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1 |
Material Type: Article
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DsbA-L prevents obesity-induced inflammation and insulin resistance by suppressing the mtDNA release-activated cGAS-cGAMP-STING pathwayProceedings of the National Academy of Sciences - PNAS, 2017-11, Vol.114 (46), p.12196-12201 [Peer Reviewed Journal]Volumes 1–89 and 106–114, copyright as a collective work only; author(s) retains copyright to individual articles ;Copyright National Academy of Sciences Nov 14, 2017 ;2017 ;ISSN: 0027-8424 ;EISSN: 1091-6490 ;DOI: 10.1073/pnas.1708744114 ;PMID: 29087318Full text available |
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Material Type: Article
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Inhibition of the cGAS‐STING pathway ameliorates the premature senescence hallmarks of Ataxia‐Telangiectasia brain organoidsAging cell, 2021-09, Vol.20 (9), p.e13468-n/a [Peer Reviewed Journal]2021 The Authors. published by Anatomical Society and John Wiley & Sons Ltd. ;2021 The Authors. Aging Cell published by Anatomical Society and John Wiley & Sons Ltd. ;COPYRIGHT 2021 John Wiley & Sons, Inc. ;2021. This article is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;ISSN: 1474-9718 ;EISSN: 1474-9726 ;DOI: 10.1111/acel.13468 ;PMID: 34459078Full text available |
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Material Type: Article
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Termination of STING responses is mediated via ESCRT‐dependent degradationThe EMBO journal, 2023-06, Vol.42 (12), p.e112712-n/a [Peer Reviewed Journal]2023 The Authors. Published under the terms of the CC BY 4.0 license. ;2023. This article is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;ISSN: 0261-4189 ;EISSN: 1460-2075 ;DOI: 10.15252/embj.2022112712 ;PMID: 37139896Full text available |
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4 |
Material Type: Article
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The cGAS–STING signaling in cardiovascular and metabolic diseases: Future novel target option for pharmacotherapyActa pharmaceutica Sinica. B, 2022-01, Vol.12 (1), p.50-75 [Peer Reviewed Journal]2022 Chinese Pharmaceutical Association and Institute of Materia Medica, Chinese Academy of Medical Sciences ;2022 Chinese Pharmaceutical Association and Institute of Materia Medica, Chinese Academy of Medical Sciences. Production and hosting by Elsevier B.V. ;2022 Chinese Pharmaceutical Association and Institute of Materia Medica, Chinese Academy of Medical Sciences. Production and hosting by Elsevier B.V. 2022 Chinese Pharmaceutical Association and Institute of Materia Medica, Chinese Academy of Medical Sciences ;ISSN: 2211-3835 ;EISSN: 2211-3843 ;DOI: 10.1016/j.apsb.2021.05.011 ;PMID: 35127372Full text available |
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Material Type: Article
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Corrigendum: The cGAS-STING pathway affects vertebral bone but does not promote intervertebral disc cell senescence or degenerationFrontiers in immunology, 2023-04, Vol.14, p.1201655-1201655 [Peer Reviewed Journal]Copyright © 2023 Ottone, Kim, Collins and Risbud. ;Copyright © 2023 Ottone, Kim, Collins and Risbud 2023 Ottone, Kim, Collins and Risbud ;ISSN: 1664-3224 ;EISSN: 1664-3224 ;DOI: 10.3389/fimmu.2023.1201655 ;PMID: 37180136Full text available |
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Material Type: Article
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The Relationship between Reactive Oxygen Species and the cGAS/STING Signaling Pathway in the Inflammaging ProcessInternational journal of molecular sciences, 2022-12, Vol.23 (23), p.15182 [Peer Reviewed Journal]2022 by the authors. Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (https://creativecommons.org/licenses/by/4.0/). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2022 by the authors. 2022 ;ISSN: 1422-0067 ;ISSN: 1661-6596 ;EISSN: 1422-0067 ;DOI: 10.3390/ijms232315182 ;PMID: 36499506Full text available |
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7 |
Material Type: Article
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Harnessing the cooperation between DNA‐PK and cGAS in cancer therapiesBioEssays, 2023-07 [Peer Reviewed Journal]Attribution - NonCommercial ;ISSN: 0265-9247 ;EISSN: 1521-1878 ;DOI: 10.1002/bies.202300045Digital Resources/Online E-Resources |
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8 |
Material Type: Article
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HDAC3 inhibition ameliorates ischemia/reperfusion-induced brain injury by regulating the microglial cGAS-STING pathwayTheranostics, 2020-01, Vol.10 (21), p.9644-9662 [Peer Reviewed Journal]2020. This work is published under https://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The author(s) 2020 ;ISSN: 1838-7640 ;EISSN: 1838-7640 ;DOI: 10.7150/thno.47651 ;PMID: 32863951Full text available |
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Material Type: Article
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Clinical and mechanistic insights into the roles of DDX41 in haematological malignanciesFEBS letters, 2022-11, Vol.596 (21), p.2736-2745 [Peer Reviewed Journal]2022 Federation of European Biochemical Societies. ;ISSN: 0014-5793 ;EISSN: 1873-3468 ;DOI: 10.1002/1873-3468.14487 ;PMID: 36036093Full text available |
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Material Type: Article
