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08 ONE HEALTH: A CROSS-SECTORAL, HOLISTIC APPROACH TO REDUCE DISEASE RISK AND BUILD RESILIENCE
08 ONE HEALTH: A CROSS-SECTORAL, HOLISTIC APPROACH TO REDUCE DISEASE RISK AND BUILD RESILIENCE
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08 ONE HEALTH: A CROSS-SECTORAL, HOLISTIC APPROACH TO REDUCE DISEASE RISK AND BUILD RESILIENCE

Development cooperation report, 2020-01, p.175-182 [Peer Reviewed Journal]

Copyright Organisation for Economic Cooperation and Development (OECD) 2020 ;ISSN: 2074-773X ;EISSN: 2074-7721

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094 Vascular targeting of phosphatidylserine causing thrombotic occlusion in an arteriovenous malformation animal model
094 Vascular targeting of phosphatidylserine causing thrombotic occlusion in an arteriovenous malformation animal model
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094 Vascular targeting of phosphatidylserine causing thrombotic occlusion in an arteriovenous malformation animal model

Journal of neurology, neurosurgery and psychiatry, 2018-06, Vol.89 (6), p.A37-A38 [Peer Reviewed Journal]

2018, Published by the BMJ Publishing Group Limited. For permission to use (where not already granted under a licence) please go to http://group.bmj.com/group/rights-licensing/permissions ;2018 2018, Published by the BMJ Publishing Group Limited. For permission to use (where not already granted under a licence) please go to http://group.bmj.com/group/rights-licensing/permissions ;ISSN: 0022-3050 ;EISSN: 1468-330X ;DOI: 10.1136/jnnp-2018-ANZAN.93

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3
[1- 11 C]-Butanol Positron Emission Tomography reveals an impaired brain to nasal turbinates pathway in aging amyloid positive subjects
[1- 11 C]-Butanol Positron Emission Tomography reveals an impaired brain to nasal turbinates pathway in aging amyloid positive subjects
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[1- 11 C]-Butanol Positron Emission Tomography reveals an impaired brain to nasal turbinates pathway in aging amyloid positive subjects

Fluids and barriers of the CNS, 2024-04, Vol.21 (1), p.30-30 [Peer Reviewed Journal]

2024. The Author(s). ;COPYRIGHT 2024 BioMed Central Ltd. ;The Author(s) 2024 ;ISSN: 2045-8118 ;EISSN: 2045-8118 ;DOI: 10.1186/s12987-024-00530-y ;PMID: 38566110

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4
1, 2 di-substituted idopyranose from Vitex negundo l. Protects against streptozotocin-induced diabetes by inhibiting nuclear factor-kappa B and inducible nitric oxide synthase expression
1, 2 di-substituted idopyranose from Vitex negundo l. Protects against streptozotocin-induced diabetes by inhibiting nuclear factor-kappa B and inducible nitric oxide synthase expression
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1, 2 di-substituted idopyranose from Vitex negundo l. Protects against streptozotocin-induced diabetes by inhibiting nuclear factor-kappa B and inducible nitric oxide synthase expression

Microscopy research and technique, 2011-04, Vol.74 (4), p.301-307 [Peer Reviewed Journal]

Copyright © 2010 Wiley‐Liss, Inc. ;Copyright © 2010 Wiley-Liss, Inc. ;ISSN: 1059-910X ;EISSN: 1097-0029 ;DOI: 10.1002/jemt.20904 ;PMID: 20687129

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5
1, 25-dihydroxy-vitamin D3 with tumor necrosis factor-alpha protects against rheumatoid arthritis by promoting p53 acetylation-mediated apoptosis via Sirt1 in synoviocytes
1, 25-dihydroxy-vitamin D3 with tumor necrosis factor-alpha protects against rheumatoid arthritis by promoting p53 acetylation-mediated apoptosis via Sirt1 in synoviocytes
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1, 25-dihydroxy-vitamin D3 with tumor necrosis factor-alpha protects against rheumatoid arthritis by promoting p53 acetylation-mediated apoptosis via Sirt1 in synoviocytes

Cell death & disease, 2016-10, Vol.7 (10), p.e2423-e2423 [Peer Reviewed Journal]

