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1
Active MLKL triggers the NLRP3 inflammasome in a cell-intrinsic manner
Active MLKL triggers the NLRP3 inflammasome in a cell-intrinsic manner
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Active MLKL triggers the NLRP3 inflammasome in a cell-intrinsic manner

Proceedings of the National Academy of Sciences - PNAS, 2017-02, Vol.114 (6), p.E961-E969 [Peer Reviewed Journal]

Volumes 1–89 and 106–114, copyright as a collective work only; author(s) retains copyright to individual articles ;Copyright National Academy of Sciences Feb 7, 2017 ;ISSN: 0027-8424 ;EISSN: 1091-6490 ;DOI: 10.1073/pnas.1613305114 ;PMID: 28096356

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2
Microbiota and adipocyte mitochondrial damage in type 2 diabetes are linked by Mmp12+ macrophages
Microbiota and adipocyte mitochondrial damage in type 2 diabetes are linked by Mmp12+ macrophages
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Microbiota and adipocyte mitochondrial damage in type 2 diabetes are linked by Mmp12+ macrophages

The Journal of experimental medicine, 2022-07, Vol.219 (7) [Peer Reviewed Journal]

2022 Li et al. ;2022 Li et al. 2022 ;ISSN: 0022-1007 ;EISSN: 1540-9538 ;DOI: 10.1084/jem.20220017 ;PMID: 35657352

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3
ATF3 Is a Key Regulator of Macrophage IFN Responses
ATF3 Is a Key Regulator of Macrophage IFN Responses
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ATF3 Is a Key Regulator of Macrophage IFN Responses

The Journal of immunology (1950), 2015-11, Vol.195 (9), p.4446-4455 [Peer Reviewed Journal]

Copyright © 2015 by The American Association of Immunologists, Inc. ;ISSN: 0022-1767 ;EISSN: 1550-6606 ;DOI: 10.4049/jimmunol.1500204 ;PMID: 26416280

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4
Connexin-Dependent Transfer of cGAMP to Phagocytes Modulates Antiviral Responses
Connexin-Dependent Transfer of cGAMP to Phagocytes Modulates Antiviral Responses
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Connexin-Dependent Transfer of cGAMP to Phagocytes Modulates Antiviral Responses

mBio, 2020-01, Vol.11 (1) [Peer Reviewed Journal]

Copyright © 2020 Pépin et al. ;Copyright © 2020 Pépin et al. 2020 Pépin et al. ;ISSN: 2161-2129 ;EISSN: 2150-7511 ;DOI: 10.1128/mBio.03187-19 ;PMID: 31992625

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5
Signaling Crosstalk during Sequential TLR4 and TLR9 Activation Amplifies the Inflammatory Response of Mouse Macrophages
Signaling Crosstalk during Sequential TLR4 and TLR9 Activation Amplifies the Inflammatory Response of Mouse Macrophages
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Signaling Crosstalk during Sequential TLR4 and TLR9 Activation Amplifies the Inflammatory Response of Mouse Macrophages

The Journal of immunology (1950), 2009-12, Vol.183 (12), p.8110-8118 [Peer Reviewed Journal]

ISSN: 0022-1767 ;EISSN: 1550-6606 ;DOI: 10.4049/jimmunol.0901031 ;PMID: 19923461

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6
Genistein Targets STING-Driven Antiviral Responses
Genistein Targets STING-Driven Antiviral Responses
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Genistein Targets STING-Driven Antiviral Responses

mBio, 2022-08, Vol.13 (4), p.e0206422-e0206422 [Peer Reviewed Journal]

Copyright © 2022 Ullah et al. ;Copyright © 2022 Ullah et al. 2022 Ullah et al. ;ISSN: 2150-7511 ;EISSN: 2150-7511 ;DOI: 10.1128/mbio.02064-22 ;PMID: 35924852

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7
The Critical Role of the Colony-Stimulating Factor-1 Receptor in the Differentiation of Myeloblastic Leukemia Cells
The Critical Role of the Colony-Stimulating Factor-1 Receptor in the Differentiation of Myeloblastic Leukemia Cells
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The Critical Role of the Colony-Stimulating Factor-1 Receptor in the Differentiation of Myeloblastic Leukemia Cells

Molecular cancer research, 2008-03, Vol.6 (3), p.458-467 [Peer Reviewed Journal]

