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Intestinal barrier dysfunction as a therapeutic target for cardiovascular disease
Intestinal barrier dysfunction as a therapeutic target for cardiovascular disease
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Intestinal barrier dysfunction as a therapeutic target for cardiovascular disease

American journal of physiology. Heart and circulatory physiology, 2020-12, Vol.319 (6), p.H1227-H1233 [Peer Reviewed Journal]

Copyright American Physiological Society Dec 2020 ;ISSN: 0363-6135 ;EISSN: 1522-1539 ;DOI: 10.1152/ajpheart.00612.2020 ;PMID: 32986965

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2
IL-4 and IL-13 induce equivalent expression of traditional M2 markers and modulation of reactive oxygen species in human macrophages
IL-4 and IL-13 induce equivalent expression of traditional M2 markers and modulation of reactive oxygen species in human macrophages
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IL-4 and IL-13 induce equivalent expression of traditional M2 markers and modulation of reactive oxygen species in human macrophages

Scientific reports, 2023-11, Vol.13 (1), p.19589-19589, Article 19589 [Peer Reviewed Journal]

The Author(s) 2023. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2023 ;ISSN: 2045-2322 ;EISSN: 2045-2322 ;DOI: 10.1038/s41598-023-46237-2 ;PMID: 37949903

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3
Distinct Redox Signalling following Macrophage Activation Influences Profibrotic Activity
Distinct Redox Signalling following Macrophage Activation Influences Profibrotic Activity
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Distinct Redox Signalling following Macrophage Activation Influences Profibrotic Activity

Journal of immunology research, 2019, Vol.2019, p.1278301-15 [Peer Reviewed Journal]

Copyright © 2019 Caitlin V. Lewis et al. ;Copyright © 2019 Caitlin V. Lewis et al. This is an open access article distributed under the Creative Commons Attribution License (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. http://creativecommons.org/licenses/by/4.0 ;Copyright © 2019 Caitlin V. Lewis et al. 2019 ;ISSN: 2314-8861 ;EISSN: 2314-7156 ;DOI: 10.1155/2019/1278301 ;PMID: 31815149

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4
Intestinal barrier dysfunction in murine sickle cell disease is associated with small intestine neutrophilic inflammation, oxidative stress, and dysbiosis
Intestinal barrier dysfunction in murine sickle cell disease is associated with small intestine neutrophilic inflammation, oxidative stress, and dysbiosis
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Intestinal barrier dysfunction in murine sickle cell disease is associated with small intestine neutrophilic inflammation, oxidative stress, and dysbiosis

FASEB bioAdvances, 2023-05, Vol.5 (5), p.199-210 [Peer Reviewed Journal]

2023 The Authors. published by Wiley Periodicals LLC on behalf of The Federation of American Societies for Experimental Biology. ;2023 The Authors. FASEB BioAdvances published by Wiley Periodicals LLC on behalf of The Federation of American Societies for Experimental Biology. ;2023. This work is published under http://creativecommons.org/licenses/by-nc-nd/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;ISSN: 2573-9832 ;EISSN: 2573-9832 ;DOI: 10.1096/fba.2022-00121 ;PMID: 37151850

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5
Obligatory Role for B Cells in the Development of Angiotensin II-Dependent Hypertension
Obligatory Role for B Cells in the Development of Angiotensin II-Dependent Hypertension
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Obligatory Role for B Cells in the Development of Angiotensin II-Dependent Hypertension

Hypertension (Dallas, Tex. 1979), 2015-11, Vol.66 (5), p.1023-1033 [Peer Reviewed Journal]

2015 American Heart Association, Inc. ;ISSN: 0194-911X ;EISSN: 1524-4563 ;DOI: 10.1161/HYPERTENSIONAHA.115.05779 ;PMID: 26351030

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6
Increasing nitric oxide bioavailability fails to improve collateral vessel formation in humanized sickle cell mice
Increasing nitric oxide bioavailability fails to improve collateral vessel formation in humanized sickle cell mice
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Increasing nitric oxide bioavailability fails to improve collateral vessel formation in humanized sickle cell mice

Laboratory investigation, 2022-08, Vol.102 (8), p.805-813 [Peer Reviewed Journal]

2022 United States & Canadian Academy of Pathology ;2022. This is a U.S. government work and not under copyright protection in the U.S.; foreign copyright protection may apply. ;This is a U.S. government work and not under copyright protection in the U.S.; foreign copyright protection may apply 2022. ;ISSN: 0023-6837 ;EISSN: 1530-0307 ;DOI: 10.1038/s41374-022-00780-0 ;PMID: 35354915

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7
Abstract 119: Ccl18 as a Potential Mediator of the Pro-Fibrotic Actions of M2 Macrophages in the Vessel Wall During Hypertension
Abstract 119: Ccl18 as a Potential Mediator of the Pro-Fibrotic Actions of M2 Macrophages in the Vessel Wall During Hypertension
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Abstract 119: Ccl18 as a Potential Mediator of the Pro-Fibrotic Actions of M2 Macrophages in the Vessel Wall During Hypertension

Hypertension (Dallas, Tex. 1979), 2019-09, Vol.74 (Suppl_1) [Peer Reviewed Journal]

ISSN: 0194-911X ;EISSN: 1524-4563 ;DOI: 10.1161/hyp.74.suppl_1.119

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