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11β‐hydroxysteroid dehydrogenase type 1 deficiency in bone marrow‐derived cells reduces atherosclerosis
11β‐hydroxysteroid dehydrogenase type 1 deficiency in bone marrow‐derived cells reduces atherosclerosis
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11β‐hydroxysteroid dehydrogenase type 1 deficiency in bone marrow‐derived cells reduces atherosclerosis

The FASEB journal, 2013-04, Vol.27 (4), p.1519-1531 [Peer Reviewed Journal]

FASEB ;FASEB 2013 ;ISSN: 0892-6638 ;EISSN: 1530-6860 ;DOI: 10.1096/fj.12-219105 ;PMID: 23303209

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2
11β-hydroxysteroid dehydrogenase type 1 gene knockout attenuates atherosclerosis and in vivo foam cell formation in hyperlipidemic apoE⁻/⁻ mice
11β-hydroxysteroid dehydrogenase type 1 gene knockout attenuates atherosclerosis and in vivo foam cell formation in hyperlipidemic apoE⁻/⁻ mice
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11β-hydroxysteroid dehydrogenase type 1 gene knockout attenuates atherosclerosis and in vivo foam cell formation in hyperlipidemic apoE⁻/⁻ mice

PloS one, 2013-02, Vol.8 (2), p.e53192-e53192 [Peer Reviewed Journal]

2013 García et al. This is an open-access article distributed under the terms of the Creative Commons Attribution License: https://creativecommons.org/licenses/by/4.0/ (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2013 García et al 2013 García et al ;ISSN: 1932-6203 ;EISSN: 1932-6203 ;DOI: 10.1371/journal.pone.0053192 ;PMID: 23383297

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3
11β-Hydroxysteroid Dehydrogenase Type 2 Deficiency Accelerates Atherogenesis and Causes Proinflammatory Changes in the Endothelium in Apoe−/− Mice
11β-Hydroxysteroid Dehydrogenase Type 2 Deficiency Accelerates Atherogenesis and Causes Proinflammatory Changes in the Endothelium in Apoe−/− Mice
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11β-Hydroxysteroid Dehydrogenase Type 2 Deficiency Accelerates Atherogenesis and Causes Proinflammatory Changes in the Endothelium in Apoe−/− Mice

Endocrinology (Philadelphia), 2011-01, Vol.152 (1), p.236-246 [Peer Reviewed Journal]

ISSN: 0013-7227 ;EISSN: 1945-7170 ;DOI: 10.1210/en.2010-0925 ;PMID: 21106873

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4
1,1′-(1,2-ethynediyl)bis-benzene derivatives as PTP 1-B inhibitors
1,1′-(1,2-ethynediyl)bis-benzene derivatives as PTP 1-B inhibitors
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Patent 		Add to My Research

1,1′-(1,2-ethynediyl)bis-benzene derivatives as PTP 1-B inhibitors

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5
12/15-lipoxygenase regulates intercellular adhesion molecule-1 expression and monocyte adhesion to endothelium through activation of RhoA and nuclear factor-kappaB
12/15-lipoxygenase regulates intercellular adhesion molecule-1 expression and monocyte adhesion to endothelium through activation of RhoA and nuclear factor-kappaB
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12/15-lipoxygenase regulates intercellular adhesion molecule-1 expression and monocyte adhesion to endothelium through activation of RhoA and nuclear factor-kappaB

Arteriosclerosis, thrombosis, and vascular biology, 2005-11, Vol.25 (11), p.2301-2307 [Peer Reviewed Journal]

ISSN: 1079-5642 ;EISSN: 1524-4636 ;PMID: 16166569

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6
1,25(OH) 2 D 3 improves blood lipid metabolism, liver function, and atherosclerosis by constraining the TGF-β/Smad signaling pathway in rats with hyperlipidemia
1,25(OH) 2 D 3 improves blood lipid metabolism, liver function, and atherosclerosis by constraining the TGF-β/Smad signaling pathway in rats with hyperlipidemia
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1,25(OH) 2 D 3 improves blood lipid metabolism, liver function, and atherosclerosis by constraining the TGF-β/Smad signaling pathway in rats with hyperlipidemia

Cell cycle (Georgetown, Tex.), 2019-11, Vol.18 (22), p.3111 [Peer Reviewed Journal]

EISSN: 1551-4005 ;PMID: 31544583

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7
17β-Estradiol Enhances Vascular Endothelial Ets-1/miR-126-3p Expression: The Possible Mechanism for Attenuation of Atherosclerosis
17β-Estradiol Enhances Vascular Endothelial Ets-1/miR-126-3p Expression: The Possible Mechanism for Attenuation of Atherosclerosis
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17β-Estradiol Enhances Vascular Endothelial Ets-1/miR-126-3p Expression: The Possible Mechanism for Attenuation of Atherosclerosis

The journal of clinical endocrinology and metabolism, 2017-02, Vol.102 (2), p.594-603 [Peer Reviewed Journal]

