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Treating Influenza Infection, From Now and Into the Future
Treating Influenza Infection, From Now and Into the Future
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Treating Influenza Infection, From Now and Into the Future

Frontiers in immunology, 2018-09, Vol.9, p.1946-1946 [Peer Reviewed Journal]

COPYRIGHT 2018 Frontiers Research Foundation ;Copyright © 2018 Davidson. 2018 Davidson ;ISSN: 1664-3224 ;EISSN: 1664-3224 ;DOI: 10.3389/fimmu.2018.01946 ;PMID: 30250466

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2
Pathogenic potential of interferon αβ in acute influenza infection
Pathogenic potential of interferon αβ in acute influenza infection
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Pathogenic potential of interferon αβ in acute influenza infection

Nature communications, 2014-05, Vol.5 (1), p.3864-3864, Article 3864 [Peer Reviewed Journal]

Copyright © 2014, Nature Publishing Group, a division of Macmillan Publishers Limited. All Rights Reserved. 2014 Nature Publishing Group, a division of Macmillan Publishers Limited. All Rights Reserved. ;ISSN: 2041-1723 ;EISSN: 2041-1723 ;DOI: 10.1038/ncomms4864 ;PMID: 24844667

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3
IFNλ is a potent anti‐influenza therapeutic without the inflammatory side effects of IFNα treatment
IFNλ is a potent anti‐influenza therapeutic without the inflammatory side effects of IFNα treatment
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IFNλ is a potent anti‐influenza therapeutic without the inflammatory side effects of IFNα treatment

EMBO molecular medicine, 2016-09, Vol.8 (9), p.1099-1112 [Peer Reviewed Journal]

2016 The Authors. Published under the terms of the CC BY 4.0 license ;2016 The Authors. Published under the terms of the CC BY 4.0 license. ;COPYRIGHT 2016 John Wiley & Sons, Inc. ;2016. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;ISSN: 1757-4676 ;EISSN: 1757-4684 ;DOI: 10.15252/emmm.201606413 ;PMID: 27520969

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4
Type I and type III interferons drive redundant amplification loops to induce a transcriptional signature in influenza-infected airway epithelia
Type I and type III interferons drive redundant amplification loops to induce a transcriptional signature in influenza-infected airway epithelia
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Type I and type III interferons drive redundant amplification loops to induce a transcriptional signature in influenza-infected airway epithelia

PLoS pathogens, 2013-11, Vol.9 (11), p.e1003773-e1003773 [Peer Reviewed Journal]

COPYRIGHT 2013 Public Library of Science ;COPYRIGHT 2013 Public Library of Science ;Attribution ;2013 Crotta et al 2013 Crotta et al ;2013 Crotta et al. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited: Crotta S, Davidson S, Mahlakoiv T, Desmet CJ, Buckwalter MR, et al. (2013) Type I and Type III Interferons Drive Redundant Amplification Loops to Induce a Transcriptional Signature in Influenza-Infected Airway Epithelia. PLoS Pathog 9(11): e1003773. doi:10.1371/journal.ppat.1003773 ;ISSN: 1553-7374 ;ISSN: 1553-7366 ;EISSN: 1553-7374 ;DOI: 10.1371/journal.ppat.1003773 ;PMID: 24278020

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5
Deficiency in coatomer complex I causes aberrant activation of STING signalling
Deficiency in coatomer complex I causes aberrant activation of STING signalling
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Deficiency in coatomer complex I causes aberrant activation of STING signalling

Nature communications, 2022-04, Vol.13 (1), p.2321-2321, Article 2321 [Peer Reviewed Journal]

2022. The Author(s). ;The Author(s) 2022. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2022 ;ISSN: 2041-1723 ;EISSN: 2041-1723 ;DOI: 10.1038/s41467-022-29946-6 ;PMID: 35484149

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6
A Mutation Outside the Dimerization Domain Causing Atypical STING-Associated Vasculopathy With Onset in Infancy
A Mutation Outside the Dimerization Domain Causing Atypical STING-Associated Vasculopathy With Onset in Infancy
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A Mutation Outside the Dimerization Domain Causing Atypical STING-Associated Vasculopathy With Onset in Infancy

