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1
Human and mouse single-nucleus transcriptomics reveal TREM2-dependent and TREM2-independent cellular responses in Alzheimer's disease
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Human and mouse single-nucleus transcriptomics reveal TREM2-dependent and TREM2-independent cellular responses in Alzheimer's disease

Nature medicine, 2020-01, Vol.26 (1), p.131-142 [Peer Reviewed Journal]

COPYRIGHT 2020 Nature Publishing Group ;COPYRIGHT 2020 Nature Publishing Group ;The Author(s), under exclusive licence to Springer Nature America, Inc. 2020. ;ISSN: 1078-8956 ;EISSN: 1546-170X ;DOI: 10.1038/s41591-019-0695-9 ;PMID: 31932797

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2
Impairment of glymphatic pathway function promotes tau pathology after traumatic brain injury
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Impairment of glymphatic pathway function promotes tau pathology after traumatic brain injury

The Journal of neuroscience, 2014-12, Vol.34 (49), p.16180-16193 [Peer Reviewed Journal]

Copyright © 2014 the authors 0270-6474/14/3416180-14$15.00/0. ;Copyright © 2014 the authors 0270-6474/14/3416180-14$15.00/0 2014 ;ISSN: 0270-6474 ;EISSN: 1529-2401 ;DOI: 10.1523/jneurosci.3020-14.2014 ;PMID: 25471560

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3
Ultrastructural Characterization of the Lower Motor System in a Mouse Model of Krabbe Disease
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Ultrastructural Characterization of the Lower Motor System in a Mouse Model of Krabbe Disease

Scientific reports, 2016-12, Vol.6 (1), p.1-15, Article 1 [Peer Reviewed Journal]

Copyright Nature Publishing Group Dec 2016 ;The Author(s) 2016 ;ISSN: 2045-2322 ;EISSN: 2045-2322 ;DOI: 10.1038/s41598-016-0001-8 ;PMID: 28442746

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4
ATN classification and clinical progression in subjective cognitive decline: The SCIENCe project
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ATN classification and clinical progression in subjective cognitive decline: The SCIENCe project

Neurology, 2020-07, Vol.95 (1), p.e46-e58 [Peer Reviewed Journal]

2020 American Academy of Neurology ;Copyright © 2020 The Author(s). Published by Wolters Kluwer Health, Inc. on behalf of the American Academy of Neurology. ;Copyright © 2020 The Author(s). Published by Wolters Kluwer Health, Inc. on behalf of the American Academy of Neurology. 2020 American Academy of Neurology ;ISSN: 0028-3878 ;ISSN: 1526-632X ;EISSN: 1526-632X ;DOI: 10.1212/WNL.0000000000009724 ;PMID: 32522798

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5
Objective subtle cognitive difficulties predict future amyloid accumulation and neurodegeneration
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Objective subtle cognitive difficulties predict future amyloid accumulation and neurodegeneration

Neurology, 2020-01, Vol.94 (4), p.e397-e406 [Peer Reviewed Journal]

2020 American Academy of Neurology ;2019 American Academy of Neurology. ;2019 American Academy of Neurology 2019 American Academy of Neurology ;ISSN: 0028-3878 ;EISSN: 1526-632X ;DOI: 10.1212/WNL.0000000000008838 ;PMID: 31888974

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6
Commensal microflora-induced T cell responses mediate progressive neurodegeneration in glaucoma
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Commensal microflora-induced T cell responses mediate progressive neurodegeneration in glaucoma

Nature communications, 2018-08, Vol.9 (1), p.3209-13, Article 3209 [Peer Reviewed Journal]

2018. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2018 ;ISSN: 2041-1723 ;EISSN: 2041-1723 ;DOI: 10.1038/s41467-018-05681-9 ;PMID: 30097565

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7
Neurodegenerative disease status and post-mortem pathology in idiopathic rapid-eye-movement sleep behaviour disorder: an observational cohort study
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Neurodegenerative disease status and post-mortem pathology in idiopathic rapid-eye-movement sleep behaviour disorder: an observational cohort study

Lancet neurology, 2013-05, Vol.12 (5), p.443-453 [Peer Reviewed Journal]

