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1
Genes from Chagas susceptibility loci that are differentially expressed in T. cruzi-resistant mice are candidates accounting for impaired immunity
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Genes from Chagas susceptibility loci that are differentially expressed in T. cruzi-resistant mice are candidates accounting for impaired immunity

PloS one, 2006-12, Vol.1 (1), p.e57-e57 [Peer Reviewed Journal]

COPYRIGHT 2006 Public Library of Science ;2006 Graefe et al. This is an open-access article distributed under the terms of the Creative Commons Attribution License (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;Graefe et al. 2006 ;ISSN: 1932-6203 ;EISSN: 1932-6203 ;DOI: 10.1371/journal.pone.0000057 ;PMID: 17183687

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2
Mitochondrial oxidative stress causes hyperphosphorylation of tau
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Mitochondrial oxidative stress causes hyperphosphorylation of tau

PloS one, 2007-06, Vol.2 (6), p.e536-e536 [Peer Reviewed Journal]

COPYRIGHT 2007 Public Library of Science ;2007 Melov et al. This is an open-access article distributed under the terms of the Creative Commons Attribution License (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;Melov et al. 2007 ;ISSN: 1932-6203 ;EISSN: 1932-6203 ;DOI: 10.1371/journal.pone.0000536 ;PMID: 17579710

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3
Genetic variation for cardiac dysfunction in Drosophila
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Genetic variation for cardiac dysfunction in Drosophila

PloS one, 2007-07, Vol.2 (7), p.e601-e601 [Peer Reviewed Journal]

COPYRIGHT 2007 Public Library of Science ;2007 Ocorr et al. This is an open-access article distributed under the terms of the Creative Commons Attribution License (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;Ocorr et al. 2007 ;ISSN: 1932-6203 ;EISSN: 1932-6203 ;DOI: 10.1371/journal.pone.0000601 ;PMID: 17622346

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4
TrpC3 regulates hypertrophy-associated gene expression without affecting myocyte beating or cell size
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TrpC3 regulates hypertrophy-associated gene expression without affecting myocyte beating or cell size

PloS one, 2007-08, Vol.2 (8), p.e802-e802 [Peer Reviewed Journal]

COPYRIGHT 2007 Public Library of Science ;2007 Brenner, Dolmetsch. This is an open-access article distributed under the terms of the Creative Commons Attribution License (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;Brenner, Dolmetsch. 2007 ;ISSN: 1932-6203 ;EISSN: 1932-6203 ;DOI: 10.1371/journal.pone.0000802 ;PMID: 17726532

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5
Variable expression of Cre recombinase transgenes precludes reliable prediction of tissue-specific gene disruption by tail-biopsy genotyping
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Variable expression of Cre recombinase transgenes precludes reliable prediction of tissue-specific gene disruption by tail-biopsy genotyping

PloS one, 2007-10, Vol.2 (10), p.e1013-e1013 [Peer Reviewed Journal]

COPYRIGHT 2007 Public Library of Science ;2007 Schulz et al. This is an open-access article distributed under the terms of the Creative Commons Attribution License (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;Schulz et al. 2007 ;ISSN: 1932-6203 ;EISSN: 1932-6203 ;DOI: 10.1371/journal.pone.0001013 ;PMID: 17925861

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6
The R403Q myosin mutation implicated in familial hypertrophic cardiomyopathy causes disorder at the actomyosin interface
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The R403Q myosin mutation implicated in familial hypertrophic cardiomyopathy causes disorder at the actomyosin interface

PloS one, 2007-11, Vol.2 (11), p.e1123-e1123 [Peer Reviewed Journal]

COPYRIGHT 2007 Public Library of Science ;2007 Volkmann et al. This is an open-access article distributed under the terms of the Creative Commons Attribution License (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;Volkmann et al. 2007 ;ISSN: 1932-6203 ;EISSN: 1932-6203 ;DOI: 10.1371/journal.pone.0001123 ;PMID: 17987111

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7
Linking gene expression and functional network data in human heart failure
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Linking gene expression and functional network data in human heart failure

PloS one, 2007-12, Vol.2 (12), p.e1347 [Peer Reviewed Journal]

COPYRIGHT 2007 Public Library of Science ;2007 Camargo, Azuaje. This is an open-access article distributed under the terms of the Creative Commons Attribution License (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;Camargo, Azuaje. 2007 ;ISSN: 1932-6203 ;EISSN: 1932-6203 ;DOI: 10.1371/journal.pone.0001347 ;PMID: 18094754

