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Contributions of Mass Spectrometry to the Identification of Low Molecular Weight Molecules Able to Reduce the Toxicity of Amyloid-β Peptide to Cell Cultures and Transgenic Mouse Models of Alzheimer's Disease
Contributions of Mass Spectrometry to the Identification of Low Molecular Weight Molecules Able to Reduce the Toxicity of Amyloid-β Peptide to Cell Cultures and Transgenic Mouse Models of Alzheimer's Disease
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Contributions of Mass Spectrometry to the Identification of Low Molecular Weight Molecules Able to Reduce the Toxicity of Amyloid-β Peptide to Cell Cultures and Transgenic Mouse Models of Alzheimer's Disease

Molecules (Basel, Switzerland), 2019-03, Vol.24 (6), p.1167 [Peer Reviewed Journal]

2019 by the authors. Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (http://creativecommons.org/licenses/by/4.0/). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2019 by the authors. 2019 ;ISSN: 1420-3049 ;EISSN: 1420-3049 ;DOI: 10.3390/molecules24061167 ;PMID: 30909659

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2
Highly localized intracellular Ca.sup.2+ signals promote optimal salivary gland fluid secretion
Highly localized intracellular Ca.sup.2+ signals promote optimal salivary gland fluid secretion
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Highly localized intracellular Ca.sup.2+ signals promote optimal salivary gland fluid secretion

eLife, 2021-07, Vol.10 [Peer Reviewed Journal]

COPYRIGHT 2021 eLife Science Publications, Ltd. ;ISSN: 2050-084X ;EISSN: 2050-084X ;DOI: 10.7554/eLife.66170

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3
Correction to: A probiotic has differential effects on allergic airway inflammation in A/J and C57BL/6 mice and is correlated with the gut microbiome
Correction to: A probiotic has differential effects on allergic airway inflammation in A/J and C57BL/6 mice and is correlated with the gut microbiome
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Correction to: A probiotic has differential effects on allergic airway inflammation in A/J and C57BL/6 mice and is correlated with the gut microbiome

Microbiome, 2021-07, Vol.9 (1), p.1-159, Article 159 [Peer Reviewed Journal]

COPYRIGHT 2021 BioMed Central Ltd. ;2021. This work is licensed under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2021 ;ISSN: 2049-2618 ;EISSN: 2049-2618 ;DOI: 10.1186/s40168-021-01116-8 ;PMID: 34261528

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4
DNA exonuclease Trex1 regulates radiotherapy-induced tumour immunogenicity
DNA exonuclease Trex1 regulates radiotherapy-induced tumour immunogenicity
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DNA exonuclease Trex1 regulates radiotherapy-induced tumour immunogenicity

Nature communications, 2017-06, Vol.8 (1), p.15618-15618, Article 15618 [Peer Reviewed Journal]

Copyright Nature Publishing Group Jun 2017 ;Copyright © 2017, The Author(s) 2017 The Author(s) ;ISSN: 2041-1723 ;EISSN: 2041-1723 ;DOI: 10.1038/ncomms15618 ;PMID: 28598415

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5
Administration of nucleoside-modified mRNA encoding broadly neutralizing antibody protects humanized mice from HIV-1 challenge
Administration of nucleoside-modified mRNA encoding broadly neutralizing antibody protects humanized mice from HIV-1 challenge
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Administration of nucleoside-modified mRNA encoding broadly neutralizing antibody protects humanized mice from HIV-1 challenge

Nature communications, 2017-03, Vol.8 (1), p.14630-8, Article 14630 [Peer Reviewed Journal]

Copyright Nature Publishing Group Mar 2017 ;Copyright © 2017, The Author(s) 2017 The Author(s) ;ISSN: 2041-1723 ;EISSN: 2041-1723 ;DOI: 10.1038/ncomms14630 ;PMID: 28251988

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6
Plasminogen alleles influence susceptibility to invasive aspergillosis
Plasminogen alleles influence susceptibility to invasive aspergillosis
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Plasminogen alleles influence susceptibility to invasive aspergillosis

PLoS genetics, 2008-06, Vol.4 (6), p.e1000101-e1000101 [Peer Reviewed Journal]

