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Refined by: subject: Oxidative Stress remove
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1
Oxidative stress: the mitochondria-dependent and mitochondria-independent pathways of apoptosis
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Oxidative stress: the mitochondria-dependent and mitochondria-independent pathways of apoptosis

Archives of toxicology, 2013-07, Vol.87 (7), p.1157-1180 [Peer Reviewed Journal]

Springer-Verlag Berlin Heidelberg 2013 ;ISSN: 0340-5761 ;EISSN: 1432-0738 ;DOI: 10.1007/s00204-013-1034-4 ;PMID: 23543009

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2
Role of mitochondrial dysfunction and altered autophagy in cardiovascular aging and disease: from mechanisms to therapeutics
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Role of mitochondrial dysfunction and altered autophagy in cardiovascular aging and disease: from mechanisms to therapeutics

American journal of physiology. Heart and circulatory physiology, 2013-08, Vol.305 (4), p.H459-H476 [Peer Reviewed Journal]

Copyright American Physiological Society Aug 15, 2013 ;Copyright © 2013 the American Physiological Society 2013 American Physiological Society ;ISSN: 0363-6135 ;EISSN: 1522-1539 ;DOI: 10.1152/ajpheart.00936.2012 ;PMID: 23748424 ;CODEN: AJPPDI

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3
Neutralization of Oxidized Phospholipids Ameliorates Non-alcoholic Steatohepatitis
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Neutralization of Oxidized Phospholipids Ameliorates Non-alcoholic Steatohepatitis

Cell metabolism, 2020-01, Vol.31 (1), p.189-206.e8 [Peer Reviewed Journal]

2019 Elsevier Inc. ;Copyright © 2019 Elsevier Inc. All rights reserved. ;ISSN: 1550-4131 ;EISSN: 1932-7420 ;DOI: 10.1016/j.cmet.2019.10.014 ;PMID: 31761566

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4
A pathway coordinated by DELE1 relays mitochondrial stress to the cytosol
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A pathway coordinated by DELE1 relays mitochondrial stress to the cytosol

Nature (London), 2020-03, Vol.579 (7799), p.433-437 [Peer Reviewed Journal]

COPYRIGHT 2020 Nature Publishing Group ;Copyright Nature Publishing Group Mar 19, 2020 ;ISSN: 0028-0836 ;EISSN: 1476-4687 ;DOI: 10.1038/s41586-020-2076-4 ;PMID: 32132706

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5
Melatonin: an ancient molecule that makes oxygen metabolically tolerable
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Melatonin: an ancient molecule that makes oxygen metabolically tolerable

Journal of pineal research, 2015-11, Vol.59 (4), p.403-419 [Peer Reviewed Journal]

2015 John Wiley & Sons A/S. Published by John Wiley & Sons Ltd ;2015 John Wiley & Sons A/S. Published by John Wiley & Sons Ltd. ;ISSN: 0742-3098 ;EISSN: 1600-079X ;DOI: 10.1111/jpi.12267 ;PMID: 26272235

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6
Mitochondrial fission links ECM mechanotransduction to metabolic redox homeostasis and metastatic chemotherapy resistance
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Mitochondrial fission links ECM mechanotransduction to metabolic redox homeostasis and metastatic chemotherapy resistance

Nature cell biology, 2022-02, Vol.24 (2), p.168-180 [Peer Reviewed Journal]

2022. The Author(s), under exclusive licence to Springer Nature Limited. ;The Author(s), under exclusive licence to Springer Nature Limited 2022. ;ISSN: 1465-7392 ;EISSN: 1476-4679 ;DOI: 10.1038/s41556-022-00843-w ;PMID: 35165418

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7
2-APQC, a small-molecule activator of Sirtuin-3 (SIRT3), alleviates myocardial hypertrophy and fibrosis by regulating mitochondrial homeostasis
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2-APQC, a small-molecule activator of Sirtuin-3 (SIRT3), alleviates myocardial hypertrophy and fibrosis by regulating mitochondrial homeostasis