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STING Agonists as Cancer TherapeuticsCancers, 2021-05, Vol.13 (11), p.2695 [Peer Reviewed Journal]2021 by the authors. Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (https://creativecommons.org/licenses/by/4.0/). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2021 by the authors. 2021 ;ISSN: 2072-6694 ;EISSN: 2072-6694 ;DOI: 10.3390/cancers13112695 ;PMID: 34070756Full text available |
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11 |
Material Type: Article
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cGAS is essential for cellular senescenceProceedings of the National Academy of Sciences - PNAS, 2017-06, Vol.114 (23), p.E4612-E4620 [Peer Reviewed Journal]Volumes 1–89 and 106–114, copyright as a collective work only; author(s) retains copyright to individual articles ;Copyright National Academy of Sciences Jun 6, 2017 ;ISSN: 0027-8424 ;EISSN: 1091-6490 ;DOI: 10.1073/pnas.1705499114 ;PMID: 28533362Full text available |
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12 |
Material Type: Article
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Research Advances in How the cGAS-STING Pathway Controls the Cellular Inflammatory ResponseFrontiers in immunology, 2020-04, Vol.11, p.615-615 [Peer Reviewed Journal]Copyright © 2020 Wan, Jiang and Hao. ;COPYRIGHT 2020 Frontiers Research Foundation ;Copyright © 2020 Wan, Jiang and Hao. 2020 Wan, Jiang and Hao ;ISSN: 1664-3224 ;EISSN: 1664-3224 ;DOI: 10.3389/fimmu.2020.00615 ;PMID: 32411126Full text available |
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13 |
Material Type: Article
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mtDNA Activates cGAS Signaling and Suppresses the YAP-Mediated Endothelial Cell Proliferation Program to Promote Inflammatory InjuryImmunity (Cambridge, Mass.), 2020-03, Vol.52 (3), p.475-486.e5 [Peer Reviewed Journal]2020 Elsevier Inc. ;Copyright © 2020 Elsevier Inc. All rights reserved. ;2020. Elsevier Inc. ;ISSN: 1074-7613 ;EISSN: 1097-4180 ;DOI: 10.1016/j.immuni.2020.02.002 ;PMID: 32164878Full text available |
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14 |
Material Type: Article
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Cytosolic DNA sensor cGAS plays an essential pathogenetic role in pressure overload-induced heart failureAmerican journal of physiology. Heart and circulatory physiology, 2020-06, Vol.318 (6), p.H1525-H1537 [Peer Reviewed Journal]ISSN: 0363-6135 ;EISSN: 1522-1539 ;DOI: 10.1152/AJPHEART.00097.2020 ;PMID: 32383996Full text available |
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15 |
Material Type: Article
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Inflammasome Activation Triggers Caspase-1-Mediated Cleavage of cGAS to Regulate Responses to DNA Virus InfectionImmunity (Cambridge, Mass.), 2017-03, Vol.46 (3), p.393-404 [Peer Reviewed Journal]2017 Elsevier Inc. ;Copyright © 2017 Elsevier Inc. All rights reserved. ;Copyright Elsevier Limited Mar 21, 2017 ;ISSN: 1074-7613 ;EISSN: 1097-4180 ;DOI: 10.1016/j.immuni.2017.02.011 ;PMID: 28314590Full text available |
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16 |
Material Type: Article
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CGAS is a micronucleophagy receptor for the clearance of micronucleiAutophagy, 2021-12, Vol.17 (12), p.3976-3991 [Peer Reviewed Journal]2021 The Author(s). Published by Informa UK Limited, trading as Taylor & Francis Group. 2021 ;2021 The Author(s). Published by Informa UK Limited, trading as Taylor & Francis Group. 2021 The Author(s) ;ISSN: 1554-8627 ;EISSN: 1554-8635 ;DOI: 10.1080/15548627.2021.1899440 ;PMID: 33752561Full text available |
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17 |
Material Type: Article
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STING orchestrates the crosstalk between polyunsaturated fatty acid metabolism and inflammatory responsesCell metabolism, 2022-01, Vol.34 (1), p.125-139.e8 [Peer Reviewed Journal]2021 The Author(s) ;Copyright © 2021 The Author(s). Published by Elsevier Inc. All rights reserved. ;Attribution - NonCommercial - NoDerivatives ;2021 The Author(s) 2021 ;ISSN: 1550-4131 ;EISSN: 1932-7420 ;DOI: 10.1016/j.cmet.2021.12.007 ;PMID: 34986331Full text available |
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18 |
Material Type: Article
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cGAS-STING Signaling Pathway and Liver Disease: From Basic Research to Clinical PracticeFrontiers in pharmacology, 2021-08, Vol.12, p.719644-719644 [Peer Reviewed Journal]Copyright © 2021 Chen, Rao, Wang, Luo, Wang, Sheng, Liu, Zhang, Jin, Chen, Sun, Xu and Du. 2021 Chen, Rao, Wang, Luo, Wang, Sheng, Liu, Zhang, Jin, Chen, Sun, Xu and Du ;ISSN: 1663-9812 ;EISSN: 1663-9812 ;DOI: 10.3389/fphar.2021.719644 ;PMID: 34483930Full text available |
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19 |
Material Type: Article
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New Insights into Neuroinflammation Involved in Pathogenic Mechanism of Alzheimer’s Disease and Its Potential for Therapeutic InterventionCells (Basel, Switzerland), 2022-06, Vol.11 (12), p.1925 [Peer Reviewed Journal]2022 by the authors. Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (https://creativecommons.org/licenses/by/4.0/). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2022 by the authors. 2022 ;ISSN: 2073-4409 ;EISSN: 2073-4409 ;DOI: 10.3390/cells11121925 ;PMID: 35741054Full text available |
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20 |
Material Type: Article
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Role of the cGAS–STING pathway in cancer development and oncotherapeutic approachesEMBO reports, 2018-12, Vol.19 (12), p.n/a [Peer Reviewed Journal]2018 The Authors ;2018 The Authors. ;2018 EMBO ;ISSN: 1469-221X ;EISSN: 1469-3178 ;DOI: 10.15252/embr.201846935 ;PMID: 30446584Full text available |