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1
Active MLKL triggers the NLRP3 inflammasome in a cell-intrinsic manner
Active MLKL triggers the NLRP3 inflammasome in a cell-intrinsic manner
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Active MLKL triggers the NLRP3 inflammasome in a cell-intrinsic manner

Proceedings of the National Academy of Sciences - PNAS, 2017-02, Vol.114 (6), p.E961-E969 [Peer Reviewed Journal]

Volumes 1–89 and 106–114, copyright as a collective work only; author(s) retains copyright to individual articles ;Copyright National Academy of Sciences Feb 7, 2017 ;ISSN: 0027-8424 ;EISSN: 1091-6490 ;DOI: 10.1073/pnas.1613305114 ;PMID: 28096356

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2
NLRP1 Inflammasome Activation Induces Pyroptosis of Hematopoietic Progenitor Cells
NLRP1 Inflammasome Activation Induces Pyroptosis of Hematopoietic Progenitor Cells
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NLRP1 Inflammasome Activation Induces Pyroptosis of Hematopoietic Progenitor Cells

Immunity (Cambridge, Mass.), 2012-12, Vol.37 (6), p.1009-1023 [Peer Reviewed Journal]

2012 Elsevier Inc. ;Copyright © 2012 Elsevier Inc. All rights reserved. ;Copyright Elsevier Limited Dec 14, 2012 ;2012 Elsevier Inc. All rights reserved. 2012 ;ISSN: 1074-7613 ;EISSN: 1097-4180 ;DOI: 10.1016/j.immuni.2012.08.027 ;PMID: 23219391

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3
Myeloid-derived miR-223 regulates intestinal inflammation via repression of the NLRP3 inflammasome
Myeloid-derived miR-223 regulates intestinal inflammation via repression of the NLRP3 inflammasome
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Myeloid-derived miR-223 regulates intestinal inflammation via repression of the NLRP3 inflammasome

The Journal of experimental medicine, 2017-06, Vol.214 (6), p.1737-1752 [Peer Reviewed Journal]

2017 Neudecker et al. ;Copyright Rockefeller University Press Jun 5, 2017 ;2017 Neudecker et al. 2017 ;ISSN: 0022-1007 ;EISSN: 1540-9538 ;DOI: 10.1084/jem.20160462 ;PMID: 28487310

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4
Human DPP9 represses NLRP1 inflammasome and protects against autoinflammatory diseases via both peptidase activity and FIIND domain binding
Human DPP9 represses NLRP1 inflammasome and protects against autoinflammatory diseases via both peptidase activity and FIIND domain binding
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Human DPP9 represses NLRP1 inflammasome and protects against autoinflammatory diseases via both peptidase activity and FIIND domain binding

The Journal of biological chemistry, 2018-12, Vol.293 (49), p.18864-18878 [Peer Reviewed Journal]

2018 © 2018 Zhong et al. ;2018 Zhong et al. ;2018 Zhong et al. 2018 Zhong et al. ;ISSN: 0021-9258 ;EISSN: 1083-351X ;DOI: 10.1074/jbc.RA118.004350 ;PMID: 30291141

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5
Germline NLRP1 Mutations Cause Skin Inflammatory and Cancer Susceptibility Syndromes via Inflammasome Activation
Germline NLRP1 Mutations Cause Skin Inflammatory and Cancer Susceptibility Syndromes via Inflammasome Activation
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Germline NLRP1 Mutations Cause Skin Inflammatory and Cancer Susceptibility Syndromes via Inflammasome Activation

Cell, 2016-09, Vol.167 (1), p.187-202.e17 [Peer Reviewed Journal]

2016 Elsevier Inc. ;Copyright © 2016 Elsevier Inc. All rights reserved. ;Distributed under a Creative Commons Attribution 4.0 International License ;ISSN: 0092-8674 ;EISSN: 1097-4172 ;DOI: 10.1016/j.cell.2016.09.001 ;PMID: 27662089

