skip to main content
Language:
Search Limited to: Search Limited to: Resource type Show Results with: Show Results with: Search type Index

Results 1 - 20 of 2,318  for All Library Resources

Results 1 2 3 4 5 next page
Show only
Result Number Material Type Add to My Shelf Action Record Details and Options
1
Mitofusin‐2 knockdown increases ER–mitochondria contact and decreases amyloid β‐peptide production
Material Type:
Article
Add to My Research

Mitofusin‐2 knockdown increases ER–mitochondria contact and decreases amyloid β‐peptide production

Journal of cellular and molecular medicine, 2016-09, Vol.20 (9), p.1686-1695 [Peer Reviewed Journal]

2016 The Authors. Journal of Cellular and Molecular Medicine published by John Wiley & Sons Ltd and Foundation for Cellular and Molecular Medicine. ;2016. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;ISSN: 1582-1838 ;ISSN: 1582-4934 ;EISSN: 1582-4934 ;DOI: 10.1111/jcmm.12863 ;PMID: 27203684

Full text available

2
The mitochondria‐targeted anti‐oxidant MitoQ protects against intervertebral disc degeneration by ameliorating mitochondrial dysfunction and redox imbalance
Material Type:
Article
Add to My Research

The mitochondria‐targeted anti‐oxidant MitoQ protects against intervertebral disc degeneration by ameliorating mitochondrial dysfunction and redox imbalance

Cell proliferation, 2020-03, Vol.53 (3), p.e12779-n/a [Peer Reviewed Journal]

2020 The Authors. Published by John Wiley & Sons Ltd. ;2020 The Authors. Cell Proliferation Published by John Wiley & Sons Ltd. ;2020. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;ISSN: 0960-7722 ;EISSN: 1365-2184 ;DOI: 10.1111/cpr.12779 ;PMID: 32020711

Full text available

3
MCU Up‐regulation contributes to myocardial ischemia‐reperfusion Injury through calpain/OPA‐1–mediated mitochondrial fusion/mitophagy Inhibition
Material Type:
Article
Add to My Research

MCU Up‐regulation contributes to myocardial ischemia‐reperfusion Injury through calpain/OPA‐1–mediated mitochondrial fusion/mitophagy Inhibition

Journal of cellular and molecular medicine, 2019-11, Vol.23 (11), p.7830-7843 [Peer Reviewed Journal]

2019 The Authors. published by John Wiley & Sons Ltd and Foundation for Cellular and Molecular Medicine. ;2019 The Authors. Journal of Cellular and Molecular Medicine published by John Wiley & Sons Ltd and Foundation for Cellular and Molecular Medicine. ;2019. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;ISSN: 1582-1838 ;EISSN: 1582-4934 ;DOI: 10.1111/jcmm.14662 ;PMID: 31502361

Full text available

4
Yeast mitochondria: an overview of mitochondrial biology and the potential of mitochondrial systems biology
Material Type:
Article
Add to My Research

Yeast mitochondria: an overview of mitochondrial biology and the potential of mitochondrial systems biology

FEMS yeast research, 2018-08, Vol.18 (5), p.1 [Peer Reviewed Journal]

COPYRIGHT 2018 Oxford University Press ;FEMS 2018. ;ISSN: 1567-1364 ;ISSN: 1567-1356 ;EISSN: 1567-1364 ;DOI: 10.1093/femsyr/foy040 ;PMID: 29788060

Full text available

5
Inhibition of Drp1/Fis1 interaction slows progression of amyotrophic lateral sclerosis
Material Type:
Article
Add to My Research

Inhibition of Drp1/Fis1 interaction slows progression of amyotrophic lateral sclerosis

EMBO molecular medicine, 2018-03, Vol.10 (3), p.n/a [Peer Reviewed Journal]

2018 The Authors. Published under the terms of the CC BY 4.0 license ;2018 The Authors. Published under the terms of the CC BY 4.0 license. ;2018. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;ISSN: 1757-4676 ;EISSN: 1757-4684 ;DOI: 10.15252/emmm.201708166 ;PMID: 29335339

Full text available

6
Internalization of isolated functional mitochondria: involvement of macropinocytosis
Material Type:
Article
Add to My Research