Copyright Nature Publishing Group Oct 2016 ;Copyright © 2016 The Author(s) 2016 The Author(s) ;ISSN: 2041-4889 ;EISSN: 2041-4889 ;DOI: 10.1038/cddis.2016.300 ;PMID: 27763638

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6
1-(3′,5′-dihydroxyphenoxy)-7-(2″,4″,6-trihydroxyphenoxy)-2,4,9-trihydroxydibenzo-1,4-dioxin Inhibits Adipocyte Differentiation of 3T3-L1 Fibroblasts
1-(3′,5′-dihydroxyphenoxy)-7-(2″,4″,6-trihydroxyphenoxy)-2,4,9-trihydroxydibenzo-1,4-dioxin Inhibits Adipocyte Differentiation of 3T3-L1 Fibroblasts
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1-(3′,5′-dihydroxyphenoxy)-7-(2″,4″,6-trihydroxyphenoxy)-2,4,9-trihydroxydibenzo-1,4-dioxin Inhibits Adipocyte Differentiation of 3T3-L1 Fibroblasts

Marine biotechnology (New York, N.Y.), 2010-06, Vol.12 (3), p.299-307 [Peer Reviewed Journal]

Springer Science+Business Media, LLC 2009 ;Springer Science+Business Media, LLC 2010 ;ISSN: 1436-2228 ;EISSN: 1436-2236 ;DOI: 10.1007/s10126-009-9224-z ;PMID: 19680725

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7
1-benzoyl-6-hydroxy-2,2,4-trimethyl-1,2-dihydroquinoline exerts a neuroprotective effect and normalises redox homeostasis in a rat model of cerebral ischemia/reperfusion
1-benzoyl-6-hydroxy-2,2,4-trimethyl-1,2-dihydroquinoline exerts a neuroprotective effect and normalises redox homeostasis in a rat model of cerebral ischemia/reperfusion
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1-benzoyl-6-hydroxy-2,2,4-trimethyl-1,2-dihydroquinoline exerts a neuroprotective effect and normalises redox homeostasis in a rat model of cerebral ischemia/reperfusion

Metabolic brain disease, 2022-04, Vol.37 (4), p.1271-1282 [Peer Reviewed Journal]

The Author(s), under exclusive licence to Springer Science+Business Media, LLC, part of Springer Nature 2022 ;2022. The Author(s), under exclusive licence to Springer Science+Business Media, LLC, part of Springer Nature. ;The Author(s), under exclusive licence to Springer Science+Business Media, LLC, part of Springer Nature 2022. ;ISSN: 0885-7490 ;EISSN: 1573-7365 ;DOI: 10.1007/s11011-022-00928-3 ;PMID: 35201554

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8
1-Deoxynojirimycin Alleviates Insulin Resistance via Activation of Insulin Signaling PI3K/AKT Pathway in Skeletal Muscle of db/db Mice
1-Deoxynojirimycin Alleviates Insulin Resistance via Activation of Insulin Signaling PI3K/AKT Pathway in Skeletal Muscle of db/db Mice
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1-Deoxynojirimycin Alleviates Insulin Resistance via Activation of Insulin Signaling PI3K/AKT Pathway in Skeletal Muscle of db/db Mice

Molecules (Basel, Switzerland), 2015-12, Vol.20 (12), p.21700-21714 [Peer Reviewed Journal]

2015. This work is licensed under https://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2015 by the authors. 2015 ;ISSN: 1420-3049 ;EISSN: 1420-3049 ;DOI: 10.3390/molecules201219794 ;PMID: 26690098

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9
1-Deoxysphingolipids cause autophagosome and lysosome accumulation and trigger NLRP3 inflammasome activation
1-Deoxysphingolipids cause autophagosome and lysosome accumulation and trigger NLRP3 inflammasome activation
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1-Deoxysphingolipids cause autophagosome and lysosome accumulation and trigger NLRP3 inflammasome activation

Autophagy, 2021-08, Vol.17 (8), p.1947-1961 [Peer Reviewed Journal]

2020 Informa UK Limited, trading as Taylor & Francis Group 2020 ;2020 Informa UK Limited, trading as Taylor & Francis Group 2020 Informa UK Limited, trading as Taylor & Francis Group ;ISSN: 1554-8627 ;EISSN: 1554-8635 ;DOI: 10.1080/15548627.2020.1804677 ;PMID: 32835606

