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21
P243 High prevalence of Ureaplasma spp. in women visiting an STI clinic although no azithromycin resistance was discovered
P243 High prevalence of Ureaplasma spp. in women visiting an STI clinic although no azithromycin resistance was discovered
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P243 High prevalence of Ureaplasma spp. in women visiting an STI clinic although no azithromycin resistance was discovered

Sexually transmitted infections, 2019-07, Vol.95 (Suppl 1), p.A149 [Peer Reviewed Journal]

Author(s) (or their employer(s)) 2019. No commercial re-use. See rights and permissions. Published by BMJ. ;2019 Author(s) (or their employer(s)) 2019. No commercial re-use. See rights and permissions. Published by BMJ. ;ISSN: 1368-4973 ;EISSN: 1472-3263 ;DOI: 10.1136/sextrans-2019-sti.377

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22
P597 Comparison of assays and specimen types for the diagnosis of mycoplasma genitalium and macrolide resistant mutations
P597 Comparison of assays and specimen types for the diagnosis of mycoplasma genitalium and macrolide resistant mutations
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P597 Comparison of assays and specimen types for the diagnosis of mycoplasma genitalium and macrolide resistant mutations

Sexually transmitted infections, 2019-07, Vol.95 (Suppl 1), p.A265 [Peer Reviewed Journal]

Author(s) (or their employer(s)) 2019. No commercial re-use. See rights and permissions. Published by BMJ. ;2019 Author(s) (or their employer(s)) 2019. No commercial re-use. See rights and permissions. Published by BMJ. ;ISSN: 1368-4973 ;EISSN: 1472-3263 ;DOI: 10.1136/sextrans-2019-sti.666

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23
Reply
Reply
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Reply

American journal of ophthalmology, 2018-02, Vol.186, p.170 [Peer Reviewed Journal]

Copyright Elsevier Limited Feb 2018 ;ISSN: 0002-9394 ;EISSN: 1879-1891 ;DOI: 10.1016/j.ajo.2017.11.005

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24
PO180 Mosaicism in neurofibromatosis type 2: a case report
PO180 Mosaicism in neurofibromatosis type 2: a case report
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PO180 Mosaicism in neurofibromatosis type 2: a case report

Journal of neurology, neurosurgery and psychiatry, 2017-12, Vol.88 (Suppl 1), p.A59-A59 [Peer Reviewed Journal]

2017, Published by the BMJ Publishing Group Limited. For permission to use (where not already granted under a licence) please go to http://group.bmj.com/group/rights-licensing/permissions ;Copyright: 2017 © 2017, Published by the BMJ Publishing Group Limited. For permission to use (where not already granted under a licence) please go to http://group.bmj.com/group/rights-licensing/permissions ;ISSN: 0022-3050 ;EISSN: 1468-330X ;DOI: 10.1136/jnnp-2017-ABN.202

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25
PO186 Hereditary leukoencephalopathy (hls) and csf1r heterogeneity
PO186 Hereditary leukoencephalopathy (hls) and csf1r heterogeneity
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PO186 Hereditary leukoencephalopathy (hls) and csf1r heterogeneity

Journal of neurology, neurosurgery and psychiatry, 2017-12, Vol.88 (Suppl 1), p.A60-A60 [Peer Reviewed Journal]

2017, Published by the BMJ Publishing Group Limited. For permission to use (where not already granted under a licence) please go to http://group.bmj.com/group/rights-licensing/permissions ;Copyright: 2017 © 2017, Published by the BMJ Publishing Group Limited. For permission to use (where not already granted under a licence) please go to http://group.bmj.com/group/rights-licensing/permissions ;ISSN: 0022-3050 ;EISSN: 1468-330X ;DOI: 10.1136/jnnp-2017-ABN.207

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26
PO202 Natural history study in hereditary sensory neuropathy type 1
PO202 Natural history study in hereditary sensory neuropathy type 1
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PO202 Natural history study in hereditary sensory neuropathy type 1

