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1
Recombinant Human Placental Growth Factor-2 Treatment in Porcine Ischemic Cardiomyopathy
Recombinant Human Placental Growth Factor-2 Treatment in Porcine Ischemic Cardiomyopathy
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Recombinant Human Placental Growth Factor-2 Treatment in Porcine Ischemic Cardiomyopathy

CIRCULATION, 2022, Vol.146

ISSN: 0009-7322 ;EISSN: 1524-4539

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2
Safety And Feasibility Of Tissue-engineered Biological Ventricular Assist Devices For Ischemic Cardiomyopathy
Safety And Feasibility Of Tissue-engineered Biological Ventricular Assist Devices For Ischemic Cardiomyopathy
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Safety And Feasibility Of Tissue-engineered Biological Ventricular Assist Devices For Ischemic Cardiomyopathy

CIRCULATION RESEARCH, 2023, Vol.133

ISSN: 0009-7330 ;EISSN: 1524-4571

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3
Prevalence and clinical consequences of multiple pathogenic variants in dilated cardiomyopathy
Prevalence and clinical consequences of multiple pathogenic variants in dilated cardiomyopathy
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Prevalence and clinical consequences of multiple pathogenic variants in dilated cardiomyopathy

EUROPEAN JOURNAL OF HUMAN GENETICS, 2023, Vol.31, p.418-418

ISSN: 1018-4813 ;EISSN: 1476-5438

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4
Clinical impact of re-evaluating genes and variants implicated in dilated cardiomyopathy
Clinical impact of re-evaluating genes and variants implicated in dilated cardiomyopathy
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Clinical impact of re-evaluating genes and variants implicated in dilated cardiomyopathy

EUROPEAN JOURNAL OF HUMAN GENETICS, 2022, Vol.30 (SUPPL 1), p.189-189

ISSN: 1018-4813 ;EISSN: 1476-5438

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5
Detecting transthyretin amyloid cardiomyopathy in a real-world heart failure population: automated data extraction from electronic health records using natural language processing
Detecting transthyretin amyloid cardiomyopathy in a real-world heart failure population: automated data extraction from electronic health records using natural language processing
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Detecting transthyretin amyloid cardiomyopathy in a real-world heart failure population: automated data extraction from electronic health records using natural language processing

EUROPEAN JOURNAL OF HEART FAILURE, 2023, Vol.25, p.122-122

ISSN: 1388-9842 ;EISSN: 1879-0844

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6
Impact of Hypertrophic Cardiomyopathy Mutations on Myosin Catalytic ATPase Activity
Impact of Hypertrophic Cardiomyopathy Mutations on Myosin Catalytic ATPase Activity
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Impact of Hypertrophic Cardiomyopathy Mutations on Myosin Catalytic ATPase Activity

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7
Biventricular imaging markers to predict outcome in non-compaction cardiomyopathy: a machine learning study
Biventricular imaging markers to predict outcome in non-compaction cardiomyopathy: a machine learning study
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Biventricular imaging markers to predict outcome in non-compaction cardiomyopathy: a machine learning study

EUROPEAN HEART JOURNAL, 2018, Vol.39, p.1376-1377

ISSN: 0195-668X ;EISSN: 1522-9645

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8
Effects of Hypertrophic Cardiomyopathy Mutations on Myosin Structural Dynamics
Effects of Hypertrophic Cardiomyopathy Mutations on Myosin Structural Dynamics
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Effects of Hypertrophic Cardiomyopathy Mutations on Myosin Structural Dynamics

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9
Hepatocyte-restricted SR-BI gene transfer corrects cardiac dysfunction in SR-BI deficient mice and improves pressure overload-induced cardiomyopathy
Hepatocyte-restricted SR-BI gene transfer corrects cardiac dysfunction in SR-BI deficient mice and improves pressure overload-induced cardiomyopathy
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Hepatocyte-restricted SR-BI gene transfer corrects cardiac dysfunction in SR-BI deficient mice and improves pressure overload-induced cardiomyopathy

European Heart Journal, 2017, Vol.38 (Supplement 1), p.259-260

ISSN: 0195-668X ;EISSN: 1522-9645

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10
Hepatocyte-specific SR-BI gene transfer corrects cardiac dysfunction in Scarb1-deficient mice and improves pressure overload-induced cardiomyopathy
Hepatocyte-specific SR-BI gene transfer corrects cardiac dysfunction in Scarb1-deficient mice and improves pressure overload-induced cardiomyopathy
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Hepatocyte-specific SR-BI gene transfer corrects cardiac dysfunction in Scarb1-deficient mice and improves pressure overload-induced cardiomyopathy

