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| Result Number | Material Type | Add to My Shelf Action | Record Details and Options |
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| 1 |
Material Type: Article
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Hypertrophic cardiomyopathy dysfunction mimicked in human engineered heart tissue and improved by sodium–glucose cotransporter 2 inhibitorsCardiovascular research, 2024-03, Vol.120 (3), p.301-317 [Peer Reviewed Journal]The Author(s) 2024. Published by Oxford University Press on behalf of the European Society of Cardiology. 2024 ;The Author(s) 2024. Published by Oxford University Press on behalf of the European Society of Cardiology. ;ISSN: 0008-6363 ;EISSN: 1755-3245 ;DOI: 10.1093/cvr/cvae004 ;PMID: 38240646Full text available |
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IKKβ stabilizes Mitofusin 2 and suppresses doxorubicin cardiomyopathyCardiovascular research, 2024-03, Vol.120 (2), p.164-173 [Peer Reviewed Journal]The Author(s) 2024. Published by Oxford University Press on behalf of the European Society of Cardiology. All rights reserved. For permissions, please e-mail: journals.permissions@oup.com 2024 ;The Author(s) 2024. Published by Oxford University Press on behalf of the European Society of Cardiology. All rights reserved. For permissions, please e-mail: journals.permissions@oup.com. ;ISSN: 0008-6363 ;EISSN: 1755-3245 ;DOI: 10.1093/cvr/cvad145 ;PMID: 38165268Full text available |
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RBPMS regulates cardiomyocyte contraction and cardiac function through RNA alternative splicingCardiovascular research, 2024-02, Vol.120 (1), p.56-68 [Peer Reviewed Journal]Published by Oxford University Press on behalf of the European Society of Cardiology 2023. 2023 ;Published by Oxford University Press on behalf of the European Society of Cardiology 2023. ;ISSN: 0008-6363 ;EISSN: 1755-3245 ;DOI: 10.1093/cvr/cvad166 ;PMID: 37890031Full text available |
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Dysregulated iron homeostasis in dystrophin-deficient cardiomyocytes: correction by gene editing and pharmacological treatmentCardiovascular research, 2024-02, Vol.120 (1), p.69-81 [Peer Reviewed Journal]The Author(s) 2023. Published by Oxford University Press on behalf of the European Society of Cardiology. 2023 ;The Author(s) 2023. Published by Oxford University Press on behalf of the European Society of Cardiology. ;ISSN: 0008-6363 ;EISSN: 1755-3245 ;DOI: 10.1093/cvr/cvad182 ;PMID: 38078368Full text available |
| 5 |
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Single-nuclear transcriptome profiling identifies persistent fibroblast activation in hypertrophic and failing human hearts of patients with longstanding diseaseCardiovascular research, 2023-11, Vol.119 (15), p.2550-2562 [Peer Reviewed Journal]The Author(s) 2023. Published by Oxford University Press on behalf of the European Society of Cardiology. All rights reserved. For permissions, please e-mail: journals.permissions@oup.com 2023 ;The Author(s) 2023. Published by Oxford University Press on behalf of the European Society of Cardiology. All rights reserved. For permissions, please e-mail: journals.permissions@oup.com. ;ISSN: 0008-6363 ;EISSN: 1755-3245 ;DOI: 10.1093/cvr/cvad140 ;PMID: 37648651Full text available |
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FAM210A regulates mitochondrial translation and maintains cardiac mitochondrial homeostasisCardiovascular research, 2023-11, Vol.119 (14), p.2441-2457 [Peer Reviewed Journal]The Author(s) 2023. Published by Oxford University Press on behalf of the European Society of Cardiology. All rights reserved. For permissions, please e-mail: journals.permissions@oup.com 2023 ;The Author(s) 2023. Published by Oxford University Press on behalf of the European Society of Cardiology. All rights reserved. For permissions, please e-mail: journals.permissions@oup.com. ;ISSN: 0008-6363 ;EISSN: 1755-3245 ;DOI: 10.1093/cvr/cvad124 ;PMID: 37522353Full text available |
| 7 |