COPYRIGHT 2008 Public Library of Science ;This is an open-access article distributed under the terms of the Creative Commons Public Domain declaration which stipulates that, once placed in the public domain, this work may be freely reproduced, distributed, transmitted, modified, built upon, or otherwise used by anyone for any lawful purpose. 2008 ;2008 Public Library of Science. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited: Citation: Zaas AK, Liao G, Chien JW, Weinberg C, Shore D, et al. (2008) Plasminogen Alleles Influence Susceptibility to Invasive Aspergillosis. PLoS Genet 4(6): e1000101. doi:10.1371/journal.pgen.1000101 ;ISSN: 1553-7404 ;ISSN: 1553-7390 ;EISSN: 1553-7404 ;DOI: 10.1371/journal.pgen.1000101 ;PMID: 18566672

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7
Chloroquine modulates antitumor immune response by resetting tumor-associated macrophages toward M1 phenotype
Chloroquine modulates antitumor immune response by resetting tumor-associated macrophages toward M1 phenotype
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Chloroquine modulates antitumor immune response by resetting tumor-associated macrophages toward M1 phenotype

Nature communications, 2018-02, Vol.9 (1), p.873-15, Article 873 [Peer Reviewed Journal]

2018. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2018 ;ISSN: 2041-1723 ;EISSN: 2041-1723 ;DOI: 10.1038/s41467-018-03225-9 ;PMID: 29491374

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8
Pulmonary Toxicity of Polystyrene, Polypropylene, and Polyvinyl Chloride Microplastics in Mice
Pulmonary Toxicity of Polystyrene, Polypropylene, and Polyvinyl Chloride Microplastics in Mice
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Pulmonary Toxicity of Polystyrene, Polypropylene, and Polyvinyl Chloride Microplastics in Mice

Molecules (Basel, Switzerland), 2022-11, Vol.27 (22), p.7926 [Peer Reviewed Journal]

COPYRIGHT 2022 MDPI AG ;2022 by the authors. Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (https://creativecommons.org/licenses/by/4.0/). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2022 by the authors. 2022 ;ISSN: 1420-3049 ;EISSN: 1420-3049 ;DOI: 10.3390/molecules27227926 ;PMID: 36432032

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9
Systemic immune challenges trigger and drive Alzheimer-like neuropathology in mice
Systemic immune challenges trigger and drive Alzheimer-like neuropathology in mice
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Systemic immune challenges trigger and drive Alzheimer-like neuropathology in mice

Journal of neuroinflammation, 2012-07, Vol.9 (1), p.151-151, Article 151 [Peer Reviewed Journal]

COPYRIGHT 2012 BioMed Central Ltd. ;2012 Krstic et al.; licensee BioMed Central Ltd. This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/2.0), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited. ;Copyright ©2012 Krstic et al.; licensee BioMed Central Ltd. 2012 Krstic et al.; licensee BioMed Central Ltd. ;ISSN: 1742-2094 ;EISSN: 1742-2094 ;DOI: 10.1186/1742-2094-9-151 ;PMID: 22747753

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10
Blockade of IDO-kynurenine-AhR metabolic circuitry abrogates IFN-γ-induced immunologic dormancy of tumor-repopulating cells
Blockade of IDO-kynurenine-AhR metabolic circuitry abrogates IFN-γ-induced immunologic dormancy of tumor-repopulating cells
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Blockade of IDO-kynurenine-AhR metabolic circuitry abrogates IFN-γ-induced immunologic dormancy of tumor-repopulating cells

Nature communications, 2017-05, Vol.8 (1), p.15207-15207, Article 15207 [Peer Reviewed Journal]

Copyright © 2017, The Author(s) 2017 The Author(s) ;ISSN: 2041-1723 ;EISSN: 2041-1723 ;DOI: 10.1038/ncomms15207 ;PMID: 28488695

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11
Using mice to model Alzheimer's dementia: an overview of the clinical disease and the preclinical behavioral changes in 10 mouse models
Using mice to model Alzheimer's dementia: an overview of the clinical disease and the preclinical behavioral changes in 10 mouse models
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Using mice to model Alzheimer's dementia: an overview of the clinical disease and the preclinical behavioral changes in 10 mouse models