Internalization of isolated functional mitochondria: involvement of macropinocytosis

Journal of cellular and molecular medicine, 2014-08, Vol.18 (8), p.1694-1703 [Peer Reviewed Journal]

2014 The Authors. Journal of Cellular and Molecular Medicine published by John Wiley & Sons Ltd and Foundation for Cellular and Molecular Medicine. ;Copyright © 2014 John Wiley & Sons Ltd and Foundation for Cellular and Molecular Medicine ;2014. This work is published under http://creativecommons.org/licenses/by/3.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2014 The Authors. Journal of Cellular and Molecular Medicine published by John Wiley & Sons Ltd and Foundation for Cellular and Molecular Medicine. 2014 ;ISSN: 1582-1838 ;EISSN: 1582-4934 ;DOI: 10.1111/jcmm.12316 ;PMID: 24912369

Full text available

7
Rapamycin rescues mitochondrial myopathy via coordinated activation of autophagy and lysosomal biogenesis
Material Type:
Article
Add to My Research

Rapamycin rescues mitochondrial myopathy via coordinated activation of autophagy and lysosomal biogenesis

EMBO molecular medicine, 2018-11, Vol.10 (11), p.n/a [Peer Reviewed Journal]

2018 The Authors. Published under the terms of the CC BY 4.0 license ;2018 The Authors. Published under the terms of the CC BY 4.0 license. ;2018. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;ISSN: 1757-4676 ;EISSN: 1757-4684 ;DOI: 10.15252/emmm.201708799 ;PMID: 30309855

Full text available

8
Branched‐chain amino acids promote endothelial dysfunction through increased reactive oxygen species generation and inflammation
Material Type:
Article
Add to My Research

Branched‐chain amino acids promote endothelial dysfunction through increased reactive oxygen species generation and inflammation

Journal of cellular and molecular medicine, 2018-10, Vol.22 (10), p.4948-4962 [Peer Reviewed Journal]

2018 The Authors. Journal of Cellular and Molecular Medicine published by John Wiley & Sons Ltd and Foundation for Cellular and Molecular Medicine. ;2018. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;ISSN: 1582-1838 ;EISSN: 1582-4934 ;DOI: 10.1111/jcmm.13759 ;PMID: 30063118

Full text available

9
Melatonin ameliorates intervertebral disc degeneration via the potential mechanisms of mitophagy induction and apoptosis inhibition
Material Type:
Article
Add to My Research

Melatonin ameliorates intervertebral disc degeneration via the potential mechanisms of mitophagy induction and apoptosis inhibition

Journal of cellular and molecular medicine, 2019-03, Vol.23 (3), p.2136-2148 [Peer Reviewed Journal]

2019 The Authors. Journal of Cellular and Molecular Medicine published by John Wiley & Sons Ltd and Foundation for Cellular and Molecular Medicine. ;Copyright © 2019 Journal of Cellular and Molecular Medicine published by John Wiley & Sons Ltd and Foundation for Cellular and Molecular Medicine ;ISSN: 1582-1838 ;EISSN: 1582-4934 ;DOI: 10.1111/jcmm.14125 ;PMID: 30609271

Full text available

10
Acylated ghrelin treatment normalizes skeletal muscle mitochondrial oxidative capacity and AKT phosphorylation in rat chronic heart failure
Material Type:
Article
Add to My Research

Acylated ghrelin treatment normalizes skeletal muscle mitochondrial oxidative capacity and AKT phosphorylation in rat chronic heart failure

Journal of cachexia, sarcopenia and muscle, 2017-12, Vol.8 (6), p.991-998 [Peer Reviewed Journal]

2017 The Authors. Journal of Cachexia, Sarcopenia and Muscle published by John Wiley & Sons Ltd on behalf of the Society on Sarcopenia, Cachexia and Wasting Disorders ;2017 The Authors. Journal of Cachexia, Sarcopenia and Muscle published by John Wiley & Sons Ltd on behalf of the Society on Sarcopenia, Cachexia and Wasting Disorders. ;2017. This work is published under http://creativecommons.org/licenses/by-nc-nd/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;ISSN: 2190-5991 ;EISSN: 2190-6009 ;DOI: 10.1002/jcsm.12254 ;PMID: 29098797