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Immune profiling of murine cardiac leukocytes identifies triggering receptor expressed on myeloid cells 2 as a novel mediator of hypertensive heart failureCardiovascular research, 2023-10, Vol.119 (13), p.2312-2328 [Peer Reviewed Journal]The Author(s) 2023. Published by Oxford University Press on behalf of the European Society of Cardiology. All rights reserved. For permissions, please e-mail: journals.permissions@oup.com 2023 ;The Author(s) 2023. Published by Oxford University Press on behalf of the European Society of Cardiology. All rights reserved. For permissions, please e-mail: journals.permissions@oup.com. ;ISSN: 0008-6363 ;EISSN: 1755-3245 ;DOI: 10.1093/cvr/cvad093 ;PMID: 37314125Full text available |
| 8 |
Material Type: Article
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Takotsubo syndrome: getting closer to its causesCardiovascular research, 2023-07, Vol.119 (7), p.1480-1494 [Peer Reviewed Journal]The Author(s) 2023. Published by Oxford University Press on behalf of the European Society of Cardiology. All rights reserved. For permissions, please e-mail: journals.permissions@oup.com 2023 ;The Author(s) 2023. Published by Oxford University Press on behalf of the European Society of Cardiology. All rights reserved. For permissions, please e-mail: journals.permissions@oup.com. ;ISSN: 0008-6363 ;EISSN: 1755-3245 ;DOI: 10.1093/cvr/cvad053 ;PMID: 37183265Full text available |
| 9 |
Material Type: Article
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Diabetic cardiomyopathy: the need for adjusting experimental models to meet clinical realityCardiovascular research, 2023-05, Vol.119 (5), p.1130-1145 [Peer Reviewed Journal]The Author(s) 2022. Published by Oxford University Press on behalf of the European Society of Cardiology. All rights reserved. For permissions, please email: journals.permissions@oup.com. 2022 ;The Author(s) 2022. Published by Oxford University Press on behalf of the European Society of Cardiology. All rights reserved. For permissions, please email: journals.permissions@oup.com. ;Distributed under a Creative Commons Attribution 4.0 International License ;ISSN: 0008-6363 ;EISSN: 1755-3245 ;DOI: 10.1093/cvr/cvac152 ;PMID: 36082907Full text available |
| 10 |
Material Type: Article
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Scientists on the spot: Relaxing the heart in hypertrophic cardiomyopathyCardiovascular research, 2023-05, Vol.119 (5), p.e131-e132 [Peer Reviewed Journal]The Author(s) 2023. Published by Oxford University Press on behalf of the European Society of Cardiology. 2023 ;ISSN: 0008-6363 ;EISSN: 1755-3245 ;DOI: 10.1093/cvr/cvad049 ;PMID: 37102235Full text available |
| 11 |
Material Type: Article
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Cellular interplay between cardiomyocytes and non-myocytes in diabetic cardiomyopathyCardiovascular research, 2023-05, Vol.119 (3), p.668-690 [Peer Reviewed Journal]The Author(s) 2022. Published by Oxford University Press on behalf of the European Society of Cardiology. 2022 ;The Author(s) 2022. Published by Oxford University Press on behalf of the European Society of Cardiology. ;ISSN: 0008-6363 ;EISSN: 1755-3245 ;DOI: 10.1093/cvr/cvac049 ;PMID: 35388880Full text available |
| 12 |
Material Type: Article
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A novel patient–patient network medicine approach to refine hypertrophic cardiomyopathy subgrouping: implications for risk stratificationCardiovascular research, 2023-05, Vol.119 (3), p.e125-e127 [Peer Reviewed Journal]Published by Oxford University Press on behalf of the European Society of Cardiology 2023. 2023 ;ISSN: 0008-6363 ;EISSN: 1755-3245 ;DOI: 10.1093/cvr/cvad036 ;PMID: 37129987Full text available |
| 13 |
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Spatial transcriptomics unveils ZBTB11 as a regulator of cardiomyocyte degeneration in arrhythmogenic cardiomyopathyCardiovascular research, 2023-03, Vol.119 (2), p.477-491 [Peer Reviewed Journal]The Author(s) 2022. Published by Oxford University Press on behalf of the European Society of Cardiology. ;The Author(s) 2022. Published by Oxford University Press on behalf of the European Society of Cardiology. 2022 ;ISSN: 0008-6363 ;EISSN: 1755-3245 ;DOI: 10.1093/cvr/cvac072 ;PMID: 35576477Full text available |
| 14 |
