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1
Metabolomic insights in advanced cardiomyopathy of chronic chagasic and idiopathic patients that underwent heart transplant
Metabolomic insights in advanced cardiomyopathy of chronic chagasic and idiopathic patients that underwent heart transplant
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Metabolomic insights in advanced cardiomyopathy of chronic chagasic and idiopathic patients that underwent heart transplant

Scientific reports, 2024-04, Vol.14 (1), p.9810-9810 [Peer Reviewed Journal]

2024. The Author(s). ;The Author(s) 2024. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2024 ;EISSN: 2045-2322 ;DOI: 10.1038/s41598-024-53875-7 ;PMID: 38684702

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2
Clinical implications of septic cardiomyopathy: A narrative review
Clinical implications of septic cardiomyopathy: A narrative review
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Clinical implications of septic cardiomyopathy: A narrative review

Medicine (Baltimore), 2024-04, Vol.103 (17), p.e37940-e37940 [Peer Reviewed Journal]

Copyright © 2024 the Author(s). Published by Wolters Kluwer Health, Inc. ;EISSN: 1536-5964 ;DOI: 10.1097/MD.0000000000037940 ;PMID: 38669408

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3
Catheter ablation versus antiarrhythmic drug therapy for sustained ventricular tachycardia in patients with hypertrophic cardiomyopathy
Catheter ablation versus antiarrhythmic drug therapy for sustained ventricular tachycardia in patients with hypertrophic cardiomyopathy
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Catheter ablation versus antiarrhythmic drug therapy for sustained ventricular tachycardia in patients with hypertrophic cardiomyopathy

BMC cardiovascular disorders, 2024-05, Vol.24 (1), p.255-255 [Peer Reviewed Journal]

2024. The Author(s). ;COPYRIGHT 2024 BioMed Central Ltd. ;ISSN: 1471-2261 ;EISSN: 1471-2261 ;DOI: 10.1186/s12872-024-03924-w ;PMID: 38755595

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4
MAP4K4 exacerbates cardiac microvascular injury in diabetes by facilitating S-nitrosylation modification of Drp1
MAP4K4 exacerbates cardiac microvascular injury in diabetes by facilitating S-nitrosylation modification of Drp1
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MAP4K4 exacerbates cardiac microvascular injury in diabetes by facilitating S-nitrosylation modification of Drp1

Cardiovascular diabetology, 2024-05, Vol.23 (1), p.164-164 [Peer Reviewed Journal]

2024. The Author(s). ;2024. This work is licensed under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;EISSN: 1475-2840 ;DOI: 10.1186/s12933-024-02254-7 ;PMID: 38724987

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5
Magnetic Resonance Imaging Characterization and Clinical Outcomes of Dilated and Arrhythmogenic Left Ventricular Cardiomyopathies
Magnetic Resonance Imaging Characterization and Clinical Outcomes of Dilated and Arrhythmogenic Left Ventricular Cardiomyopathies
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Magnetic Resonance Imaging Characterization and Clinical Outcomes of Dilated and Arrhythmogenic Left Ventricular Cardiomyopathies

Journal of the American College of Cardiology, 2024-05, Vol.83 (19), p.1841-1851 [Peer Reviewed Journal]

2024 American College of Cardiology Foundation ;Copyright © 2024 American College of Cardiology Foundation. Published by Elsevier Inc. All rights reserved. ;ISSN: 0735-1097 ;EISSN: 1558-3597 ;DOI: 10.1016/j.jacc.2024.02.041 ;PMID: 38719365

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6
Predictors of Arrhythmic Outcomes in Patients With Dilated and Nondilated Left Ventricular Cardiomyopathies
Predictors of Arrhythmic Outcomes in Patients With Dilated and Nondilated Left Ventricular Cardiomyopathies
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Predictors of Arrhythmic Outcomes in Patients With Dilated and Nondilated Left Ventricular Cardiomyopathies

Journal of the American College of Cardiology, 2024-05, Vol.83 (19), p.1852-1854 [Peer Reviewed Journal]

2024 American College of Cardiology Foundation ;ISSN: 0735-1097 ;EISSN: 1558-3597 ;DOI: 10.1016/j.jacc.2024.03.404 ;PMID: 38719366

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7
Modeling cardiomyocyte signaling and metabolism predicts genotype-to-phenotype mechanisms in hypertrophic cardiomyopathy
Modeling cardiomyocyte signaling and metabolism predicts genotype-to-phenotype mechanisms in hypertrophic cardiomyopathy
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Modeling cardiomyocyte signaling and metabolism predicts genotype-to-phenotype mechanisms in hypertrophic cardiomyopathy

Computers in biology and medicine, 2024-06, Vol.175, p.108499-108499, Article 108499 [Peer Reviewed Journal]

