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1
Rare cell variability and drug-induced reprogramming as a mode of cancer drug resistance
Rare cell variability and drug-induced reprogramming as a mode of cancer drug resistance
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Rare cell variability and drug-induced reprogramming as a mode of cancer drug resistance

Nature (London), 2017-06, Vol.546 (7658), p.431-435 [Peer Reviewed Journal]

COPYRIGHT 2017 Nature Publishing Group ;COPYRIGHT 2017 Nature Publishing Group ;Copyright Nature Publishing Group Jun 15, 2017 ;ISSN: 0028-0836 ;EISSN: 1476-4687 ;DOI: 10.1038/nature22794 ;PMID: 28607484

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2
Roles of the Raf/MEK/ERK and PI3K/PTEN/Akt/mTOR pathways in controlling growth and sensitivity to therapy-implications for cancer and aging
Roles of the Raf/MEK/ERK and PI3K/PTEN/Akt/mTOR pathways in controlling growth and sensitivity to therapy-implications for cancer and aging
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Roles of the Raf/MEK/ERK and PI3K/PTEN/Akt/mTOR pathways in controlling growth and sensitivity to therapy-implications for cancer and aging

Aging (Albany, NY.), 2011-03, Vol.3 (3), p.192-222

Copyright: © 2011 Steelman et al. 2011 ;ISSN: 1945-4589 ;EISSN: 1945-4589 ;DOI: 10.18632/aging.100296 ;PMID: 21422497

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3
The Notch signalling system: recent insights into the complexity of a conserved pathway
The Notch signalling system: recent insights into the complexity of a conserved pathway
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The Notch signalling system: recent insights into the complexity of a conserved pathway

Nature reviews. Genetics, 2012-09, Vol.13 (9), p.654-666 [Peer Reviewed Journal]

2015 INIST-CNRS ;COPYRIGHT 2012 Nature Publishing Group ;COPYRIGHT 2012 Nature Publishing Group ;Copyright Nature Publishing Group Sep 2012 ;2012 Macmillan Publishers Limited. All rights reserved 2012 ;ISSN: 1471-0056 ;EISSN: 1471-0064 ;DOI: 10.1038/nrg3272 ;PMID: 22868267

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4
Stress, sex hormones, inflammation, and major depressive disorder: Extending Social Signal Transduction Theory of Depression to account for sex differences in mood disorders
Stress, sex hormones, inflammation, and major depressive disorder: Extending Social Signal Transduction Theory of Depression to account for sex differences in mood disorders
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Stress, sex hormones, inflammation, and major depressive disorder: Extending Social Signal Transduction Theory of Depression to account for sex differences in mood disorders

Psychopharmacology, 2019-10, Vol.236 (10), p.3063-3079 [Peer Reviewed Journal]

Springer-Verlag GmbH Germany, part of Springer Nature 2019 ;COPYRIGHT 2019 Springer ;Psychopharmacology is a copyright of Springer, (2019). All Rights Reserved. ;ISSN: 0033-3158 ;EISSN: 1432-2072 ;DOI: 10.1007/s00213-019-05326-9 ;PMID: 31359117

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5
NOD1 and NOD2 signalling links ER stress with inflammation
NOD1 and NOD2 signalling links ER stress with inflammation
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NOD1 and NOD2 signalling links ER stress with inflammation

Nature (London), 2016-04, Vol.532 (7599), p.394-397 [Peer Reviewed Journal]

COPYRIGHT 2016 Nature Publishing Group ;COPYRIGHT 2016 Nature Publishing Group ;Copyright Nature Publishing Group Apr 21, 2016 ;ISSN: 0028-0836 ;EISSN: 1476-4687 ;DOI: 10.1038/nature17631 ;PMID: 27007849 ;CODEN: NATUAS

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6
The role of hypoxia-inducible factors in metabolic diseases
The role of hypoxia-inducible factors in metabolic diseases
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The role of hypoxia-inducible factors in metabolic diseases

Nature reviews. Endocrinology, 2019-01, Vol.15 (1), p.21-32 [Peer Reviewed Journal]

COPYRIGHT 2019 Nature Publishing Group ;Copyright Nature Publishing Group Jan 2019 ;ISSN: 1759-5029 ;EISSN: 1759-5037 ;DOI: 10.1038/s41574-018-0096-z ;PMID: 30275460

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7
T-cell dysfunction in the glioblastoma microenvironment is mediated by myeloid cells releasing interleukin-10
T-cell dysfunction in the glioblastoma microenvironment is mediated by myeloid cells releasing interleukin-10
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T-cell dysfunction in the glioblastoma microenvironment is mediated by myeloid cells releasing interleukin-10

Nature communications, 2022-02, Vol.13 (1), p.925-925, Article 925 [Peer Reviewed Journal]

