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1
11β-HSD1 inhibition ameliorates diabetes-induced cardiomyocyte hypertrophy and cardiac fibrosis through modulation of EGFR activity
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11β-HSD1 inhibition ameliorates diabetes-induced cardiomyocyte hypertrophy and cardiac fibrosis through modulation of EGFR activity

Oncotarget, 2017-11, Vol.8 (56), p.96263-96275

Copyright: © 2017 Zou et al. 2017 ;ISSN: 1949-2553 ;EISSN: 1949-2553 ;DOI: 10.18632/oncotarget.22015 ;PMID: 29221204

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2
14-3-3 proteins block apoptosis and differentially regulate MAPK cascades
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14-3-3 proteins block apoptosis and differentially regulate MAPK cascades

The EMBO Journal, 2000-02, Vol.19 (3), p.349-358 [Peer Reviewed Journal]

Copyright © 2000 European Molecular Biology Organization ;Copyright Oxford University Press(England) Feb 01, 2000 ;Copyright © 2000 European Molecular Biology Organization 2000 ;ISSN: 0261-4189 ;ISSN: 1460-2075 ;EISSN: 1460-2075 ;DOI: 10.1093/emboj/19.3.349 ;PMID: 10654934 ;CODEN: EMJODG

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3
2-Aminobutyric acid modulates glutathione homeostasis in the myocardium
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2-Aminobutyric acid modulates glutathione homeostasis in the myocardium

Scientific reports, 2016-11, Vol.6 (1), p.36749-36749, Article 36749 [Peer Reviewed Journal]

Copyright Nature Publishing Group Nov 2016 ;Copyright © 2016, The Author(s) 2016 The Author(s) ;ISSN: 2045-2322 ;EISSN: 2045-2322 ;DOI: 10.1038/srep36749 ;PMID: 27827456

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4
2014 Korean guidelines for appropriate utilization of cardiovascular magnetic resonance imaging: a joint report of the Korean Society of Cardiology and the Korean Society of Radiology
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2014 Korean guidelines for appropriate utilization of cardiovascular magnetic resonance imaging: a joint report of the Korean Society of Cardiology and the Korean Society of Radiology

Korean Journal of Radiology, 2014, 15(6), , pp.659-688

2014. This work is published under http://creativecommons.org/licenses/by-nc/3.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;Copyright © 2014 The Korean Society of Radiology 2014 ;ISSN: 1229-6929 ;EISSN: 2005-8330 ;DOI: 10.3348/kjr.2014.15.6.659 ;PMID: 25469078

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5
2019 HRS/EHRA/APHRS/LAHRS expert consensus statement on catheter ablation of ventricular arrhythmias: Executive summary
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2019 HRS/EHRA/APHRS/LAHRS expert consensus statement on catheter ablation of ventricular arrhythmias: Executive summary

Journal of arrhythmia, 2020-02, Vol.36 (1), p.1-58 [Peer Reviewed Journal]

2020 The Authors. published by John Wiley & Sons Australia, Ltd on behalf of the Japanese Heart Rhythm Society. ;2020 The Authors. Journal of Arrhythmia published by John Wiley & Sons Australia, Ltd on behalf of the Japanese Heart Rhythm Society. ;COPYRIGHT 2020 John Wiley & Sons, Inc. ;2020. This work is published under http://creativecommons.org/licenses/by-nc-nd/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;ISSN: 1880-4276 ;EISSN: 1883-2148 ;DOI: 10.1002/joa3.12264 ;PMID: 32071620

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6
3',4'-Dihydroxyflavonol antioxidant attenuates diastolic dysfunction and cardiac remodeling in streptozotocin-induced diabetic m(Ren2)27 rats
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3',4'-Dihydroxyflavonol antioxidant attenuates diastolic dysfunction and cardiac remodeling in streptozotocin-induced diabetic m(Ren2)27 rats

PloS one, 2011-07, Vol.6 (7), p.e22777-e22777 [Peer Reviewed Journal]

2011 Khong et al. This is an open-access article distributed under the terms of the Creative Commons Attribution License: https://creativecommons.org/licenses/by/4.0/ (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;Khong et al. 2011 ;ISSN: 1932-6203 ;EISSN: 1932-6203 ;DOI: 10.1371/journal.pone.0022777 ;PMID: 21829510

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7
4‐O‐methylhonokiol protects against diabetic cardiomyopathy in type 2 diabetic mice by activation of AMPK‐mediated cardiac lipid metabolism improvement
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4‐O‐methylhonokiol protects against diabetic cardiomyopathy in type 2 diabetic mice by activation of AMPK‐mediated cardiac lipid metabolism improvement

