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| Result Number | Material Type | Add to My Shelf Action | Record Details and Options |
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Mild Hypoxia Accelerates Cerebral Cavernous Malformation Disease Through CX3CR1-CX3CL1 SignalingArteriosclerosis, thrombosis, and vascular biology, 2024-06, Vol.44 (6), p.1246-1264 [Peer Reviewed Journal]EISSN: 1524-4636 ;DOI: 10.1161/ATVBAHA.123.320367 ;PMID: 38660801Full text available |
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Shear stress and pathophysiological PI3K involvement in vascular malformationsThe Journal of clinical investigation, 2024-05, Vol.134 (10) [Peer Reviewed Journal]COPYRIGHT 2024 American Society for Clinical Investigation ;2024 Abdelilah-Seyfried et al. 2024 Abdelilah-Seyfried et al. ;ISSN: 0021-9738 ;EISSN: 1558-8238 ;DOI: 10.1172/JCI172843 ;PMID: 38747293Full text available |
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Somatic GJA4 mutation in intracranial extra-axial cavernous hemangiomasStroke and vascular neurology, 2023-12, Vol.8 (6), p.453-462 [Peer Reviewed Journal]Author(s) (or their employer(s)) 2023. Re-use permitted under CC BY-NC. No commercial re-use. See rights and permissions. Published by BMJ. ;2023 Author(s) (or their employer(s)) 2023. Re-use permitted under CC BY-NC. No commercial re-use. See rights and permissions. Published by BMJ. http://creativecommons.org/licenses/by-nc/4.0/ This is an open access article distributed in accordance with the Creative Commons Attribution Non Commercial (CC BY-NC 4.0) license, which permits others to distribute, remix, adapt, build upon this work non-commercially, and license their derivative works on different terms, provided the original work is properly cited, appropriate credit is given, any changes made indicated, and the use is non-commercial. See: http://creativecommons.org/licenses/by-nc/4.0/ . Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;Author(s) (or their employer(s)) 2023. Re-use permitted under CC BY-NC. No commercial re-use. See rights and permissions. Published by BMJ. 2023 ;ISSN: 2059-8688 ;EISSN: 2059-8696 ;DOI: 10.1136/svn-2022-002227 ;PMID: 37072338Full text available |
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KRIT1: A Traffic Warden at the Busy Crossroads Between Redox Signaling and the Pathogenesis of Cerebral Cavernous Malformation DiseaseAntioxidants & redox signaling, 2023-03, Vol.38 (7-9), p.496-528 [Peer Reviewed Journal]2023, Mary Ann Liebert, Inc., publishers ;Copyright Mary Ann Liebert, Inc. Mar 1, 2023 ;Copyright 2023, Mary Ann Liebert, Inc., publishers 2023 Mary Ann Liebert, Inc., publishers ;ISSN: 1523-0864 ;EISSN: 1557-7716 ;DOI: 10.1089/ars.2021.0263 ;PMID: 36047808Full text available |
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β1 integrin monoclonal antibody treatment ameliorates cerebral cavernous malformationsThe FASEB journal, 2022-12, Vol.36 (12), p.e22629-n/a [Peer Reviewed Journal]2022 Federation of American Societies for Experimental Biology. ;Distributed under a Creative Commons Attribution 4.0 International License ;ISSN: 0892-6638 ;EISSN: 1530-6860 ;DOI: 10.1096/fj.202200907RR ;PMID: 36349990Full text available |
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Immunothrombosis and vascular heterogeneity in cerebral cavernous malformationBlood, 2022-11, Vol.140 (20), p.2154-2169 [Peer Reviewed Journal]2022 The American Society of Hematology ;2022 by The American Society of Hematology. Licensed under Creative Commons Attribution-NonCommercial-NoDerivatives 4.0 International (CC BY-NC-ND 4.0), permitting only noncommercial, nonderivative use with attribution. All other rights reserved. ;ISSN: 0006-4971 ;ISSN: 1528-0020 ;EISSN: 1528-0020 ;DOI: 10.1182/blood.2021015350 ;PMID: 35981497Full text available |
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Deciphering impact of single nucleotide polymorphisms on cotranscriptional modification in CCM gene mRNAsAmerican Journal of Physiology: Cell Physiology, 2022-10, Vol.323 (4), p.C1274-C1284 [Peer Reviewed Journal]ISSN: 0363-6143 ;EISSN: 1522-1563 ;DOI: 10.1152/ajpcell.00279.2022 ;PMID: 36094437Full text available |
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Inflammation and neutrophil extracellular traps in cerebral cavernous malformationCellular and molecular life sciences : CMLS, 2022-04, Vol.79 (4), p.206-206, Article 206 [Peer Reviewed Journal]The Author(s) 2022. corrected publication 2022 ;2022. The Author(s). ;The Author(s) 2022. corrected publication 2022. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2022, corrected publication 2022 ;ISSN: 1420-682X ;ISSN: 1420-9071 ;EISSN: 1420-9071 ;DOI: 10.1007/s00018-022-04224-2 ;PMID: 35333979Full text available |
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GCKIII (Germinal Center Kinase III) Kinases STK24 and STK25 (Serine/Threonine Kinase 24 and 25) Inhibit Cavernoma DevelopmentStroke (1970), 2022-03, Vol.53 (3), p.976-986 [Peer Reviewed Journal]ISSN: 0039-2499 ;EISSN: 1524-4628 ;DOI: 10.1161/STROKEAHA.121.036940 ;PMID: 35130716Full text available |
| 10 |