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10
1,1-Dichloroethylene nephrotoxicity in the rat
1,1-Dichloroethylene nephrotoxicity in the rat
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1,1-Dichloroethylene nephrotoxicity in the rat

Toxicology and applied pharmacology, 1978-10, Vol.46 (1), p.131-141 [Peer Reviewed Journal]

1978 ;ISSN: 0041-008X ;EISSN: 1096-0333 ;DOI: 10.1016/0041-008X(78)90144-8 ;PMID: 725939

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11
1.2 Aspects of test assessment
1.2 Aspects of test assessment
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1.2 Aspects of test assessment

Human & experimental toxicology, 2009-02, Vol.28 (2-3), p.95-96 [Peer Reviewed Journal]

SAGE Publications © Feb/Mar 2009 ;ISSN: 0960-3271 ;EISSN: 1477-0903 ;DOI: 10.1177/0960327109105757 ;PMID: 19713368

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12
1,2-Octanediol, a novel surfactant, for treating head louse infestation: identification of activity, formulation, and randomised, controlled trials
1,2-Octanediol, a novel surfactant, for treating head louse infestation: identification of activity, formulation, and randomised, controlled trials
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1,2-Octanediol, a novel surfactant, for treating head louse infestation: identification of activity, formulation, and randomised, controlled trials

PloS one, 2012-04, Vol.7 (4), p.e35419 [Peer Reviewed Journal]

COPYRIGHT 2012 Public Library of Science ;COPYRIGHT 2012 Public Library of Science ;2012 Burgess et al. This is an open-access article distributed under the terms of the Creative Commons Attribution License: https://creativecommons.org/licenses/by/4.0/ (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;Burgess et al. 2012 ;ISSN: 1932-6203 ;EISSN: 1932-6203 ;DOI: 10.1371/journal.pone.0035419 ;PMID: 22523593

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13
1,2-propanediol and the type of cryopreservation procedure adversely affect mouse oocyte physiology
1,2-propanediol and the type of cryopreservation procedure adversely affect mouse oocyte physiology
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1,2-propanediol and the type of cryopreservation procedure adversely affect mouse oocyte physiology

Human reproduction (Oxford), 2007-01, Vol.22 (1), p.250-259 [Peer Reviewed Journal]

The Author 2006. Published by Oxford University Press on behalf of the European Society of Human Reproduction and Embryology. All rights reserved. For Permissions, please email: journals.permissions@oxfordjournals.org 2007 ;2007 INIST-CNRS ;Copyright Oxford University Press(England) Jan 2007 ;ISSN: 0268-1161 ;EISSN: 1460-2350 ;DOI: 10.1093/humrep/del319 ;PMID: 16905767 ;CODEN: HUREEE

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14
1,25-Dihydroxycholecalciferol down-regulates 3-mercaptopyruvate sulfur transferase and caspase-3 in rat model of non-alcoholic fatty liver disease
1,25-Dihydroxycholecalciferol down-regulates 3-mercaptopyruvate sulfur transferase and caspase-3 in rat model of non-alcoholic fatty liver disease
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1,25-Dihydroxycholecalciferol down-regulates 3-mercaptopyruvate sulfur transferase and caspase-3 in rat model of non-alcoholic fatty liver disease

Journal of molecular histology, 2023-04, Vol.54 (2), p.119-134 [Peer Reviewed Journal]

The Author(s), under exclusive licence to Springer Nature B.V. 2023. Springer Nature or its licensor (e.g. a society or other partner) holds exclusive rights to this article under a publishing agreement with the author(s) or other rightsholder(s); author self-archiving of the accepted manuscript version of this article is solely governed by the terms of such publishing agreement and applicable law. ;2023. The Author(s), under exclusive licence to Springer Nature B.V. ;ISSN: 1567-2379 ;EISSN: 1567-2387 ;DOI: 10.1007/s10735-023-10118-9 ;PMID: 36930413

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15
1,25-Dihydroxyvitamin D improved the free fatty-acid-induced insulin resistance in cultured C2C12 cells
1,25-Dihydroxyvitamin D improved the free fatty-acid-induced insulin resistance in cultured C2C12 cells
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1,25-Dihydroxyvitamin D improved the free fatty-acid-induced insulin resistance in cultured C2C12 cells

Diabetes/metabolism research and reviews, 2008-09, Vol.24 (6), p.459-464 [Peer Reviewed Journal]