Signal transduction and targeted therapy, 2024-05, Vol.9 (1), p.133-133 [Peer Reviewed Journal]

2024. The Author(s). ;The Author(s) 2024. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2024 ;ISSN: 2095-9907 ;EISSN: 2059-3635 ;DOI: 10.1038/s41392-024-01816-1 ;PMID: 38744811

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8
SLC25A51 is a mammalian mitochondrial NAD + transporter
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SLC25A51 is a mammalian mitochondrial NAD + transporter

Nature (London), 2020-12, Vol.588 (7836), p.174-179 [Peer Reviewed Journal]

Copyright Nature Publishing Group Dec 3, 2020 ;ISSN: 0028-0836 ;EISSN: 1476-4687 ;DOI: 10.1038/s41586-020-2741-7 ;PMID: 32906142

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9
The epigenetic regulators and metabolic changes in ferroptosis-associated cancer progression
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The epigenetic regulators and metabolic changes in ferroptosis-associated cancer progression

Molecular cancer, 2020-02, Vol.19 (1), p.39-17, Article 39 [Peer Reviewed Journal]

COPYRIGHT 2020 BioMed Central Ltd. ;2020. This work is licensed under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2020 ;ISSN: 1476-4598 ;EISSN: 1476-4598 ;DOI: 10.1186/s12943-020-01157-x ;PMID: 32103754

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10
MOFs-based nanoagent enables dual mitochondrial damage in synergistic antitumor therapy via oxidative stress and calcium overload
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MOFs-based nanoagent enables dual mitochondrial damage in synergistic antitumor therapy via oxidative stress and calcium overload

Nature communications, 2021-11, Vol.12 (1), p.6399-6399, Article 6399 [Peer Reviewed Journal]

2021. The Author(s). ;The Author(s) 2021. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2021 ;ISSN: 2041-1723 ;EISSN: 2041-1723 ;DOI: 10.1038/s41467-021-26655-4 ;PMID: 34737274

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11
Cytotoxic Potential and Molecular Pathway Analysis of Silver Nanoparticles in Human Colon Cancer Cells HCT116
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Cytotoxic Potential and Molecular Pathway Analysis of Silver Nanoparticles in Human Colon Cancer Cells HCT116

International journal of molecular sciences, 2018-08, Vol.19 (8), p.2269 [Peer Reviewed Journal]

2018. This work is licensed under https://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2018 by the authors. 2018 ;ISSN: 1422-0067 ;ISSN: 1661-6596 ;EISSN: 1422-0067 ;DOI: 10.3390/ijms19082269 ;PMID: 30072642

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12
Short-term starvation is a strategy to unravel the cellular capacity of oxidizing specific exogenous/endogenous substrates in mitochondria
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Short-term starvation is a strategy to unravel the cellular capacity of oxidizing specific exogenous/endogenous substrates in mitochondria

The Journal of biological chemistry, 2017-08, Vol.292 (34), p.14176-14187 [Peer Reviewed Journal]

2017 © 2017 ASBMB. Currently published by Elsevier Inc; originally published by American Society for Biochemistry and Molecular Biology. ;2017 by The American Society for Biochemistry and Molecular Biology, Inc. ;2017 by The American Society for Biochemistry and Molecular Biology, Inc. 2017 The American Society for Biochemistry and Molecular Biology, Inc. ;ISSN: 0021-9258 ;EISSN: 1083-351X ;DOI: 10.1074/jbc.M117.786582 ;PMID: 28663370

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13
Altered Cardiac Energetics and Mitochondrial Dysfunction in Hypertrophic Cardiomyopathy
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Altered Cardiac Energetics and Mitochondrial Dysfunction in Hypertrophic Cardiomyopathy

Circulation (New York, N.Y.), 2021-11, Vol.144 (21), p.1714-1731 [Peer Reviewed Journal]

ISSN: 0009-7322 ;EISSN: 1524-4539 ;DOI: 10.1161/circulationaha.121.053575 ;PMID: 34672721

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14
The Significance of Mitochondrial Dysfunction in Cancer
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The Significance of Mitochondrial Dysfunction in Cancer

