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1
Exploration and validation of signature genes and immune associations in septic cardiomyopathy
Exploration and validation of signature genes and immune associations in septic cardiomyopathy
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Exploration and validation of signature genes and immune associations in septic cardiomyopathy

Clinical and experimental hypertension (1993), 2024-12, Vol.46 (1), p.2302570-2302570 [Peer Reviewed Journal]

ISSN: 1064-1963 ;EISSN: 1525-6006 ;DOI: 10.1080/10641963.2024.2302570 ;PMID: 38281072

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2
Molecular mechanism of triptolide in myocardial fibrosis through the Wnt/β-catenin signaling pathway
Molecular mechanism of triptolide in myocardial fibrosis through the Wnt/β-catenin signaling pathway
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Molecular mechanism of triptolide in myocardial fibrosis through the Wnt/β-catenin signaling pathway

Scandinavian cardiovascular journal : SCJ, 2024-12, Vol.58 (1), p.2295785 [Peer Reviewed Journal]

EISSN: 1651-2006 ;DOI: 10.1080/14017431.2023.2295785 ;PMID: 38164796

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3
Catheter Ablation for Ventricular Tachycardia in Patients With Desmoplakin Cardiomyopathy
Catheter Ablation for Ventricular Tachycardia in Patients With Desmoplakin Cardiomyopathy
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Catheter Ablation for Ventricular Tachycardia in Patients With Desmoplakin Cardiomyopathy

ISSN: 2405-500X ;EISSN: 2405-5018 ;DOI: 10.1016/j.jacep.2023.11.017 ;DOI: 10.5167/uzh-257657

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4
The MMP-9 promoter genetic variant rs3918242, mRNA and protein expression in advanced carotid plaque tissue
The MMP-9 promoter genetic variant rs3918242, mRNA and protein expression in advanced carotid plaque tissue
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The MMP-9 promoter genetic variant rs3918242, mRNA and protein expression in advanced carotid plaque tissue

Molecular biology reports, 2024-12, Vol.51 (1), p.540-540 [Peer Reviewed Journal]

The Author(s), under exclusive licence to Springer Nature B.V. 2024. Springer Nature or its licensor (e.g. a society or other partner) holds exclusive rights to this article under a publishing agreement with the author(s) or other rightsholder(s); author self-archiving of the accepted manuscript version of this article is solely governed by the terms of such publishing agreement and applicable law. ;2024. The Author(s), under exclusive licence to Springer Nature B.V. ;ISSN: 0301-4851 ;EISSN: 1573-4978 ;DOI: 10.1007/s11033-024-09458-w ;PMID: 38642151

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5
Post translational modifications of connexin 43 in ventricular arrhythmias after myocardial infarction
Post translational modifications of connexin 43 in ventricular arrhythmias after myocardial infarction
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Post translational modifications of connexin 43 in ventricular arrhythmias after myocardial infarction

Molecular biology reports, 2024-12, Vol.51 (1), p.329-329 [Peer Reviewed Journal]

The Author(s), under exclusive licence to Springer Nature B.V. 2024. Springer Nature or its licensor (e.g. a society or other partner) holds exclusive rights to this article under a publishing agreement with the author(s) or other rightsholder(s); author self-archiving of the accepted manuscript version of this article is solely governed by the terms of such publishing agreement and applicable law. ;2024. The Author(s), under exclusive licence to Springer Nature B.V. ;ISSN: 0301-4851 ;EISSN: 1573-4978 ;DOI: 10.1007/s11033-024-09290-2 ;PMID: 38393658

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6
Does epigenetic markers of HLA gene show association with coronary artery disease in Indian subjects?
Does epigenetic markers of HLA gene show association with coronary artery disease in Indian subjects?
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Does epigenetic markers of HLA gene show association with coronary artery disease in Indian subjects?