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Frequent premature atrial contractions as a signalling marker of atrial cardiomyopathy, incident atrial fibrillation, and strokeCardiovascular research, 2023-03, Vol.119 (2), p.429-439 [Peer Reviewed Journal]The Author(s) 2022. Published by Oxford University Press on behalf of the European Society of Cardiology. ;The Author(s) 2022. Published by Oxford University Press on behalf of the European Society of Cardiology. 2022 ;ISSN: 0008-6363 ;EISSN: 1755-3245 ;DOI: 10.1093/cvr/cvac054 ;PMID: 35388889Full text available |
| 15 |
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Transthyretin cardiac amyloidosisCardiovascular research, 2023-02, Vol.118 (18), p.3517-3535 [Peer Reviewed Journal]The Author(s) 2022. Published by Oxford University Press on behalf of the European Society of Cardiology. ;The Author(s) 2022. Published by Oxford University Press on behalf of the European Society of Cardiology. 2022 ;ISSN: 0008-6363 ;EISSN: 1755-3245 ;DOI: 10.1093/cvr/cvac119 ;PMID: 35929637Full text available |
| 16 |
Material Type: Article
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Gene editing reverses arrhythmia susceptibility in humanized PLN-R14del mice: modelling a European cardiomyopathy with global impactCardiovascular research, 2022-12, Vol.118 (15), p.3140-3150 [Peer Reviewed Journal]Published on behalf of the European Society of Cardiology. All rights reserved. © The Author(s) 2022. For permissions, please email: journals.permissions@oup.com. ;Published on behalf of the European Society of Cardiology. All rights reserved. © The Author(s) 2022. For permissions, please email: journals.permissions@oup.com. 2022 ;ISSN: 0008-6363 ;EISSN: 1755-3245 ;DOI: 10.1093/cvr/cvac021 ;PMID: 35191471Full text available |
| 17 |
Material Type: Article
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Titin (TTN): from molecule to modifications, mechanics, and medical significanceCardiovascular research, 2022-11, Vol.118 (14), p.2903-2918 [Peer Reviewed Journal]The Author(s) 2021. Published by Oxford University Press on behalf of the European Society of Cardiology. ;The Author(s) 2021. Published by Oxford University Press on behalf of the European Society of Cardiology. 2021 ;ISSN: 0008-6363 ;EISSN: 1755-3245 ;DOI: 10.1093/cvr/cvab328 ;PMID: 34662387Full text available |
| 18 |
Material Type: Article
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Sporadic dilated cardiomyopathy is often familialCardiovascular research, 2022-07, Vol.118 (10), p.e69-e71 [Peer Reviewed Journal]The Author(s) 2022. Published by Oxford University Press on behalf of the European Society of Cardiology. All rights reserved. For permissions, please email: journals.permissions@oup.com. 2022 ;ISSN: 0008-6363 ;EISSN: 1755-3245 ;DOI: 10.1093/cvr/cvac075 ;PMID: 35726845Full text available |
| 19 |
Material Type: Article
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The EP300/TP53 pathway, a suppressor of the Hippo and canonical WNT pathways, is activated in human hearts with arrhythmogenic cardiomyopathy in the absence of overt heart failureCardiovascular research, 2022-05, Vol.118 (6), p.1466-1478 [Peer Reviewed Journal]Published on behalf of the European Society of Cardiology. All rights reserved. © The Author(s) 2021. For permissions, please email: journals.permissions@oup.com. 2021 ;Published on behalf of the European Society of Cardiology. All rights reserved. © The Author(s) 2021. For permissions, please email: journals.permissions@oup.com. ;ISSN: 0008-6363 ;EISSN: 1755-3245 ;DOI: 10.1093/cvr/cvab197 ;PMID: 34132777Full text available |
| 20 |
Material Type: Article
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Human BIN1 isoforms grow, maintain, and regenerate excitation–contraction couplons in adult rat and human stem cell-derived cardiomyocytesCardiovascular research, 2022-05, Vol.118 (6), p.1479-1491 [Peer Reviewed Journal]Published on behalf of the European Society of Cardiology. All rights reserved. © The Author(s) 2021. For permissions, please email: journals.permissions@oup.com. 2021 ;Published on behalf of the European Society of Cardiology. All rights reserved. © The Author(s) 2021. For permissions, please email: journals.permissions@oup.com. ;ISSN: 0008-6363 ;EISSN: 1755-3245 ;DOI: 10.1093/cvr/cvab195 ;PMID: 34152414Full text available |
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