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CuII(atsm) improves the neurological phenotype and survival of SOD1G93A mice and selectively increases enzymatically active SOD1 in the spinal cord
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CuII(atsm) improves the neurological phenotype and survival of SOD1G93A mice and selectively increases enzymatically active SOD1 in the spinal cord

Scientific reports, 2017-02, Vol.7 (1), p.42292-42292, Article 42292 [Peer Reviewed Journal]

Copyright Nature Publishing Group Feb 2017 ;Copyright © 2017, The Author(s) 2017 The Author(s) ;ISSN: 2045-2322 ;EISSN: 2045-2322 ;DOI: 10.1038/srep42292 ;PMID: 28205575

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2
Evidence for disrupted copper availability in human spinal cord supports CuII(atsm) as a treatment option for sporadic cases of ALS
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Article
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Evidence for disrupted copper availability in human spinal cord supports CuII(atsm) as a treatment option for sporadic cases of ALS

Scientific reports, 2024-03, Vol.14 (1), p.5929-5929 [Peer Reviewed Journal]

The Author(s) 2024. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2024 ;EISSN: 2045-2322 ;DOI: 10.1038/s41598-024-55832-w

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3
Evidence for disrupted copper availability in human spinal cord supports Cu II (atsm) as a treatment option for sporadic cases of ALS
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Article
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Evidence for disrupted copper availability in human spinal cord supports Cu II (atsm) as a treatment option for sporadic cases of ALS

Scientific reports, 2024-03, Vol.14 (1), p.5929 [Peer Reviewed Journal]

2024. The Author(s). ;EISSN: 2045-2322 ;PMID: 38467696

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4
Cu II (atsm) improves the neurological phenotype and survival of SOD1 G93A mice and selectively increases enzymatically active SOD1 in the spinal cord
Material Type:
Article
Add to My Research

Cu II (atsm) improves the neurological phenotype and survival of SOD1 G93A mice and selectively increases enzymatically active SOD1 in the spinal cord

Scientific reports, 2017-02, Vol.7, p.42292 [Peer Reviewed Journal]

EISSN: 2045-2322 ;PMID: 28205575

Full text available

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