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1
Molecular discrimination of structurally equivalent Lys 63-linked and linear polyubiquitin chains
Molecular discrimination of structurally equivalent Lys 63-linked and linear polyubiquitin chains
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Molecular discrimination of structurally equivalent Lys 63-linked and linear polyubiquitin chains

EMBO reports, 2009-05, Vol.10 (5), p.466-473 [Peer Reviewed Journal]

Copyright © 2009 European Molecular Biology Organization ;Copyright © 2009, European Molecular Biology Organization 2009 ;ISSN: 1469-221X ;EISSN: 1469-3178 ;DOI: 10.1038/embor.2009.55 ;PMID: 19373254 ;CODEN: ERMEAX

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2
Chronic and Cycling Hypoxia: Drivers of Cancer Chronic Inflammation through HIF-1 and NF-κB Activation: A Review of the Molecular Mechanisms
Chronic and Cycling Hypoxia: Drivers of Cancer Chronic Inflammation through HIF-1 and NF-κB Activation: A Review of the Molecular Mechanisms
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Chronic and Cycling Hypoxia: Drivers of Cancer Chronic Inflammation through HIF-1 and NF-κB Activation: A Review of the Molecular Mechanisms

International journal of molecular sciences, 2021-10, Vol.22 (19), p.10701 [Peer Reviewed Journal]

2021 by the authors. Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (https://creativecommons.org/licenses/by/4.0/). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2021 by the authors. 2021 ;ISSN: 1422-0067 ;ISSN: 1661-6596 ;EISSN: 1422-0067 ;DOI: 10.3390/ijms221910701 ;PMID: 34639040

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3
miR-301a as an NF-κB activator in pancreatic cancer cells
miR-301a as an NF-κB activator in pancreatic cancer cells
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miR-301a as an NF-κB activator in pancreatic cancer cells

The EMBO journal, 2011-01, Vol.30 (1), p.57-67 [Peer Reviewed Journal]

Copyright © 2011 European Molecular Biology Organization ;Copyright © 2011, European Molecular Biology Organization 2011 European Molecular Biology Organization ;ISSN: 0261-4189 ;EISSN: 1460-2075 ;DOI: 10.1038/emboj.2010.296 ;PMID: 21113131

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4
Targeting NF-κB-Inducing Kinase (NIK) in Immunity, Inflammation, and Cancer
Targeting NF-κB-Inducing Kinase (NIK) in Immunity, Inflammation, and Cancer
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Targeting NF-κB-Inducing Kinase (NIK) in Immunity, Inflammation, and Cancer

International journal of molecular sciences, 2020-11, Vol.21 (22), p.8470 [Peer Reviewed Journal]

2020. This work is licensed under http://creativecommons.org/licenses/by/3.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2020 by the authors. 2020 ;ISSN: 1422-0067 ;ISSN: 1661-6596 ;EISSN: 1422-0067 ;DOI: 10.3390/ijms21228470 ;PMID: 33187137

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5
p62/SQSTM1: ‘Jack of all trades’ in health and cancer
p62/SQSTM1: ‘Jack of all trades’ in health and cancer
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p62/SQSTM1: ‘Jack of all trades’ in health and cancer

The FEBS journal, 2019-01, Vol.286 (1), p.8-23 [Peer Reviewed Journal]

2018 The Authors. published by John Wiley & Sons Ltd on behalf of Federation of European Biochemical Societies. ;2018 The Authors. The FEBS Journal published by John Wiley & Sons Ltd on behalf of Federation of European Biochemical Societies. ;Copyright © 2019 Federation of European Biochemical Societies ;ISSN: 1742-464X ;EISSN: 1742-4658 ;DOI: 10.1111/febs.14712 ;PMID: 30499183

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6
NF-κB as a potential molecular target for cancer therapy
NF-κB as a potential molecular target for cancer therapy
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NF-κB as a potential molecular target for cancer therapy

BioFactors (Oxford), 2007, Vol.29 (1), p.19-35 [Peer Reviewed Journal]

Copyright © 2007 International Union of Biochemistry and Molecular Biology, Inc. ;ISSN: 0951-6433 ;EISSN: 1872-8081 ;DOI: 10.1002/biof.5520290103

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7
Activation of IKKα target genes depends on recognition of specific κB binding sites by RelB:p52 dimers
Activation of IKKα target genes depends on recognition of specific κB binding sites by RelB:p52 dimers
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Activation of IKKα target genes depends on recognition of specific κB binding sites by RelB:p52 dimers

The EMBO journal, 2004-10, Vol.23 (21), p.4202-4210 [Peer Reviewed Journal]

