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1
A Review of Sudden Cardiac Death in Young Athletes and Strategies for Preparticipation Cardiovascular Screening
A Review of Sudden Cardiac Death in Young Athletes and Strategies for Preparticipation Cardiovascular Screening
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A Review of Sudden Cardiac Death in Young Athletes and Strategies for Preparticipation Cardiovascular Screening

Journal of athletic training, 2001-04, Vol.36 (2), p.197-204 [Peer Reviewed Journal]

Copyright National Athletic Trainers Association Apr-Jun 2001 ;by the National Athletic Trainers' Association, Inc 2001 ;ISSN: 1062-6050 ;EISSN: 1938-162X ;PMID: 12937463

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2
Development: kinase crucial for cardiac development
Development: kinase crucial for cardiac development
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Development: kinase crucial for cardiac development

Disease models & mechanisms, 2008-01, Vol.1 (4-5), p.184-185 [Peer Reviewed Journal]

2008. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the associated terms available at https://journals.biologists.com/dmm/pages/rights-permissions . ;2008 ;ISSN: 1754-8403 ;EISSN: 1754-8411

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3
A Case of Takotsubo Cardiomyopathy after Surgery for Common Bile Duct Stones
A Case of Takotsubo Cardiomyopathy after Surgery for Common Bile Duct Stones
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A Case of Takotsubo Cardiomyopathy after Surgery for Common Bile Duct Stones

Case reports in gastroenterology, 2008-03, Vol.2 (1), p.91-95 [Peer Reviewed Journal]

2008 S. Karger AG, Basel ;Copyright © 2008 by S. Karger AG, Basel 2008 ;ISSN: 1662-0631 ;EISSN: 1662-0631 ;DOI: 10.1159/000118799 ;PMID: 21490845

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4
Cardiac disease: Ca signaling mechanism underlies cardiomyopathy and arrhythmia
Cardiac disease: Ca signaling mechanism underlies cardiomyopathy and arrhythmia
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Cardiac disease: Ca signaling mechanism underlies cardiomyopathy and arrhythmia

Disease models & mechanisms, 2009-01, Vol.2 (1-2), p.2-2 [Peer Reviewed Journal]

2009. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the associated terms available at https://journals.biologists.com/dmm/pages/rights-permissions . ;2009 ;ISSN: 1754-8403 ;EISSN: 1754-8411

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5
Fishing for the genetic basis of cardiovascular disease
Fishing for the genetic basis of cardiovascular disease
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Fishing for the genetic basis of cardiovascular disease

Disease models & mechanisms, 2009-01, Vol.2 (1-2), p.18-22 [Peer Reviewed Journal]

2009. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the associated terms available at https://journals.biologists.com/dmm/pages/rights-permissions . ;2009 ;ISSN: 1754-8403 ;EISSN: 1754-8411 ;DOI: 10.1242/dmm.000687 ;PMID: 19132116

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6
Muscular dystrophy begins early in embryonic development deriving from stem cell loss and disrupted skeletal muscle formation
Muscular dystrophy begins early in embryonic development deriving from stem cell loss and disrupted skeletal muscle formation
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Muscular dystrophy begins early in embryonic development deriving from stem cell loss and disrupted skeletal muscle formation

Disease models & mechanisms, 2009-07, Vol.2 (7-8), p.374-388 [Peer Reviewed Journal]

2009. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the associated terms available at https://journals.biologists.com/dmm/pages/rights-permissions . ;2009 ;ISSN: 1754-8403 ;EISSN: 1754-8411 ;DOI: 10.1242/dmm.001008 ;PMID: 19535499

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7
Cardiac repair and regeneration: the Rubik's cube of cell therapy for heart disease
Cardiac repair and regeneration: the Rubik's cube of cell therapy for heart disease
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Cardiac repair and regeneration: the Rubik's cube of cell therapy for heart disease

Disease models & mechanisms, 2009-07, Vol.2 (7-8), p.344-358 [Peer Reviewed Journal]

2009. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the associated terms available at https://journals.biologists.com/dmm/pages/rights-permissions . ;2009 ;ISSN: 1754-8403 ;EISSN: 1754-8411 ;DOI: 10.1242/dmm.000240 ;PMID: 19553696

