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1
Constitutive Activity among Orphan Class-A G Protein Coupled Receptors
Constitutive Activity among Orphan Class-A G Protein Coupled Receptors
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Constitutive Activity among Orphan Class-A G Protein Coupled Receptors

PloS one, 2015-09, Vol.10 (9), p.e0138463-e0138463 [Peer Reviewed Journal]

COPYRIGHT 2015 Public Library of Science ;COPYRIGHT 2015 Public Library of Science ;2015 Martin et al. This is an open access article distributed under the terms of the Creative Commons Attribution License: http://creativecommons.org/licenses/by/4.0/ (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2015 Martin et al 2015 Martin et al ;ISSN: 1932-6203 ;EISSN: 1932-6203 ;DOI: 10.1371/journal.pone.0138463 ;PMID: 26384023

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2
Plant hormone jasmonate prioritizes defense over growth by interfering with gibberellin signaling cascade
Plant hormone jasmonate prioritizes defense over growth by interfering with gibberellin signaling cascade
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Plant hormone jasmonate prioritizes defense over growth by interfering with gibberellin signaling cascade

Proceedings of the National Academy of Sciences - PNAS, 2012-05, Vol.109 (19), p.E1192-E1200 [Peer Reviewed Journal]

Copyright National Academy of Sciences May 8, 2012 ;ISSN: 0027-8424 ;EISSN: 1091-6490 ;DOI: 10.1073/pnas.1201616109 ;PMID: 22529386

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3
Adenosine integrates light and sleep signalling for the regulation of circadian timing in mice
Adenosine integrates light and sleep signalling for the regulation of circadian timing in mice
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Adenosine integrates light and sleep signalling for the regulation of circadian timing in mice

Nature communications, 2021-04, Vol.12 (1), p.2113-11, Article 2113 [Peer Reviewed Journal]

The Author(s) 2021. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2021 ;ISSN: 2041-1723 ;EISSN: 2041-1723 ;DOI: 10.1038/s41467-021-22179-z ;PMID: 33837202

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4
Rare cell variability and drug-induced reprogramming as a mode of cancer drug resistance
Rare cell variability and drug-induced reprogramming as a mode of cancer drug resistance
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Rare cell variability and drug-induced reprogramming as a mode of cancer drug resistance

Nature (London), 2017-06, Vol.546 (7658), p.431-435 [Peer Reviewed Journal]

COPYRIGHT 2017 Nature Publishing Group ;COPYRIGHT 2017 Nature Publishing Group ;Copyright Nature Publishing Group Jun 15, 2017 ;ISSN: 0028-0836 ;EISSN: 1476-4687 ;DOI: 10.1038/nature22794 ;PMID: 28607484

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5
Identification of EMT signaling cross-talk and gene regulatory networks by single-cell RNA sequencing
Identification of EMT signaling cross-talk and gene regulatory networks by single-cell RNA sequencing
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Identification of EMT signaling cross-talk and gene regulatory networks by single-cell RNA sequencing

Proceedings of the National Academy of Sciences - PNAS, 2021-05, Vol.118 (19), p.1 [Peer Reviewed Journal]

Copyright National Academy of Sciences May 11, 2021 ;Copyright © 2021 the Author(s). Published by PNAS. 2021 ;ISSN: 0027-8424 ;EISSN: 1091-6490 ;DOI: 10.1073/pnas.2102050118 ;PMID: 33941680

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6
Tumour and host cell PD-L1 is required to mediate suppression of anti-tumour immunity in mice
Tumour and host cell PD-L1 is required to mediate suppression of anti-tumour immunity in mice
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Tumour and host cell PD-L1 is required to mediate suppression of anti-tumour immunity in mice

Nature communications, 2017-02, Vol.8 (1), p.14572-14572, Article 14572 [Peer Reviewed Journal]

Copyright Nature Publishing Group Feb 2017 ;Copyright © 2017, The Author(s) 2017 The Author(s) ;ISSN: 2041-1723 ;EISSN: 2041-1723 ;DOI: 10.1038/ncomms14572 ;PMID: 28220772

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7
NOD1 and NOD2 signalling links ER stress with inflammation
NOD1 and NOD2 signalling links ER stress with inflammation
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NOD1 and NOD2 signalling links ER stress with inflammation

Nature (London), 2016-04, Vol.532 (7599), p.394-397 [Peer Reviewed Journal]

COPYRIGHT 2016 Nature Publishing Group ;COPYRIGHT 2016 Nature Publishing Group ;Copyright Nature Publishing Group Apr 21, 2016 ;ISSN: 0028-0836 ;EISSN: 1476-4687 ;DOI: 10.1038/nature17631 ;PMID: 27007849 ;CODEN: NATUAS