Molecular biology reports, 2024-12, Vol.51 (1), p.173-173, Article 173 [Peer Reviewed Journal]

The Author(s), under exclusive licence to Springer Nature B.V. 2024. Springer Nature or its licensor (e.g. a society or other partner) holds exclusive rights to this article under a publishing agreement with the author(s) or other rightsholder(s); author self-archiving of the accepted manuscript version of this article is solely governed by the terms of such publishing agreement and applicable law. ;2024. The Author(s), under exclusive licence to Springer Nature B.V. ;ISSN: 0301-4851 ;EISSN: 1573-4978 ;DOI: 10.1007/s11033-023-08974-5 ;PMID: 38252175

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7
Trisomy 21-driven metabolite alterations are linked to cellular injuries in Down syndrome
Trisomy 21-driven metabolite alterations are linked to cellular injuries in Down syndrome
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Trisomy 21-driven metabolite alterations are linked to cellular injuries in Down syndrome

Cellular and molecular life sciences : CMLS, 2024-12, Vol.81 (1), p.112-112 [Peer Reviewed Journal]

The Author(s) 2024 ;2024. The Author(s). ;The Author(s) 2024. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;ISSN: 1420-682X ;EISSN: 1420-9071 ;DOI: 10.1007/s00018-024-05127-0 ;PMID: 38433139

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8
Emerging roles for the ADAMTS-like family of matricellular proteins in cardiovascular disease through regulation of the extracellular microenvironment
Emerging roles for the ADAMTS-like family of matricellular proteins in cardiovascular disease through regulation of the extracellular microenvironment
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Emerging roles for the ADAMTS-like family of matricellular proteins in cardiovascular disease through regulation of the extracellular microenvironment

Molecular biology reports, 2024-12, Vol.51 (1), p.280-280 [Peer Reviewed Journal]

The Author(s) 2024 ;2024. The Author(s). ;The Author(s) 2024. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;ISSN: 0301-4851 ;EISSN: 1573-4978 ;DOI: 10.1007/s11033-024-09255-5 ;PMID: 38324186

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9
Over-expression of mir-181a-3p in serum of breast cancer patients as diagnostic biomarker
Over-expression of mir-181a-3p in serum of breast cancer patients as diagnostic biomarker
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Over-expression of mir-181a-3p in serum of breast cancer patients as diagnostic biomarker

Molecular biology reports, 2024-12, Vol.51 (1), p.372-372 [Peer Reviewed Journal]

The Author(s), under exclusive licence to Springer Nature B.V. 2024. Springer Nature or its licensor (e.g. a society or other partner) holds exclusive rights to this article under a publishing agreement with the author(s) or other rightsholder(s); author self-archiving of the accepted manuscript version of this article is solely governed by the terms of such publishing agreement and applicable law. ;2024. The Author(s), under exclusive licence to Springer Nature B.V. ;ISSN: 0301-4851 ;EISSN: 1573-4978 ;DOI: 10.1007/s11033-024-09272-4 ;PMID: 38411870

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10
Evaluating left atrial function changes by speckle tracking echocardiography in type 2 diabetes patients in Central Vietnam: a cross-sectional comparative study
Evaluating left atrial function changes by speckle tracking echocardiography in type 2 diabetes patients in Central Vietnam: a cross-sectional comparative study
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Evaluating left atrial function changes by speckle tracking echocardiography in type 2 diabetes patients in Central Vietnam: a cross-sectional comparative study

The Egyptian heart journal, 2024-12, Vol.76 (1), p.38-38 [Peer Reviewed Journal]

The Author(s) 2024 ;2024. The Author(s). ;COPYRIGHT 2024 Springer ;The Author(s) 2024. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;ISSN: 1110-2608 ;EISSN: 2090-911X ;DOI: 10.1186/s43044-024-00470-w ;PMID: 38546901

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11
Left atrial vortex flow and its relationship with left atrial functions in patients with congenital heart disease
Left atrial vortex flow and its relationship with left atrial functions in patients with congenital heart disease
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Left atrial vortex flow and its relationship with left atrial functions in patients with congenital heart disease