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8
Rodent models of diabetic cardiomyopathy
Rodent models of diabetic cardiomyopathy
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Rodent models of diabetic cardiomyopathy

ISSN: 1754-8411 ;ISSN: 1754-8403 ;EISSN: 1754-8411 ;DOI: 10.1242/dmm.001941

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9
Rodent models of diabetic cardiomyopathy
Rodent models of diabetic cardiomyopathy
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Rodent models of diabetic cardiomyopathy

Disease models & mechanisms, 2009-09, Vol.2 (9-10), p.454-466 [Peer Reviewed Journal]

2009. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the associated terms available at https://journals.biologists.com/dmm/pages/rights-permissions . ;ISSN: 1754-8403 ;EISSN: 1754-8411 ;DOI: 10.1242/dmm.001941 ;PMID: 19726805

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10
Taking translational research to heart: an interview with Sir Magdi Yacoub
Taking translational research to heart: an interview with Sir Magdi Yacoub
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Taking translational research to heart: an interview with Sir Magdi Yacoub

Disease models & mechanisms, 2009-09, Vol.2 (9-10), p.433-435 [Peer Reviewed Journal]

2009. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the associated terms available at https://journals.biologists.com/dmm/pages/rights-permissions . ;ISSN: 1754-8403 ;EISSN: 1754-8411 ;DOI: 10.1242/dmm.004176 ;PMID: 19726801

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11
Cardiomyopathy: myozap for the heart
Cardiomyopathy: myozap for the heart
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Cardiomyopathy: myozap for the heart

Disease models & mechanisms, 2010-01, Vol.3 (3-4), p.122 [Peer Reviewed Journal]

2010. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the associated terms available at https://journals.biologists.com/dmm/pages/rights-permissions . ;ISSN: 1754-8403 ;EISSN: 1754-8411

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12
Human cardiomyopathy mutations induce myocyte hyperplasia and activate hypertrophic pathways during cardiogenesis in zebrafish
Human cardiomyopathy mutations induce myocyte hyperplasia and activate hypertrophic pathways during cardiogenesis in zebrafish
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Human cardiomyopathy mutations induce myocyte hyperplasia and activate hypertrophic pathways during cardiogenesis in zebrafish

Disease models & mechanisms, 2011-05, Vol.4 (3), p.400-410 [Peer Reviewed Journal]

2011. This work is licensed under http://creativecommons.org/licenses/by-nc-sa/3.0 (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2011. Published by The Company of Biologists Ltd 2011 ;ISSN: 1754-8403 ;EISSN: 1754-8411 ;DOI: 10.1242/dmm.006148 ;PMID: 21245263

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13
LMNA cardiomyopathy: cell biology and genetics meet clinical medicine
LMNA cardiomyopathy: cell biology and genetics meet clinical medicine
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LMNA cardiomyopathy: cell biology and genetics meet clinical medicine

Disease models & mechanisms, 2011-09, Vol.4 (5), p.562-568 [Peer Reviewed Journal]

2011. This work is licensed under http://creativecommons.org/licenses/by-nc-sa/3.0 (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2011. Published by The Company of Biologists Ltd 2011 ;ISSN: 1754-8403 ;EISSN: 1754-8411 ;DOI: 10.1242/dmm.006346 ;PMID: 21810905

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14
Understanding the genetic and molecular pathogenesis of Friedreich's ataxia through animal and cellular models
Understanding the genetic and molecular pathogenesis of Friedreich's ataxia through animal and cellular models
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Understanding the genetic and molecular pathogenesis of Friedreich's ataxia through animal and cellular models

Disease models & mechanisms, 2012-03, Vol.5 (2), p.165-176 [Peer Reviewed Journal]

2012. This work is licensed under http://creativecommons.org/licenses/by-nc-sa/3.0 (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2012. Published by The Company of Biologists Ltd 2012 ;ISSN: 1754-8403 ;EISSN: 1754-8411 ;DOI: 10.1242/dmm.008706 ;PMID: 22382366

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15
A composite polymer nanoparticle overcomes multidrug resistance and ameliorates doxorubicin-associated cardiomyopathy
A composite polymer nanoparticle overcomes multidrug resistance and ameliorates doxorubicin-associated cardiomyopathy
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A composite polymer nanoparticle overcomes multidrug resistance and ameliorates doxorubicin-associated cardiomyopathy

