Language:

Results 1 - 20 of 63 for All Library Resources

Sorted by: Sorted by: RelevanceOr hit Enter to replace sort method

Results 1 2 3 4

Refined by: resource type: Text Resources

Result Number	Material Type	Record Details and Options
1	Material Type: Text Resource	Investigating the Molecular Mechanisms Underlying Lipo-toxicity in Diabetic Cardiomyopathy: KLB and FGFR1 Interaction and Signaling Digital Resources/Online E-Resources View Online Details Recommendations Reviews Times Cited External Links Citations Cited by
2	Material Type: Text Resource	Human iPSC-Based Model Reveals NOX4 as Therapeutic Target in Duchenne Cardiomyopathy bioRxiv, 2021 Digital Resources/Online E-Resources View Online Details Recommendations Reviews Times Cited External Links Citations Cited by
3	Material Type: Text Resource	Myocyte [Na+]i Dysregulation in Heart Failure and Diabetic Cardiomyopathy DOI: 10.3389/fphys.2018.01303 Digital Resources/Online E-Resources View Online Details Recommendations Reviews Times Cited External Links Citations Cited by
4	Material Type: Text Resource	Increased C-reactive protein is associated with the severity of thoracic radiotherapy-induced cardiomyopathy The Author(s). 2020. Open Access. This article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated. Digital Resources/Online E-Resources View Online Details Recommendations Reviews Times Cited External Links Citations Cited by
5	Material Type: Text Resource	Additional file 13 of Multi-omics integration identifies key upstream regulators of pathomechanisms in hypertrophic cardiomyopathy due to truncating MYBPC3 mutations DOI: 10.6084/m9.figshare.14302788.v1 Digital Resources/Online E-Resources View Online Details Recommendations Reviews Times Cited External Links Citations Cited by
6	Material Type: Text Resource	Additional file 10 of Multi-omics integration identifies key upstream regulators of pathomechanisms in hypertrophic cardiomyopathy due to truncating MYBPC3 mutations DOI: 10.6084/m9.figshare.14302779.v1 Digital Resources/Online E-Resources View Online Details Recommendations Reviews Times Cited External Links Citations Cited by
7	Material Type: Text Resource	Additional file 9 of Multi-omics integration identifies key upstream regulators of pathomechanisms in hypertrophic cardiomyopathy due to truncating MYBPC3 mutations DOI: 10.6084/m9.figshare.14302830.v1 Digital Resources/Online E-Resources View Online Details Recommendations Reviews Times Cited External Links Citations Cited by
8	Material Type: Text Resource	Additional file 17 of Multi-omics integration identifies key upstream regulators of pathomechanisms in hypertrophic cardiomyopathy due to truncating MYBPC3 mutations DOI: 10.6084/m9.figshare.14302800.v1 Digital Resources/Online E-Resources View Online Details Recommendations Reviews Times Cited External Links Citations Cited by
9	Material Type: Text Resource	Additional file 9 of Multi-omics integration identifies key upstream regulators of pathomechanisms in hypertrophic cardiomyopathy due to truncating MYBPC3 mutations DOI: 10.6084/m9.figshare.14302830.v1 Digital Resources/Online E-Resources View Online Details Recommendations Reviews Times Cited External Links Citations Cited by
10	Material Type: Text Resource	Additional file 14 of Multi-omics integration identifies key upstream regulators of pathomechanisms in hypertrophic cardiomyopathy due to truncating MYBPC3 mutations DOI: 10.6084/m9.figshare.14302791.v1 Digital Resources/Online E-Resources View Online Details Recommendations Reviews Times Cited External Links Citations Cited by
11	Material Type: Text Resource	Additional file 16 of Multi-omics integration identifies key upstream regulators of pathomechanisms in hypertrophic cardiomyopathy due to truncating MYBPC3 mutations DOI: 10.6084/m9.figshare.14302797.v1 Digital Resources/Online E-Resources View Online Details Recommendations Reviews Times Cited External Links Citations Cited by
12	Material Type: Text Resource	Additional file 11 of Multi-omics integration identifies key upstream regulators of pathomechanisms in hypertrophic cardiomyopathy due to truncating MYBPC3 mutations DOI: 10.6084/m9.figshare.14302782.v1 Digital Resources/Online E-Resources View Online Details Recommendations Reviews Times Cited External Links Citations Cited by
13	Material Type: Text Resource	Additional file 18 of Multi-omics integration identifies key upstream regulators of pathomechanisms in hypertrophic cardiomyopathy due to truncating MYBPC3 mutations DOI: 10.6084/m9.figshare.14302803.v1 Digital Resources/Online E-Resources View Online Details Recommendations Reviews Times Cited External Links Citations Cited by
14	Material Type: Text Resource	Additional file 17 of Multi-omics integration identifies key upstream regulators of pathomechanisms in hypertrophic cardiomyopathy due to truncating MYBPC3 mutations DOI: 10.6084/m9.figshare.14302800.v1 Digital Resources/Online E-Resources View Online Details Recommendations Reviews Times Cited External Links Citations Cited by
15	Material Type: Text Resource	Additional file 16 of Multi-omics integration identifies key upstream regulators of pathomechanisms in hypertrophic cardiomyopathy due to truncating MYBPC3 mutations DOI: 10.6084/m9.figshare.14302797.v1 Digital Resources/Online E-Resources View Online Details Recommendations Reviews Times Cited External Links Citations Cited by
16	Material Type: Text Resource	Additional file 18 of Multi-omics integration identifies key upstream regulators of pathomechanisms in hypertrophic cardiomyopathy due to truncating MYBPC3 mutations DOI: 10.6084/m9.figshare.14302803.v1 Digital Resources/Online E-Resources View Online Details Recommendations Reviews Times Cited External Links Citations Cited by
17	Material Type: Text Resource	Additional file 15 of Multi-omics integration identifies key upstream regulators of pathomechanisms in hypertrophic cardiomyopathy due to truncating MYBPC3 mutations DOI: 10.6084/m9.figshare.14302794.v1 Digital Resources/Online E-Resources View Online Details Recommendations Reviews Times Cited External Links Citations Cited by
18	Material Type: Text Resource	Additional file 15 of Multi-omics integration identifies key upstream regulators of pathomechanisms in hypertrophic cardiomyopathy due to truncating MYBPC3 mutations DOI: 10.6084/m9.figshare.14302794.v1 Digital Resources/Online E-Resources View Online Details Recommendations Reviews Times Cited External Links Citations Cited by
19	Material Type: Text Resource	Additional file 13 of Multi-omics integration identifies key upstream regulators of pathomechanisms in hypertrophic cardiomyopathy due to truncating MYBPC3 mutations DOI: 10.6084/m9.figshare.14302788.v1 Digital Resources/Online E-Resources View Online Details Recommendations Reviews Times Cited External Links Citations Cited by
20	Material Type: Text Resource	Additional file 14 of Multi-omics integration identifies key upstream regulators of pathomechanisms in hypertrophic cardiomyopathy due to truncating MYBPC3 mutations DOI: 10.6084/m9.figshare.14302791.v1 Digital Resources/Online E-Resources View Online Details Recommendations Reviews Times Cited External Links Citations Cited by