ARTERIOSCLEROSIS THROMBOSIS AND VASCULAR BIOLOGY, 2017, Vol.In press, p.259-260

ISSN: 1079-5642 ;EISSN: 1524-4636

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11
Proliferating myofibroblasts contribute to extracellular matrix remodeling in a porcine model of ischemic cardiomyopathy
Proliferating myofibroblasts contribute to extracellular matrix remodeling in a porcine model of ischemic cardiomyopathy
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Proliferating myofibroblasts contribute to extracellular matrix remodeling in a porcine model of ischemic cardiomyopathy

European Heart Journal, 2015, Vol.36, p.952-953

ISSN: 0195-668X ;EISSN: 1522-9645

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12
Neovascularisation Potential of Blood Outgrowth Endothelial Cells From Patients With Severe Ischemic Cardiomyopathy
Neovascularisation Potential of Blood Outgrowth Endothelial Cells From Patients With Severe Ischemic Cardiomyopathy
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Neovascularisation Potential of Blood Outgrowth Endothelial Cells From Patients With Severe Ischemic Cardiomyopathy

Circulation, 2013, Vol.128 (22)

ISSN: 0009-7322 ;EISSN: 1524-4539

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13
Targeted capture sequencing identifies mutations in a substantial proportion of prior genotype negative - phenotype positive patients with inherited primary arrhythmia syndrome or cardiomyopathy
Targeted capture sequencing identifies mutations in a substantial proportion of prior genotype negative - phenotype positive patients with inherited primary arrhythmia syndrome or cardiomyopathy
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Targeted capture sequencing identifies mutations in a substantial proportion of prior genotype negative - phenotype positive patients with inherited primary arrhythmia syndrome or cardiomyopathy

European Heart Journal. Supplements, 2015, Vol.36 (1), p.531-531

ISSN: 0195-668X ;EISSN: 1522-9645

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14
Prognostic value of cardiac fibrosis in patients with non-ischemic cardiomyopathy without congestive heart failure: a multicentre magnetic resonance study
Prognostic value of cardiac fibrosis in patients with non-ischemic cardiomyopathy without congestive heart failure: a multicentre magnetic resonance study
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Prognostic value of cardiac fibrosis in patients with non-ischemic cardiomyopathy without congestive heart failure: a multicentre magnetic resonance study

European Heart Journal, 2014, Vol.35, p.870-871

ISSN: 0195-668X ;EISSN: 1522-9645

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15
CMR of LV non-compaction cardiomyopathy: association of clinical presentation and prognosis with cardiac phenotype
CMR of LV non-compaction cardiomyopathy: association of clinical presentation and prognosis with cardiac phenotype
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CMR of LV non-compaction cardiomyopathy: association of clinical presentation and prognosis with cardiac phenotype

DOI: 10.1186/1532-429X-13-S1-P291

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16
Profound hypocalcaemia presenting with a dilated cardiomyopathy and possible mitochondrial dysfunction in a 3 week old infant
Profound hypocalcaemia presenting with a dilated cardiomyopathy and possible mitochondrial dysfunction in a 3 week old infant
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Profound hypocalcaemia presenting with a dilated cardiomyopathy and possible mitochondrial dysfunction in a 3 week old infant

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17
Hypertensive cardiomyopathy in the practice of primary therapist and cardiologist (lecture)
Hypertensive cardiomyopathy in the practice of primary therapist and cardiologist (lecture)
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Hypertensive cardiomyopathy in the practice of primary therapist and cardiologist (lecture)

Terapevticheskii arkhiv, 2006, Vol.78 (1), p.28-35

ISSN: 0040-3660

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18
Dilated cardiomyopathy in the practice of a therapist and cardiologist (lecture)
Dilated cardiomyopathy in the practice of a therapist and cardiologist (lecture)
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Dilated cardiomyopathy in the practice of a therapist and cardiologist (lecture)

Terapevticheskii arkhiv, 2004, Vol.76 (1), p.12-17

ISSN: 0040-3660

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19
Genetic analysis of multifactorial cardiac diseases: idiopathic dilated cardiomyopathy and myocardial infarction
Genetic analysis of multifactorial cardiac diseases: idiopathic dilated cardiomyopathy and myocardial infarction
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Genetic analysis of multifactorial cardiac diseases: idiopathic dilated cardiomyopathy and myocardial infarction

Nihon Ika Daigaku zasshi, 1999, Vol.66 (5), p.332-335

ISSN: 0048-0444

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20
Transcriptional differences in fibroblasts underlie distinct fibrotic patterns within dilated and Ischemic cardiomyopathies
Transcriptional differences in fibroblasts underlie distinct fibrotic patterns within dilated and Ischemic cardiomyopathies
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Transcriptional differences in fibroblasts underlie distinct fibrotic patterns within dilated and Ischemic cardiomyopathies

JOURNAL OF MOLECULAR AND CELLULAR CARDIOLOGY, 2022, Vol.173, p.S149-S149

ISSN: 0022-2828 ;EISSN: 1095-8584

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