Oncotarget, 2012-06, Vol.3 (6), p.640-650

Copyright: © 2012 Pramanik et al. 2012 ;EISSN: 1949-2553 ;DOI: 10.18632/oncotarget.543 ;PMID: 22791660

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16
Understanding cardiovascular disease: a journey through the genome (and what we found there)
Understanding cardiovascular disease: a journey through the genome (and what we found there)
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Understanding cardiovascular disease: a journey through the genome (and what we found there)

Disease models & mechanisms, 2012-07, Vol.5 (4), p.434-443 [Peer Reviewed Journal]

2012. This work is licensed under http://creativecommons.org/licenses/by-nc-sa/3.0 (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2012. Published by The Company of Biologists Ltd 2012 ;ISSN: 1754-8403 ;EISSN: 1754-8411 ;DOI: 10.1242/dmm.009787 ;PMID: 22730474

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17
Locally expressed IGF1 propeptide improves mouse heart function in induced dilated cardiomyopathy by blocking myocardial fibrosis and SRF-dependent CTGF induction
Locally expressed IGF1 propeptide improves mouse heart function in induced dilated cardiomyopathy by blocking myocardial fibrosis and SRF-dependent CTGF induction
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Locally expressed IGF1 propeptide improves mouse heart function in induced dilated cardiomyopathy by blocking myocardial fibrosis and SRF-dependent CTGF induction

Disease models & mechanisms, 2012-07, Vol.5 (4), p.481-491 [Peer Reviewed Journal]

2012. This work is licensed under http://creativecommons.org/licenses/by-nc-sa/3.0 (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;Distributed under a Creative Commons Attribution 4.0 International License ;2012. Published by The Company of Biologists Ltd 2012 ;ISSN: 1754-8403 ;EISSN: 1754-8411 ;DOI: 10.1242/dmm.009456 ;PMID: 22563064

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18
Cardiac-specific deletion of the microtubule-binding protein CENP-F causes dilated cardiomyopathy
Cardiac-specific deletion of the microtubule-binding protein CENP-F causes dilated cardiomyopathy
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Cardiac-specific deletion of the microtubule-binding protein CENP-F causes dilated cardiomyopathy

Disease models & mechanisms, 2012-07, Vol.5 (4), p.468-480 [Peer Reviewed Journal]

2012. This work is licensed under http://creativecommons.org/licenses/by-nc-sa/3.0 (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2012. Published by The Company of Biologists Ltd 2012 ;ISSN: 1754-8403 ;EISSN: 1754-8411 ;DOI: 10.1242/dmm.008680 ;PMID: 22563055

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19
Characterization of a canine model of glycogen storage disease type IIIa
Characterization of a canine model of glycogen storage disease type IIIa
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Characterization of a canine model of glycogen storage disease type IIIa

Disease models & mechanisms, 2012-11, Vol.5 (6), p.804-811 [Peer Reviewed Journal]

2012. This work is licensed under http://creativecommons.org/licenses/by-nc-sa/3.0 (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2012. Published by The Company of Biologists Ltd 2012 ;ISSN: 1754-8403 ;EISSN: 1754-8411 ;DOI: 10.1242/dmm.009712 ;PMID: 22736456

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20
Clinical data and characterization of the liver conditional mouse model exclude neoplasia as a non-neurological manifestation associated with Friedreich's ataxia
Clinical data and characterization of the liver conditional mouse model exclude neoplasia as a non-neurological manifestation associated with Friedreich's ataxia
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Clinical data and characterization of the liver conditional mouse model exclude neoplasia as a non-neurological manifestation associated with Friedreich's ataxia

Disease models & mechanisms, 2012-11, Vol.5 (6), p.860-869 [Peer Reviewed Journal]

2012. This work is licensed under http://creativecommons.org/licenses/by-nc-sa/3.0 (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2012. Published by The Company of Biologists Ltd 2012 ;ISSN: 1754-8403 ;EISSN: 1754-8411 ;DOI: 10.1242/dmm.009829 ;PMID: 22736457

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