The Egyptian heart journal, 2024-12, Vol.76 (1), p.53-53 [Peer Reviewed Journal]

The Author(s) 2024 ;2024. The Author(s). ;COPYRIGHT 2024 Springer ;The Author(s) 2024. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;ISSN: 1110-2608 ;EISSN: 2090-911X ;DOI: 10.1186/s43044-024-00486-2 ;PMID: 38696068

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12
Mitochondrial impairment, decreased sirtuin activity and protein acetylation in dorsal root ganglia in Friedreich Ataxia models
Mitochondrial impairment, decreased sirtuin activity and protein acetylation in dorsal root ganglia in Friedreich Ataxia models
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Article 		Add to My Research

Mitochondrial impairment, decreased sirtuin activity and protein acetylation in dorsal root ganglia in Friedreich Ataxia models

Cellular and molecular life sciences : CMLS, 2024-12, Vol.81 (1), p.12-12, Article 12 [Peer Reviewed Journal]

The Author(s) 2023 ;2023. The Author(s). ;The Author(s) 2023. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;ISSN: 1420-682X ;EISSN: 1420-9071 ;DOI: 10.1007/s00018-023-05064-4 ;PMID: 38129330

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13
STAT protein family and cardiovascular diseases: overview of pathological mechanisms and therapeutic implications
STAT protein family and cardiovascular diseases: overview of pathological mechanisms and therapeutic implications
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Article 		Add to My Research

STAT protein family and cardiovascular diseases: overview of pathological mechanisms and therapeutic implications

Molecular biology reports, 2024-12, Vol.51 (1), p.440-440 [Peer Reviewed Journal]

The Author(s), under exclusive licence to Springer Nature B.V. 2024. Springer Nature or its licensor (e.g. a society or other partner) holds exclusive rights to this article under a publishing agreement with the author(s) or other rightsholder(s); author self-archiving of the accepted manuscript version of this article is solely governed by the terms of such publishing agreement and applicable law. ;2024. The Author(s), under exclusive licence to Springer Nature B.V. ;ISSN: 0301-4851 ;EISSN: 1573-4978 ;DOI: 10.1007/s11033-024-09371-2 ;PMID: 38520542

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14
Short-term regulation of TSFM level does not alter amyloidogenesis and mitochondrial function in type-specific cells
Short-term regulation of TSFM level does not alter amyloidogenesis and mitochondrial function in type-specific cells
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Short-term regulation of TSFM level does not alter amyloidogenesis and mitochondrial function in type-specific cells

Molecular biology reports, 2024-12, Vol.51 (1), p.484-484 [Peer Reviewed Journal]

The Author(s), under exclusive licence to Springer Nature B.V. 2024. Springer Nature or its licensor (e.g. a society or other partner) holds exclusive rights to this article under a publishing agreement with the author(s) or other rightsholder(s); author self-archiving of the accepted manuscript version of this article is solely governed by the terms of such publishing agreement and applicable law. ;2024. The Author(s), under exclusive licence to Springer Nature B.V. ;ISSN: 0301-4851 ;EISSN: 1573-4978 ;DOI: 10.1007/s11033-024-09426-4 ;PMID: 38578353

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15
CACA guidelines for holistic integrative management of adult acute myeloid leukemia
CACA guidelines for holistic integrative management of adult acute myeloid leukemia
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CACA guidelines for holistic integrative management of adult acute myeloid leukemia

Holistic Integrative Oncology, 2024-12, Vol.3 (1), p.12

The Author(s) 2024 ;The Author(s) 2024. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;EISSN: 2731-4529 ;DOI: 10.1007/s44178-024-00078-9

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16
An update of new/potential cardiovascular markers: a narrative review
An update of new/potential cardiovascular markers: a narrative review
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An update of new/potential cardiovascular markers: a narrative review

Molecular biology reports, 2024-12, Vol.51 (1), p.179-179, Article 179 [Peer Reviewed Journal]

