 2017, Published by the BMJ Publishing Group Limited. For permission to use (where not already granted under a licence) please go to http://group.bmj.com/group/rights-licensing/permissions ;ISSN: 0003-4967 ;EISSN: 1468-2060 ;DOI: 10.1136/annrheumdis-2016-211048.13 ;CODEN: ARDIAO

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17
01.16Amp-activated protein kinase: An anti-inflammatory target for methotrexate in macrophages
01.16Amp-activated protein kinase: An anti-inflammatory target for methotrexate in macrophages
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01.16Amp-activated protein kinase: An anti-inflammatory target for methotrexate in macrophages

Annals of the rheumatic diseases, 2017-03, Vol.76 (Suppl 1), p.A7-A8 [Peer Reviewed Journal]

ISSN: 0003-4967 ;EISSN: 1468-2060 ;DOI: 10.1136/annrheumdis-2016-211048.16

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18
01.16 Amp-activated protein kinase: An anti-inflammatory target for methotrexate in macrophages
01.16 Amp-activated protein kinase: An anti-inflammatory target for methotrexate in macrophages
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01.16 Amp-activated protein kinase: An anti-inflammatory target for methotrexate in macrophages

Annals of the rheumatic diseases, 2017-03, Vol.76 (Suppl 1), p.A7 [Peer Reviewed Journal]

2017, Published by the BMJ Publishing Group Limited. For permission to use (where not already granted under a licence) please go to http://group.bmj.com/group/rights-licensing/permissions ;Copyright: 2017 (c) 2017, Published by the BMJ Publishing Group Limited. For permission to use (where not already granted under a licence) please go to http://group.bmj.com/group/rights-licensing/permissions ;ISSN: 0003-4967 ;EISSN: 1468-2060 ;DOI: 10.1136/annrheumdis-2016-211048.16 ;CODEN: ARDIAO

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19
011 A new molecular mechanism for familial dilated cardiomyopathy based on studies with intact mutant transgenic mouse and human explanted heart muscle
011 A new molecular mechanism for familial dilated cardiomyopathy based on studies with intact mutant transgenic mouse and human explanted heart muscle
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011 A new molecular mechanism for familial dilated cardiomyopathy based on studies with intact mutant transgenic mouse and human explanted heart muscle

Heart (British Cardiac Society), 2010-02, Vol.96 (4), p.e4-e4 [Peer Reviewed Journal]

2009, Published by the BMJ Publishing Group Limited For permission to use, (where not already granted under a licence) please go to http://group.bmj.com/group/rights-licensing/permissions. ;Copyright: 2009 (c) 2009, Published by the BMJ Publishing Group Limited For permission to use, (where not already granted under a licence) please go to http://group.bmj.com/group/rights-licensing/permissions. ;ISSN: 1355-6037 ;EISSN: 1468-201X ;DOI: 10.1136/hrt.2009.191049k

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20
014 Thin filaments reconstituted with troponin extracted from patients with hypertrophic obstructive cardiomyopathy are functionally abnormal
014 Thin filaments reconstituted with troponin extracted from patients with hypertrophic obstructive cardiomyopathy are functionally abnormal
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014 Thin filaments reconstituted with troponin extracted from patients with hypertrophic obstructive cardiomyopathy are functionally abnormal

Heart (British Cardiac Society), 2010-02, Vol.96 (4), p.e4-e5 [Peer Reviewed Journal]

2009, Published by the BMJ Publishing Group Limited For permission to use, (where not already granted under a licence) please go to http://group.bmj.com/group/rights-licensing/permissions. ;Copyright: 2009 (c) 2009, Published by the BMJ Publishing Group Limited For permission to use, (where not already granted under a licence) please go to http://group.bmj.com/group/rights-licensing/permissions. ;ISSN: 1355-6037 ;EISSN: 1468-201X ;DOI: 10.1136/hrt.2009.191064b

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