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1 |
Material Type: Article
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Neurotoxic microglia promote TDP-43 proteinopathy in progranulin deficiencyNature (London), 2020-12, Vol.588 (7838), p.459-465 [Peer Reviewed Journal]COPYRIGHT 2020 Nature Publishing Group ;Copyright Nature Publishing Group Dec 17, 2020 ;ISSN: 0028-0836 ;EISSN: 1476-4687 ;DOI: 10.1038/s41586-020-2709-7 ;PMID: 32866962Full text available |
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Material Type: Article
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Structural Transformation of the Amyloidogenic Core Region of TDP-43 Protein Initiates Its Aggregation and Cytoplasmic InclusionThe Journal of biological chemistry, 2013-07, Vol.288 (27), p.19614-19624 [Peer Reviewed Journal]2013 © 2013 ASBMB. Currently published by Elsevier Inc; originally published by American Society for Biochemistry and Molecular Biology. ;2013 by The American Society for Biochemistry and Molecular Biology, Inc. 2013 ;ISSN: 0021-9258 ;EISSN: 1083-351X ;DOI: 10.1074/jbc.M113.463828 ;PMID: 23689371Full text available |
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3 |
Material Type: Article
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TDP-43 Proteinopathy and Motor Neuron Disease in Chronic Traumatic EncephalopathyJournal of neuropathology and experimental neurology, 2010-09, Vol.69 (9), p.918-929 [Peer Reviewed Journal]2010 American Association of Neuropathologists, Inc ;ISSN: 0022-3069 ;EISSN: 1554-6578 ;DOI: 10.1097/NEN.0b013e3181ee7d85 ;PMID: 20720505 ;CODEN: JNENADFull text available |
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4 |
Material Type: Article
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Exosome secretion is a key pathway for clearance of pathological TDP-43Brain (London, England : 1878), 2016-12, Vol.139 (Pt 12), p.3187-3201 [Peer Reviewed Journal]The Author (2016). Published by Oxford University Press on behalf of the Guarantors of Brain. All rights reserved. For Permissions, please email: journals.permissions@oup.com. ;The Author (2016). Published by Oxford University Press on behalf of the Guarantors of Brain. All rights reserved. For Permissions, please email: journals.permissions@oup.com 2016 ;ISSN: 0006-8950 ;EISSN: 1460-2156 ;DOI: 10.1093/brain/aww237 ;PMID: 27679482Full text available |
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5 |
Material Type: Article
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Aberrant deposition of stress granule-resident proteins linked to C9orf72-associated TDP-43 proteinopathyMolecular neurodegeneration, 2019-02, Vol.14 (1), p.9-15, Article 9 [Peer Reviewed Journal]COPYRIGHT 2019 BioMed Central Ltd. ;Copyright © 2019. This work is licensed under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s). 2019 ;ISSN: 1750-1326 ;EISSN: 1750-1326 ;DOI: 10.1186/s13024-019-0310-z ;PMID: 30767771Full text available |
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Material Type: Article
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Cortical influences drive amyotrophic lateral sclerosisJournal of neurology, neurosurgery and psychiatry, 2017-11, Vol.88 (11), p.917-924 [Peer Reviewed Journal]Article author(s) (or their employer(s) unless otherwise stated in the text of the article) 2017. All rights reserved. No commercial use is permitted unless otherwise expressly granted. ;Copyright: 2017 © Article author(s) (or their employer(s) unless otherwise stated in the text of the article) 2017. All rights reserved. No commercial use is permitted unless otherwise expressly granted. ;ISSN: 0022-3050 ;EISSN: 1468-330X ;DOI: 10.1136/jnnp-2017-315573 ;PMID: 28710326Full text available |
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Material Type: Article
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TDP-43 induces mitochondrial damage and activates the mitochondrial unfolded protein responsePLoS genetics, 2019-05, Vol.15 (5), p.e1007947-e1007947 [Peer Reviewed Journal]COPYRIGHT 2019 Public Library of Science ;COPYRIGHT 2019 Public Library of Science ;2019 Wang et al. This is an open access article distributed under the terms of the Creative Commons Attribution License: http://creativecommons.org/licenses/by/4.0/ (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2019 Wang et al 2019 Wang et al ;ISSN: 1553-7404 ;ISSN: 1553-7390 ;EISSN: 1553-7404 ;DOI: 10.1371/journal.pgen.1007947 ;PMID: 31100073Full text available |
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Material Type: Article
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Emerging Therapies and Novel Targets for TDP-43 Proteinopathy in ALS/FTDNeurotherapeutics, 2022-07, Vol.19 (4), p.1061-1084 [Peer Reviewed Journal]The American Society for Experimental NeuroTherapeutics, Inc. 2022 ;2022. The American Society for Experimental NeuroTherapeutics, Inc. ;The American Society for Experimental NeuroTherapeutics, Inc. 2022. ;ISSN: 1933-7213 ;ISSN: 1878-7479 ;EISSN: 1878-7479 ;DOI: 10.1007/s13311-022-01260-5 ;PMID: 35790708Full text available |
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9 |
Material Type: Article
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CDC7 inhibition blocks pathological TDP-43 phosphorylation and neurodegenerationAnnals of neurology, 2013-07, Vol.74 (1), p.39-52 [Peer Reviewed Journal]Copyright © 2013 American Neurological Association ;Copyright © 2013 American Neurological Association. ;ISSN: 0364-5134 ;EISSN: 1531-8249 ;DOI: 10.1002/ana.23870 ;PMID: 23424178Full text available |
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10 |
Material Type: Article
