Result Number | Material Type | Add to My Shelf Action | Record Details and Options |
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1 |
Material Type: Article
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The matricellular protein CCN3 supports lung endothelial homeostasis and functionAmerican journal of physiology. Lung cellular and molecular physiology, 2023-02, Vol.324 (2), p.L154-L168 [Peer Reviewed Journal]Copyright © 2023 the American Physiological Society. 2023 American Physiological Society ;ISSN: 1040-0605 ;EISSN: 1522-1504 ;DOI: 10.1152/ajplung.00248.2022 ;PMID: 36573684Full text available |
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2 |
Material Type: Article
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Connective tissue growth factor (CCN2) is a matricellular preproprotein controlled by proteolytic activationThe Journal of biological chemistry, 2018-11, Vol.293 (46), p.17953-17970 [Peer Reviewed Journal]2018 © 2018 Kaasbøll et al. ;2018 Kaasbøll et al. ;info:eu-repo/semantics/openAccess ;2018 Kaasbøll et al. 2018 Kaasbøll et al. ;ISSN: 0021-9258 ;EISSN: 1083-351X ;DOI: 10.1074/jbc.RA118.004559 ;PMID: 30262666Full text available |
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3 |
Material Type: Article
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Role of Omentin, Vaspin, Cardiotrophin-1, TWEAK and NOV/CCN3 in Obesity and Diabetes DevelopmentInternational journal of molecular sciences, 2017-08, Vol.18 (8), p.1770 [Peer Reviewed Journal]Copyright MDPI AG 2017 ;2017 by the authors. 2017 ;ISSN: 1422-0067 ;ISSN: 1661-6596 ;EISSN: 1422-0067 ;DOI: 10.3390/ijms18081770 ;PMID: 28809783Full text available |
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4 |
Material Type: Article
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Decreased CCN3 in Systemic Sclerosis Endothelial Cells Contributes to Impaired AngiogenesisJournal of investigative dermatology, 2020-07, Vol.140 (7), p.1427-1434.e5 [Peer Reviewed Journal]2020 The Authors ;Copyright © 2020 The Authors. Published by Elsevier Inc. All rights reserved. ;Attribution - NonCommercial ;ISSN: 0022-202X ;EISSN: 1523-1747 ;DOI: 10.1016/j.jid.2019.11.026 ;PMID: 31954725Full text available |
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5 |
Material Type: Article
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EET intervention on Wnt1, NOV, and HO-1 signaling prevents obesity-induced cardiomyopathy in obese miceAmerican journal of physiology. Heart and circulatory physiology, 2017-08, Vol.313 (2), p.H368-H380 [Peer Reviewed Journal]Copyright © 2017 the American Physiological Society. ;Copyright American Physiological Society Aug 2017 ;Copyright © 2017 the American Physiological Society 2017 American Physiological Society ;ISSN: 0363-6135 ;EISSN: 1522-1539 ;DOI: 10.1152/ajpheart.00093.2017 ;PMID: 28576832Full text available |
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6 |
Material Type: Article
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NOV/CCN3 induces cartilage protection by inhibiting PI3K/AKT/mTOR pathwayJournal of cellular and molecular medicine, 2019-11, Vol.23 (11), p.7525-7534 [Peer Reviewed Journal]2019 The Authors. published by Foundation for Cellular and Molecular Medicine and John Wiley & Sons Ltd ;2019 The Authors. Journal of Cellular and Molecular Medicine published by Foundation for Cellular and Molecular Medicine and John Wiley & Sons Ltd. ;2019. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;ISSN: 1582-1838 ;EISSN: 1582-4934 ;DOI: 10.1111/jcmm.14621 ;PMID: 31454155Full text available |
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7 |
Material Type: Article
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NOV/CCN3: A New Adipocytokine Involved in Obesity-Associated Insulin ResistanceDiabetes (New York, N.Y.), 2016-09, Vol.65 (9), p.2502-2515 [Peer Reviewed Journal]2016 by the American Diabetes Association. ;Copyright American Diabetes Association Sep 1, 2016 ;Distributed under a Creative Commons Attribution 4.0 International License ;ISSN: 0012-1797 ;EISSN: 1939-327X ;DOI: 10.2337/db15-0617 ;PMID: 27284105 ;CODEN: DIAEAZFull text available |