Journal of neurology, neurosurgery and psychiatry, 2017-12, Vol.88 (Suppl 1), p.A65-A65 [Peer Reviewed Journal]

2017, Published by the BMJ Publishing Group Limited. For permission to use (where not already granted under a licence) please go to http://group.bmj.com/group/rights-licensing/permissions ;Copyright: 2017 © 2017, Published by the BMJ Publishing Group Limited. For permission to use (where not already granted under a licence) please go to http://group.bmj.com/group/rights-licensing/permissions ;ISSN: 0022-3050 ;EISSN: 1468-330X ;DOI: 10.1136/jnnp-2017-ABN.223

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27
CORRIGENDUM
CORRIGENDUM
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CORRIGENDUM

Genetics (Austin), 2019-07, Vol.212 (3), p.953-953 [Peer Reviewed Journal]

Copyright Genetics Society of America Jul 2019 ;ISSN: 0016-6731 ;EISSN: 1943-2631 ;DOI: 10.1534/genetics.119.302298

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28
Abstract 49: A single centre experience on nodular adrenocortical cushings syndrome
Abstract 49: A single centre experience on nodular adrenocortical cushings syndrome
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Abstract 49: A single centre experience on nodular adrenocortical cushings syndrome

Indian journal of endocrinology and metabolism, 2022-12, Vol.26 (8), p.21-21 [Peer Reviewed Journal]

COPYRIGHT 2022 Medknow Publications and Media Pvt. Ltd. ;2022. This article is published under (http://creativecommons.org/licenses/by-nc-sa/3.0/) (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;ISSN: 2230-8210 ;EISSN: 2230-9500 ;DOI: 10.4103/2230-8210.363736

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29
Correction: Association of SMAD4 mutation with patient demographics, tumor characteristics, and clinical outcomes in colorectal cancer
Correction: Association of SMAD4 mutation with patient demographics, tumor characteristics, and clinical outcomes in colorectal cancer
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Correction: Association of SMAD4 mutation with patient demographics, tumor characteristics, and clinical outcomes in colorectal cancer

PloS one, 2017-05, Vol.12 (5), p.e0178275-e0178275 [Peer Reviewed Journal]

COPYRIGHT 2017 Public Library of Science ;COPYRIGHT 2017 Public Library of Science ;2017 The PLOS ONE Staff. This is an open access article distributed under the terms of the Creative Commons Attribution License: http://creativecommons.org/licenses/by/4.0/ (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2017 The PLOS ONE Staff 2017 The PLOS ONE Staff ;ISSN: 1932-6203 ;EISSN: 1932-6203 ;DOI: 10.1371/journal.pone.0178275 ;PMID: 28545046

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30
Correction: A Novel Quantitative Hemolytic Assay Coupled with Restriction Fragment Length Polymorphisms Analysis Enabled Early Diagnosis of Atypical Hemolytic Uremic Syndrome and Identified Unique Predisposing Mutations in Japan
Correction: A Novel Quantitative Hemolytic Assay Coupled with Restriction Fragment Length Polymorphisms Analysis Enabled Early Diagnosis of Atypical Hemolytic Uremic Syndrome and Identified Unique Predisposing Mutations in Japan
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Correction: A Novel Quantitative Hemolytic Assay Coupled with Restriction Fragment Length Polymorphisms Analysis Enabled Early Diagnosis of Atypical Hemolytic Uremic Syndrome and Identified Unique Predisposing Mutations in Japan

PloS one, 2017-05, Vol.12 (5), p.e0178015-e0178015 [Peer Reviewed Journal]

COPYRIGHT 2017 Public Library of Science ;COPYRIGHT 2017 Public Library of Science ;2017 Yoshida et al. This is an open access article distributed under the terms of the Creative Commons Attribution License: http://creativecommons.org/licenses/by/4.0/ (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2017 Yoshida et al 2017 Yoshida et al ;ISSN: 1932-6203 ;EISSN: 1932-6203 ;DOI: 10.1371/journal.pone.0178015 ;PMID: 28520790