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8
Noncompaction of the ventricular myocardium is associated with a de novo mutation in the beta-myosin heavy chain gene
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Noncompaction of the ventricular myocardium is associated with a de novo mutation in the beta-myosin heavy chain gene

PloS one, 2007-12, Vol.2 (12), p.e1362-e1362 [Peer Reviewed Journal]

2007 Budde et al. This is an open-access article distributed under the terms of the Creative Commons Attribution License (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;Budde et al. 2007 ;ISSN: 1932-6203 ;EISSN: 1932-6203 ;DOI: 10.1371/journal.pone.0001362 ;PMID: 18159245

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9
Paracrine factors of mesenchymal stem cells recruit macrophages and endothelial lineage cells and enhance wound healing
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Paracrine factors of mesenchymal stem cells recruit macrophages and endothelial lineage cells and enhance wound healing

PloS one, 2008-04, Vol.3 (4), p.e1886-e1886 [Peer Reviewed Journal]

COPYRIGHT 2008 Public Library of Science ;2008 Chen et al. This is an open-access article distributed under the terms of the Creative Commons Attribution License (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;Chen et al. 2008 ;ISSN: 1932-6203 ;EISSN: 1932-6203 ;DOI: 10.1371/journal.pone.0001886 ;PMID: 18382669

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10
NS1 specific CD8+ T-cells with effector function and TRBV11 dominance in a patient with parvovirus B19 associated inflammatory cardiomyopathy
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Article
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NS1 specific CD8+ T-cells with effector function and TRBV11 dominance in a patient with parvovirus B19 associated inflammatory cardiomyopathy

PloS one, 2008-06, Vol.3 (6), p.e2361-e2361 [Peer Reviewed Journal]

2008 Streitz et al. This is an open-access article distributed under the terms of the Creative Commons Attribution License (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;Streitz et al. 2008 ;ISSN: 1932-6203 ;EISSN: 1932-6203 ;DOI: 10.1371/journal.pone.0002361 ;PMID: 18523634

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11
NS1 Specific CD8.sup.+ T-Cells with Effector Function and TRBV11 Dominance in a Patient with Parvovirus B19 Associated Inflammatory Cardiomyopathy
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NS1 Specific CD8.sup.+ T-Cells with Effector Function and TRBV11 Dominance in a Patient with Parvovirus B19 Associated Inflammatory Cardiomyopathy

PloS one, 2008-06, Vol.3 (6), p.e2361 [Peer Reviewed Journal]

COPYRIGHT 2008 Public Library of Science ;ISSN: 1932-6203 ;EISSN: 1932-6203 ;DOI: 10.1371/journal.pone.0002361

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12
Destabilization of the dystrophin-glycoprotein complex without functional deficits in alpha-dystrobrevin null muscle
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Destabilization of the dystrophin-glycoprotein complex without functional deficits in alpha-dystrobrevin null muscle

PloS one, 2008-07, Vol.3 (7), p.e2604-e2604 [Peer Reviewed Journal]

2008 Bunnell et al. This is an open-access article distributed under the terms of the Creative Commons Attribution License (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;Bunnell et al. 2008 ;ISSN: 1932-6203 ;EISSN: 1932-6203 ;DOI: 10.1371/journal.pone.0002604 ;PMID: 18596960

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13
Transcriptome alteration in the diabetic heart by rosiglitazone: implications for cardiovascular mortality
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Transcriptome alteration in the diabetic heart by rosiglitazone: implications for cardiovascular mortality

PloS one, 2008-07, Vol.3 (7), p.e2609-e2609 [Peer Reviewed Journal]

COPYRIGHT 2008 Public Library of Science ;2008 Wilson et al. This is an open-access article distributed under the terms of the Creative Commons Attribution License (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;Wilson et al. 2008 ;ISSN: 1932-6203 ;EISSN: 1932-6203 ;DOI: 10.1371/journal.pone.0002609 ;PMID: 18648539

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14
The role of cardiac troponin T quantity and function in cardiac development and dilated cardiomyopathy
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The role of cardiac troponin T quantity and function in cardiac development and dilated cardiomyopathy

PloS one, 2008-07, Vol.3 (7), p.e2642-e2642 [Peer Reviewed Journal]