Copyright © Oxford University Press 2015 ;Copyright © 2017 by the Endocrine Society ;ISSN: 0021-972X ;EISSN: 1945-7197 ;DOI: 10.1210/jc.2016-2974 ;PMID: 27870587

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8
17β-estradiol induces vasorelaxation by stimulating endothelial hydrogen sulfide release
17β-estradiol induces vasorelaxation by stimulating endothelial hydrogen sulfide release
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17β-estradiol induces vasorelaxation by stimulating endothelial hydrogen sulfide release

Molecular human reproduction, 2013-03, Vol.19 (3-4), p.169-176 [Peer Reviewed Journal]

2014 INIST-CNRS ;ISSN: 1360-9947 ;EISSN: 1460-2407 ;DOI: 10.1093/molehr/gas044 ;PMID: 23041593

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9
18-kDa translocator protein ligand 18 F-FEMPA: Biodistribution and uptake into atherosclerotic plaques in mice
18-kDa translocator protein ligand 18 F-FEMPA: Biodistribution and uptake into atherosclerotic plaques in mice
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18-kDa translocator protein ligand 18 F-FEMPA: Biodistribution and uptake into atherosclerotic plaques in mice

Journal of nuclear cardiology, 2017-06, Vol.24 (3), p.862 [Peer Reviewed Journal]

EISSN: 1532-6551 ;PMID: 27225517

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10
(18)F-FDG PET and vascular inflammation: time to refine the paradigm?
(18)F-FDG PET and vascular inflammation: time to refine the paradigm?
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(18)F-FDG PET and vascular inflammation: time to refine the paradigm?

Journal of nuclear cardiology, 2015-04, Vol.22 (2), p.319 [Peer Reviewed Journal]

EISSN: 1532-6551 ;DOI: 10.1007/s12350-014-9917-1 ;PMID: 24925623

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11
2'-5' oligoadenylate synthetase‑like 1 (OASL1) protects against atherosclerosis by maintaining endothelial nitric oxide synthase mRNA stability
2'-5' oligoadenylate synthetase‑like 1 (OASL1) protects against atherosclerosis by maintaining endothelial nitric oxide synthase mRNA stability
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2'-5' oligoadenylate synthetase‑like 1 (OASL1) protects against atherosclerosis by maintaining endothelial nitric oxide synthase mRNA stability

Nature communications, 2022-11, Vol.13 (1), p.6647-6647, Article 6647 [Peer Reviewed Journal]

2022. The Author(s). ;The Author(s) 2022. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2022 ;ISSN: 2041-1723 ;EISSN: 2041-1723 ;DOI: 10.1038/s41467-022-34433-z ;PMID: 36333342

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12
2013 Russell Ross Memorial Lecture in Vascular Biology: Cellular and Molecular Mechanisms of Diabetes Mellitus–Accelerated Atherosclerosis
2013 Russell Ross Memorial Lecture in Vascular Biology: Cellular and Molecular Mechanisms of Diabetes Mellitus–Accelerated Atherosclerosis
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2013 Russell Ross Memorial Lecture in Vascular Biology: Cellular and Molecular Mechanisms of Diabetes Mellitus–Accelerated Atherosclerosis

Arteriosclerosis, thrombosis, and vascular biology, 2014-04, Vol.34 (4), p.705-714 [Peer Reviewed Journal]

2014 American Heart Association, Inc. ;ISSN: 1079-5642 ;EISSN: 1524-4636 ;DOI: 10.1161/ATVBAHA.113.301928 ;PMID: 24665124

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13
2015 Russell Ross Memorial Lecture in Vascular Biology: Protective Autoimmunity in Atherosclerosis
2015 Russell Ross Memorial Lecture in Vascular Biology: Protective Autoimmunity in Atherosclerosis
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2015 Russell Ross Memorial Lecture in Vascular Biology: Protective Autoimmunity in Atherosclerosis

Arteriosclerosis, thrombosis, and vascular biology, 2016-03, Vol.36 (3), p.429-438 [Peer Reviewed Journal]

2016 American Heart Association, Inc. ;ISSN: 1079-5642 ;EISSN: 1524-4636 ;DOI: 10.1161/ATVBAHA.115.306009 ;PMID: 26821946

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14
2016 Russell Ross Memorial Lecture in Vascular Biology: Molecular-Cellular Mechanisms in the Progression of Atherosclerosis
2016 Russell Ross Memorial Lecture in Vascular Biology: Molecular-Cellular Mechanisms in the Progression of Atherosclerosis
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2016 Russell Ross Memorial Lecture in Vascular Biology: Molecular-Cellular Mechanisms in the Progression of Atherosclerosis

Arteriosclerosis, thrombosis, and vascular biology, 2017-02, Vol.37 (2), p.183-189 [Peer Reviewed Journal]

2016 American Heart Association, Inc. ;ISSN: 1079-5642 ;EISSN: 1524-4636 ;DOI: 10.1161/ATVBAHA.116.308036 ;PMID: 27979856