The Author(s), under exclusive licence to Springer Nature B.V. 2024. Springer Nature or its licensor (e.g. a society or other partner) holds exclusive rights to this article under a publishing agreement with the author(s) or other rightsholder(s); author self-archiving of the accepted manuscript version of this article is solely governed by the terms of such publishing agreement and applicable law. ;2024. The Author(s), under exclusive licence to Springer Nature B.V. ;ISSN: 0301-4851 ;EISSN: 1573-4978 ;DOI: 10.1007/s11033-023-08978-1 ;PMID: 38252393

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17
Genetically confirmed coexistence of neurofibromatosis type 1 and Cherubism in a pediatric patient
Genetically confirmed coexistence of neurofibromatosis type 1 and Cherubism in a pediatric patient
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Article 		Add to My Research

Genetically confirmed coexistence of neurofibromatosis type 1 and Cherubism in a pediatric patient

Molecular biology reports, 2024-12, Vol.51 (1), p.216-216, Article 216 [Peer Reviewed Journal]

The Author(s) 2024 ;2024. The Author(s). ;The Author(s) 2024. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;ISSN: 0301-4851 ;EISSN: 1573-4978 ;DOI: 10.1007/s11033-024-09214-0 ;PMID: 38281202

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18
A rare clinical case of systemic AA amyloidosis with cardiac involvement complicating ankylosing spondylitis: a case report
A rare clinical case of systemic AA amyloidosis with cardiac involvement complicating ankylosing spondylitis: a case report
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Article 		Add to My Research

A rare clinical case of systemic AA amyloidosis with cardiac involvement complicating ankylosing spondylitis: a case report

The Egyptian heart journal, 2024-12, Vol.76 (1), p.40-40 [Peer Reviewed Journal]

The Author(s) 2024 ;2024. The Author(s). ;COPYRIGHT 2024 Springer ;The Author(s) 2024. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;ISSN: 1110-2608 ;EISSN: 2090-911X ;DOI: 10.1186/s43044-024-00471-9 ;PMID: 38546927

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19
Epicardial fat thickness among neonates of diabetic mothers attending the neonatal intensive care unit at Fayoum University Hospital: a case control study
Epicardial fat thickness among neonates of diabetic mothers attending the neonatal intensive care unit at Fayoum University Hospital: a case control study
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Article 		Add to My Research

Epicardial fat thickness among neonates of diabetic mothers attending the neonatal intensive care unit at Fayoum University Hospital: a case control study

The Gazette of the Egyptian Paediatric Association, 2024-12, Vol.72 (1), p.13-10 [Peer Reviewed Journal]

The Author(s) 2024 ;COPYRIGHT 2024 Springer ;The Author(s) 2024. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;ISSN: 1110-6638 ;EISSN: 2090-9942 ;DOI: 10.1186/s43054-024-00254-6

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20
Revealing the role of epigenetic and post-translational modulations of autophagy proteins in the regulation of autophagy and cancer: a therapeutic approach
Revealing the role of epigenetic and post-translational modulations of autophagy proteins in the regulation of autophagy and cancer: a therapeutic approach
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Article 		Add to My Research

Revealing the role of epigenetic and post-translational modulations of autophagy proteins in the regulation of autophagy and cancer: a therapeutic approach

Molecular biology reports, 2024-12, Vol.51 (1), p.3-3, Article 3 [Peer Reviewed Journal]

The Author(s), under exclusive licence to Springer Nature B.V. 2023. Springer Nature or its licensor (e.g. a society or other partner) holds exclusive rights to this article under a publishing agreement with the author(s) or other rightsholder(s); author self-archiving of the accepted manuscript version of this article is solely governed by the terms of such publishing agreement and applicable law. ;2023. The Author(s), under exclusive licence to Springer Nature B.V. ;ISSN: 0301-4851 ;EISSN: 1573-4978 ;DOI: 10.1007/s11033-023-08961-w ;PMID: 38063905

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