Elsevier Ltd ;2013 Elsevier Ltd ;Copyright © 2013 Elsevier Ltd. All rights reserved. ;Copyright Elsevier Limited May 2013 ;ISSN: 1474-4422 ;EISSN: 1474-4465 ;DOI: 10.1016/S1474-4422(13)70056-5 ;PMID: 23562390 ;CODEN: LANCAO

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8
Basal forebrain volume reliably predicts the cortical spread of Alzheimer's degeneration
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Basal forebrain volume reliably predicts the cortical spread of Alzheimer's degeneration

Brain (London, England : 1878), 2020-03, Vol.143 (3), p.993-1009 [Peer Reviewed Journal]

The Author(s) (2020). Published by Oxford University Press on behalf of the Guarantors of Brain. ;The Author(s) (2020). Published by Oxford University Press on behalf of the Guarantors of Brain. 2020 ;ISSN: 0006-8950 ;EISSN: 1460-2156 ;DOI: 10.1093/brain/awaa012 ;PMID: 32203580

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9
Entorhinal Tau Pathology, Episodic Memory Decline, and Neurodegeneration in Aging
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Entorhinal Tau Pathology, Episodic Memory Decline, and Neurodegeneration in Aging

The Journal of neuroscience, 2018-01, Vol.38 (3), p.530-543 [Peer Reviewed Journal]

Copyright © 2018 the authors 0270-6474/18/380530-14$15.00/0. ;Copyright Society for Neuroscience Jan 17, 2018 ;Copyright © 2018 the authors 0270-6474/18/380530-14$15.00/0 2018 ;ISSN: 0270-6474 ;EISSN: 1529-2401 ;DOI: 10.1523/jneurosci.2028-17.2017 ;PMID: 29192126

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10
Intrastriatal injection of pre-formed mouse α-synuclein fibrils into rats triggers α-synuclein pathology and bilateral nigrostriatal degeneration
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Intrastriatal injection of pre-formed mouse α-synuclein fibrils into rats triggers α-synuclein pathology and bilateral nigrostriatal degeneration

Neurobiology of disease, 2015-10, Vol.82, p.185-199 [Peer Reviewed Journal]

EISSN: 1095-953X ;DOI: 10.1016/j.nbd.2015.06.003

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11
Rescue of a lysosomal storage disorder caused by Grn loss of function with a brain penetrant progranulin biologic
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Article
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Rescue of a lysosomal storage disorder caused by Grn loss of function with a brain penetrant progranulin biologic

Cell, 2021-09, Vol.184 (18), p.4651-4668.e25 [Peer Reviewed Journal]

2021 Elsevier Inc. ;Copyright © 2021 Elsevier Inc. All rights reserved. ;ISSN: 0092-8674 ;EISSN: 1097-4172 ;DOI: 10.1016/j.cell.2021.08.002 ;PMID: 34450028

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12
MHCII is required for α-synuclein-induced activation of microglia, CD4 T cell proliferation, and dopaminergic neurodegeneration
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MHCII is required for α-synuclein-induced activation of microglia, CD4 T cell proliferation, and dopaminergic neurodegeneration

The Journal of neuroscience, 2013-06, Vol.33 (23), p.9592-9600 [Peer Reviewed Journal]

Copyright © 2013 the authors 0270-6474/13/339592-09$15.00/0 2013 ;ISSN: 1529-2401 ;ISSN: 0270-6474 ;EISSN: 1529-2401 ;DOI: 10.1523/JNEUROSCI.5610-12.2013 ;PMID: 23739956

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13
Glymphatic system clears extracellular tau and protects from tau aggregation and neurodegeneration
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Article
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Glymphatic system clears extracellular tau and protects from tau aggregation and neurodegeneration

The Journal of experimental medicine, 2022-03, Vol.219 (3) [Peer Reviewed Journal]

2022 Ishida et al. ;2022 Ishida et al. 2022 ;ISSN: 0022-1007 ;EISSN: 1540-9538 ;DOI: 10.1084/jem.20211275 ;PMID: 35212707

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14
Regional brain iron and gene expression provide insights into neurodegeneration in Parkinson's disease
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Regional brain iron and gene expression provide insights into neurodegeneration in Parkinson's disease