International journal of molecular sciences, 2020-08, Vol.21 (16), p.5598 [Peer Reviewed Journal]

2020. This work is licensed under http://creativecommons.org/licenses/by/3.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2020 by the authors. 2020 ;ISSN: 1422-0067 ;ISSN: 1661-6596 ;EISSN: 1422-0067 ;DOI: 10.3390/ijms21165598 ;PMID: 32764295

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15
Insulin resistance in brain alters dopamine turnover and causes behavioral disorders
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Insulin resistance in brain alters dopamine turnover and causes behavioral disorders

Proceedings of the National Academy of Sciences - PNAS, 2015-03, Vol.112 (11), p.3463-3468 [Peer Reviewed Journal]

Volumes 1–89 and 106–112, copyright as a collective work only; author(s) retains copyright to individual articles ;Copyright National Academy of Sciences Mar 17, 2015 ;ISSN: 0027-8424 ;EISSN: 1091-6490 ;DOI: 10.1073/pnas.1500877112 ;PMID: 25733901

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16
Extracellular vesicle-based interorgan transport of mitochondria from energetically stressed adipocytes
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Extracellular vesicle-based interorgan transport of mitochondria from energetically stressed adipocytes

Cell metabolism, 2021-09, Vol.33 (9), p.1853-1868.e11 [Peer Reviewed Journal]

2021 Elsevier Inc. ;Copyright © 2021 Elsevier Inc. All rights reserved. ;ISSN: 1550-4131 ;EISSN: 1932-7420 ;DOI: 10.1016/j.cmet.2021.08.002 ;PMID: 34418352

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17
First‐in‐class cardiolipin‐protective compound as a therapeutic agent to restore mitochondrial bioenergetics
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First‐in‐class cardiolipin‐protective compound as a therapeutic agent to restore mitochondrial bioenergetics

British journal of pharmacology, 2014-04, Vol.171 (8), p.2029-2050 [Peer Reviewed Journal]

2013 The British Pharmacological Society ;2013 The British Pharmacological Society. ;Copyright © 2014 The British Pharmacological Society ;2013 The British Pharmacological Society 2013 ;ISSN: 0007-1188 ;EISSN: 1476-5381 ;DOI: 10.1111/bph.12461 ;PMID: 24117165

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18
Mitochondrial Determinants of Doxorubicin-Induced Cardiomyopathy
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Article
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Mitochondrial Determinants of Doxorubicin-Induced Cardiomyopathy

Circulation research, 2020-03, Vol.126 (7), p.926-941 [Peer Reviewed Journal]

2020 American Heart Association, Inc. ;ISSN: 0009-7330 ;EISSN: 1524-4571 ;DOI: 10.1161/CIRCRESAHA.119.314681 ;PMID: 32213135

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19
Mitochondrial Dysfunctions: A Red Thread across Neurodegenerative Diseases
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Mitochondrial Dysfunctions: A Red Thread across Neurodegenerative Diseases

International journal of molecular sciences, 2020-05, Vol.21 (10), p.3719 [Peer Reviewed Journal]

2020. This work is licensed under http://creativecommons.org/licenses/by/3.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2020 by the authors. 2020 ;ISSN: 1422-0067 ;ISSN: 1661-6596 ;EISSN: 1422-0067 ;DOI: 10.3390/ijms21103719 ;PMID: 32466216

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20
Pharmacologic activation of mitochondrial biogenesis exerts widespread beneficial effects in a transgenic mouse model of Huntington's disease
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Pharmacologic activation of mitochondrial biogenesis exerts widespread beneficial effects in a transgenic mouse model of Huntington's disease

Human molecular genetics, 2012-03, Vol.21 (5), p.1124-1137 [Peer Reviewed Journal]

The Author 2011. Published by Oxford University Press. All rights reserved. For Permissions, please email: journals.permissions@oup.com 2012 ;2015 INIST-CNRS ;ISSN: 0964-6906 ;EISSN: 1460-2083 ;DOI: 10.1093/hmg/ddr541 ;PMID: 22095692

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