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Refined by: subject: Mitochondria remove
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1
Endolysosomal Targeting of Mitochondria Is Integral to BAX-Mediated Mitochondrial Permeabilization during Apoptosis Signaling
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Endolysosomal Targeting of Mitochondria Is Integral to BAX-Mediated Mitochondrial Permeabilization during Apoptosis Signaling

Developmental cell, 2020-06, Vol.53 (6), p.627-645.e7 [Peer Reviewed Journal]

2020 Elsevier Inc. ;Copyright © 2020 Elsevier Inc. All rights reserved. ;ISSN: 1534-5807 ;EISSN: 1878-1551 ;DOI: 10.1016/j.devcel.2020.05.014 ;PMID: 32504557

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2
Targeting the Mitochondrial Permeability Transition Pore to Prevent Age-Associated Cell Damage and Neurodegeneration
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Targeting the Mitochondrial Permeability Transition Pore to Prevent Age-Associated Cell Damage and Neurodegeneration

Oxidative medicine and cellular longevity, 2021, Vol.2021, p.6626484-15 [Peer Reviewed Journal]

Copyright © 2021 Andrew C. Kent et al. ;Copyright © 2021 Andrew C. Kent et al. This is an open access article distributed under the Creative Commons Attribution License (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. https://creativecommons.org/licenses/by/4.0 ;Copyright © 2021 Andrew C. Kent et al. 2021 ;ISSN: 1942-0900 ;EISSN: 1942-0994 ;DOI: 10.1155/2021/6626484 ;PMID: 33574977

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3
Reduction of elevated proton leak rejuvenates mitochondria in the aged cardiomyocyte
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Reduction of elevated proton leak rejuvenates mitochondria in the aged cardiomyocyte

eLife, 2020-12, Vol.9 [Peer Reviewed Journal]

2020, Zhang et al. ;COPYRIGHT 2020 eLife Science Publications, Ltd. ;2020, Zhang et al 2020 Zhang et al ;ISSN: 2050-084X ;EISSN: 2050-084X ;DOI: 10.7554/eLife.60827 ;PMID: 33319746

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4
MCUb Induction Protects the Heart From Postischemic Remodeling
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MCUb Induction Protects the Heart From Postischemic Remodeling

Circulation research, 2020-07, Vol.127 (3), p.379-390 [Peer Reviewed Journal]

2020 American Heart Association, Inc. ;ISSN: 0009-7330 ;EISSN: 1524-4571 ;DOI: 10.1161/CIRCRESAHA.119.316369 ;PMID: 32299299

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5
The RISK pathway leading to mitochondria and cardioprotection: how everything started
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The RISK pathway leading to mitochondria and cardioprotection: how everything started

Basic research in cardiology, 2023-05, Vol.118 (1), p.22-22, Article 22 [Peer Reviewed Journal]

The Author(s) 2023 ;2023. The Author(s). ;The Author(s) 2023. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;ISSN: 1435-1803 ;ISSN: 0300-8428 ;EISSN: 1435-1803 ;DOI: 10.1007/s00395-023-00992-5 ;PMID: 37233787

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6
TDP-43 Triggers Mitochondrial DNA Release via mPTP to Activate cGAS/STING in ALS
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TDP-43 Triggers Mitochondrial DNA Release via mPTP to Activate cGAS/STING in ALS

Cell, 2020-10, Vol.183 (3), p.636-649.e18 [Peer Reviewed Journal]

2020 The Author(s) ;Copyright © 2020 The Author(s). Published by Elsevier Inc. All rights reserved. ;2020 The Author(s) 2020 ;ISSN: 0092-8674 ;EISSN: 1097-4172 ;DOI: 10.1016/j.cell.2020.09.020 ;PMID: 33031745

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7
Promising Strategy of mPTP Modulation in Cancer Therapy: An Emerging Progress and Future Insight
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Promising Strategy of mPTP Modulation in Cancer Therapy: An Emerging Progress and Future Insight

International journal of molecular sciences, 2023-03, Vol.24 (6), p.5564 [Peer Reviewed Journal]

COPYRIGHT 2023 MDPI AG ;2023 by the authors. Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (https://creativecommons.org/licenses/by/4.0/). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2023 by the authors. 2023 ;ISSN: 1422-0067 ;ISSN: 1661-6596 ;EISSN: 1422-0067 ;DOI: 10.3390/ijms24065564 ;PMID: 36982637