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8 |
Material Type: Article
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Periostin and its interacting proteins in the construction of extracellular architecturesCellular and molecular life sciences : CMLS, 2017-12, Vol.74 (23), p.4269-4277 [Peer Reviewed Journal]Springer International Publishing AG 2017 ;Cellular and Molecular Life Sciences is a copyright of Springer, 2017. ;ISSN: 1420-682X ;EISSN: 1420-9071 ;DOI: 10.1007/s00018-017-2644-4 ;PMID: 28887577Full text available |
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9 |
Material Type: Article
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Dynamic CCN3 expression in the murine CNS does not confer essential roles in myelination or remyelinationProceedings of the National Academy of Sciences - PNAS, 2020-07, Vol.117 (30), p.18018-18028 [Peer Reviewed Journal]Copyright © 2020 the Author(s). Published by PNAS. ;Copyright National Academy of Sciences Jul 28, 2020 ;Copyright © 2020 the Author(s). Published by PNAS. 2020 ;ISSN: 0027-8424 ;EISSN: 1091-6490 ;DOI: 10.1073/pnas.1922089117 ;PMID: 32651278Full text available |
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10 |
Material Type: Article
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Matricellular protein CCN3 mitigates abdominal aortic aneurysmThe Journal of clinical investigation, 2016-04, Vol.126 (4), p.1282-1299 [Peer Reviewed Journal]COPYRIGHT 2016 American Society for Clinical Investigation ;COPYRIGHT 2016 American Society for Clinical Investigation ;Copyright American Society for Clinical Investigation Apr 2016 ;Copyright © 2016, American Society for Clinical Investigation 2016 American Society for Clinical Investigation ;ISSN: 0021-9738 ;EISSN: 1558-8238 ;DOI: 10.1172/JCI82337 ;PMID: 26974158Full text available |
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11 |
Material Type: Article
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Emerging role of CCN family proteins in tumorigenesis and cancer metastasis (Review)International journal of molecular medicine, 2015-12, Vol.36 (6), p.1451-1463 [Peer Reviewed Journal]Copyright: © Li et al. ;COPYRIGHT 2015 Spandidos Publications ;Copyright: © Li et al. 2015 ;ISSN: 1107-3756 ;EISSN: 1791-244X ;DOI: 10.3892/ijmm.2015.2390 ;PMID: 26498181Full text available |
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12 |
Material Type: Article
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Structural analyses of von Willebrand factor C domains of collagen 2A and CCN3 reveal an alternative mode of binding to bone morphogenetic protein-2The Journal of biological chemistry, 2017-07, Vol.292 (30), p.12516-12527 [Peer Reviewed Journal]2017 © 2017 ASBMB. Currently published by Elsevier Inc; originally published by American Society for Biochemistry and Molecular Biology ;2017 by The American Society for Biochemistry and Molecular Biology, Inc. ;2017 by The American Society for Biochemistry and Molecular Biology, Inc. 2017 The American Society for Biochemistry and Molecular Biology, Inc. ;ISSN: 0021-9258 ;EISSN: 1083-351X ;DOI: 10.1074/jbc.M117.788992 ;PMID: 28584056Full text available |
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13 |
Material Type: Article
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Huaier restrains proliferative and invasive potential of human hepatoma SKHEP-1 cells partially through decreased Lamin B1 and elevated NOVScientific reports, 2016-08, Vol.6 (1), p.31298-31298, Article 31298 [Peer Reviewed Journal]Copyright Nature Publishing Group Aug 2016 ;Copyright © 2016, The Author(s) 2016 The Author(s) ;ISSN: 2045-2322 ;EISSN: 2045-2322 ;DOI: 10.1038/srep31298 ;PMID: 27503760Full text available |