2022. The Author(s). ;The Author(s) 2022. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2022 ;ISSN: 2041-1723 ;EISSN: 2041-1723 ;DOI: 10.1038/s41467-022-28523-1 ;PMID: 35177622

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8
A multiprotein supercomplex controlling oncogenic signalling in lymphoma
A multiprotein supercomplex controlling oncogenic signalling in lymphoma
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A multiprotein supercomplex controlling oncogenic signalling in lymphoma

Nature (London), 2018-08, Vol.560 (7718), p.387-391 [Peer Reviewed Journal]

COPYRIGHT 2018 Nature Publishing Group ;Copyright Nature Publishing Group Aug 16, 2018 ;Distributed under a Creative Commons Attribution 4.0 International License ;ISSN: 0028-0836 ;EISSN: 1476-4687 ;DOI: 10.1038/s41586-018-0290-0 ;PMID: 29925955

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9
Deconvoluting global cytokine signaling networks in natural killer cells
Deconvoluting global cytokine signaling networks in natural killer cells
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Deconvoluting global cytokine signaling networks in natural killer cells

Nature immunology, 2021-05, Vol.22 (5), p.627-638 [Peer Reviewed Journal]

COPYRIGHT 2021 Nature Publishing Group ;The Author(s), under exclusive licence to Springer Nature America, Inc. 2021. ;ISSN: 1529-2908 ;EISSN: 1529-2916 ;DOI: 10.1038/s41590-021-00909-1 ;PMID: 33859404

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10
Direct migration of follicular melanocyte stem cells to the epidermis after wounding or UVB irradiation is dependent on Mc1r signaling
Direct migration of follicular melanocyte stem cells to the epidermis after wounding or UVB irradiation is dependent on Mc1r signaling
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Direct migration of follicular melanocyte stem cells to the epidermis after wounding or UVB irradiation is dependent on Mc1r signaling

Nature medicine, 2013-07, Vol.19 (7), p.924-929 [Peer Reviewed Journal]

COPYRIGHT 2013 Nature Publishing Group ;COPYRIGHT 2013 Nature Publishing Group ;Copyright Nature Publishing Group Jul 2013 ;ISSN: 1078-8956 ;EISSN: 1546-170X ;DOI: 10.1038/nm.3194 ;PMID: 23749232

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11
Astrocytes control synaptic strength by two distinct v-SNARE-dependent release pathways
Astrocytes control synaptic strength by two distinct v-SNARE-dependent release pathways
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Astrocytes control synaptic strength by two distinct v-SNARE-dependent release pathways

Nature neuroscience, 2017-11, Vol.20 (11), p.1529-1539 [Peer Reviewed Journal]

COPYRIGHT 2017 Nature Publishing Group ;COPYRIGHT 2017 Nature Publishing Group ;Copyright Nature Publishing Group Nov 2017 ;ISSN: 1097-6256 ;EISSN: 1546-1726 ;DOI: 10.1038/nn.4647 ;PMID: 28945220

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12
Transcriptome networks identify mechanisms of viral and nonviral asthma exacerbations in children
Transcriptome networks identify mechanisms of viral and nonviral asthma exacerbations in children
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Transcriptome networks identify mechanisms of viral and nonviral asthma exacerbations in children

Nature immunology, 2019-05, Vol.20 (5), p.637-651 [Peer Reviewed Journal]

COPYRIGHT 2019 Nature Publishing Group ;COPYRIGHT 2019 Nature Publishing Group ;The Author(s), under exclusive licence to Springer Nature America, Inc. 2019. ;The Author(s), under exclusive licence to Springer Nature America, Inc. 2019 ;ISSN: 1529-2908 ;EISSN: 1529-2916 ;DOI: 10.1038/s41590-019-0347-8 ;PMID: 30962590

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13
Adipocyte Piezo1 mediates obesogenic adipogenesis through the FGF1/FGFR1 signaling pathway in mice
Adipocyte Piezo1 mediates obesogenic adipogenesis through the FGF1/FGFR1 signaling pathway in mice
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Adipocyte Piezo1 mediates obesogenic adipogenesis through the FGF1/FGFR1 signaling pathway in mice

Nature communications, 2020-05, Vol.11 (1), p.2303-2303, Article 2303 [Peer Reviewed Journal]

The Author(s) 2020. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2020. corrected publication 2022. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;Distributed under a Creative Commons Attribution 4.0 International License ;The Author(s) 2020, corrected publication 2022 ;ISSN: 2041-1723 ;EISSN: 2041-1723 ;DOI: 10.1038/s41467-020-16026-w ;PMID: 32385276