Frontiers in genetics, 2014-04, Vol.5, p.88-88 [Peer Reviewed Journal]

Copyright © 2014 Webster, Bachstetter, Nelson, Schmitt and Van Eldik. 2014 ;ISSN: 1664-8021 ;EISSN: 1664-8021 ;DOI: 10.3389/fgene.2014.00088 ;PMID: 24795750

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12
IFN-γ-dependent NK cell activation is essential to metastasis suppression by engineered Salmonella
IFN-γ-dependent NK cell activation is essential to metastasis suppression by engineered Salmonella
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IFN-γ-dependent NK cell activation is essential to metastasis suppression by engineered Salmonella

Nature communications, 2021-05, Vol.12 (1), p.2537-2537, Article 2537 [Peer Reviewed Journal]

The Author(s) 2021. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2021 ;ISSN: 2041-1723 ;EISSN: 2041-1723 ;DOI: 10.1038/s41467-021-22755-3 ;PMID: 33953170

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13
Sheltering behavior and locomotor activity in 11 genetically diverse common inbred mouse strains using home-cage monitoring
Sheltering behavior and locomotor activity in 11 genetically diverse common inbred mouse strains using home-cage monitoring
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Sheltering behavior and locomotor activity in 11 genetically diverse common inbred mouse strains using home-cage monitoring

PloS one, 2014-09, Vol.9 (9), p.e108563-e108563 [Peer Reviewed Journal]

2014 Loos et al. This is an open-access article distributed under the terms of the Creative Commons Attribution License: http://creativecommons.org/licenses/by/4.0/ (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2014 Loos et al 2014 Loos et al ;ISSN: 1932-6203 ;EISSN: 1932-6203 ;DOI: 10.1371/journal.pone.0108563 ;PMID: 25264768

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14
MAIT cells launch a rapid, robust and distinct hyperinflammatory response to bacterial superantigens and quickly acquire an anergic phenotype that impedes their cognate antimicrobial function: Defining a novel mechanism of superantigen-induced immunopathology and immunosuppression
MAIT cells launch a rapid, robust and distinct hyperinflammatory response to bacterial superantigens and quickly acquire an anergic phenotype that impedes their cognate antimicrobial function: Defining a novel mechanism of superantigen-induced immunopathology and immunosuppression
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MAIT cells launch a rapid, robust and distinct hyperinflammatory response to bacterial superantigens and quickly acquire an anergic phenotype that impedes their cognate antimicrobial function: Defining a novel mechanism of superantigen-induced immunopathology and immunosuppression

PLoS biology, 2017-06, Vol.15 (6), p.e2001930-e2001930 [Peer Reviewed Journal]

2017 Public Library of Science. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited: Shaler CR, Choi J, Rudak PT, Memarnejadian A, Szabo PA, Tun-Abraham ME, et al. (2017) MAIT cells launch a rapid, robust and distinct hyperinflammatory response to bacterial superantigens and quickly acquire an anergic phenotype that impedes their cognate antimicrobial function: Defining a novel mechanism of superantigen-induced immunopathology and immunosuppression. PLoS Biol 15(6): e2001930. https://doi.org/10.1371/journal.pbio.2001930 ;2017 Shaler et al 2017 Shaler et al ;2017 Public Library of Science. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited: Shaler CR, Choi J, Rudak PT, Memarnejadian A, Szabo PA, Tun-Abraham ME, et al. (2017) MAIT cells launch a rapid, robust and distinct hyperinflammatory response to bacterial superantigens and quickly acquire an anergic phenotype that impedes their cognate antimicrobial function: Defining a novel mechanism of superantigen-induced immunopathology and immunosuppression. PLoS Biol 15(6): e2001930. https://doi.org/10.1371/journal.pbio.2001930 ;ISSN: 1545-7885 ;ISSN: 1544-9173 ;EISSN: 1545-7885 ;DOI: 10.1371/journal.pbio.2001930 ;PMID: 28632753

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15
Microbiota-driven interleukin-17-producing cells and eosinophils synergize to accelerate multiple myeloma progression
Microbiota-driven interleukin-17-producing cells and eosinophils synergize to accelerate multiple myeloma progression
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Microbiota-driven interleukin-17-producing cells and eosinophils synergize to accelerate multiple myeloma progression