Full text available

11
Altered bioenergetics and mitochondrial dysfunction of monocytes in patients with COVID‐19 pneumonia
Material Type:
Article
Add to My Research

Altered bioenergetics and mitochondrial dysfunction of monocytes in patients with COVID‐19 pneumonia

EMBO molecular medicine, 2020-12, Vol.12 (12), p.e13001-n/a [Peer Reviewed Journal]

2020 The Authors. Published under the terms of the CC BY 4.0 license ;2020 The Authors. Published under the terms of the CC BY 4.0 license. ;2020. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;ISSN: 1757-4676 ;EISSN: 1757-4684 ;DOI: 10.15252/emmm.202013001 ;PMID: 33078545

Full text available

12
Mitochondrial and mitochondrial‐independent pathways of myocardial cell death during ischaemia and reperfusion injury
Material Type:
Article
Add to My Research

Mitochondrial and mitochondrial‐independent pathways of myocardial cell death during ischaemia and reperfusion injury

Journal of cellular and molecular medicine, 2020-04, Vol.24 (7), p.3795-3806 [Peer Reviewed Journal]

2020 The Authors. published by Foundation for Cellular and Molecular Medicine and John Wiley & Sons Ltd ;2020 The Authors. Journal of Cellular and Molecular Medicine published by Foundation for Cellular and Molecular Medicine and John Wiley & Sons Ltd. ;2020. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;info:eu-repo/semantics/openAccess ;ISSN: 1582-1838 ;EISSN: 1582-4934 ;DOI: 10.1111/jcmm.15127 ;PMID: 32155321

Full text available

13
Stem cell‐derived mitochondria transplantation: A promising therapy for mitochondrial encephalomyopathy
Material Type:
Article
Add to My Research

Stem cell‐derived mitochondria transplantation: A promising therapy for mitochondrial encephalomyopathy

CNS neuroscience & therapeutics, 2021-07, Vol.27 (7), p.733-742 [Peer Reviewed Journal]

2021 The Authors. Published by John Wiley & Sons Ltd. ;2021 The Authors. CNS Neuroscience & Therapeutics Published by John Wiley & Sons Ltd. ;2021. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;ISSN: 1755-5930 ;EISSN: 1755-5949 ;DOI: 10.1111/cns.13618 ;PMID: 33538116

Full text available

14
Metformin upregulates mitophagy in patients with T2DM: A randomized placebo‐controlled study
Material Type:
Article
Add to My Research

Metformin upregulates mitophagy in patients with T2DM: A randomized placebo‐controlled study

Journal of cellular and molecular medicine, 2020-03, Vol.24 (5), p.2832-2846 [Peer Reviewed Journal]

2020 The Authors. published by John Wiley & Sons Ltd and Foundation for Cellular and Molecular Medicine. ;2020 The Authors. Journal of Cellular and Molecular Medicine published by John Wiley & Sons Ltd and Foundation for Cellular and Molecular Medicine. ;2020. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;ISSN: 1582-1838 ;EISSN: 1582-4934 ;DOI: 10.1111/jcmm.14834 ;PMID: 31975558

Full text available

15
Mitochondria-associated endoplasmic reticulum membranes allow adaptation of mitochondrial metabolism to glucose availability in the liver
Material Type:
Article
Add to My Research

Mitochondria-associated endoplasmic reticulum membranes allow adaptation of mitochondrial metabolism to glucose availability in the liver

Journal of molecular cell biology, 2016-04, Vol.8 (2), p.129-143 [Peer Reviewed Journal]

The Author (2016). Published by Oxford University Press on behalf of Journal of Molecular Cell Biology, IBCB, SIBS, CAS. All rights reserved. ;Distributed under a Creative Commons Attribution 4.0 International License ;ISSN: 1674-2788 ;EISSN: 1759-4685 ;DOI: 10.1093/jmcb/mjw004 ;PMID: 26892023

Full text available

16
Charcot–Marie‐Tooth type 2A in vivo models: Current updates
Material Type:
Article
Add to My Research

Charcot–Marie‐Tooth type 2A in vivo models: Current updates

Journal of cellular and molecular medicine, 2024-05, Vol.28 (9), p.e18293-n/a [Peer Reviewed Journal]

