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1
Tranilast directly targets NLRP3 to treat inflammasome‐driven diseases
Tranilast directly targets NLRP3 to treat inflammasome‐driven diseases
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Tranilast directly targets NLRP3 to treat inflammasome‐driven diseases

EMBO molecular medicine, 2018-04, Vol.10 (4), p.n/a [Peer Reviewed Journal]

2018 The Authors. Published under the terms of the CC BY 4.0 license ;2018 The Authors. Published under the terms of the CC BY 4.0 license. ;ISSN: 1757-4676 ;EISSN: 1757-4684 ;DOI: 10.15252/emmm.201708689 ;PMID: 29531021

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2
Exosomes derived from pro‐inflammatory bone marrow‐derived mesenchymal stem cells reduce inflammation and myocardial injury via mediating macrophage polarization
Exosomes derived from pro‐inflammatory bone marrow‐derived mesenchymal stem cells reduce inflammation and myocardial injury via mediating macrophage polarization
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Exosomes derived from pro‐inflammatory bone marrow‐derived mesenchymal stem cells reduce inflammation and myocardial injury via mediating macrophage polarization

Journal of cellular and molecular medicine, 2019-11, Vol.23 (11), p.7617-7631 [Peer Reviewed Journal]

2019 The Authors. Journal of Cellular and Molecular Medicine published by John Wiley & Sons Ltd and Foundation for Cellular and Molecular Medicine. ;2019. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;ISSN: 1582-1838 ;EISSN: 1582-4934 ;DOI: 10.1111/jcmm.14635 ;PMID: 31557396

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3
Exosomes derived from miR‐181‐5p‐modified adipose‐derived mesenchymal stem cells prevent liver fibrosis via autophagy activation
Exosomes derived from miR‐181‐5p‐modified adipose‐derived mesenchymal stem cells prevent liver fibrosis via autophagy activation
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Exosomes derived from miR‐181‐5p‐modified adipose‐derived mesenchymal stem cells prevent liver fibrosis via autophagy activation

Journal of cellular and molecular medicine, 2017-10, Vol.21 (10), p.2491-2502 [Peer Reviewed Journal]

2017 The Authors. Journal of Cellular and Molecular Medicine published by John Wiley & Sons Ltd and Foundation for Cellular and Molecular Medicine. ;2017. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;ISSN: 1582-1838 ;EISSN: 1582-4934 ;DOI: 10.1111/jcmm.13170 ;PMID: 28382720

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4
Lipid droplet‐dependent fatty acid metabolism controls the immune suppressive phenotype of tumor‐associated macrophages
Lipid droplet‐dependent fatty acid metabolism controls the immune suppressive phenotype of tumor‐associated macrophages
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Lipid droplet‐dependent fatty acid metabolism controls the immune suppressive phenotype of tumor‐associated macrophages

EMBO molecular medicine, 2019-11, Vol.11 (11), p.e10698-n/a [Peer Reviewed Journal]

2019 The Authors. Published under the terms of the CC BY 4.0 license ;2019 The Authors. Published under the terms of the CC BY 4.0 license. ;2019. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;ISSN: 1757-4676 ;EISSN: 1757-4684 ;DOI: 10.15252/emmm.201910698 ;PMID: 31602788

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5
Phenotypical changes of satellite glial cells in a murine model of G M1 -gangliosidosis
Phenotypical changes of satellite glial cells in a murine model of G M1 -gangliosidosis
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Phenotypical changes of satellite glial cells in a murine model of G M1 -gangliosidosis

Journal of cellular and molecular medicine, 2022-01, Vol.26 (2), p.527-539 [Peer Reviewed Journal]

2021 The Authors. Journal of Cellular and Molecular Medicine published by Foundation for Cellular and Molecular Medicine and John Wiley & Sons Ltd. ;ISSN: 1582-1838 ;EISSN: 1582-4934 ;DOI: 10.1111/jcmm.17113 ;PMID: 34877779

