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Material Type: Article
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ALS-associated TDP-43 induces endoplasmic reticulum stress, which drives cytoplasmic TDP-43 accumulation and stress granule formationPloS one, 2013-11, Vol.8 (11), p.e81170-e81170 [Peer Reviewed Journal]COPYRIGHT 2013 Public Library of Science ;2013 Walker et al. This is an open-access article distributed under the terms of the Creative Commons Attribution License: http://creativecommons.org/licenses/by/3.0/ (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2013 Walker et al 2013 Walker et al ;ISSN: 1932-6203 ;EISSN: 1932-6203 ;DOI: 10.1371/journal.pone.0081170 ;PMID: 24312274Full text available |
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Material Type: Article
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Comment: Cu-ATSM to treat and image amyotrophic lateral sclerosisNeurology, 2015-05, Vol.84 (20), p.2038-2038 [Peer Reviewed Journal]2015 American Academy of Neurology ;ISSN: 0028-3878 ;EISSN: 1526-632X ;DOI: 10.1212/WNL.0000000000001600 ;PMID: 25904689Full text available |
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Material Type: Article
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impaired mitochondrial electron transport chain increases retention of the hypoxia imaging agent diacetylbis(4-methylthiosemicarbazonato)copperIIProceedings of the National Academy of Sciences - PNAS, 2012-01, Vol.109 (1), p.47-52 [Peer Reviewed Journal]copyright © 1993—2008 the National Academy of Sciences of the United States of America ;Copyright National Academy of Sciences Jan 3, 2012 ;ISSN: 0027-8424 ;EISSN: 1091-6490 ;DOI: 10.1073/pnas.1116227108 ;PMID: 22173633Full text available |
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Material Type: Article
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Diacetylbis(N(4)-methylthiosemicarbazonato) Copper(II) (CuII(atsm)) Protects against Peroxynitrite-induced Nitrosative Damage and Prolongs Survival in Amyotrophic Lateral Sclerosis Mouse ModelThe Journal of biological chemistry, 2011-12, Vol.286 (51), p.44035-44044 [Peer Reviewed Journal]2011 © 2011 ASBMB. Currently published by Elsevier Inc; originally published by American Society for Biochemistry and Molecular Biology. ;2011 by The American Society for Biochemistry and Molecular Biology, Inc. 2011 ;ISSN: 0021-9258 ;EISSN: 1083-351X ;DOI: 10.1074/jbc.M111.274407 ;PMID: 22033929Full text available |
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Material Type: Article
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Iron accumulation in skeletal muscles of old mice is associated with impaired regeneration after ischaemia–reperfusion damageJournal of cachexia, sarcopenia and muscle, 2021-04, Vol.12 (2), p.476-492 [Peer Reviewed Journal]2021 The Authors. Journal of Cachexia, Sarcopenia and Muscle published by John Wiley & Sons Ltd on behalf of Society on Sarcopenia, Cachexia and Wasting Disorders ;2021 The Authors. Journal of Cachexia, Sarcopenia and Muscle published by John Wiley & Sons Ltd on behalf of Society on Sarcopenia, Cachexia and Wasting Disorders. ;2021. This work is published under http://creativecommons.org/licenses/by-nc-nd/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;ISSN: 2190-5991 ;EISSN: 2190-6009 ;DOI: 10.1002/jcsm.12685 ;PMID: 33665974Full text available |
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Material Type: Article
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C-Jun N-terminal kinase controls TDP-43 accumulation in stress granules induced by oxidative stressMolecular neurodegeneration, 2011-08, Vol.6 (1), p.57-57, Article 57 [Peer Reviewed Journal]COPYRIGHT 2011 BioMed Central Ltd. ;Copyright ©2011 Meyerowitz et al; licensee BioMed Central Ltd. 2011 Meyerowitz et al; licensee BioMed Central Ltd. ;ISSN: 1750-1326 ;EISSN: 1750-1326 ;DOI: 10.1186/1750-1326-6-57 ;PMID: 21819629Full text available |
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Material Type: Article
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Iron overload and impaired iron handling contribute to the dystrophic pathology in models of Duchenne muscular dystrophyJournal of cachexia, sarcopenia and muscle, 2022-06, Vol.13 (3), p.1541-1553 [Peer Reviewed Journal]2022 The Authors. Journal of Cachexia, Sarcopenia and Muscle published by John Wiley & Sons Ltd on behalf of Society on Sarcopenia, Cachexia and Wasting Disorders. ;2022. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;ISSN: 2190-5991 ;EISSN: 2190-6009 ;DOI: 10.1002/jcsm.12950 ;PMID: 35249268Full text available |
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Material Type: Article
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Laser ablation-inductively coupled plasma-mass spectrometry imaging of white and gray matter iron distribution in Alzheimer's disease frontal cortexNeuroImage (Orlando, Fla.), 2016-08, Vol.137, p.124-131 [Peer Reviewed Journal]2016 ;Crown Copyright © 2016. Published by Elsevier Inc. All rights reserved. ;Copyright Elsevier Limited Aug 15, 2016 ;ISSN: 1053-8119 ;EISSN: 1095-9572 ;DOI: 10.1016/j.neuroimage.2016.05.057 ;PMID: 27233149Full text available |
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Material Type: Article
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The modulation of metal bio‐availability as a therapeutic strategy for the treatment of Alzheimer's diseaseThe FEBS journal, 2007-08, Vol.274 (15), p.3775-3783 [Peer Reviewed Journal]ISSN: 1742-464X ;EISSN: 1742-4658 ;DOI: 10.1111/j.1742-4658.2007.05918.x ;PMID: 17617225Full text available |
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Material Type: Article
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Editorial: Metals and neurodegeneration: restoring the balanceFrontiers in aging neuroscience, 2015-07, Vol.7, p.127-127 [Peer Reviewed Journal]2015. This work is licensed under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;Copyright © 2015 White, Kanninen and Crouch. 2015 White, Kanninen and Crouch ;ISSN: 1663-4365 ;EISSN: 1663-4365 ;DOI: 10.3389/fnagi.2015.00127 ;PMID: 26191002Full text available |
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Material Type: Article
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Metallo-complex activation of neuroprotective signalling pathways as a therapeutic treatment for Alzheimer's diseaseMolecular bioSystems, 2009-01, Vol.5 (2), p.134-142 [Peer Reviewed Journal]ISSN: 1742-206X ;EISSN: 1742-2051 ;DOI: 10.1039/b816577g ;PMID: 19156258Full text available |
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Material Type: Article
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Membrane‐targeted strategies for modulating APP and Aβ‐mediated toxicityJournal of cellular and molecular medicine, 2009-02, Vol.13 (2), p.249-261 [Peer Reviewed Journal]2009 The Authors Journal compilation © 2009 Foundation for Cellular and Molecular Medicine/Blackwell Publishing Ltd ;Copyright John Wiley & Sons, Inc. Feb 2009 ;2009 The Authors Journal compilation © 2009 Foundation for Cellular and Molecular Medicine/Blackwell Publishing Ltd 2009 ;ISSN: 1582-1838 ;EISSN: 1582-4934 ;DOI: 10.1111/j.1582-4934.2008.00642.x ;PMID: 19278455Digital Resources/Online E-Resources |