Journal of cellular and molecular medicine, 2019-08, Vol.23 (8), p.5771-5781 [Peer Reviewed Journal]

2019 The Authors. Journal of Cellular and Molecular Medicine published by John Wiley & Sons Ltd and Foundation for Cellular and Molecular Medicine. ;2019. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;ISSN: 1582-1838 ;EISSN: 1582-4934 ;DOI: 10.1111/jcmm.14493 ;PMID: 31199069

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8
99mTc-DPD uptake reflects amyloid fibril composition in hereditary transthyretin amyloidosis
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99mTc-DPD uptake reflects amyloid fibril composition in hereditary transthyretin amyloidosis

Upsala journal of medical sciences, 2016-01, Vol.121 (1), p.17-24 [Peer Reviewed Journal]

2016 Taylor & Francis 2016 ;2016 Taylor & Francis. This work is licensed under the Creative Commons Attribution License http://creativecommons.org/Licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;ISSN: 0300-9734 ;EISSN: 2000-1967 ;DOI: 10.3109/03009734.2015.1122687 ;PMID: 26849806

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9
99mTechnetium‐pyrophosphate scintigraphy: a practical guide for early diagnosis of transthyretin amyloid cardiomyopathy
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99mTechnetium‐pyrophosphate scintigraphy: a practical guide for early diagnosis of transthyretin amyloid cardiomyopathy

ESC Heart Failure, 2022-02, Vol.9 (1), p.251-262 [Peer Reviewed Journal]

2021 The Authors. ESC Heart Failure published by John Wiley & Sons Ltd on behalf of European Society of Cardiology. ;2022. This work is published under http://creativecommons.org/licenses/by-nc-nd/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;ISSN: 2055-5822 ;EISSN: 2055-5822 ;DOI: 10.1002/ehf2.13693 ;PMID: 34841715

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10
Aberrant activation of AMP-activated protein kinase remodels metabolic network in favor of cardiac glycogen storage
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Aberrant activation of AMP-activated protein kinase remodels metabolic network in favor of cardiac glycogen storage

The Journal of clinical investigation, 2007-05, Vol.117 (5), p.1432-1439 [Peer Reviewed Journal]

COPYRIGHT 2007 American Society for Clinical Investigation ;Copyright American Society for Clinical Investigation May 2007 ;Copyright © 2007, American Society for Clinical Investigation 2007 ;ISSN: 0021-9738 ;EISSN: 1558-8238 ;DOI: 10.1172/jci30658 ;PMID: 17431505

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11
Ablation of ALCAT1 Mitigates Hypertrophic Cardiomyopathy through Effects on Oxidative Stress and Mitophagy
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Ablation of ALCAT1 Mitigates Hypertrophic Cardiomyopathy through Effects on Oxidative Stress and Mitophagy

Molecular and Cellular Biology, 2012-11, Vol.32 (21), p.4493-4504 [Peer Reviewed Journal]

Copyright © 2012, American Society for Microbiology 2012 ;Copyright © 2012, American Society for Microbiology. All Rights Reserved. 2012 American Society for Microbiology ;ISSN: 0270-7306 ;ISSN: 1098-5549 ;EISSN: 1098-5549 ;DOI: 10.1128/MCB.01092-12 ;PMID: 22949503

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12
Ablation of cyclase-associated protein 2 (CAP2) leads to cardiomyopathy
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Ablation of cyclase-associated protein 2 (CAP2) leads to cardiomyopathy

Cellular and molecular life sciences : CMLS, 2013-02, Vol.70 (3), p.527-543 [Peer Reviewed Journal]

Springer Basel AG 2012 ;Springer Basel 2013 ;ISSN: 1420-682X ;EISSN: 1420-9071 ;DOI: 10.1007/s00018-012-1142-y ;PMID: 22945801

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13
Ablation of lysophosphatidic acid receptor 1 attenuates hypertrophic cardiomyopathy in a mouse model
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Ablation of lysophosphatidic acid receptor 1 attenuates hypertrophic cardiomyopathy in a mouse model

Proceedings of the National Academy of Sciences - PNAS, 2022-07, Vol.119 (28), p.e2204174119-e2204174119 [Peer Reviewed Journal]

Copyright National Academy of Sciences Jul 12, 2022 ;Copyright © 2022 the Author(s). Published by PNAS. 2022 ;ISSN: 0027-8424 ;EISSN: 1091-6490 ;DOI: 10.1073/pnas.2204174119 ;PMID: 35787042