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31
A44 INTERVENTIONAL PULMONARY: CASE REPORTS: C.1309c/t Endoglin (eng) Gene Mutation Associated Multiple Pulmonary Arteriovenous Malformations Successfully Treated By Endovascular Coiling And Vascular Plugs
A44 INTERVENTIONAL PULMONARY: CASE REPORTS: C.1309c/t Endoglin (eng) Gene Mutation Associated Multiple Pulmonary Arteriovenous Malformations Successfully Treated By Endovascular Coiling And Vascular Plugs
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A44 INTERVENTIONAL PULMONARY: CASE REPORTS: C.1309c/t Endoglin (eng) Gene Mutation Associated Multiple Pulmonary Arteriovenous Malformations Successfully Treated By Endovascular Coiling And Vascular Plugs

American journal of respiratory and critical care medicine, 2017-01, Vol.195 [Peer Reviewed Journal]

Copyright American Thoracic Society 2017 ;ISSN: 1073-449X ;EISSN: 1535-4970

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32
C25 NON-TUBERCULOUS MYCOBACTERIA: FROM BENCH TO CLINIC: A Bna-Pcr Based Rapid Identification System For The Detection Of Macrolide Resistant Mycobacterium Avium-Intracellulare Complex
C25 NON-TUBERCULOUS MYCOBACTERIA: FROM BENCH TO CLINIC: A Bna-Pcr Based Rapid Identification System For The Detection Of Macrolide Resistant Mycobacterium Avium-Intracellulare Complex
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C25 NON-TUBERCULOUS MYCOBACTERIA: FROM BENCH TO CLINIC: A Bna-Pcr Based Rapid Identification System For The Detection Of Macrolide Resistant Mycobacterium Avium-Intracellulare Complex

American journal of respiratory and critical care medicine, 2017-01, Vol.195 [Peer Reviewed Journal]

Copyright American Thoracic Society 2017 ;ISSN: 1073-449X ;EISSN: 1535-4970

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33
C80-J THORACIC ONCOLOGY CASE REPORTS IV: Histologic Transformation From Non-Small Cell Lung Cancer (nsclc) Adenocarcinoma With EGFR-Mutant Exons 20(s768i) And 21(l858r) To Small Cell Lung Carcinoma (sclc): Case Report
C80-J THORACIC ONCOLOGY CASE REPORTS IV: Histologic Transformation From Non-Small Cell Lung Cancer (nsclc) Adenocarcinoma With EGFR-Mutant Exons 20(s768i) And 21(l858r) To Small Cell Lung Carcinoma (sclc): Case Report
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C80-J THORACIC ONCOLOGY CASE REPORTS IV: Histologic Transformation From Non-Small Cell Lung Cancer (nsclc) Adenocarcinoma With EGFR-Mutant Exons 20(s768i) And 21(l858r) To Small Cell Lung Carcinoma (sclc): Case Report

American journal of respiratory and critical care medicine, 2017-01, Vol.195 [Peer Reviewed Journal]

Copyright American Thoracic Society 2017 ;ISSN: 1073-449X ;EISSN: 1535-4970

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34
CORRECTION
CORRECTION
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CORRECTION

Nature (London), 2018-11, Vol.563 (7732), p.E27-E27 [Peer Reviewed Journal]

Copyright Nature Publishing Group Nov 22, 2018 ;ISSN: 0028-0836 ;EISSN: 1476-4687 ;DOI: 10.1038/s41586-018-0580-6

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35
Genotype/Phenotype Correlation of Cases with PTPN11 Gene Mutation: Eastern Black Sea Experience
Genotype/Phenotype Correlation of Cases with PTPN11 Gene Mutation: Eastern Black Sea Experience
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Genotype/Phenotype Correlation of Cases with PTPN11 Gene Mutation: Eastern Black Sea Experience