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14 |
Material Type: Article
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CCN3/Nephroblastoma Overexpressed Is a Functional Mediator of Prostate Cancer Bone Metastasis That Is Associated with Poor Patient PrognosisThe American journal of pathology, 2019-07, Vol.189 (7), p.1451-1461 [Peer Reviewed Journal]2019 American Society for Investigative Pathology ;Copyright © 2019 American Society for Investigative Pathology. Published by Elsevier Inc. All rights reserved. ;ISSN: 0002-9440 ;EISSN: 1525-2191 ;DOI: 10.1016/j.ajpath.2019.04.006 ;PMID: 31202437Full text available |
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15 |
Material Type: Article
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CCN3 promotes prostate cancer bone metastasis by modulating the tumor-bone microenvironment through RANKL-dependent pathwayCarcinogenesis (New York), 2013-07, Vol.34 (7), p.1669-1679 [Peer Reviewed Journal]ISSN: 0143-3334 ;EISSN: 1460-2180 ;DOI: 10.1093/carcin/bgt103 ;PMID: 23536580Full text available |
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16 |
Material Type: Article
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Polycomb-Mediated Disruption of an Androgen Receptor Feedback Loop Drives Castration-Resistant Prostate CancerCancer research (Chicago, Ill.), 2017-01, Vol.77 (2), p.412-422 [Peer Reviewed Journal]2016 American Association for Cancer Research. ;Copyright American Association for Cancer Research, Inc. Jan 15, 2017 ;ISSN: 0008-5472 ;EISSN: 1538-7445 ;DOI: 10.1158/0008-5472.can-16-1949 ;PMID: 27815387Full text available |
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17 |
Material Type: Article
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CCN3 (NOV) Drives Degradative Changes in Aging Articular CartilageInternational journal of molecular sciences, 2020-10, Vol.21 (20), p.7556 [Peer Reviewed Journal]2020 by the authors. Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (http://creativecommons.org/licenses/by/4.0/). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2020 by the authors. 2020 ;ISSN: 1422-0067 ;ISSN: 1661-6596 ;EISSN: 1422-0067 ;DOI: 10.3390/ijms21207556 ;PMID: 33066270Full text available |
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18 |
Material Type: Article
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Prostate cancer-derived CCN3 induces M2 macrophage infiltration and contributes to angiogenesis in prostate cancer microenvironmentOncotarget, 2014-03, Vol.5 (6), p.1595-1608Copyright: © 2014 Chen et al. 2014 ;ISSN: 1949-2553 ;EISSN: 1949-2553 ;DOI: 10.18632/oncotarget.1570 ;PMID: 24721786Full text available |
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19 |
Material Type: Article
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Reduced NOV/CCN3 Expression Limits Inflammation and Interstitial Renal Fibrosis after Obstructive Nephropathy in MicePloS one, 2015-09, Vol.10 (9), p.e0137876-e0137876 [Peer Reviewed Journal]COPYRIGHT 2015 Public Library of Science ;COPYRIGHT 2015 Public Library of Science ;2015 Marchal et al. This is an open access article distributed under the terms of the Creative Commons Attribution License: http://creativecommons.org/licenses/by/4.0/ (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;Attribution ;2015 Marchal et al 2015 Marchal et al ;ISSN: 1932-6203 ;EISSN: 1932-6203 ;DOI: 10.1371/journal.pone.0137876 ;PMID: 26367310Full text available |
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20 |
Material Type: Article
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CCN3 suppresses TGF-β1-induced extracellular matrix accumulation in human mesangial cells in vitroActa pharmacologica Sinica, 2018-02, Vol.39 (2), p.222-229 [Peer Reviewed Journal]Copyright © 2018 CPS and SIMM 2018 CPS and SIMM ;ISSN: 1671-4083 ;EISSN: 1745-7254 ;DOI: 10.1038/aps.2017.87 ;PMID: 28858296Full text available |