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6
RIPK3 promotes cell death and NLRP3 inflammasome activation in the absence of MLKL
RIPK3 promotes cell death and NLRP3 inflammasome activation in the absence of MLKL
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RIPK3 promotes cell death and NLRP3 inflammasome activation in the absence of MLKL

Nature communications, 2015-02, Vol.6 (1), p.6282-6282, Article 6282 [Peer Reviewed Journal]

Copyright Nature Publishing Group Feb 2015 ;Copyright © 2015, Nature Publishing Group, a division of Macmillan Publishers Limited. All Rights Reserved. 2015 Nature Publishing Group, a division of Macmillan Publishers Limited. All Rights Reserved. ;ISSN: 2041-1723 ;EISSN: 2041-1723 ;DOI: 10.1038/ncomms7282 ;PMID: 25693118

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7
Adipose Tissue Macrophages Promote Myelopoiesis and Monocytosis in Obesity
Adipose Tissue Macrophages Promote Myelopoiesis and Monocytosis in Obesity
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Adipose Tissue Macrophages Promote Myelopoiesis and Monocytosis in Obesity

Cell metabolism, 2014-05, Vol.19 (5), p.821-835 [Peer Reviewed Journal]

2014 Elsevier Inc. ;Copyright © 2014 Elsevier Inc. All rights reserved. ;2014 Elsevier Inc. All rights reserved. 2014 ;ISSN: 1550-4131 ;EISSN: 1932-7420 ;DOI: 10.1016/j.cmet.2014.03.029 ;PMID: 24807222

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8
IL-18 Production from the NLRP1 Inflammasome Prevents Obesity and Metabolic Syndrome
IL-18 Production from the NLRP1 Inflammasome Prevents Obesity and Metabolic Syndrome
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IL-18 Production from the NLRP1 Inflammasome Prevents Obesity and Metabolic Syndrome

Cell metabolism, 2016-01, Vol.23 (1), p.155-164 [Peer Reviewed Journal]

2016 Elsevier Inc. ;Copyright © 2016 Elsevier Inc. All rights reserved. ;ISSN: 1550-4131 ;EISSN: 1932-7420 ;DOI: 10.1016/j.cmet.2015.09.024 ;PMID: 26603191

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9
The RNA-binding protein Tristetraprolin (TTP) is a critical negative regulator of the NLRP3 inflammasome
The RNA-binding protein Tristetraprolin (TTP) is a critical negative regulator of the NLRP3 inflammasome
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The RNA-binding protein Tristetraprolin (TTP) is a critical negative regulator of the NLRP3 inflammasome

The Journal of biological chemistry, 2017-04, Vol.292 (17), p.6869-6881 [Peer Reviewed Journal]

2017 © 2017 ASBMB. Currently published by Elsevier Inc; originally published by American Society for Biochemistry and Molecular Biology. ;2017 by The American Society for Biochemistry and Molecular Biology, Inc. ;2017 by The American Society for Biochemistry and Molecular Biology, Inc. 2017 The American Society for Biochemistry and Molecular Biology, Inc. ;ISSN: 0021-9258 ;EISSN: 1083-351X ;DOI: 10.1074/jbc.M116.772947 ;PMID: 28302726

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10
Transcriptional analysis of the three Nlrp1 paralogs in mice
Transcriptional analysis of the three Nlrp1 paralogs in mice
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Transcriptional analysis of the three Nlrp1 paralogs in mice

BMC genomics, 2013-03, Vol.14 (1), p.188-188 [Peer Reviewed Journal]

COPYRIGHT 2013 BioMed Central Ltd. ;COPYRIGHT 2013 BioMed Central Ltd. ;2013 Sastalla et al.; licensee BioMed Central Ltd. This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/2.0), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited. ;Copyright © 2013 Sastalla et al.; licensee BioMed Central Ltd. 2013 Sastalla et al.; licensee BioMed Central Ltd. ;ISSN: 1471-2164 ;EISSN: 1471-2164 ;DOI: 10.1186/1471-2164-14-188 ;PMID: 23506131