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8
Genetic deletion of p66shc and/or cyclophilin D results in decreased pulmonary vascular tone
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Genetic deletion of p66shc and/or cyclophilin D results in decreased pulmonary vascular tone

Cardiovascular research, 2022-01, Vol.118 (1), p.305-315 [Peer Reviewed Journal]

The Author(s) 2020. Published by Oxford University Press on behalf of the European Society of Cardiology. 2020 ;The Author(s) 2020. Published by Oxford University Press on behalf of the European Society of Cardiology. ;ISSN: 0008-6363 ;EISSN: 1755-3245 ;DOI: 10.1093/cvr/cvaa310 ;PMID: 33119054

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9
Recent progress in the use of mitochondrial membrane permeability transition pore in mitochondrial dysfunction-related disease therapies
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Recent progress in the use of mitochondrial membrane permeability transition pore in mitochondrial dysfunction-related disease therapies

Molecular and cellular biochemistry, 2021-01, Vol.476 (1), p.493-506 [Peer Reviewed Journal]

Springer Science+Business Media, LLC, part of Springer Nature 2020 ;COPYRIGHT 2020 Springer ;Springer Science+Business Media, LLC, part of Springer Nature 2020. ;ISSN: 0300-8177 ;EISSN: 1573-4919 ;DOI: 10.1007/s11010-020-03926-0 ;PMID: 33000352

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10
Calpain-mediated protein targets in cardiac mitochondria following ischemia-reperfusion
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Calpain-mediated protein targets in cardiac mitochondria following ischemia-reperfusion

Scientific reports, 2022-01, Vol.12 (1), p.138-17, Article 138 [Peer Reviewed Journal]

2022. The Author(s). ;The Author(s) 2022. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2022 ;ISSN: 2045-2322 ;EISSN: 2045-2322 ;DOI: 10.1038/s41598-021-03947-9 ;PMID: 34997008

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11
Mitochondrial Calcium Deregulation in the Mechanism of Beta-Amyloid and Tau Pathology
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Mitochondrial Calcium Deregulation in the Mechanism of Beta-Amyloid and Tau Pathology

Cells (Basel, Switzerland), 2020-09, Vol.9 (9), p.2135 [Peer Reviewed Journal]

COPYRIGHT 2020 MDPI AG ;2020 by the authors. 2020 ;ISSN: 2073-4409 ;EISSN: 2073-4409 ;DOI: 10.3390/cells9092135 ;PMID: 32967303

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12
The mitochondrial NO‐synthase/guanylate cyclase/protein kinase G signaling system underpins the dual effects of nitric oxide on mitochondrial respiration and opening of the permeability transition pore
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The mitochondrial NO‐synthase/guanylate cyclase/protein kinase G signaling system underpins the dual effects of nitric oxide on mitochondrial respiration and opening of the permeability transition pore

The FEBS journal, 2020-04, Vol.287 (8), p.1525-1536 [Peer Reviewed Journal]

2019 Federation of European Biochemical Societies ;2019 Federation of European Biochemical Societies. ;Copyright © 2020 Federation of European Biochemical Societies ;ISSN: 1742-464X ;EISSN: 1742-4658 ;DOI: 10.1111/febs.15090 ;PMID: 31602795

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13
Peptides Targeting the IF1-ATP Synthase Complex Modulate the Permeability Transition Pore in Cancer HeLa Cells
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Peptides Targeting the IF1-ATP Synthase Complex Modulate the Permeability Transition Pore in Cancer HeLa Cells

International journal of molecular sciences, 2024-05, Vol.25 (9), p.4655 [Peer Reviewed Journal]

COPYRIGHT 2024 MDPI AG ;2024 by the authors. Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (https://creativecommons.org/licenses/by/4.0/). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;ISSN: 1422-0067 ;ISSN: 1661-6596 ;EISSN: 1422-0067 ;DOI: 10.3390/ijms25094655 ;PMID: 38731874

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14
A naturally occurring mutation in ATP synthase subunit c is associated with increased damage following hypoxia/reoxygenation in STEMI patients
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A naturally occurring mutation in ATP synthase subunit c is associated with increased damage following hypoxia/reoxygenation in STEMI patients