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14
Ablation of Ventricular Tachycardia Originating from the Right Ventricle Associated with Scleroderma Cardiomyopathy
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Ablation of Ventricular Tachycardia Originating from the Right Ventricle Associated with Scleroderma Cardiomyopathy

Journal of Arrhythmia, 2005, Vol.21 (5), p.542-545 [Peer Reviewed Journal]

ISSN: 1880-4276 ;EISSN: 1883-2148

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15
Abnormal T2-STIR magnetic resonance in hypertrophic cardiomyopathy: a marker of advanced disease and electrical myocardial instability
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Abnormal T2-STIR magnetic resonance in hypertrophic cardiomyopathy: a marker of advanced disease and electrical myocardial instability

PloS one, 2014-10, Vol.9 (10), p.e111366-e111366 [Peer Reviewed Journal]

COPYRIGHT 2014 Public Library of Science ;COPYRIGHT 2014 Public Library of Science ;2014 Todiere et al. This is an open-access article distributed under the terms of the Creative Commons Attribution License: http://creativecommons.org/licenses/by/4.0/ (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2014 Todiere et al 2014 Todiere et al ;ISSN: 1932-6203 ;EISSN: 1932-6203 ;DOI: 10.1371/journal.pone.0111366 ;PMID: 25356653

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16
Abrupt aggravation of atrioventricular block and syncope in hypertrophic cardiomyopathy
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Abrupt aggravation of atrioventricular block and syncope in hypertrophic cardiomyopathy

Archives of disease in childhood, 1995-12, Vol.73 (6), p.536-537 [Peer Reviewed Journal]

1996 INIST-CNRS ;ISSN: 0003-9888 ;EISSN: 1468-2044 ;DOI: 10.1136/adc.73.6.536 ;PMID: 8546514 ;CODEN: ADCHAK

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17
Absence of linkage between idiopathic dilated cardiomyopathy and candidate genes involved in the immune function in a large Italian pedigree
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Absence of linkage between idiopathic dilated cardiomyopathy and candidate genes involved in the immune function in a large Italian pedigree

Journal of medical genetics, 1994-10, Vol.31 (10), p.766-771 [Peer Reviewed Journal]

Copyright BMJ Publishing Group LTD Oct 1994 ;ISSN: 0022-2593 ;ISSN: 1468-6244 ;EISSN: 1468-6244 ;DOI: 10.1136/jmg.31.10.766 ;PMID: 7837253 ;CODEN: JMDGAE

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18
Absence of Mannose-Binding Lectin Prevents Hyperglycemic Cardiovascular Complications
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Absence of Mannose-Binding Lectin Prevents Hyperglycemic Cardiovascular Complications

The American journal of pathology, 2012, Vol.180 (1), p.104-112 [Peer Reviewed Journal]

American Society for Investigative Pathology ;2012 American Society for Investigative Pathology ;2015 INIST-CNRS ;Copyright © 2012 American Society for Investigative Pathology. Published by Elsevier Inc. All rights reserved. ;2012 American Society for Investigative Pathology. Published by Elsevier Inc. All rights reserved. 2012 American Society for Investigative Pathology ;ISSN: 0002-9440 ;EISSN: 1525-2191 ;DOI: 10.1016/j.ajpath.2011.09.026 ;PMID: 22079428 ;CODEN: AJPAA4

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19
Accelerating the Mdx Heart Histo-Pathology through Physical Exercise
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Accelerating the Mdx Heart Histo-Pathology through Physical Exercise

Life (Basel, Switzerland), 2021-07, Vol.11 (7), p.706 [Peer Reviewed Journal]

2021 by the authors. Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (https://creativecommons.org/licenses/by/4.0/). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2021 by the authors. 2021 ;ISSN: 2075-1729 ;EISSN: 2075-1729 ;DOI: 10.3390/life11070706 ;PMID: 34357078

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20
Access and Outcomes Among Hypertrophic Cardiomyopathy Patients in a Large Integrated Health System
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Access and Outcomes Among Hypertrophic Cardiomyopathy Patients in a Large Integrated Health System

Journal of the American Heart Association, 2020-02, Vol.9 (3), p.e014095-e014095 [Peer Reviewed Journal]

2020 The Authors. Published on behalf of the American Heart Association, Inc., by Wiley. ;ISSN: 2047-9980 ;EISSN: 2047-9980 ;DOI: 10.1161/jaha.119.014095 ;PMID: 31973610

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