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8
T-cell dysfunction in the glioblastoma microenvironment is mediated by myeloid cells releasing interleukin-10
T-cell dysfunction in the glioblastoma microenvironment is mediated by myeloid cells releasing interleukin-10
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T-cell dysfunction in the glioblastoma microenvironment is mediated by myeloid cells releasing interleukin-10

Nature communications, 2022-02, Vol.13 (1), p.925-925, Article 925 [Peer Reviewed Journal]

2022. The Author(s). ;The Author(s) 2022. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2022 ;ISSN: 2041-1723 ;EISSN: 2041-1723 ;DOI: 10.1038/s41467-022-28523-1 ;PMID: 35177622

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9
Butyrate modifies intestinal barrier function in IPEC-J2 cells through a selective upregulation of tight junction proteins and activation of the Akt signaling pathway
Butyrate modifies intestinal barrier function in IPEC-J2 cells through a selective upregulation of tight junction proteins and activation of the Akt signaling pathway
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Butyrate modifies intestinal barrier function in IPEC-J2 cells through a selective upregulation of tight junction proteins and activation of the Akt signaling pathway

PloS one, 2017-06, Vol.12 (6), p.e0179586-e0179586 [Peer Reviewed Journal]

COPYRIGHT 2017 Public Library of Science ;COPYRIGHT 2017 Public Library of Science ;2017 Yan, Ajuwon. This is an open access article distributed under the terms of the Creative Commons Attribution License: http://creativecommons.org/licenses/by/4.0/ (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2017 Yan, Ajuwon 2017 Yan, Ajuwon ;ISSN: 1932-6203 ;EISSN: 1932-6203 ;DOI: 10.1371/journal.pone.0179586 ;PMID: 28654658

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10
A multiprotein supercomplex controlling oncogenic signalling in lymphoma
A multiprotein supercomplex controlling oncogenic signalling in lymphoma
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A multiprotein supercomplex controlling oncogenic signalling in lymphoma

Nature (London), 2018-08, Vol.560 (7718), p.387-391 [Peer Reviewed Journal]

COPYRIGHT 2018 Nature Publishing Group ;Copyright Nature Publishing Group Aug 16, 2018 ;Distributed under a Creative Commons Attribution 4.0 International License ;ISSN: 0028-0836 ;EISSN: 1476-4687 ;DOI: 10.1038/s41586-018-0290-0 ;PMID: 29925955

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11
Early immune responses are independent of RGC dysfunction in glaucoma with complement component C3 being protective
Early immune responses are independent of RGC dysfunction in glaucoma with complement component C3 being protective
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Early immune responses are independent of RGC dysfunction in glaucoma with complement component C3 being protective

Proceedings of the National Academy of Sciences - PNAS, 2017-05, Vol.114 (19), p.E3839-E3848 [Peer Reviewed Journal]

Volumes 1–89 and 106–114, copyright as a collective work only; author(s) retains copyright to individual articles ;Copyright National Academy of Sciences May 9, 2017 ;ISSN: 0027-8424 ;EISSN: 1091-6490 ;DOI: 10.1073/pnas.1608769114 ;PMID: 28446616

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12
Species-specific disruption of STING-dependent antiviral cellular defenses by the Zika virus NS2B3 protease
Species-specific disruption of STING-dependent antiviral cellular defenses by the Zika virus NS2B3 protease
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Species-specific disruption of STING-dependent antiviral cellular defenses by the Zika virus NS2B3 protease

Proceedings of the National Academy of Sciences - PNAS, 2018-07, Vol.115 (27), p.E6310-E6318 [Peer Reviewed Journal]

Volumes 1–89 and 106–115, copyright as a collective work only; author(s) retains copyright to individual articles ;Copyright National Academy of Sciences Jul 3, 2018 ;2018 ;ISSN: 0027-8424 ;EISSN: 1091-6490 ;DOI: 10.1073/pnas.1803406115 ;PMID: 29915078

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13
ssGSEA score-based Ras dependency indexes derived from gene expression data reveal potential Ras addiction mechanisms with possible clinical implications
ssGSEA score-based Ras dependency indexes derived from gene expression data reveal potential Ras addiction mechanisms with possible clinical implications
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ssGSEA score-based Ras dependency indexes derived from gene expression data reveal potential Ras addiction mechanisms with possible clinical implications

Scientific reports, 2020-06, Vol.10 (1), p.10258, Article 10258 [Peer Reviewed Journal]

The Author(s) 2020. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2020 ;ISSN: 2045-2322 ;EISSN: 2045-2322 ;DOI: 10.1038/s41598-020-66986-8 ;PMID: 32581224