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Selective Motor Neuron Resistance and Recovery in a New Inducible Mouse Model of TDP-43 ProteinopathyThe Journal of neuroscience, 2016-07, Vol.36 (29), p.7707-7717 [Peer Reviewed Journal]Copyright © 2016 the authors 0270-6474/16/367707-11$15.00/0. ;Copyright © 2016 the authors 0270-6474/16/367707-11$15.00/0 2016 ;ISSN: 0270-6474 ;EISSN: 1529-2401 ;DOI: 10.1523/JNEUROSCI.1457-16.2016 ;PMID: 27445147Full text available |
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Material Type: Article
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Autophagy activators rescue and alleviate pathogenesis of a mouse model with proteinopathies of the TAR DNA-binding protein 43Proceedings of the National Academy of Sciences - PNAS, 2012-09, Vol.109 (37), p.15024-15029 [Peer Reviewed Journal]copyright © 1993-2008 National Academy of Sciences of the United States of America ;Copyright National Academy of Sciences Sep 11, 2012 ;ISSN: 0027-8424 ;EISSN: 1091-6490 ;DOI: 10.1073/pnas.1206362109 ;PMID: 22932872Full text available |
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Material Type: Article
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Traumatic injury compromises nucleocytoplasmic transport and leads to TDP-43 pathologyeLife, 2021-06, Vol.10 [Peer Reviewed Journal]2021, Anderson et al. ;2021, Anderson et al. This work is published under https://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2021, Anderson et al 2021 Anderson et al ;ISSN: 2050-084X ;EISSN: 2050-084X ;DOI: 10.7554/eLife.67587 ;PMID: 34060470Full text available |
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Material Type: Article
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RNA Binding Antagonizes Neurotoxic Phase Transitions of TDP-43Neuron (Cambridge, Mass.), 2019-04, Vol.102 (2), p.321-338.e8 [Peer Reviewed Journal]2019 Elsevier Inc. ;Copyright © 2019 Elsevier Inc. All rights reserved. ;2019. Elsevier Inc. ;ISSN: 0896-6273 ;EISSN: 1097-4199 ;DOI: 10.1016/j.neuron.2019.01.048 ;PMID: 30826182Full text available |
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Material Type: Article
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Cytoplasmic TDP-43 De-mixing Independent of Stress Granules Drives Inhibition of Nuclear Import, Loss of Nuclear TDP-43, and Cell DeathNeuron (Cambridge, Mass.), 2019-04, Vol.102 (2), p.339-357.e7 [Peer Reviewed Journal]2019 Elsevier Inc. ;Copyright © 2019 Elsevier Inc. All rights reserved. ;2019. Elsevier Inc. ;ISSN: 0896-6273 ;EISSN: 1097-4199 ;DOI: 10.1016/j.neuron.2019.02.038 ;PMID: 30853299Full text available |
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Material Type: Article
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Effective Inhibition of TDP‐43 Aggregation by Native State StabilizationAngewandte Chemie International Edition, 2024-01 [Peer Reviewed Journal]Attribution - NonCommercial ;ISSN: 1433-7851 ;EISSN: 1521-3773 ;DOI: 10.1002/anie.202314587 ;PMID: 37949836Digital Resources/Online E-Resources |
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16 |
Material Type: Article
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Alternatively spliced ELAVL3 cryptic exon 4a causes ELAVL3 downregulation in ALS TDP-43 proteinopathyActa neuropathologica, 2024-05, Vol.147 (1) [Peer Reviewed Journal]The Author(s) 2024 ;ISSN: 0001-6322 ;EISSN: 1432-0533 ;DOI: 10.1007/s00401-024-02732-yDigital Resources/Online E-Resources |
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17 |
Material Type: Article
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Mitigating a TDP-43 proteinopathy by targeting ataxin-2 using RNA-targeting CRISPR effector proteinsNature communications, 2023-10, Vol.14 (1), p.6492-6492, Article 6492 [Peer Reviewed Journal]2023. Springer Nature Limited. ;The Author(s) 2023. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;Springer Nature Limited 2023 ;ISSN: 2041-1723 ;EISSN: 2041-1723 ;DOI: 10.1038/s41467-023-42147-z ;PMID: 37838698Full text available |
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18 |
Material Type: Article
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Pathological mechanisms underlying TDP-43 driven neurodegeneration in FTLD-ALS spectrum disordersHuman molecular genetics, 2013-10, Vol.22 (R1), p.R77-R87 [Peer Reviewed Journal]The Author 2013. Published by Oxford University Press. 2013 ;ISSN: 0964-6906 ;EISSN: 1460-2083 ;DOI: 10.1093/hmg/ddt349 ;PMID: 23900071Full text available |
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19 |
Material Type: Article
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TDP-43 proteinopathy in ALS is triggered by loss of ASRGL1 and associated with HML-2 expressionNature communications, 2024-05, Vol.15 (1), p.4163-4163 [Peer Reviewed Journal]2024. This is a U.S. Government work and not under copyright protection in the US; foreign copyright protection may apply. ;This is a U.S. Government work and not under copyright protection in the US; foreign copyright protection may apply 2024. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;This is a U.S. Government work and not under copyright protection in the US; foreign copyright protection may apply 2024 ;EISSN: 2041-1723 ;DOI: 10.1038/s41467-024-48488-7 ;PMID: 38755145Full text available |
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20 |
Material Type: Article
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Microglia-mediated recovery from ALS-relevant motor neuron degeneration in a mouse model of TDP-43 proteinopathyNature neuroscience, 2018-03, Vol.21 (3), p.329-340 [Peer Reviewed Journal]COPYRIGHT 2018 Nature Publishing Group ;COPYRIGHT 2018 Nature Publishing Group ;Copyright Nature Publishing Group Mar 2018 ;ISSN: 1097-6256 ;EISSN: 1546-1726 ;DOI: 10.1038/s41593-018-0083-7 ;PMID: 29463850Full text available |