Brain (London, England : 1878), 2021-07, Vol.144 (6), p.1787-1798 [Peer Reviewed Journal]

The Author(s) (2021). Published by Oxford University Press on behalf of the Guarantors of Brain. ;The Author(s) (2021). Published by Oxford University Press on behalf of the Guarantors of Brain. 2021 ;ISSN: 0006-8950 ;EISSN: 1460-2156 ;DOI: 10.1093/brain/awab084 ;PMID: 33704443

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15
Neuronal ApoE upregulates MHC-I expression to drive selective neurodegeneration in Alzheimer's disease
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Neuronal ApoE upregulates MHC-I expression to drive selective neurodegeneration in Alzheimer's disease

Nature neuroscience, 2021-06, Vol.24 (6), p.786-798 [Peer Reviewed Journal]

The Author(s), under exclusive licence to Springer Nature America, Inc. 2021. ;ISSN: 1097-6256 ;EISSN: 1546-1726 ;DOI: 10.1038/s41593-021-00851-3 ;PMID: 33958804

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16
Much of late life cognitive decline is not due to common neurodegenerative pathologies
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Much of late life cognitive decline is not due to common neurodegenerative pathologies

Annals of neurology, 2013-09, Vol.74 (3), p.478-489 [Peer Reviewed Journal]

Copyright © 2013 American Neurological Association ;Copyright © 2013 American Neurological Association. ;ISSN: 0364-5134 ;EISSN: 1531-8249 ;DOI: 10.1002/ana.23964 ;PMID: 23798485

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17
EEG evidence of compensatory mechanisms in preclinical Alzheimer's disease
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EEG evidence of compensatory mechanisms in preclinical Alzheimer's disease

Brain (London, England : 1878), 2019-07, Vol.142 (7), p.2096-2112 [Peer Reviewed Journal]

The Author(s) (2019). Published by Oxford University Press on behalf of the Guarantors of Brain. All rights reserved. For Permissions, please email: journals.permissions@oup.com. ;Distributed under a Creative Commons Attribution 4.0 International License ;ISSN: 0006-8950 ;ISSN: 1460-2156 ;EISSN: 1460-2156 ;DOI: 10.1093/brain/awz150 ;PMID: 31211359

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18
Parietal white matter lesions in Alzheimer’s disease are associated with cortical neurodegenerative pathology, but not with small vessel disease
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Parietal white matter lesions in Alzheimer’s disease are associated with cortical neurodegenerative pathology, but not with small vessel disease

Acta neuropathologica, 2017-09, Vol.134 (3), p.459-473 [Peer Reviewed Journal]

The Author(s) 2017 ;Acta Neuropathologica is a copyright of Springer, 2017. ;ISSN: 0001-6322 ;EISSN: 1432-0533 ;DOI: 10.1007/s00401-017-1738-2 ;PMID: 28638989

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19
Patient-Tailored, Connectivity-Based Forecasts of Spreading Brain Atrophy
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Article
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Patient-Tailored, Connectivity-Based Forecasts of Spreading Brain Atrophy

Neuron (Cambridge, Mass.), 2019-12, Vol.104 (5), p.856-868.e5 [Peer Reviewed Journal]

2019 Elsevier Inc. ;Copyright © 2019 Elsevier Inc. All rights reserved. ;2019. Elsevier Inc. ;ISSN: 0896-6273 ;EISSN: 1097-4199 ;DOI: 10.1016/j.neuron.2019.08.037 ;PMID: 31623919

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20
Variants in saposin D domain of prosaposin gene linked to Parkinson's disease
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Article
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Variants in saposin D domain of prosaposin gene linked to Parkinson's disease

Brain (London, England : 1878), 2020-04, Vol.143 (4), p.1190-1205 [Peer Reviewed Journal]

The Author(s) (2020). Published by Oxford University Press on behalf of the Guarantors of Brain. All rights reserved. For permissions, please email: journals.permissions@oup.com. ;ISSN: 0006-8950 ;EISSN: 1460-2156 ;DOI: 10.1093/brain/awaa064 ;PMID: 32201884

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