COPYRIGHT 2008 Public Library of Science ;2008 Ahmad et al. This is an open-access article distributed under the terms of the Creative Commons Attribution License (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;Ahmad et al. 2008 ;ISSN: 1932-6203 ;EISSN: 1932-6203 ;DOI: 10.1371/journal.pone.0002642 ;PMID: 18612386

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15
Modulation of serotonin transporter function during fetal development causes dilated heart cardiomyopathy and lifelong behavioral abnormalities
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Modulation of serotonin transporter function during fetal development causes dilated heart cardiomyopathy and lifelong behavioral abnormalities

PloS one, 2008-07, Vol.3 (7), p.e2782 [Peer Reviewed Journal]

COPYRIGHT 2008 Public Library of Science ;2008 Noorlander et al. This is an open-access article distributed under the terms of the Creative Commons Attribution License (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;Noorlander et al. 2008 ;ISSN: 1932-6203 ;EISSN: 1932-6203 ;DOI: 10.1371/journal.pone.0002782 ;PMID: 18716672

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16
Emergence of Xin demarcates a key innovation in heart evolution
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Emergence of Xin demarcates a key innovation in heart evolution

PloS one, 2008-08, Vol.3 (8), p.e2857-e2857 [Peer Reviewed Journal]

COPYRIGHT 2008 Public Library of Science ;2008 Grosskurth et al. This is an open-access article distributed under the terms of the Creative Commons Attribution License (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;Grosskurth et al. 2008 ;ISSN: 1932-6203 ;EISSN: 1932-6203 ;DOI: 10.1371/journal.pone.0002857 ;PMID: 18682726

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17
Choice of cell-delivery route for skeletal myoblast transplantation for treating post-infarction chronic heart failure in rat
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Choice of cell-delivery route for skeletal myoblast transplantation for treating post-infarction chronic heart failure in rat

PloS one, 2008-08, Vol.3 (8), p.e3071-e3071 [Peer Reviewed Journal]

COPYRIGHT 2008 Public Library of Science ;2008 Fukushima et al. This is an open-access article distributed under the terms of the Creative Commons Attribution License (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;Fukushima et al. 2008 ;ISSN: 1932-6203 ;EISSN: 1932-6203 ;DOI: 10.1371/journal.pone.0003071 ;PMID: 18728781

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18
The variability of the harlequin mouse phenotype resembles that of human mitochondrial-complex I-deficiency syndromes
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The variability of the harlequin mouse phenotype resembles that of human mitochondrial-complex I-deficiency syndromes

PloS one, 2008-09, Vol.3 (9), p.e3208-e3208 [Peer Reviewed Journal]

COPYRIGHT 2008 Public Library of Science ;2008 Bénit et al. This is an open-access article distributed under the terms of the Creative Commons Attribution License (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;Bénit et al. 2008 ;ISSN: 1932-6203 ;EISSN: 1932-6203 ;DOI: 10.1371/journal.pone.0003208 ;PMID: 18791645

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19
Magic-factor 1, a partial agonist of Met, induces muscle hypertrophy by protecting myogenic progenitors from apoptosis
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Magic-factor 1, a partial agonist of Met, induces muscle hypertrophy by protecting myogenic progenitors from apoptosis

PloS one, 2008-09, Vol.3 (9), p.e3223-e3223 [Peer Reviewed Journal]

COPYRIGHT 2008 Public Library of Science ;2008 Cassano et al. This is an open-access article distributed under the terms of the Creative Commons Attribution License (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;Cassano et al. 2008 ;ISSN: 1932-6203 ;EISSN: 1932-6203 ;DOI: 10.1371/journal.pone.0003223 ;PMID: 18795097

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20
Effects of lysosomal membrane protein depletion on the Salmonella-containing vacuole
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Effects of lysosomal membrane protein depletion on the Salmonella-containing vacuole

PloS one, 2008-10, Vol.3 (10), p.e3538-e3538 [Peer Reviewed Journal]

COPYRIGHT 2008 Public Library of Science ;2008 Roark et al. This is an open-access article distributed under the terms of the Creative Commons Attribution License (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;Roark et al. 2008 ;ISSN: 1932-6203 ;EISSN: 1932-6203 ;DOI: 10.1371/journal.pone.0003538 ;PMID: 18958159

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