Copyright © 2004 European Molecular Biology Organization ;Copyright © 2004, European Molecular Biology Organization 2004 ;ISSN: 0261-4189 ;EISSN: 1460-2075 ;DOI: 10.1038/sj.emboj.7600391 ;PMID: 15470505

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8
Chicoric acid supplementation prevents systemic inflammation‐induced memory impairment and amyloidogenesis via inhibition of NF‐κB
Chicoric acid supplementation prevents systemic inflammation‐induced memory impairment and amyloidogenesis via inhibition of NF‐κB
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Chicoric acid supplementation prevents systemic inflammation‐induced memory impairment and amyloidogenesis via inhibition of NF‐κB

The FASEB journal, 2017-04, Vol.31 (4), p.1494-1507 [Peer Reviewed Journal]

FASEB ;FASEB. ;Copyright Federation of American Societies for Experimental Biology (FASEB) Apr 2017 ;ISSN: 0892-6638 ;EISSN: 1530-6860 ;DOI: 10.1096/fj.201601071R ;PMID: 28003341

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9
NFKB1: a suppressor of inflammation, ageing and cancer
NFKB1: a suppressor of inflammation, ageing and cancer
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NFKB1: a suppressor of inflammation, ageing and cancer

The FEBS journal, 2016-05, Vol.283 (10), p.1812-1822 [Peer Reviewed Journal]

2015 FEBS ;2015 FEBS. ;Copyright © 2016 Federation of European Biochemical Societies ;ISSN: 1742-464X ;EISSN: 1742-4658 ;DOI: 10.1111/febs.13627 ;PMID: 26663363

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10
m6A Reader YTHDF2 Regulates LPS-Induced Inflammatory Response
m6A Reader YTHDF2 Regulates LPS-Induced Inflammatory Response
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m6A Reader YTHDF2 Regulates LPS-Induced Inflammatory Response

International journal of molecular sciences, 2019-03, Vol.20 (6), p.1323 [Peer Reviewed Journal]

2019. This work is licensed under https://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2019 by the authors. 2019 ;ISSN: 1422-0067 ;ISSN: 1661-6596 ;EISSN: 1422-0067 ;DOI: 10.3390/ijms20061323 ;PMID: 30875984

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11
p38MAPK is a novel DNA damage response-independent regulator of the senescence-associated secretory phenotype
p38MAPK is a novel DNA damage response-independent regulator of the senescence-associated secretory phenotype
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p38MAPK is a novel DNA damage response-independent regulator of the senescence-associated secretory phenotype

The EMBO journal, 2011-04, Vol.30 (8), p.1536-1548 [Peer Reviewed Journal]

Copyright © 2011 European Molecular Biology Organization ;Copyright © 2011, European Molecular Biology Organization 2011 European Molecular Biology Organization ;ISSN: 0261-4189 ;EISSN: 1460-2075 ;DOI: 10.1038/emboj.2011.69 ;PMID: 21399611 ;CODEN: EMJODG

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12
DAI/ZBP1 recruits RIP1 and RIP3 through RIP homotypic interaction motifs to activate NF-κB
DAI/ZBP1 recruits RIP1 and RIP3 through RIP homotypic interaction motifs to activate NF-κB
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DAI/ZBP1 recruits RIP1 and RIP3 through RIP homotypic interaction motifs to activate NF-κB

EMBO reports, 2009-08, Vol.10 (8), p.916-922 [Peer Reviewed Journal]

Copyright © 2009 European Molecular Biology Organization ;Copyright © 2009, European Molecular Biology Organization 2009 ;ISSN: 1469-221X ;EISSN: 1469-3178 ;DOI: 10.1038/embor.2009.109 ;PMID: 19590578

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13
Hypoxia‐induced HE4 in tubular epithelial cells promotes extracellular matrix accumulation and renal fibrosis via NF‐κB
Hypoxia‐induced HE4 in tubular epithelial cells promotes extracellular matrix accumulation and renal fibrosis via NF‐κB
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Hypoxia‐induced HE4 in tubular epithelial cells promotes extracellular matrix accumulation and renal fibrosis via NF‐κB

The FASEB journal, 2020-02, Vol.34 (2), p.2554-2567 [Peer Reviewed Journal]

2019 Federation of American Societies for Experimental Biology ;2019 Federation of American Societies for Experimental Biology. ;ISSN: 0892-6638 ;EISSN: 1530-6860 ;DOI: 10.1096/fj.201901950R ;PMID: 31909536

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14
NF‐κB and HIF crosstalk in immune responses
NF‐κB and HIF crosstalk in immune responses
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NF‐κB and HIF crosstalk in immune responses

The FEBS journal, 2016-02, Vol.283 (3), p.413-424 [Peer Reviewed Journal]