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14
GLUTAMATE RECEPTOR-LIKE genes mediate leaf-to-leaf wound signalling
GLUTAMATE RECEPTOR-LIKE genes mediate leaf-to-leaf wound signalling
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GLUTAMATE RECEPTOR-LIKE genes mediate leaf-to-leaf wound signalling

Nature (London), 2013-08, Vol.500 (7463), p.422-426 [Peer Reviewed Journal]

COPYRIGHT 2013 Nature Publishing Group ;Copyright Nature Publishing Group Aug 22, 2013 ;ISSN: 0028-0836 ;EISSN: 1476-4687 ;DOI: 10.1038/nature12478 ;PMID: 23969459 ;CODEN: NATUAS

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15
Directed self-assembly of herbal small molecules into sustained release hydrogels for treating neural inflammation
Directed self-assembly of herbal small molecules into sustained release hydrogels for treating neural inflammation
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Directed self-assembly of herbal small molecules into sustained release hydrogels for treating neural inflammation

Nature communications, 2019-04, Vol.10 (1), p.1604-1604, Article 1604 [Peer Reviewed Journal]

The Author(s) 2019. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2019 ;ISSN: 2041-1723 ;EISSN: 2041-1723 ;DOI: 10.1038/s41467-019-09601-3 ;PMID: 30962431

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16
Adipocyte Piezo1 mediates obesogenic adipogenesis through the FGF1/FGFR1 signaling pathway in mice
Adipocyte Piezo1 mediates obesogenic adipogenesis through the FGF1/FGFR1 signaling pathway in mice
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Adipocyte Piezo1 mediates obesogenic adipogenesis through the FGF1/FGFR1 signaling pathway in mice

Nature communications, 2020-05, Vol.11 (1), p.2303-2303, Article 2303 [Peer Reviewed Journal]

The Author(s) 2020. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2020. corrected publication 2022. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;Distributed under a Creative Commons Attribution 4.0 International License ;The Author(s) 2020, corrected publication 2022 ;ISSN: 2041-1723 ;EISSN: 2041-1723 ;DOI: 10.1038/s41467-020-16026-w ;PMID: 32385276

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17
Inference and analysis of cell-cell communication using CellChat
Inference and analysis of cell-cell communication using CellChat
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Inference and analysis of cell-cell communication using CellChat

Nature communications, 2021-02, Vol.12 (1), p.1088-1088, Article 1088 [Peer Reviewed Journal]

The Author(s) 2021. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2021 ;ISSN: 2041-1723 ;EISSN: 2041-1723 ;DOI: 10.1038/s41467-021-21246-9 ;PMID: 33597522

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18
Signal amplification and transduction in phytochrome photosensors
Signal amplification and transduction in phytochrome photosensors
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Signal amplification and transduction in phytochrome photosensors

Nature (London), 2014-05, Vol.509 (7499), p.245-248 [Peer Reviewed Journal]

COPYRIGHT 2014 Nature Publishing Group ;COPYRIGHT 2014 Nature Publishing Group ;Copyright Nature Publishing Group May 8, 2014 ;ISSN: 0028-0836 ;EISSN: 1476-4687 ;DOI: 10.1038/nature13310 ;PMID: 24776794 ;CODEN: NATUAS

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19
A LATS biosensor screen identifies VEGFR as a regulator of the Hippo pathway in angiogenesis
A LATS biosensor screen identifies VEGFR as a regulator of the Hippo pathway in angiogenesis
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A LATS biosensor screen identifies VEGFR as a regulator of the Hippo pathway in angiogenesis

Nature communications, 2018-03, Vol.9 (1), p.1061-15, Article 1061 [Peer Reviewed Journal]

2018. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2018 ;ISSN: 2041-1723 ;EISSN: 2041-1723 ;DOI: 10.1038/s41467-018-03278-w ;PMID: 29535383

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20
Epicatechin modulates stress-resistance in C. elegans via insulin/IGF-1 signaling pathway
Epicatechin modulates stress-resistance in C. elegans via insulin/IGF-1 signaling pathway
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Epicatechin modulates stress-resistance in C. elegans via insulin/IGF-1 signaling pathway

PloS one, 2019-01, Vol.14 (1), p.e0199483-e0199483 [Peer Reviewed Journal]

COPYRIGHT 2019 Public Library of Science ;COPYRIGHT 2019 Public Library of Science ;2019 Ayuda-Durán et al. This is an open access article distributed under the terms of the Creative Commons Attribution License: http://creativecommons.org/licenses/by/4.0/ (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2019 Ayuda-Durán et al 2019 Ayuda-Durán et al ;ISSN: 1932-6203 ;EISSN: 1932-6203 ;DOI: 10.1371/journal.pone.0199483 ;PMID: 30689636

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