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11
The Mitochondrial Apoptotic Effectors BAX/BAK Activate Caspase-3 and -7 to Trigger NLRP3 Inflammasome and Caspase-8 Driven IL-1β Activation
The Mitochondrial Apoptotic Effectors BAX/BAK Activate Caspase-3 and -7 to Trigger NLRP3 Inflammasome and Caspase-8 Driven IL-1β Activation
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The Mitochondrial Apoptotic Effectors BAX/BAK Activate Caspase-3 and -7 to Trigger NLRP3 Inflammasome and Caspase-8 Driven IL-1β Activation

Cell reports (Cambridge), 2018-11, Vol.25 (9), p.2339-2353.e4 [Peer Reviewed Journal]

2018 The Authors ;Copyright © 2018 The Authors. Published by Elsevier Inc. All rights reserved. ;ISSN: 2211-1247 ;EISSN: 2211-1247 ;DOI: 10.1016/j.celrep.2018.10.103 ;PMID: 30485804

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12
Inflammasome sensor NLRP1 disease variant M1184V promotes autoproteolysis and DPP9 complex formation by stabilizing the FIIND domain
Inflammasome sensor NLRP1 disease variant M1184V promotes autoproteolysis and DPP9 complex formation by stabilizing the FIIND domain
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Inflammasome sensor NLRP1 disease variant M1184V promotes autoproteolysis and DPP9 complex formation by stabilizing the FIIND domain

The Journal of biological chemistry, 2022-12, Vol.298 (12), p.102645-102645, Article 102645 [Peer Reviewed Journal]

2022 The Authors ;Copyright © 2022 The Authors. Published by Elsevier Inc. All rights reserved. ;2022 The Authors 2022 ;ISSN: 0021-9258 ;EISSN: 1083-351X ;DOI: 10.1016/j.jbc.2022.102645 ;PMID: 36309085

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13
Membrane vesicles from Pseudomonas aeruginosa activate the noncanonical inflammasome through caspase‐5 in human monocytes
Membrane vesicles from Pseudomonas aeruginosa activate the noncanonical inflammasome through caspase‐5 in human monocytes
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Membrane vesicles from Pseudomonas aeruginosa activate the noncanonical inflammasome through caspase‐5 in human monocytes

Immunology and cell biology, 2018-11, Vol.96 (10), p.1120-1130 [Peer Reviewed Journal]

2018 Australasian Society for Immunology Inc. ;Copyright Blackwell Science Ltd. Nov/Dec 2018 ;ISSN: 0818-9641 ;EISSN: 1440-1711 ;DOI: 10.1111/imcb.12190 ;PMID: 30003588

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14
Caspase‐8‐driven apoptotic and pyroptotic crosstalk causes cell death and IL‐1β release in X‐linked inhibitor of apoptosis (XIAP) deficiency
Caspase‐8‐driven apoptotic and pyroptotic crosstalk causes cell death and IL‐1β release in X‐linked inhibitor of apoptosis (XIAP) deficiency
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Caspase‐8‐driven apoptotic and pyroptotic crosstalk causes cell death and IL‐1β release in X‐linked inhibitor of apoptosis (XIAP) deficiency

The EMBO journal, 2023-03, Vol.42 (5), p.e110468-n/a [Peer Reviewed Journal]

2023 The Authors ;2023 The Authors. ;2023 EMBO ;ISSN: 0261-4189 ;EISSN: 1460-2075 ;DOI: 10.15252/embj.2021110468 ;PMID: 36647737

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15
Recessive NLRC4-Autoinflammatory Disease Reveals an Ulcerative Colitis Locus
Recessive NLRC4-Autoinflammatory Disease Reveals an Ulcerative Colitis Locus
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Recessive NLRC4-Autoinflammatory Disease Reveals an Ulcerative Colitis Locus

Journal of clinical immunology, 2022-02, Vol.42 (2), p.325-335 [Peer Reviewed Journal]

The Author(s) 2021 ;2021. The Author(s). ;The Author(s) 2021. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;ISSN: 0271-9142 ;EISSN: 1573-2592 ;DOI: 10.1007/s10875-021-01175-4 ;PMID: 34783940

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