Cell reports (Cambridge), 2021-04, Vol.35 (2), p.108983-108983, Article 108983 [Peer Reviewed Journal]

2021 The Authors ;Copyright © 2021 The Authors. Published by Elsevier Inc. All rights reserved. ;ISSN: 2211-1247 ;EISSN: 2211-1247 ;DOI: 10.1016/j.celrep.2021.108983 ;PMID: 33852870

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15
Modulation and Pharmacology of the Mitochondrial Permeability Transition: A Journey from F-ATP Synthase to ANT
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Modulation and Pharmacology of the Mitochondrial Permeability Transition: A Journey from F-ATP Synthase to ANT

Molecules (Basel, Switzerland), 2021-10, Vol.26 (21), p.6463 [Peer Reviewed Journal]

2021 by the authors. Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (https://creativecommons.org/licenses/by/4.0/). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2021 by the authors. 2021 ;ISSN: 1420-3049 ;EISSN: 1420-3049 ;DOI: 10.3390/molecules26216463 ;PMID: 34770872

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16
Cryptotanshinone Ameliorates Doxorubicin-Induced Cardiotoxicity by Targeting Akt-GSK-3β-mPTP Pathway In Vitro
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Cryptotanshinone Ameliorates Doxorubicin-Induced Cardiotoxicity by Targeting Akt-GSK-3β-mPTP Pathway In Vitro

Molecules (Basel, Switzerland), 2021-03, Vol.26 (5), p.1460 [Peer Reviewed Journal]

2021. This work is licensed under http://creativecommons.org/licenses/by/3.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2021 by the authors. 2021 ;ISSN: 1420-3049 ;EISSN: 1420-3049 ;DOI: 10.3390/molecules26051460 ;PMID: 33800264

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17
Formation of High-Conductive C Subunit Channels upon Interaction with Cyclophilin D
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Formation of High-Conductive C Subunit Channels upon Interaction with Cyclophilin D

International journal of molecular sciences, 2021-10, Vol.22 (20), p.11022 [Peer Reviewed Journal]

2021 by the authors. Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (https://creativecommons.org/licenses/by/4.0/). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2021 by the authors. 2021 ;ISSN: 1422-0067 ;ISSN: 1661-6596 ;EISSN: 1422-0067 ;DOI: 10.3390/ijms222011022 ;PMID: 34681682

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18
Opening of mitochondrial permeability transition pore in cardiomyocytes: is ferutinin a suitable tool for its assessment?
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Opening of mitochondrial permeability transition pore in cardiomyocytes: is ferutinin a suitable tool for its assessment?

Fundamental & clinical pharmacology, 2023-08 [Peer Reviewed Journal]

Distributed under a Creative Commons Attribution 4.0 International License ;ISSN: 0767-3981 ;EISSN: 1472-8206 ;DOI: 10.1111/fcp.12879 ;PMID: 36797226

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19
Amorphous SiO2 nanoparticles promote cardiac dysfunction via the opening of the mitochondrial permeability transition pore in rat heart and human cardiomyocytes
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Amorphous SiO2 nanoparticles promote cardiac dysfunction via the opening of the mitochondrial permeability transition pore in rat heart and human cardiomyocytes

Particle and fibre toxicology, 2020-05, Vol.17 (1), p.15-16, Article 15 [Peer Reviewed Journal]

COPYRIGHT 2020 BioMed Central Ltd. ;2020. This work is licensed under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2020 ;ISSN: 1743-8977 ;EISSN: 1743-8977 ;DOI: 10.1186/s12989-020-00346-2 ;PMID: 32381100

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20
Cardioprotection of Immature Heart by Simultaneous Activation of PKA and Epac: A Role for the Mitochondrial Permeability Transition Pore
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Cardioprotection of Immature Heart by Simultaneous Activation of PKA and Epac: A Role for the Mitochondrial Permeability Transition Pore

International journal of molecular sciences, 2022-02, Vol.23 (3), p.1720 [Peer Reviewed Journal]

2022 by the authors. Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (https://creativecommons.org/licenses/by/4.0/). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2022 by the authors. 2022 ;ISSN: 1422-0067 ;ISSN: 1661-6596 ;EISSN: 1422-0067 ;DOI: 10.3390/ijms23031720 ;PMID: 35163640

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