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21	Material Type: Text Resource	Adapting evidence-based clinical practice guidelines for people with attention deficit hyperactivity disorder in Saudi Arabia: process and outputs of a national initiative Child and Adolescent Psychiatry and Mental Health, 2020 2020. This work is published under https://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;DOI: 10.21203/rs.3.rs-42254/v2 Digital Resources/Online E-Resources Services Details Recommendations Reviews Times Cited Citations Cited by
22	Material Type: Text Resource	Addition of routine blood biomarkers to TIMI risk score improves predictive performance of 1-year mortality in patients with ST-segment elevation myocardial infarction BMC Cardiovascular Disorders, 2020 2020. This work is published under https://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;DOI: 10.21203/rs.3.rs-41698/v3 Digital Resources/Online E-Resources Services Details Recommendations Reviews Times Cited Citations Cited by
23	Material Type: Text Resource	Additional file 10 of Multi-omics integration identifies key upstream regulators of pathomechanisms in hypertrophic cardiomyopathy due to truncating MYBPC3 mutations DOI: 10.6084/m9.figshare.14302779.v1 Digital Resources/Online E-Resources View Online Details Recommendations Reviews Times Cited External Links Citations Cited by
24	Material Type: Text Resource	Additional file 10 of Multi-omics integration identifies key upstream regulators of pathomechanisms in hypertrophic cardiomyopathy due to truncating MYBPC3 mutations DOI: 10.6084/m9.figshare.14302779.v1 Digital Resources/Online E-Resources View Online Details Recommendations Reviews Times Cited External Links Citations Cited by
25	Material Type: Text Resource	Additional file 11 of Multi-omics integration identifies key upstream regulators of pathomechanisms in hypertrophic cardiomyopathy due to truncating MYBPC3 mutations DOI: 10.6084/m9.figshare.14302782.v1 Digital Resources/Online E-Resources View Online Details Recommendations Reviews Times Cited External Links Citations Cited by
26	Material Type: Text Resource	Additional file 11 of Multi-omics integration identifies key upstream regulators of pathomechanisms in hypertrophic cardiomyopathy due to truncating MYBPC3 mutations DOI: 10.6084/m9.figshare.14302782.v1 Digital Resources/Online E-Resources View Online Details Recommendations Reviews Times Cited External Links Citations Cited by
27	Material Type: Text Resource	Additional file 12 of Multi-omics integration identifies key upstream regulators of pathomechanisms in hypertrophic cardiomyopathy due to truncating MYBPC3 mutations DOI: 10.6084/m9.figshare.14302785.v1 Digital Resources/Online E-Resources View Online Details Recommendations Reviews Times Cited External Links Citations Cited by
28	Material Type: Text Resource	Additional file 12 of Multi-omics integration identifies key upstream regulators of pathomechanisms in hypertrophic cardiomyopathy due to truncating MYBPC3 mutations DOI: 10.6084/m9.figshare.14302785.v1 Digital Resources/Online E-Resources View Online Details Recommendations Reviews Times Cited External Links Citations Cited by
29	Material Type: Text Resource	Additional file 13 of Multi-omics integration identifies key upstream regulators of pathomechanisms in hypertrophic cardiomyopathy due to truncating MYBPC3 mutations DOI: 10.6084/m9.figshare.14302788.v1 Digital Resources/Online E-Resources View Online Details Recommendations Reviews Times Cited External Links Citations Cited by
30	Material Type: Text Resource	Additional file 13 of Multi-omics integration identifies key upstream regulators of pathomechanisms in hypertrophic cardiomyopathy due to truncating MYBPC3 mutations DOI: 10.6084/m9.figshare.14302788.v1 Digital Resources/Online E-Resources View Online Details Recommendations Reviews Times Cited External Links Citations Cited by
31	Material Type: Text Resource	Additional file 14 of Multi-omics integration identifies key upstream regulators of pathomechanisms in hypertrophic cardiomyopathy due to truncating MYBPC3 mutations DOI: 10.6084/m9.figshare.14302791.v1 Digital Resources/Online E-Resources View Online Details Recommendations Reviews Times Cited External Links Citations Cited by
32	Material Type: Text Resource	Additional file 14 of Multi-omics integration identifies key upstream regulators of pathomechanisms in hypertrophic cardiomyopathy due to truncating MYBPC3 mutations DOI: 10.6084/m9.figshare.14302791.v1 Digital Resources/Online E-Resources View Online Details Recommendations Reviews Times Cited External Links Citations Cited by
33	Material Type: Text Resource	Additional file 15 of Multi-omics integration identifies key upstream regulators of pathomechanisms in hypertrophic cardiomyopathy due to truncating MYBPC3 mutations DOI: 10.6084/m9.figshare.14302794.v1 Digital Resources/Online E-Resources View Online Details Recommendations Reviews Times Cited External Links Citations Cited by
34	Material Type: Text Resource	Additional file 15 of Multi-omics integration identifies key upstream regulators of pathomechanisms in hypertrophic cardiomyopathy due to truncating MYBPC3 mutations DOI: 10.6084/m9.figshare.14302794.v1 Digital Resources/Online E-Resources View Online Details Recommendations Reviews Times Cited External Links Citations Cited by
35	Material Type: Text Resource	Additional file 16 of Multi-omics integration identifies key upstream regulators of pathomechanisms in hypertrophic cardiomyopathy due to truncating MYBPC3 mutations DOI: 10.6084/m9.figshare.14302797.v1 Digital Resources/Online E-Resources View Online Details Recommendations Reviews Times Cited External Links Citations Cited by
36	Material Type: Text Resource	Additional file 16 of Multi-omics integration identifies key upstream regulators of pathomechanisms in hypertrophic cardiomyopathy due to truncating MYBPC3 mutations DOI: 10.6084/m9.figshare.14302797.v1 Digital Resources/Online E-Resources View Online Details Recommendations Reviews Times Cited External Links Citations Cited by
37	Material Type: Text Resource	Additional file 17 of Multi-omics integration identifies key upstream regulators of pathomechanisms in hypertrophic cardiomyopathy due to truncating MYBPC3 mutations DOI: 10.6084/m9.figshare.14302800.v1 Digital Resources/Online E-Resources View Online Details Recommendations Reviews Times Cited External Links Citations Cited by
38	Material Type: Text Resource	Additional file 17 of Multi-omics integration identifies key upstream regulators of pathomechanisms in hypertrophic cardiomyopathy due to truncating MYBPC3 mutations DOI: 10.6084/m9.figshare.14302800.v1 Digital Resources/Online E-Resources View Online Details Recommendations Reviews Times Cited External Links Citations Cited by
39	Material Type: Text Resource	Additional file 18 of Multi-omics integration identifies key upstream regulators of pathomechanisms in hypertrophic cardiomyopathy due to truncating MYBPC3 mutations DOI: 10.6084/m9.figshare.14302803.v1 Digital Resources/Online E-Resources View Online Details Recommendations Reviews Times Cited External Links Citations Cited by
40	Material Type: Text Resource	Additional file 18 of Multi-omics integration identifies key upstream regulators of pathomechanisms in hypertrophic cardiomyopathy due to truncating MYBPC3 mutations DOI: 10.6084/m9.figshare.14302803.v1 Digital Resources/Online E-Resources View Online Details Recommendations Reviews Times Cited External Links Citations Cited by

