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1
Human DPP9 represses NLRP1 inflammasome and protects against autoinflammatory diseases via both peptidase activity and FIIND domain binding
Human DPP9 represses NLRP1 inflammasome and protects against autoinflammatory diseases via both peptidase activity and FIIND domain binding
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Human DPP9 represses NLRP1 inflammasome and protects against autoinflammatory diseases via both peptidase activity and FIIND domain binding

The Journal of biological chemistry, 2018-12, Vol.293 (49), p.18864-18878 [Peer Reviewed Journal]

2018 © 2018 Zhong et al. ;2018 Zhong et al. ;2018 Zhong et al. 2018 Zhong et al. ;ISSN: 0021-9258 ;EISSN: 1083-351X ;DOI: 10.1074/jbc.RA118.004350 ;PMID: 30291141

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Caspase‐8‐driven apoptotic and pyroptotic crosstalk causes cell death and IL‐1β release in X‐linked inhibitor of apoptosis (XIAP) deficiency
Caspase‐8‐driven apoptotic and pyroptotic crosstalk causes cell death and IL‐1β release in X‐linked inhibitor of apoptosis (XIAP) deficiency
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Caspase‐8‐driven apoptotic and pyroptotic crosstalk causes cell death and IL‐1β release in X‐linked inhibitor of apoptosis (XIAP) deficiency

The EMBO journal, 2023-03, Vol.42 (5), p.e110468-n/a [Peer Reviewed Journal]

2023 The Authors ;2023 The Authors. ;2023 EMBO ;ISSN: 0261-4189 ;EISSN: 1460-2075 ;DOI: 10.15252/embj.2021110468 ;PMID: 36647737

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3
Deficiency in coatomer complex I causes aberrant activation of STING signalling
Deficiency in coatomer complex I causes aberrant activation of STING signalling
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Deficiency in coatomer complex I causes aberrant activation of STING signalling

Nature communications, 2022-04, Vol.13 (1), p.2321-2321, Article 2321 [Peer Reviewed Journal]

2022. The Author(s). ;The Author(s) 2022. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2022 ;ISSN: 2041-1723 ;EISSN: 2041-1723 ;DOI: 10.1038/s41467-022-29946-6 ;PMID: 35484149

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4
T-cell autoreactivity to citrullinated autoantigenic peptides in rheumatoid arthritis patients carrying HLA-DRB1 shared epitope alleles
T-cell autoreactivity to citrullinated autoantigenic peptides in rheumatoid arthritis patients carrying HLA-DRB1 shared epitope alleles
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T-cell autoreactivity to citrullinated autoantigenic peptides in rheumatoid arthritis patients carrying HLA-DRB1 shared epitope alleles

Arthritis research & therapy, 2012-05, Vol.14 (3), p.R118-R118 [Peer Reviewed Journal]

COPYRIGHT 2012 BioMed Central Ltd. ;Copyright ©2012 Law et al.; licensee BioMed Central Ltd. 2012 Law et al.; licensee BioMed Central Ltd. ;ISSN: 1478-6354 ;EISSN: 1478-6362 ;DOI: 10.1186/ar3848 ;PMID: 22594821

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5
Organellar homeostasis and innate immune sensing
Organellar homeostasis and innate immune sensing
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Organellar homeostasis and innate immune sensing

Nature reviews. Immunology, 2022-09, Vol.22 (9), p.535-549 [Peer Reviewed Journal]

2022. Springer Nature Limited. ;Springer Nature Limited 2022. ;ISSN: 1474-1733 ;EISSN: 1474-1741 ;DOI: 10.1038/s41577-022-00682-8 ;PMID: 35197578

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6
TDP-43 Triggers Mitochondrial DNA Release via mPTP to Activate cGAS/STING in ALS
TDP-43 Triggers Mitochondrial DNA Release via mPTP to Activate cGAS/STING in ALS
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TDP-43 Triggers Mitochondrial DNA Release via mPTP to Activate cGAS/STING in ALS

Cell, 2020-10, Vol.183 (3), p.636-649.e18 [Peer Reviewed Journal]

2020 The Author(s) ;Copyright © 2020 The Author(s). Published by Elsevier Inc. All rights reserved. ;2020 The Author(s) 2020 ;ISSN: 0092-8674 ;EISSN: 1097-4172 ;DOI: 10.1016/j.cell.2020.09.020 ;PMID: 33031745

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7
RP105 Engages Phosphatidylinositol 3-Kinase p110δ To Facilitate the Trafficking and Secretion of Cytokines in Macrophages during Mycobacterial Infection
RP105 Engages Phosphatidylinositol 3-Kinase p110δ To Facilitate the Trafficking and Secretion of Cytokines in Macrophages during Mycobacterial Infection
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RP105 Engages Phosphatidylinositol 3-Kinase p110δ To Facilitate the Trafficking and Secretion of Cytokines in Macrophages during Mycobacterial Infection

The Journal of immunology (1950), 2015-10, Vol.195 (8), p.3890-3900 [Peer Reviewed Journal]