Ankara Ueniversitesi Tip Fakültesi mecmuasi, 2022-10, Vol.75 (3), p.368-372 [Peer Reviewed Journal]

2022. This work is published under https://creativecommons.org/licenses/by-nc-nd/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;ISSN: 1307-5608 ;ISSN: 0365-8104 ;EISSN: 1307-5608 ;DOI: 10.4274/atfm.galenos.2022.06978

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36
The WNT1G177C mutation specifically affects skeletal integrity in a mouse model of osteogenesis imperfecta type XV
The WNT1G177C mutation specifically affects skeletal integrity in a mouse model of osteogenesis imperfecta type XV
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The WNT1G177C mutation specifically affects skeletal integrity in a mouse model of osteogenesis imperfecta type XV

Bone research, 2021-11, Vol.9 (1), p.48-48 [Peer Reviewed Journal]

The Author(s) 2021. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2021 ;ISSN: 2095-4700 ;EISSN: 2095-6231 ;DOI: 10.1038/s41413-021-00170-0 ;PMID: 34759273

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37
Erratum: Whole-genome mutational landscape and characterization of noncoding and structural mutations in liver cancer
Erratum: Whole-genome mutational landscape and characterization of noncoding and structural mutations in liver cancer
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Erratum: Whole-genome mutational landscape and characterization of noncoding and structural mutations in liver cancer

Nature genetics, 2016-05, Vol.48 (6), p.700-700 [Peer Reviewed Journal]

ISSN: 1061-4036 ;EISSN: 1546-1718 ;DOI: 10.1038/ng0616-700a ;PMID: 27230686

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38
Correction: A Novel GJA8 Mutation (p.V44A) Causing Autosomal Dominant Congenital Cataract
Correction: A Novel GJA8 Mutation (p.V44A) Causing Autosomal Dominant Congenital Cataract
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Correction: A Novel GJA8 Mutation (p.V44A) Causing Autosomal Dominant Congenital Cataract

PloS one, 2015-05, Vol.10 (5), p.e0125949-e0125949 [Peer Reviewed Journal]

COPYRIGHT 2015 Public Library of Science ;COPYRIGHT 2015 Public Library of Science ;2015 Zhu et al. This is an open access article distributed under the terms of the Creative Commons Attribution License: http://creativecommons.org/licenses/by/4.0/ (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2015 Zhu et al 2015 Zhu et al ;ISSN: 1932-6203 ;EISSN: 1932-6203 ;DOI: 10.1371/journal.pone.0125949 ;PMID: 25970271

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39
The landscape of genomic alterations across childhood cancers
The landscape of genomic alterations across childhood cancers
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The landscape of genomic alterations across childhood cancers

Nature (London), 2018-03, Vol.555 (7696), p.321-327 [Peer Reviewed Journal]

COPYRIGHT 2018 Nature Publishing Group ;COPYRIGHT 2018 Nature Publishing Group ;Copyright Nature Publishing Group Mar 15, 2018 ;Distributed under a Creative Commons Attribution 4.0 International License ;ISSN: 0028-0836 ;EISSN: 1476-4687 ;DOI: 10.1038/nature25480 ;PMID: 29489754

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40
Two male patients from an extended seven generation Turkish family diagnosed with Renpenning syndrome: identifying the causative mutation and review of the literature
Two male patients from an extended seven generation Turkish family diagnosed with Renpenning syndrome: identifying the causative mutation and review of the literature
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Two male patients from an extended seven generation Turkish family diagnosed with Renpenning syndrome: identifying the causative mutation and review of the literature

The European research journal, 2022-05, Vol.8 (3), p.420-427 [Peer Reviewed Journal]

Copyright The Association of Health Research & Strategy 2022 ;ISSN: 2149-3189 ;EISSN: 2149-3189 ;DOI: 10.18621/eurj.924346

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