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6
Sorafenib attenuates liver fibrosis by triggering hepatic stellate cell ferroptosis via HIF‐1α/SLC7A11 pathway
Sorafenib attenuates liver fibrosis by triggering hepatic stellate cell ferroptosis via HIF‐1α/SLC7A11 pathway
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Sorafenib attenuates liver fibrosis by triggering hepatic stellate cell ferroptosis via HIF‐1α/SLC7A11 pathway

Cell proliferation, 2022-01, Vol.55 (1), p.e13158-n/a [Peer Reviewed Journal]

2021 The Authors. published by John Wiley & Sons Ltd. ;2021 The Authors. Cell Proliferation published by John Wiley & Sons Ltd. ;2022. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;ISSN: 0960-7722 ;EISSN: 1365-2184 ;DOI: 10.1111/cpr.13158 ;PMID: 34811833

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7
Mesenchymal stem cells‐derived exosomes ameliorate intervertebral disc degeneration through inhibiting pyroptosis
Mesenchymal stem cells‐derived exosomes ameliorate intervertebral disc degeneration through inhibiting pyroptosis
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Mesenchymal stem cells‐derived exosomes ameliorate intervertebral disc degeneration through inhibiting pyroptosis

Journal of cellular and molecular medicine, 2020-10, Vol.24 (20), p.11742-11754 [Peer Reviewed Journal]

2020 The Authors. published by John Wiley & Sons Ltd and Foundation for Cellular and Molecular Medicine. ;2020 The Authors. Journal of Cellular and Molecular Medicine published by John Wiley & Sons Ltd and Foundation for Cellular and Molecular Medicine. ;2020. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;ISSN: 1582-1838 ;EISSN: 1582-4934 ;DOI: 10.1111/jcmm.15784 ;PMID: 32860495

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8
Red blood cell-derived nanoerythrosome for antigen delivery with enhanced cancer immunotherapy
Red blood cell-derived nanoerythrosome for antigen delivery with enhanced cancer immunotherapy
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Red blood cell-derived nanoerythrosome for antigen delivery with enhanced cancer immunotherapy

Science advances, 2019-10, Vol.5 (10), p.eaaw6870 [Peer Reviewed Journal]

Copyright © 2019 The Authors, some rights reserved; exclusive licensee American Association for the Advancement of Science. No claim to original U.S. Government Works. Distributed under a Creative Commons Attribution NonCommercial License 4.0 (CC BY-NC). ;Copyright © 2019 The Authors, some rights reserved; exclusive licensee American Association for the Advancement of Science. No claim to original U.S. Government Works. Distributed under a Creative Commons Attribution NonCommercial License 4.0 (CC BY-NC). 2019 The Authors ;ISSN: 2375-2548 ;EISSN: 2375-2548 ;DOI: 10.1126/sciadv.aaw6870 ;PMID: 31681841

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9
Inhibition of ferroptosis attenuates tissue damage and improves long‐term outcomes after traumatic brain injury in mice
Inhibition of ferroptosis attenuates tissue damage and improves long‐term outcomes after traumatic brain injury in mice
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Inhibition of ferroptosis attenuates tissue damage and improves long‐term outcomes after traumatic brain injury in mice

CNS neuroscience & therapeutics, 2019-04, Vol.25 (4), p.465-475 [Peer Reviewed Journal]

2018 John Wiley & Sons Ltd ;2018 John Wiley & Sons Ltd. ;Copyright © 2019 John Wiley & Sons Ltd ;ISSN: 1755-5930 ;EISSN: 1755-5949 ;DOI: 10.1111/cns.13069 ;PMID: 30264934

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10
Role of chronic neuroinflammation in neuroplasticity and cognitive function: A hypothesis
Role of chronic neuroinflammation in neuroplasticity and cognitive function: A hypothesis
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Role of chronic neuroinflammation in neuroplasticity and cognitive function: A hypothesis

Alzheimer's & dementia, 2022-11, Vol.18 (11), p.2327 [Peer Reviewed Journal]