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Elevated proportion of TLR2- and TLR4-expressing Th17-like cells and activated memory B cells was associated with clinical activity of cerebral cavernous malformationsJournal of neuroinflammation, 2022-02, Vol.19 (1), p.28-28, Article 28 [Peer Reviewed Journal]2022. The Author(s). ;COPYRIGHT 2022 BioMed Central Ltd. ;2022. This work is licensed under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2022 ;ISSN: 1742-2094 ;EISSN: 1742-2094 ;DOI: 10.1186/s12974-022-02385-2 ;PMID: 35109870Full text available |
| 11 |
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Cerebral Cavernous Malformation: From Mechanism to TherapyCirculation research, 2021-06, Vol.129 (1), p.195-215 [Peer Reviewed Journal]ISSN: 0009-7330 ;EISSN: 1524-4571 ;DOI: 10.1161/circresaha.121.318174 ;PMID: 34166073Full text available |
| 12 |
Material Type: Article
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PIK3CA and CCM mutations fuel cavernomas through a cancer-like mechanismNature (London), 2021-06, Vol.594 (7862), p.271-276 [Peer Reviewed Journal]COPYRIGHT 2021 Nature Publishing Group ;Copyright Nature Publishing Group Jun 10, 2021 ;ISSN: 0028-0836 ;EISSN: 1476-4687 ;DOI: 10.1038/s41586-021-03562-8 ;PMID: 33910229Full text available |
| 13 |
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Abortive intussusceptive angiogenesis causes multi-cavernous vascular malformationseLife, 2021-05, Vol.10 [Peer Reviewed Journal]2021, Li et al. ;2021, Li et al. This work is published under https://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2021, Li et al 2021 Li et al ;ISSN: 2050-084X ;EISSN: 2050-084X ;DOI: 10.7554/eLife.62155 ;PMID: 34013885Full text available |
| 14 |
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Propranolol inhibits cavernous vascular malformations by β1 adrenergic receptor antagonism in animal modelsThe Journal of clinical investigation, 2021-02, Vol.131 (3) [Peer Reviewed Journal]2021 American Society for Clinical Investigation 2021 American Society for Clinical Investigation ;ISSN: 0021-9738 ;EISSN: 1558-8238 ;DOI: 10.1172/JCI144893 ;PMID: 33301422Full text available |
| 15 |
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Caveolae-mediated Tie2 signaling contributes to CCM pathogenesis in a brain endothelial cell-specific Pdcd10-deficient mouse modelNature communications, 2021-01, Vol.12 (1), p.504-504, Article 504 [Peer Reviewed Journal]The Author(s) 2021. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2021 ;ISSN: 2041-1723 ;EISSN: 2041-1723 ;DOI: 10.1038/s41467-020-20774-0 ;PMID: 33495460Full text available |
| 16 |
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Cerebral cavernous malformations are driven by ADAMTS5 proteolysis of versicanThe Journal of experimental medicine, 2020-10, Vol.217 (10) [Peer Reviewed Journal]2020 Hong et al. ;2020 Hong et al. 2020 ;ISSN: 0022-1007 ;EISSN: 1540-9538 ;DOI: 10.1084/jem.20200140 ;PMID: 32648916Full text available |
| 17 |
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VEGF signalling enhances lesion burden in KRIT1 deficient miceJournal of cellular and molecular medicine, 2020-01, Vol.24 (1), p.632-639 [Peer Reviewed Journal]2019 The Authors. Journal of Cellular and Molecular Medicine published by Foundation for cellular and Molecular Medicine and John Wiley & Sons Ltd ;2019 The Authors. Journal of Cellular and Molecular Medicine published by Foundation for cellular and Molecular Medicine and John Wiley & Sons Ltd. ;2020. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;ISSN: 1582-1838 ;EISSN: 1582-4934 ;DOI: 10.1111/jcmm.14773 ;PMID: 31746130Full text available |
| 18 |
Material Type: Article
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Loss of the transcription factor RBPJ induces disease-promoting properties in brain pericytesNature communications, 2019-06, Vol.10 (1), p.2817-19, Article 2817 [Peer Reviewed Journal]2019. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2019 ;ISSN: 2041-1723 ;EISSN: 2041-1723 ;DOI: 10.1038/s41467-019-10643-w ;PMID: 31249304Full text available |
| 19 |
Material Type: Article
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Building Blood Vessels-One Rho GTPase at a TimeCells (Basel, Switzerland), 2019-06, Vol.8 (6), p.545 [Peer Reviewed Journal]2019. This work is licensed under https://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2019 by the authors. 2019 ;ISSN: 2073-4409 ;EISSN: 2073-4409 ;DOI: 10.3390/cells8060545 ;PMID: 31174284Full text available |
| 20 |
Material Type: Article
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CCM3 and cerebral cavernous malformation diseaseStroke and vascular neurology, 2019-06, Vol.4 (2), p.67-70 [Peer Reviewed Journal]2019 Author(s) (or their employer(s)) 2019. Re-use permitted under CC BY-NC. No commercial re-use. See rights and permissions. Published by BMJ. This is an open access article distributed in accordance with the Creative Commons Attribution Non Commercial (CC BY-NC 4.0) license, which permits others to distribute, remix, adapt, build upon this work non-commercially, and license their derivative works on different terms, provided the original work is properly cited, appropriate credit is given, any changes made indicated, and the use is non-commercial. See: http://creativecommons.org/licenses/by-nc/4.0/ . Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;Author(s) (or their employer(s)) 2019. Re-use permitted under CC BY-NC. No commercial re-use. See rights and permissions. Published by BMJ. 2019 ;ISSN: 2059-8688 ;EISSN: 2059-8696 ;DOI: 10.1136/svn-2018-000195 ;PMID: 31338212Full text available |
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