Frontiers in immunology, 2018-07, Vol.9, p.1535-1535 [Peer Reviewed Journal]

COPYRIGHT 2018 Frontiers Research Foundation ;Copyright © 2018 Saldanha, Balka, Davidson, Wainstein, Wong, Macintosh, Loo, Weber, Kamath, CIRCA, AADRY, Moghaddas, De Nardo, Gray and Masters. 2018 Saldanha, Balka, Davidson, Wainstein, Wong, Macintosh, Loo, Weber, Kamath, CIRCA, AADRY, Moghaddas, De Nardo, Gray and Masters ;ISSN: 1664-3224 ;EISSN: 1664-3224 ;DOI: 10.3389/fimmu.2018.01535 ;PMID: 30038614

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7
Transcriptional profiling unveils type I and II interferon networks in blood and tissues across diseases
Transcriptional profiling unveils type I and II interferon networks in blood and tissues across diseases
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Transcriptional profiling unveils type I and II interferon networks in blood and tissues across diseases

Nature communications, 2019-06, Vol.10 (1), p.2887-21, Article 2887 [Peer Reviewed Journal]

2019. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2019 ;ISSN: 2041-1723 ;EISSN: 2041-1723 ;DOI: 10.1038/s41467-019-10601-6 ;PMID: 31253760

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8
Natural amines inhibit activation of human plasmacytoid dendritic cells through CXCR4 engagement
Natural amines inhibit activation of human plasmacytoid dendritic cells through CXCR4 engagement
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Natural amines inhibit activation of human plasmacytoid dendritic cells through CXCR4 engagement

Nature communications, 2017-02, Vol.8 (1), p.14253-14253, Article 14253 [Peer Reviewed Journal]

Copyright Nature Publishing Group Feb 2017 ;Attribution ;Copyright © 2017, The Author(s) 2017 The Author(s) ;ISSN: 2041-1723 ;EISSN: 2041-1723 ;DOI: 10.1038/ncomms14253 ;PMID: 28181493

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9
A necroptosis-independent function of RIPK3 promotes immune dysfunction and prevents control of chronic LCMV infection
A necroptosis-independent function of RIPK3 promotes immune dysfunction and prevents control of chronic LCMV infection
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A necroptosis-independent function of RIPK3 promotes immune dysfunction and prevents control of chronic LCMV infection

Cell death & disease, 2023-02, Vol.14 (2), p.123-123, Article 123 [Peer Reviewed Journal]

2023. The Author(s). ;The Author(s) 2023. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2023 ;ISSN: 2041-4889 ;EISSN: 2041-4889 ;DOI: 10.1038/s41419-023-05635-0 ;PMID: 36792599

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10
Type I and Type III Interferons Drive Redundant Amplification Loops to Induce a Transcriptional Signature in Influenza-Infected Airway Epithelia: e1003773
Type I and Type III Interferons Drive Redundant Amplification Loops to Induce a Transcriptional Signature in Influenza-Infected Airway Epithelia: e1003773
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Type I and Type III Interferons Drive Redundant Amplification Loops to Induce a Transcriptional Signature in Influenza-Infected Airway Epithelia: e1003773

PLoS pathogens, 2013-11, Vol.9 (11) [Peer Reviewed Journal]

2013 Crotta et al. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited: Crotta S, Davidson S, Mahlakoiv T, Desmet CJ, Buckwalter MR, et al. (2013) Type I and Type III Interferons Drive Redundant Amplification Loops to Induce a Transcriptional Signature in Influenza-Infected Airway Epithelia. PLoS Pathog 9(11): e1003773. doi:10.1371/journal.ppat.1003773 ;ISSN: 1553-7366 ;EISSN: 1553-7374 ;DOI: 10.1371/journal.ppat.1003773

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11
Pathogenic potential of interferon [alpha][beta] in acute influenza infection
Pathogenic potential of interferon [alpha][beta] in acute influenza infection
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Article 		Add to My Research

Pathogenic potential of interferon [alpha][beta] in acute influenza infection

Nature communications, 2014-05, Vol.5, p.3864 [Peer Reviewed Journal]

Copyright Nature Publishing Group May 2014 ;EISSN: 2041-1723 ;DOI: 10.1038/ncomms4864

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