2024 Elsevier Ltd ;Copyright © 2024 Elsevier Ltd. All rights reserved. ;2024. Elsevier Ltd ;ISSN: 0010-4825 ;EISSN: 1879-0534 ;DOI: 10.1016/j.compbiomed.2024.108499 ;PMID: 38677172

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8
Recent advancements and applications of deep learning in heart failure: Α systematic review
Recent advancements and applications of deep learning in heart failure: Α systematic review
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Recent advancements and applications of deep learning in heart failure: Α systematic review

Computers in biology and medicine, 2024-05, Vol.176, p.108557-108557, Article 108557 [Peer Reviewed Journal]

2024 Elsevier Ltd ;Copyright © 2024 Elsevier Ltd. All rights reserved. ;ISSN: 0010-4825 ;EISSN: 1879-0534 ;DOI: 10.1016/j.compbiomed.2024.108557 ;PMID: 38728995

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9
A therapeutic leap: how myosin inhibitors moved from cardiac interventions to skeletal muscle myopathy solutions
A therapeutic leap: how myosin inhibitors moved from cardiac interventions to skeletal muscle myopathy solutions
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A therapeutic leap: how myosin inhibitors moved from cardiac interventions to skeletal muscle myopathy solutions

The Journal of clinical investigation, 2024-05, Vol.134 (9), p.1-3 [Peer Reviewed Journal]

Copyright American Society for Clinical Investigation May 2024 ;ISSN: 0021-9738 ;EISSN: 1558-8238 ;DOI: 10.1172/JCI172599 ;PMID: 38690729

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10
Single-Cell RNA Sequencing Reveals Cardiac Fibroblast-Specific Transcriptomic Changes in Dilated Cardiomyopathy
Single-Cell RNA Sequencing Reveals Cardiac Fibroblast-Specific Transcriptomic Changes in Dilated Cardiomyopathy
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Single-Cell RNA Sequencing Reveals Cardiac Fibroblast-Specific Transcriptomic Changes in Dilated Cardiomyopathy

Cells (Basel, Switzerland), 2024-05, Vol.13 (9), p.752 [Peer Reviewed Journal]

COPYRIGHT 2024 MDPI AG ;2024 by the authors. Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (https://creativecommons.org/licenses/by/4.0/). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;ISSN: 2073-4409 ;EISSN: 2073-4409 ;DOI: 10.3390/cells13090752 ;PMID: 38727290

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11
Incidence and risk factors for development of left ventricular hypertrophy in Fabry disease
Incidence and risk factors for development of left ventricular hypertrophy in Fabry disease
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Incidence and risk factors for development of left ventricular hypertrophy in Fabry disease

Heart (British Cardiac Society), 2024-04, p.heartjnl-2023-323783 [Peer Reviewed Journal]

Author(s) (or their employer(s)) 2024. No commercial re-use. See rights and permissions. Published by BMJ. ;2024 Author(s) (or their employer(s)) 2024. No commercial re-use. See rights and permissions. Published by BMJ. ;ISSN: 1355-6037 ;EISSN: 1468-201X ;DOI: 10.1136/heartjnl-2023-323783 ;PMID: 38688703

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12
Incomplete-penetrant hypertrophic cardiomyopathy MYH7 G256E mutation causes hypercontractility and elevated mitochondrial respiration
Incomplete-penetrant hypertrophic cardiomyopathy MYH7 G256E mutation causes hypercontractility and elevated mitochondrial respiration
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Incomplete-penetrant hypertrophic cardiomyopathy MYH7 G256E mutation causes hypercontractility and elevated mitochondrial respiration

Proceedings of the National Academy of Sciences - PNAS, 2024-05, Vol.121 (19), p.e2318413121-e2318413121 [Peer Reviewed Journal]

Copyright National Academy of Sciences May 7, 2024 ;ISSN: 0027-8424 ;EISSN: 1091-6490 ;DOI: 10.1073/pnas.2318413121 ;PMID: 38683993

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13
Single-cell insights: pioneering an integrated atlas of chromatin accessibility and transcriptomic landscapes in diabetic cardiomyopathy
Single-cell insights: pioneering an integrated atlas of chromatin accessibility and transcriptomic landscapes in diabetic cardiomyopathy
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Single-cell insights: pioneering an integrated atlas of chromatin accessibility and transcriptomic landscapes in diabetic cardiomyopathy

Cardiovascular diabetology, 2024-04, Vol.23 (1), p.139-139 [Peer Reviewed Journal]

2024. The Author(s). ;2024. This work is licensed under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;EISSN: 1475-2840 ;DOI: 10.1186/s12933-024-02233-y ;PMID: 38664790