Copyright © 2015 by The American Association of Immunologists, Inc. ;ISSN: 0022-1767 ;EISSN: 1550-6606 ;DOI: 10.4049/jimmunol.1500017 ;PMID: 26371254

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8
NLRP1 restricts butyrate producing commensals to exacerbate inflammatory bowel disease
NLRP1 restricts butyrate producing commensals to exacerbate inflammatory bowel disease
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NLRP1 restricts butyrate producing commensals to exacerbate inflammatory bowel disease

Nature communications, 2018-09, Vol.9 (1), p.3728-11, Article 3728 [Peer Reviewed Journal]

2018. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2018 ;ISSN: 2041-1723 ;EISSN: 2041-1723 ;DOI: 10.1038/s41467-018-06125-0 ;PMID: 30214011

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9
Whole-genome sequencing reveals that variants in the Interleukin 18 Receptor Accessory Protein 3′UTR protect against ALS
Whole-genome sequencing reveals that variants in the Interleukin 18 Receptor Accessory Protein 3′UTR protect against ALS
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Whole-genome sequencing reveals that variants in the Interleukin 18 Receptor Accessory Protein 3′UTR protect against ALS

Nature neuroscience, 2022-04, Vol.25 (4), p.433-445 [Peer Reviewed Journal]

The Author(s), under exclusive licence to Springer Nature America, Inc. 2022. ;ISSN: 1097-6256 ;EISSN: 1546-1726 ;DOI: 10.1038/s41593-022-01040-6 ;PMID: 35361972

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10
Whole-genome sequencing reveals that variants in the Interleukin 18 Receptor Accessory Protein 3 ' UTR protect against ALS
Whole-genome sequencing reveals that variants in the Interleukin 18 Receptor Accessory Protein 3 ' UTR protect against ALS
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Whole-genome sequencing reveals that variants in the Interleukin 18 Receptor Accessory Protein 3 ' UTR protect against ALS

NATURE NEUROSCIENCE, 2022-04, Vol.25 (4), p.433-+ [Peer Reviewed Journal]

ISSN: 1097-6256 ;EISSN: 1546-1726

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11
NLRP1 restricts butyrate producing commensals to exacerbate inflammatory bowel disease
NLRP1 restricts butyrate producing commensals to exacerbate inflammatory bowel disease
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NLRP1 restricts butyrate producing commensals to exacerbate inflammatory bowel disease

info:eu-repo/semantics/OpenAccess ;ISSN: 2041-1723 ;EISSN: 2041-1723

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12
Whole-genome sequencing reveals that variants in the Interleukin 18 Receptor Accessory Protein 3'UTR protect against ALS
Whole-genome sequencing reveals that variants in the Interleukin 18 Receptor Accessory Protein 3'UTR protect against ALS
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Whole-genome sequencing reveals that variants in the Interleukin 18 Receptor Accessory Protein 3'UTR protect against ALS

Nature neuroscience, 2022-04, Vol.25 (4), p.433 [Peer Reviewed Journal]

2022. The Author(s), under exclusive licence to Springer Nature America, Inc. ;EISSN: 1546-1726 ;DOI: 10.1038/s41593-022-01040-6 ;PMID: 35361972

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13
Characterisation of RP105's divergent functions in macrophage responses to mycobacteria and LPS
Characterisation of RP105's divergent functions in macrophage responses to mycobacteria and LPS
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Characterisation of RP105's divergent functions in macrophage responses to mycobacteria and LPS

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14
Methods of Treatment, Prevention and Diagnosis
Methods of Treatment, Prevention and Diagnosis
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Methods of Treatment, Prevention and Diagnosis

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15
Non-Coding Genetic Analysis Implicates Interleukin 18 Receptor Accessory Protein 3′UTR in Amyotrophic Lateral Sclerosis
Non-Coding Genetic Analysis Implicates Interleukin 18 Receptor Accessory Protein 3′UTR in Amyotrophic Lateral Sclerosis
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Non-Coding Genetic Analysis Implicates Interleukin 18 Receptor Accessory Protein 3′UTR in Amyotrophic Lateral Sclerosis

bioRxiv, 2021

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16
Method for controlling display
Method for controlling display
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Method for controlling display

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17
METHOD FOR CONTROLLING DISPLAY
METHOD FOR CONTROLLING DISPLAY
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METHOD FOR CONTROLLING DISPLAY

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18
METHODS OF TREATMENT, PREVENTION AND DIAGNOSIS
METHODS OF TREATMENT, PREVENTION AND DIAGNOSIS
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METHODS OF TREATMENT, PREVENTION AND DIAGNOSIS

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19
METHODS OF TREATMENT, PREVENTION AND DIAGNOSIS
METHODS OF TREATMENT, PREVENTION AND DIAGNOSIS
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METHODS OF TREATMENT, PREVENTION AND DIAGNOSIS

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20
Methods of treatment, prevention and diagnosis
Methods of treatment, prevention and diagnosis
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Methods of treatment, prevention and diagnosis

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