Results 1 - 20 of 63 for All Library Resources

Sorted by: Sorted by: RelevanceOr hit Enter to replace sort method

Results 1 2 3 4

Personalize your results

Edit

Add page to My Research

Refine Search Results

Expand My Results

Expand My Results

on this subject:


INSPIRE LIBRARY - TON DUC THANG UNIVERSITY	(84-028) 37 755 057	Feedback
19 Nguyen Huu Tho St. Dist.7, HCM	thuvien@tdtu.edu.vn	Feedback

Results 1 - 20 of 63 for All Library Resources

DOI: 10.3389/fphys.2018.01303

DOI: 10.6084/m9.figshare.14302788.v1

DOI: 10.6084/m9.figshare.14302779.v1

DOI: 10.6084/m9.figshare.14302830.v1

DOI: 10.6084/m9.figshare.14302800.v1

DOI: 10.6084/m9.figshare.14302830.v1

DOI: 10.6084/m9.figshare.14302791.v1

DOI: 10.6084/m9.figshare.14302797.v1

DOI: 10.6084/m9.figshare.14302782.v1

DOI: 10.6084/m9.figshare.14302803.v1

DOI: 10.6084/m9.figshare.14302800.v1

DOI: 10.6084/m9.figshare.14302797.v1

DOI: 10.6084/m9.figshare.14302803.v1

DOI: 10.6084/m9.figshare.14302794.v1

DOI: 10.6084/m9.figshare.14302794.v1

DOI: 10.6084/m9.figshare.14302788.v1

DOI: 10.6084/m9.figshare.14302791.v1

Results 1 - 20 of 63 for All Library Resources

Personalize your results

Refine Search Results

Expand My Results

Refine My Results

Creation Date

New Records

Subject

Databases

on this subject:

Searching Remote Databases, Please Wait