2024 The Authors. published by Foundation for Cellular and Molecular Medicine and John Wiley & Sons Ltd. ;2024 The Authors. Journal of Cellular and Molecular Medicine published by Foundation for Cellular and Molecular Medicine and John Wiley & Sons Ltd. ;2024. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;ISSN: 1582-1838 ;EISSN: 1582-4934 ;DOI: 10.1111/jcmm.18293 ;PMID: 38722298

Full text available

17
Melatonin attenuates vascular calcification by inhibiting mitochondria fission via an AMPK/Drp1 signalling pathway
Material Type:
Article
Add to My Research

Melatonin attenuates vascular calcification by inhibiting mitochondria fission via an AMPK/Drp1 signalling pathway

Journal of cellular and molecular medicine, 2020-06, Vol.24 (11), p.6043-6054 [Peer Reviewed Journal]

2020 The Authors. published by Foundation for Cellular and Molecular Medicine and John Wiley & Sons Ltd. ;2020 The Authors. Journal of Cellular and Molecular Medicine published by Foundation for Cellular and Molecular Medicine and John Wiley & Sons Ltd. ;2020. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;ISSN: 1582-1838 ;EISSN: 1582-4934 ;DOI: 10.1111/jcmm.15157 ;PMID: 32368857

Full text available

18
Mitochondrial fission and cristae disruption increase the response of cell models of Huntington's disease to apoptotic stimuli
Material Type:
Article
Add to My Research

Mitochondrial fission and cristae disruption increase the response of cell models of Huntington's disease to apoptotic stimuli

EMBO molecular medicine, 2010-12, Vol.2 (12), p.490-503 [Peer Reviewed Journal]

Copyright © 2010 EMBO Molecular Medicine ;Copyright John Wiley & Sons, Inc. Dec 2010 ;Copyright © 2010 EMBO Molecular Medicine 2010 ;ISSN: 1757-4676 ;EISSN: 1757-4684 ;DOI: 10.1002/emmm.201000102 ;PMID: 21069748

Full text available

19
Novel mitochondrion‐targeting copper(II) complex induces HK2 malfunction and inhibits glycolysis via Drp1‐mediating mitophagy in HCC
Material Type:
Article
Add to My Research

Novel mitochondrion‐targeting copper(II) complex induces HK2 malfunction and inhibits glycolysis via Drp1‐mediating mitophagy in HCC

Journal of cellular and molecular medicine, 2020-03, Vol.24 (5), p.3091-3107 [Peer Reviewed Journal]

2020 The Authors. Journal of Cellular and Molecular Medicine published by John Wiley & Sons Ltd and Foundation for Cellular and Molecular Medicine. ;2020. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;ISSN: 1582-1838 ;EISSN: 1582-4934 ;DOI: 10.1111/jcmm.14971 ;PMID: 31994339

Full text available

20
Reduction of Early Reperfusion Injury With the Mitochondria-Targeting Peptide Bendavia
Material Type:
Article
Add to My Research

Reduction of Early Reperfusion Injury With the Mitochondria-Targeting Peptide Bendavia

Journal of cardiovascular pharmacology and therapeutics, 2014-01, Vol.19 (1), p.121-132

The Author(s) 2013 ;ISSN: 1074-2484 ;EISSN: 1940-4034 ;DOI: 10.1177/1074248413508003 ;PMID: 24288396

Full text available

Results 1 - 20 of 2,318  for All Library Resources

Results 1 2 3 4 5 next page

Personalize your results

  1. Edit

Refine Search Results

Expand My Results

  1.   

Show only

  1. Peer-reviewed Journals (2,300)

Refine My Results

Creation Date 

From To
  1. Before 1984  (13)
  2. 1984 To 1994  (25)
  3. 1995 To 2003  (140)
  4. 2004 To 2013  (528)
  5. After 2013  (1,613)
  6. More options open sub menu

Resource Type 

  1. Articles  (2,316)
  2. Book Chapters  (2)
  3. More options open sub menu

Language 

  1. Japanese  (350)
  2. Norwegian  (7)
  3. Russian  (4)
  4. French  (3)
  5. German  (3)
  6. Chinese  (1)
  7. More options open sub menu

Searching Remote Databases, Please Wait