2015 The Authors. Journal published by John Wiley & Sons Ltd on behalf of Federation of European Biochemical Societies. ;2015 The Authors. The FEBS Journal published by John Wiley & Sons Ltd on behalf of Federation of European Biochemical Societies. ;ISSN: 1742-464X ;EISSN: 1742-4658 ;DOI: 10.1111/febs.13578 ;PMID: 26513405

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15
Leishmania donovani inhibits inflammasome‐dependent macrophage activation by exploiting the negative regulatory proteins A20 and UCP2
Leishmania donovani inhibits inflammasome‐dependent macrophage activation by exploiting the negative regulatory proteins A20 and UCP2
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Leishmania donovani inhibits inflammasome‐dependent macrophage activation by exploiting the negative regulatory proteins A20 and UCP2

The FASEB journal, 2017-11, Vol.31 (11), p.5087-5101 [Peer Reviewed Journal]

FASEB ;FASEB. ;Copyright Federation of American Societies for Experimental Biology (FASEB) Nov 2017 ;ISSN: 0892-6638 ;EISSN: 1530-6860 ;DOI: 10.1096/fj.201700407R ;PMID: 28765172

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16
Relevance of SIRT1-NF-κB Axis as Therapeutic Target to Ameliorate Inflammation in Liver Disease
Relevance of SIRT1-NF-κB Axis as Therapeutic Target to Ameliorate Inflammation in Liver Disease
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Relevance of SIRT1-NF-κB Axis as Therapeutic Target to Ameliorate Inflammation in Liver Disease

International journal of molecular sciences, 2020-05, Vol.21 (11), p.3858 [Peer Reviewed Journal]

2020. This work is licensed under http://creativecommons.org/licenses/by/3.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2020 by the authors. 2020 ;ISSN: 1422-0067 ;ISSN: 1661-6596 ;EISSN: 1422-0067 ;DOI: 10.3390/ijms21113858 ;PMID: 32485811

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17
NF-κB Signaling Regulates Physiological and Pathological Chondrogenesis
NF-κB Signaling Regulates Physiological and Pathological Chondrogenesis
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NF-κB Signaling Regulates Physiological and Pathological Chondrogenesis

International journal of molecular sciences, 2019-12, Vol.20 (24), p.6275 [Peer Reviewed Journal]

2019 by the authors. Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (http://creativecommons.org/licenses/by/4.0/). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2019 by the authors. 2019 ;ISSN: 1422-0067 ;ISSN: 1661-6596 ;EISSN: 1422-0067 ;DOI: 10.3390/ijms20246275 ;PMID: 31842396

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18
The long noncoding RNA MALAT1 regulates the lipopolysaccharide‐induced inflammatory response through its interaction with NF‐κB
The long noncoding RNA MALAT1 regulates the lipopolysaccharide‐induced inflammatory response through its interaction with NF‐κB
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The long noncoding RNA MALAT1 regulates the lipopolysaccharide‐induced inflammatory response through its interaction with NF‐κB

FEBS letters, 2016-09, Vol.590 (17), p.2884-2895 [Peer Reviewed Journal]

2016 Federation of European Biochemical Societies ;2016 Federation of European Biochemical Societies. ;ISSN: 0014-5793 ;EISSN: 1873-3468 ;DOI: 10.1002/1873-3468.12315 ;PMID: 27434861

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19
Fish NF‐κB couples TCR and IL‐17 signals to regulate ancestral T‐cell immune response against bacterial infection
Fish NF‐κB couples TCR and IL‐17 signals to regulate ancestral T‐cell immune response against bacterial infection
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Fish NF‐κB couples TCR and IL‐17 signals to regulate ancestral T‐cell immune response against bacterial infection

The FASEB journal, 2021-04, Vol.35 (4), p.e21457-n/a [Peer Reviewed Journal]

2021 Federation of American Societies for Experimental Biology ;2021 Federation of American Societies for Experimental Biology. ;ISSN: 0892-6638 ;EISSN: 1530-6860 ;DOI: 10.1096/fj.202002393RR ;PMID: 33689192

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20
Aneuploid senescent cells activate NF‐κB to promote their immune clearance by NK cells
Aneuploid senescent cells activate NF‐κB to promote their immune clearance by NK cells
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Aneuploid senescent cells activate NF‐κB to promote their immune clearance by NK cells

EMBO reports, 2021-08, Vol.22 (8), p.e52032-n/a [Peer Reviewed Journal]

2021 The Authors. Published under the terms of the CC BY 4.0 license ;2021. This article is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;ISSN: 1469-221X ;EISSN: 1469-3178 ;DOI: 10.15252/embr.202052032 ;PMID: 34105235

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