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15
22016 ATVB Plenary Lecture: Receptor for Advanced Glycation Endproducts and Implications for the Pathogenesis and Treatment of Cardiometabolic Disorders: Spotlight on the Macrophage
22016 ATVB Plenary Lecture: Receptor for Advanced Glycation Endproducts and Implications for the Pathogenesis and Treatment of Cardiometabolic Disorders: Spotlight on the Macrophage
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22016 ATVB Plenary Lecture: Receptor for Advanced Glycation Endproducts and Implications for the Pathogenesis and Treatment of Cardiometabolic Disorders: Spotlight on the Macrophage

Arteriosclerosis, thrombosis, and vascular biology, 2017-04, Vol.37 (4), p.613-621 [Peer Reviewed Journal]

2017 American Heart Association, Inc. ;EISSN: 1524-4636 ;DOI: 10.1161/ATVBAHA.117.307263 ;PMID: 28183700

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16
2,3,4′,5-Tetrahydroxystilbene-2-O-β-D-glucoside Suppresses Expression of Adhesion Molecules in Aortic Wall of Dietary Atherosclerotic Rats and Promonocytic U937 Cells
2,3,4′,5-Tetrahydroxystilbene-2-O-β-D-glucoside Suppresses Expression of Adhesion Molecules in Aortic Wall of Dietary Atherosclerotic Rats and Promonocytic U937 Cells
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2,3,4′,5-Tetrahydroxystilbene-2-O-β-D-glucoside Suppresses Expression of Adhesion Molecules in Aortic Wall of Dietary Atherosclerotic Rats and Promonocytic U937 Cells

Cell biochemistry and biophysics, 2013-12, Vol.67 (3), p.997-1004 [Peer Reviewed Journal]

Springer Science+Business Media New York 2013 ;ISSN: 1085-9195 ;EISSN: 1559-0283 ;DOI: 10.1007/s12013-013-9595-7 ;PMID: 23575939

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17
2H2O-Based High-Density Lipoprotein Turnover Method for the Assessment of Dynamic High-Density Lipoprotein Function in Mice
2H2O-Based High-Density Lipoprotein Turnover Method for the Assessment of Dynamic High-Density Lipoprotein Function in Mice
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2H2O-Based High-Density Lipoprotein Turnover Method for the Assessment of Dynamic High-Density Lipoprotein Function in Mice

Arteriosclerosis, thrombosis, and vascular biology, 2013-08, Vol.33 (8), p.1994-2003 [Peer Reviewed Journal]

2013 American Heart Association, Inc. ;ISSN: 1079-5642 ;EISSN: 1524-4636 ;DOI: 10.1161/ATVBAHA.113.301700 ;PMID: 23766259

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18
4‐Hydroxynonenal and cholesterol oxidation products in atherosclerosis
4‐Hydroxynonenal and cholesterol oxidation products in atherosclerosis
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4‐Hydroxynonenal and cholesterol oxidation products in atherosclerosis

Molecular nutrition & food research, 2005-11, Vol.49 (11), p.1044-1049 [Peer Reviewed Journal]

Copyright © 2005 WILEY‐VCH Verlag GmbH & Co. KGaA, Weinheim ;ISSN: 1613-4125 ;EISSN: 1613-4133 ;DOI: 10.1002/mnfr.200500090 ;PMID: 16270277

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19
4‐Phenylbutyrate protects against atherosclerotic lesion growth by increasing the expression of HSP25 in macrophages and in the circulation of Apoe−/− mice
4‐Phenylbutyrate protects against atherosclerotic lesion growth by increasing the expression of HSP25 in macrophages and in the circulation of Apoe−/− mice
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4‐Phenylbutyrate protects against atherosclerotic lesion growth by increasing the expression of HSP25 in macrophages and in the circulation of Apoe−/− mice

The FASEB journal, 2019-07, Vol.33 (7), p.8406-8422 [Peer Reviewed Journal]

FASEB ;ISSN: 0892-6638 ;EISSN: 1530-6860 ;DOI: 10.1096/fj.201802293RR ;PMID: 30964709

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20
5-Aminolevulinic Acid-Mediated Sonodynamic Therapy Alleviates Atherosclerosis via Enhancing Efferocytosis and Facilitating a Shift in the Th1/Th2 Balance Toward Th2 Polarization
5-Aminolevulinic Acid-Mediated Sonodynamic Therapy Alleviates Atherosclerosis via Enhancing Efferocytosis and Facilitating a Shift in the Th1/Th2 Balance Toward Th2 Polarization
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5-Aminolevulinic Acid-Mediated Sonodynamic Therapy Alleviates Atherosclerosis via Enhancing Efferocytosis and Facilitating a Shift in the Th1/Th2 Balance Toward Th2 Polarization

Cellular physiology and biochemistry, 2018-01, Vol.47 (1), p.83-96 [Peer Reviewed Journal]

2018 The Author(s). Published by S. Karger AG, Basel ;2018 The Author(s). Published by S. Karger AG, Basel. ;ISSN: 1015-8987 ;ISSN: 1421-9778 ;EISSN: 1421-9778 ;DOI: 10.1159/000489751 ;PMID: 29763901

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