Copyright © 2008 John Wiley & Sons, Ltd. ;2008 INIST-CNRS ;ISSN: 1520-7552 ;EISSN: 1520-7560 ;DOI: 10.1002/dmrr.873 ;PMID: 18551686 ;CODEN: DMRRFM

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16
1,25-dihydroxyvitamin D in the pathogenesis of Barrett's esophagus and esophageal adenocarcinoma
1,25-dihydroxyvitamin D in the pathogenesis of Barrett's esophagus and esophageal adenocarcinoma
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1,25-dihydroxyvitamin D in the pathogenesis of Barrett's esophagus and esophageal adenocarcinoma

Expert review of clinical immunology, 2013-06, Vol.9 (6), p.517-533 [Peer Reviewed Journal]

2013 Expert Reviews Ltd 2013 ;COPYRIGHT 2013 Expert Reviews Ltd. ;ISSN: 1744-666X ;EISSN: 1744-8409 ;DOI: 10.1586/eci.13.38 ;PMID: 23730883

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17
1,25-Dihydroxyvitamin D modulates L-type voltage-gated calcium channels in a subset of neurons in the developing mouse prefrontal cortex
1,25-Dihydroxyvitamin D modulates L-type voltage-gated calcium channels in a subset of neurons in the developing mouse prefrontal cortex
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1,25-Dihydroxyvitamin D modulates L-type voltage-gated calcium channels in a subset of neurons in the developing mouse prefrontal cortex

Translational psychiatry, 2019-11, Vol.9 (1), p.281-10, Article 281 [Peer Reviewed Journal]

2019. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2019 ;ISSN: 2158-3188 ;EISSN: 2158-3188 ;DOI: 10.1038/s41398-019-0626-z ;PMID: 31712549

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18
1,25-Dihydroxyvitamin D3 acts directly on the T lymphocyte vitamin D receptor to inhibit experimental autoimmune encephalomyelitis
1,25-Dihydroxyvitamin D3 acts directly on the T lymphocyte vitamin D receptor to inhibit experimental autoimmune encephalomyelitis
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1,25-Dihydroxyvitamin D3 acts directly on the T lymphocyte vitamin D receptor to inhibit experimental autoimmune encephalomyelitis

European journal of immunology, 2011-03, Vol.41 (3), p.822 [Peer Reviewed Journal]

Copyright © 2011 WILEY-VCH Verlag GmbH & Co. KGaA, Weinheim. ;EISSN: 1521-4141 ;DOI: 10.1002/eji.201040632 ;PMID: 21287548

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19
1,25-Dihydroxyvitamin D3 administration to 6-hydroxydopamine-lesioned rats increases glial cell line-derived neurotrophic factor and partially restores tyrosine hydroxylase expression in substantia nigra and striatum
1,25-Dihydroxyvitamin D3 administration to 6-hydroxydopamine-lesioned rats increases glial cell line-derived neurotrophic factor and partially restores tyrosine hydroxylase expression in substantia nigra and striatum
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1,25-Dihydroxyvitamin D3 administration to 6-hydroxydopamine-lesioned rats increases glial cell line-derived neurotrophic factor and partially restores tyrosine hydroxylase expression in substantia nigra and striatum

Journal of neuroscience research, 2009-02, Vol.87 (3), p.723-732 [Peer Reviewed Journal]

Copyright © 2008 Wiley‐Liss, Inc. ;(c) 2008 Wiley-Liss, Inc. ;ISSN: 0360-4012 ;EISSN: 1097-4547 ;DOI: 10.1002/jnr.21878 ;PMID: 18816795

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20
1,25-dihydroxyvitamin D3 and its nuclear receptor repress human α1(I) collagen expression and type I collagen formation
1,25-dihydroxyvitamin D3 and its nuclear receptor repress human α1(I) collagen expression and type I collagen formation
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1,25-dihydroxyvitamin D3 and its nuclear receptor repress human α1(I) collagen expression and type I collagen formation

Liver international, 2013-05, Vol.33 (5), p.677-686 [Peer Reviewed Journal]

2013 John Wiley & Sons A/S ;2013 John Wiley & Sons A/S. ;2013 John Wiley & Sons A/S 2013 ;ISSN: 1478-3223 ;EISSN: 1478-3231 ;DOI: 10.1111/liv.12122 ;PMID: 23413886

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