ISSN: 1552-5279 ;EISSN: 1552-5279 ;DOI: 10.1002/alz.12610

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11
Aucubin inhibited lipid accumulation and oxidative stress via Nrf2/HO‐1 and AMPK signalling pathways
Aucubin inhibited lipid accumulation and oxidative stress via Nrf2/HO‐1 and AMPK signalling pathways
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Aucubin inhibited lipid accumulation and oxidative stress via Nrf2/HO‐1 and AMPK signalling pathways

Journal of cellular and molecular medicine, 2019-06, Vol.23 (6), p.4063-4075 [Peer Reviewed Journal]

2019 The Authors. Journal of Cellular and Molecular Medicine published by John Wiley & Sons Ltd and Foundation for Cellular and Molecular Medicine. ;Copyright © 2019 Journal of Cellular and Molecular Medicine published by John Wiley & Sons Ltd and Foundation for Cellular and Molecular Medicine ;ISSN: 1582-1838 ;EISSN: 1582-4934 ;DOI: 10.1111/jcmm.14293 ;PMID: 30950217

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12
Exosomes derived from mature dendritic cells increase endothelial inflammation and atherosclerosis via membrane TNF‐α mediated NF‐κB pathway
Exosomes derived from mature dendritic cells increase endothelial inflammation and atherosclerosis via membrane TNF‐α mediated NF‐κB pathway
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Exosomes derived from mature dendritic cells increase endothelial inflammation and atherosclerosis via membrane TNF‐α mediated NF‐κB pathway

Journal of cellular and molecular medicine, 2016-12, Vol.20 (12), p.2318-2327 [Peer Reviewed Journal]

2016 The Authors. Journal of Cellular and Molecular Medicine published by John Wiley & Sons Ltd and Foundation for Cellular and Molecular Medicine. ;2016. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;ISSN: 1582-1838 ;EISSN: 1582-4934 ;DOI: 10.1111/jcmm.12923 ;PMID: 27515767

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13
Identification of a novel mechanism of blood–brain communication during peripheral inflammation via choroid plexus‐derived extracellular vesicles
Identification of a novel mechanism of blood–brain communication during peripheral inflammation via choroid plexus‐derived extracellular vesicles
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Identification of a novel mechanism of blood–brain communication during peripheral inflammation via choroid plexus‐derived extracellular vesicles

EMBO molecular medicine, 2016-10, Vol.8 (10), p.1162-1183 [Peer Reviewed Journal]

2016 The Authors. Published under the terms of the CC BY 4.0 license ;2016 The Authors. Published under the terms of the CC BY 4.0 license. ;2016. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;ISSN: 1757-4676 ;EISSN: 1757-4684 ;DOI: 10.15252/emmm.201606271 ;PMID: 27596437

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14
MiR-124 Regulates Apoptosis and Autophagy Process in MPTP Model of Parkinson's Disease by Targeting to Bim
MiR-124 Regulates Apoptosis and Autophagy Process in MPTP Model of Parkinson's Disease by Targeting to Bim
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MiR-124 Regulates Apoptosis and Autophagy Process in MPTP Model of Parkinson's Disease by Targeting to Bim

Brain pathology (Zurich, Switzerland), 2016-03, Vol.26 (2), p.167-176 [Peer Reviewed Journal]

2015 International Society of Neuropathology ;2015 International Society of Neuropathology. ;2016 International Society of Neuropathology ;ISSN: 1015-6305 ;EISSN: 1750-3639 ;DOI: 10.1111/bpa.12267 ;PMID: 25976060

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15
Trimethylamine N -oxide-derived zwitterionic polymers: A new class of ultralow fouling bioinspired materials
Trimethylamine N -oxide-derived zwitterionic polymers: A new class of ultralow fouling bioinspired materials
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Trimethylamine N -oxide-derived zwitterionic polymers: A new class of ultralow fouling bioinspired materials

Science advances, 2019-06, Vol.5 (6), p.eaaw9562-eaaw9562 [Peer Reviewed Journal]