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14
Myosin-Catalyzed ATP Hydrolysis in the Presence of Disease-Causing Mutations: Mavacamten as a Way to Repair Mechanism
Myosin-Catalyzed ATP Hydrolysis in the Presence of Disease-Causing Mutations: Mavacamten as a Way to Repair Mechanism
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Myosin-Catalyzed ATP Hydrolysis in the Presence of Disease-Causing Mutations: Mavacamten as a Way to Repair Mechanism

The journal of physical chemistry. B, 2024-05, Vol.128 (19), p.4716 [Peer Reviewed Journal]

EISSN: 1520-5207 ;DOI: 10.1021/acs.jpcb.4c01601 ;PMID: 38708944

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15
Prevalence and Disease Expression of Pathogenic and Likely Pathogenic Variants Associated with Inherited Cardiomyopathies in the General Population
Prevalence and Disease Expression of Pathogenic and Likely Pathogenic Variants Associated with Inherited Cardiomyopathies in the General Population
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Prevalence and Disease Expression of Pathogenic and Likely Pathogenic Variants Associated with Inherited Cardiomyopathies in the General Population

info:eu-repo/semantics/OpenAccess ;ISSN: 2574-8300 ;EISSN: 2574-8300

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16
Current Perspectives of Mitochondria in Sepsis-Induced Cardiomyopathy
Current Perspectives of Mitochondria in Sepsis-Induced Cardiomyopathy
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Current Perspectives of Mitochondria in Sepsis-Induced Cardiomyopathy

International journal of molecular sciences, 2024-05, Vol.25 (9), p.4710 [Peer Reviewed Journal]

COPYRIGHT 2024 MDPI AG ;2024 by the authors. Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (https://creativecommons.org/licenses/by/4.0/). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;ISSN: 1422-0067 ;ISSN: 1661-6596 ;EISSN: 1422-0067 ;DOI: 10.3390/ijms25094710 ;PMID: 38731929

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17
Potential clinical value of fibrinogen-like protein 1 as a serum biomarker for the identification of diabetic cardiomyopathy
Potential clinical value of fibrinogen-like protein 1 as a serum biomarker for the identification of diabetic cardiomyopathy
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Potential clinical value of fibrinogen-like protein 1 as a serum biomarker for the identification of diabetic cardiomyopathy

Scientific reports, 2024-05, Vol.14 (1), p.10311-10311 [Peer Reviewed Journal]

2024. The Author(s). ;The Author(s) 2024. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2024 ;EISSN: 2045-2322 ;DOI: 10.1038/s41598-024-57580-3 ;PMID: 38705920

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18
Fabry disease: development and progression of left ventricular hypertrophy despite long-term enzyme replacement therapy
Fabry disease: development and progression of left ventricular hypertrophy despite long-term enzyme replacement therapy
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Fabry disease: development and progression of left ventricular hypertrophy despite long-term enzyme replacement therapy

Heart (British Cardiac Society), 2024-05, p.heartjnl-2024-323975 [Peer Reviewed Journal]

Author(s) (or their employer(s)) 2024. No commercial re-use. See rights and permissions. Published by BMJ. ;2024 Author(s) (or their employer(s)) 2024. No commercial re-use. See rights and permissions. Published by BMJ. ;ISSN: 1355-6037 ;EISSN: 1468-201X ;DOI: 10.1136/heartjnl-2024-323975 ;PMID: 38749654

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19
Growth differentiation factor 11 regulates high glucose-induced cardiomyocyte pyroptosis and diabetic cardiomyopathy by inhibiting inflammasome activation
Growth differentiation factor 11 regulates high glucose-induced cardiomyocyte pyroptosis and diabetic cardiomyopathy by inhibiting inflammasome activation
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Growth differentiation factor 11 regulates high glucose-induced cardiomyocyte pyroptosis and diabetic cardiomyopathy by inhibiting inflammasome activation

Cardiovascular diabetology, 2024-05, Vol.23 (1), p.160-160 [Peer Reviewed Journal]

2024. The Author(s). ;2024. This work is licensed under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2024 ;EISSN: 1475-2840 ;DOI: 10.1186/s12933-024-02258-3 ;PMID: 38715043

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20
Tail length and E525K dilated cardiomyopathy mutant alter human β-cardiac myosin super-relaxed state
Tail length and E525K dilated cardiomyopathy mutant alter human β-cardiac myosin super-relaxed state
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Tail length and E525K dilated cardiomyopathy mutant alter human β-cardiac myosin super-relaxed state

The Journal of general physiology, 2024-06, Vol.156 (6) [Peer Reviewed Journal]

2024 Duno-Miranda et al. ;EISSN: 1540-7748 ;DOI: 10.1085/jgp.202313522 ;PMID: 38709176

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