Nature communications, 2018-12, Vol.9 (1), p.4832-13, Article 4832 [Peer Reviewed Journal]

2018. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2018 ;ISSN: 2041-1723 ;EISSN: 2041-1723 ;DOI: 10.1038/s41467-018-07305-8 ;PMID: 30510245

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16
Genetic Ablation of Inositol 1,4,5-Trisphosphate Receptor Type 2 (IP 3 R2) Fails to Modify Disease Progression in a Mouse Model of Spinocerebellar Ataxia Type 3
Genetic Ablation of Inositol 1,4,5-Trisphosphate Receptor Type 2 (IP 3 R2) Fails to Modify Disease Progression in a Mouse Model of Spinocerebellar Ataxia Type 3
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Genetic Ablation of Inositol 1,4,5-Trisphosphate Receptor Type 2 (IP 3 R2) Fails to Modify Disease Progression in a Mouse Model of Spinocerebellar Ataxia Type 3

International journal of molecular sciences, 2023-06, Vol.24 (13), p.10606 [Peer Reviewed Journal]

COPYRIGHT 2023 MDPI AG ;2023 by the authors. Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (https://creativecommons.org/licenses/by/4.0/). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2023 by the authors. 2023 ;ISSN: 1422-0067 ;ISSN: 1661-6596 ;EISSN: 1422-0067 ;DOI: 10.3390/ijms241310606 ;PMID: 37445783

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17
In vivo conversion of astrocytes to neurons in the injured adult spinal cord
In vivo conversion of astrocytes to neurons in the injured adult spinal cord
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In vivo conversion of astrocytes to neurons in the injured adult spinal cord

Nature communications, 2014-02, Vol.5 (1), p.3338-3338, Article 3338 [Peer Reviewed Journal]

Copyright Nature Publishing Group Feb 2014 ;ISSN: 2041-1723 ;EISSN: 2041-1723 ;DOI: 10.1038/ncomms4338 ;PMID: 24569435

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18
Anti-quenching NIR-II molecular fluorophores for in vivo high-contrast imaging and pH sensing
Anti-quenching NIR-II molecular fluorophores for in vivo high-contrast imaging and pH sensing
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Anti-quenching NIR-II molecular fluorophores for in vivo high-contrast imaging and pH sensing

Nature communications, 2019-03, Vol.10 (1), p.1058-1058, Article 1058 [Peer Reviewed Journal]

This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2019 ;ISSN: 2041-1723 ;EISSN: 2041-1723 ;DOI: 10.1038/s41467-019-09043-x ;PMID: 30837470

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19
MSC-derived Extracellular Vesicles Attenuate Immune Responses in Two Autoimmune Murine Models: Type 1 Diabetes and Uveoretinitis
MSC-derived Extracellular Vesicles Attenuate Immune Responses in Two Autoimmune Murine Models: Type 1 Diabetes and Uveoretinitis
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MSC-derived Extracellular Vesicles Attenuate Immune Responses in Two Autoimmune Murine Models: Type 1 Diabetes and Uveoretinitis

Stem cell reports, 2017-05, Vol.8 (5), p.1214-1225 [Peer Reviewed Journal]

2017 The Authors ;Copyright © 2017 The Authors. Published by Elsevier Inc. All rights reserved. ;2017 The Authors 2017 ;ISSN: 2213-6711 ;EISSN: 2213-6711 ;DOI: 10.1016/j.stemcr.2017.04.008 ;PMID: 28494937

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20
Mitochondrial reactive oxygen species regulate the induction of CD8 + T cells by plasmacytoid dendritic cells
Mitochondrial reactive oxygen species regulate the induction of CD8 + T cells by plasmacytoid dendritic cells
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Mitochondrial reactive oxygen species regulate the induction of CD8 + T cells by plasmacytoid dendritic cells

Nature communications, 2018-06, Vol.9 (1), p.2241-14, Article 2241 [Peer Reviewed Journal]

2018. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;Attribution ;The Author(s) 2018 ;ISSN: 2041-1723 ;EISSN: 2041-1723 ;DOI: 10.1038/s41467-018-04686-8 ;PMID: 29884826

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