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INSPIRE LIBRARY - TON DUC THANG UNIVERSITY	(84-028) 37 755 057	Feedback
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Results 21 - 40 of 1,522 for All Library Resources

2020. This work is published under https://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;DOI: 10.21203/rs.3.rs-42254/v2

2020. This work is published under https://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;DOI: 10.21203/rs.3.rs-41698/v3

DOI: 10.6084/m9.figshare.14302779.v1

DOI: 10.6084/m9.figshare.14302779.v1

DOI: 10.6084/m9.figshare.14302782.v1

DOI: 10.6084/m9.figshare.14302782.v1

DOI: 10.6084/m9.figshare.14302785.v1

DOI: 10.6084/m9.figshare.14302785.v1

DOI: 10.6084/m9.figshare.14302788.v1

DOI: 10.6084/m9.figshare.14302788.v1

DOI: 10.6084/m9.figshare.14302791.v1

DOI: 10.6084/m9.figshare.14302791.v1

DOI: 10.6084/m9.figshare.14302794.v1

DOI: 10.6084/m9.figshare.14302794.v1

DOI: 10.6084/m9.figshare.14302797.v1

DOI: 10.6084/m9.figshare.14302797.v1

DOI: 10.6084/m9.figshare.14302800.v1

DOI: 10.6084/m9.figshare.14302800.v1

DOI: 10.6084/m9.figshare.14302803.v1

DOI: 10.6084/m9.figshare.14302803.v1

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