ISSN: 1541-7786 ;EISSN: 1557-3125 ;DOI: 10.1158/1541-7786.MCR-07-0361 ;PMID: 18337452

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8
Interleukin-1 receptor–associated kinase 4 (IRAK4) plays a dual role in myddosome formation and Toll-like receptor signaling
Interleukin-1 receptor–associated kinase 4 (IRAK4) plays a dual role in myddosome formation and Toll-like receptor signaling
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Interleukin-1 receptor–associated kinase 4 (IRAK4) plays a dual role in myddosome formation and Toll-like receptor signaling

The Journal of biological chemistry, 2018-09, Vol.293 (39), p.15195-15207 [Peer Reviewed Journal]

2018 © 2018 De Nardo et al. ;2018 De Nardo et al. ;2018 De Nardo et al. 2018 De Nardo et al. ;ISSN: 0021-9258 ;EISSN: 1083-351X ;DOI: 10.1074/jbc.RA118.003314 ;PMID: 30076215

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9
A novel Pyrin-Associated Autoinflammation with Neutrophilic Dermatosis mutation further defines 14-3-3 binding of pyrin and distinction to Familial Mediterranean Fever
A novel Pyrin-Associated Autoinflammation with Neutrophilic Dermatosis mutation further defines 14-3-3 binding of pyrin and distinction to Familial Mediterranean Fever
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A novel Pyrin-Associated Autoinflammation with Neutrophilic Dermatosis mutation further defines 14-3-3 binding of pyrin and distinction to Familial Mediterranean Fever

Annals of the rheumatic diseases, 2017-12, Vol.76 (12), p.2085-2094 [Peer Reviewed Journal]

Article author(s) (or their employer(s) unless otherwise stated in the text of the article) 2017. All rights reserved. No commercial use is permitted unless otherwise expressly granted. ;Copyright: 2017 © Article author(s) (or their employer(s) unless otherwise stated in the text of the article) 2017. All rights reserved. No commercial use is permitted unless otherwise expressly granted. ;ISSN: 0003-4967 ;EISSN: 1468-2060 ;DOI: 10.1136/annrheumdis-2017-211473 ;PMID: 28835462

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10
Posttranslational Modification as a Critical Determinant of Cytoplasmic Innate Immune Recognition
Posttranslational Modification as a Critical Determinant of Cytoplasmic Innate Immune Recognition
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Posttranslational Modification as a Critical Determinant of Cytoplasmic Innate Immune Recognition

Physiological reviews, 2017-07, Vol.97 (3), p.1165-1209 [Peer Reviewed Journal]

Copyright © 2017 the American Physiological Society. ;ISSN: 0031-9333 ;EISSN: 1522-1210 ;DOI: 10.1152/physrev.00026.2016 ;PMID: 28615462

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11
Aberrant actin depolymerization triggers the pyrin inflammasome and autoinflammatory disease that is dependent on IL-18, not IL-1β
Aberrant actin depolymerization triggers the pyrin inflammasome and autoinflammatory disease that is dependent on IL-18, not IL-1β
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Aberrant actin depolymerization triggers the pyrin inflammasome and autoinflammatory disease that is dependent on IL-18, not IL-1β

The Journal of experimental medicine, 2015-06, Vol.212 (6), p.927-938 [Peer Reviewed Journal]

2015 Kim et al. ;2015 Kim et al. 2015 ;ISSN: 0022-1007 ;EISSN: 1540-9538 ;DOI: 10.1084/jem.20142384 ;PMID: 26008898

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12
Activation of stimulator of interferon genes in dendritic cells induces interferon-lambda and subset- and species-specific dendritic cell death
Activation of stimulator of interferon genes in dendritic cells induces interferon-lambda and subset- and species-specific dendritic cell death
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Activation of stimulator of interferon genes in dendritic cells induces interferon-lambda and subset- and species-specific dendritic cell death

The Journal of immunology (1950), 2021-05, Vol.206 (1_Supplement), p.111-111.28 [Peer Reviewed Journal]

ISSN: 0022-1767 ;EISSN: 1550-6606 ;DOI: 10.4049/jimmunol.206.Supp.111.28

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13
Interferon Regulatory Factor 6 Differentially Regulates Toll-like Receptor 2-dependent Chemokine Gene Expression in Epithelial Cells
Interferon Regulatory Factor 6 Differentially Regulates Toll-like Receptor 2-dependent Chemokine Gene Expression in Epithelial Cells
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Interferon Regulatory Factor 6 Differentially Regulates Toll-like Receptor 2-dependent Chemokine Gene Expression in Epithelial Cells

The Journal of biological chemistry, 2014-07, Vol.289 (28), p.19758-19768 [Peer Reviewed Journal]