Copyright © 2019 The Authors, some rights reserved; exclusive licensee American Association for the Advancement of Science. No claim to original U.S. Government Works. Distributed under a Creative Commons Attribution NonCommercial License 4.0 (CC BY-NC). 2019 The Authors ;ISSN: 2375-2548 ;EISSN: 2375-2548 ;DOI: 10.1126/sciadv.aaw9562 ;PMID: 31214655

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16
Iron accumulation in microglia triggers a cascade of events that leads to altered metabolism and compromised function in APP/PS1 mice
Iron accumulation in microglia triggers a cascade of events that leads to altered metabolism and compromised function in APP/PS1 mice
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Iron accumulation in microglia triggers a cascade of events that leads to altered metabolism and compromised function in APP/PS1 mice

Brain pathology (Zurich, Switzerland), 2019-09, Vol.29 (5), p.606-621 [Peer Reviewed Journal]

2019 International Society of Neuropathology ;2019 International Society of Neuropathology. ;ISSN: 1015-6305 ;EISSN: 1750-3639 ;DOI: 10.1111/bpa.12704 ;PMID: 30661261

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17
Performing label‐fusion‐based segmentation using multiple automatically generated templates
Performing label‐fusion‐based segmentation using multiple automatically generated templates
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Performing label‐fusion‐based segmentation using multiple automatically generated templates

Human brain mapping, 2013-10, Vol.34 (10), p.2635-2654 [Peer Reviewed Journal]

Copyright © 2012 Wiley Periodicals, Inc. ;ISSN: 1065-9471 ;EISSN: 1097-0193 ;DOI: 10.1002/hbm.22092 ;PMID: 22611030

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18
LncRNA MALAT1 is dysregulated in diabetic nephropathy and involved in high glucose‐induced podocyte injury via its interplay with β‐catenin
LncRNA MALAT1 is dysregulated in diabetic nephropathy and involved in high glucose‐induced podocyte injury via its interplay with β‐catenin
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LncRNA MALAT1 is dysregulated in diabetic nephropathy and involved in high glucose‐induced podocyte injury via its interplay with β‐catenin

Journal of cellular and molecular medicine, 2017-11, Vol.21 (11), p.2732-2747 [Peer Reviewed Journal]

2017 The Authors. Journal of Cellular and Molecular Medicine published by John Wiley & Sons Ltd and Foundation for Cellular and Molecular Medicine. ;ISSN: 1582-1838 ;EISSN: 1582-4934 ;DOI: 10.1111/jcmm.13189 ;PMID: 28444861

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19
D‐mannose alleviates osteoarthritis progression by inhibiting chondrocyte ferroptosis in a HIF‐2α‐dependent manner
D‐mannose alleviates osteoarthritis progression by inhibiting chondrocyte ferroptosis in a HIF‐2α‐dependent manner
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D‐mannose alleviates osteoarthritis progression by inhibiting chondrocyte ferroptosis in a HIF‐2α‐dependent manner

Cell proliferation, 2021-11, Vol.54 (11), p.e13134-n/a [Peer Reviewed Journal]

2021 The Authors. Published by John Wiley & Sons Ltd. ;2021 The Authors. Cell Proliferation Published by John Wiley & Sons Ltd. ;2021. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;ISSN: 0960-7722 ;EISSN: 1365-2184 ;DOI: 10.1111/cpr.13134 ;PMID: 34561933

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20
CPT1A‐mediated succinylation of S100A10 increases human gastric cancer invasion
CPT1A‐mediated succinylation of S100A10 increases human gastric cancer invasion
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CPT1A‐mediated succinylation of S100A10 increases human gastric cancer invasion

Journal of cellular and molecular medicine, 2019-01, Vol.23 (1), p.293-305 [Peer Reviewed Journal]

2018 The Authors. Journal of Cellular and Molecular Medicine published by John Wiley & Sons Ltd and Foundation for Cellular and Molecular Medicine. ;ISSN: 1582-1838 ;EISSN: 1582-4934 ;DOI: 10.1111/jcmm.13920 ;PMID: 30394687

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