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14
A LATS biosensor screen identifies VEGFR as a regulator of the Hippo pathway in angiogenesis
A LATS biosensor screen identifies VEGFR as a regulator of the Hippo pathway in angiogenesis
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A LATS biosensor screen identifies VEGFR as a regulator of the Hippo pathway in angiogenesis

Nature communications, 2018-03, Vol.9 (1), p.1061-15, Article 1061 [Peer Reviewed Journal]

2018. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2018 ;ISSN: 2041-1723 ;EISSN: 2041-1723 ;DOI: 10.1038/s41467-018-03278-w ;PMID: 29535383

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15
B cell receptor isotypes differentially associate with cell signaling, kinetics, and outcome in chronic lymphocytic leukemia
B cell receptor isotypes differentially associate with cell signaling, kinetics, and outcome in chronic lymphocytic leukemia
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B cell receptor isotypes differentially associate with cell signaling, kinetics, and outcome in chronic lymphocytic leukemia

The Journal of clinical investigation, 2022-01, Vol.132 (2), p.0_1-14 [Peer Reviewed Journal]

COPYRIGHT 2022 American Society for Clinical Investigation ;Copyright American Society for Clinical Investigation Jan 2022 ;2022 Mazzarello et al. 2022 Mazzarello et al. ;ISSN: 1558-8238 ;ISSN: 0021-9738 ;EISSN: 1558-8238 ;DOI: 10.1172/JCI149308 ;PMID: 34813501

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16
MNK2 governs the macrophage antiinflammatory phenotype
MNK2 governs the macrophage antiinflammatory phenotype
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MNK2 governs the macrophage antiinflammatory phenotype

Proceedings of the National Academy of Sciences - PNAS, 2020-11, Vol.117 (44), p.27556-27565 [Peer Reviewed Journal]

Copyright National Academy of Sciences Nov 3, 2020 ;2020 ;ISSN: 0027-8424 ;ISSN: 1091-6490 ;EISSN: 1091-6490 ;DOI: 10.1073/pnas.1920377117 ;PMID: 33077599

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17
Lung-resident tissue macrophages generate Foxp3+ regulatory T cells and promote airway tolerance
Lung-resident tissue macrophages generate Foxp3+ regulatory T cells and promote airway tolerance
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Lung-resident tissue macrophages generate Foxp3+ regulatory T cells and promote airway tolerance

The Journal of experimental medicine, 2013-04, Vol.210 (4), p.775-788 [Peer Reviewed Journal]

2013 Soroosh et al. 2013 ;ISSN: 0022-1007 ;EISSN: 1540-9538 ;DOI: 10.1084/jem.20121849 ;PMID: 23547101

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18
Layered reward signalling through octopamine and dopamine in Drosophila
Layered reward signalling through octopamine and dopamine in Drosophila
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Layered reward signalling through octopamine and dopamine in Drosophila

Nature (London), 2012-12, Vol.492 (7429), p.433-437 [Peer Reviewed Journal]

2014 INIST-CNRS ;COPYRIGHT 2012 Nature Publishing Group ;COPYRIGHT 2012 Nature Publishing Group ;Copyright Nature Publishing Group Dec 20-Dec 27, 2012 ;ISSN: 0028-0836 ;EISSN: 1476-4687 ;DOI: 10.1038/nature11614 ;PMID: 23103875 ;CODEN: NATUAS

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19
Plasmalemma vesicle-associated protein promotes angiogenesis in cholangiocarcinoma via the DKK1/CKAP4/PI3K signaling pathway
Plasmalemma vesicle-associated protein promotes angiogenesis in cholangiocarcinoma via the DKK1/CKAP4/PI3K signaling pathway
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Plasmalemma vesicle-associated protein promotes angiogenesis in cholangiocarcinoma via the DKK1/CKAP4/PI3K signaling pathway

Oncogene, 2021-06, Vol.40 (25), p.4324-4337 [Peer Reviewed Journal]

COPYRIGHT 2021 Nature Publishing Group ;The Author(s), under exclusive licence to Springer Nature Limited 2021. ;ISSN: 0950-9232 ;EISSN: 1476-5594 ;DOI: 10.1038/s41388-021-01844-z ;PMID: 34079085

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20
Nanog signaling in cancer promotes stem-like phenotype and immune evasion
Nanog signaling in cancer promotes stem-like phenotype and immune evasion
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Article 		Add to My Research

Nanog signaling in cancer promotes stem-like phenotype and immune evasion

The Journal of clinical investigation, 2012-11, Vol.122 (11), p.4077-4093 [Peer Reviewed Journal]

COPYRIGHT 2012 American Society for Clinical Investigation ;Copyright American Society for Clinical Investigation Nov 2012 ;Copyright © 2012, American Society for Clinical Investigation 2012 ;ISSN: 0021-9738 ;EISSN: 1558-8238 ;DOI: 10.1172/jci64057 ;PMID: 23093782

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