2014 © 2014 ASBMB. Currently published by Elsevier Inc; originally published by American Society for Biochemistry and Molecular Biology. ;2014 by The American Society for Biochemistry and Molecular Biology, Inc. ;2014 by The American Society for Biochemistry and Molecular Biology, Inc. 2014 ;ISSN: 0021-9258 ;EISSN: 1083-351X ;DOI: 10.1074/jbc.M114.584540 ;PMID: 24872416

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14
Comprehensive RNAi-based screening of human and mouse TLR pathways identifies species-specific preferences in signaling protein use
Comprehensive RNAi-based screening of human and mouse TLR pathways identifies species-specific preferences in signaling protein use
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Comprehensive RNAi-based screening of human and mouse TLR pathways identifies species-specific preferences in signaling protein use

The Journal of immunology (1950), 2016-05, Vol.196 (1_Supplement), p.203-203.22 [Peer Reviewed Journal]

ISSN: 0022-1767 ;EISSN: 1550-6606 ;DOI: 10.4049/jimmunol.196.Supp.203.22

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15
Aberrant actin depolymerization triggers the pyrin inflammasome and autoinflammatory disease that is dependent on IL-18, not IL-1β
Aberrant actin depolymerization triggers the pyrin inflammasome and autoinflammatory disease that is dependent on IL-18, not IL-1β
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Article 		Add to My Research

Aberrant actin depolymerization triggers the pyrin inflammasome and autoinflammatory disease that is dependent on IL-18, not IL-1β

The Journal of cell biology, 2015-06, Vol.209 (5), p.2095 [Peer Reviewed Journal]

ISSN: 0021-9525 ;EISSN: 1540-8140 ;DOI: 10.1083/jcb.2095OIA104

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16
Aberrant actin depolymerization triggers the pyrin inflammasome and autoinflammatory disease that is dependent on IL-18, not IL-1β
Aberrant actin depolymerization triggers the pyrin inflammasome and autoinflammatory disease that is dependent on IL-18, not IL-1β
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Article 		Add to My Research

Aberrant actin depolymerization triggers the pyrin inflammasome and autoinflammatory disease that is dependent on IL-18, not IL-1β

ISSN: 0022-1007 ;EISSN: 1540-9538 ;DOI: 10.1084/jem.20142384

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17
A Central Role for the Hsp90·Cdc37 Molecular Chaperone Module in Interleukin-1 Receptor-associated-kinase-dependent Signaling by Toll-like Receptors
A Central Role for the Hsp90·Cdc37 Molecular Chaperone Module in Interleukin-1 Receptor-associated-kinase-dependent Signaling by Toll-like Receptors
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A Central Role for the Hsp90·Cdc37 Molecular Chaperone Module in Interleukin-1 Receptor-associated-kinase-dependent Signaling by Toll-like Receptors

The Journal of biological chemistry, 2005-03, Vol.280 (11), p.9813-9822 [Peer Reviewed Journal]

2005 © 2005 ASBMB. Currently published by Elsevier Inc; originally published by American Society for Biochemistry and Molecular Biology. ;ISSN: 0021-9258 ;EISSN: 1083-351X ;DOI: 10.1074/jbc.M409745200

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18
A Central Role for the Hsp90·Cdc37 Molecular Chaperone Module in Interleukin-1 Receptor-associated-kinase-dependent Signaling by Toll-like Receptors
A Central Role for the Hsp90·Cdc37 Molecular Chaperone Module in Interleukin-1 Receptor-associated-kinase-dependent Signaling by Toll-like Receptors
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A Central Role for the Hsp90·Cdc37 Molecular Chaperone Module in Interleukin-1 Receptor-associated-kinase-dependent Signaling by Toll-like Receptors

The Journal of biological chemistry, 2005-03, Vol.280 (11), p.9813 [Peer Reviewed Journal]

ISSN: 0021-9258 ;EISSN: 1083-351X ;DOI: 10.1074/jbc.M409745200 ;PMID: 15647277

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19
A central role for the Hsp90.Cdc37 molecular chaperone module in interleukin-1 receptor-associated-kinase-dependent signaling by toll-like receptors
A central role for the Hsp90.Cdc37 molecular chaperone module in interleukin-1 receptor-associated-kinase-dependent signaling by toll-like receptors
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A central role for the Hsp90.Cdc37 molecular chaperone module in interleukin-1 receptor-associated-kinase-dependent signaling by toll-like receptors

The Journal of biological chemistry, 2005-03, Vol.280 (11), p.9813-9822 [Peer Reviewed Journal]

ISSN: 0021-9258 ;EISSN: 1083-351X ;PMID: 15647277

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