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1
MicroRNAs are minor constituents of extracellular vesicles that are rarely delivered to target cells
MicroRNAs are minor constituents of extracellular vesicles that are rarely delivered to target cells
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MicroRNAs are minor constituents of extracellular vesicles that are rarely delivered to target cells

PLoS genetics, 2021-12, Vol.17 (12), p.e1009951 [Peer Reviewed Journal]

COPYRIGHT 2021 Public Library of Science ;2021 Albanese et al 2021 Albanese et al ;ISSN: 1553-7404 ;ISSN: 1553-7390 ;EISSN: 1553-7404 ;DOI: 10.1371/journal.pgen.1009951 ;PMID: 34871319

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Furin cleavage of SARS-CoV-2 Spike promotes but is not essential for infection and cell-cell fusion
Furin cleavage of SARS-CoV-2 Spike promotes but is not essential for infection and cell-cell fusion
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Furin cleavage of SARS-CoV-2 Spike promotes but is not essential for infection and cell-cell fusion

PLoS pathogens, 2021-01, Vol.17 (1), p.e1009246-e1009246 [Peer Reviewed Journal]

COPYRIGHT 2021 Public Library of Science ;2021 Papa et al. This is an open access article distributed under the terms of the Creative Commons Attribution License: http://creativecommons.org/licenses/by/4.0/ (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2021 Papa et al 2021 Papa et al ;ISSN: 1553-7374 ;ISSN: 1553-7366 ;EISSN: 1553-7374 ;DOI: 10.1371/journal.ppat.1009246 ;PMID: 33493182

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3
Mitochondria are physiologically maintained at close to 50 °C
Mitochondria are physiologically maintained at close to 50 °C
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Mitochondria are physiologically maintained at close to 50 °C

PLoS biology, 2018-01, Vol.16 (1), p.e2003992-e2003992 [Peer Reviewed Journal]

2018 Public Library of Science. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited: Chrétien D, Bénit P, Ha H-H, Keipert S, El-Khoury R, Chang Y-T, et al. (2018) Mitochondria are physiologically maintained at close to 50 °C. PLoS Biol 16(1): e2003992. https://doi.org/10.1371/journal.pbio.2003992 ;Distributed under a Creative Commons Attribution 4.0 International License ;2018 Chrétien et al 2018 Chrétien et al ;2018 Public Library of Science. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited: Chrétien D, Bénit P, Ha H-H, Keipert S, El-Khoury R, Chang Y-T, et al. (2018) Mitochondria are physiologically maintained at close to 50 °C. PLoS Biol 16(1): e2003992. https://doi.org/10.1371/journal.pbio.2003992 ;ISSN: 1545-7885 ;ISSN: 1544-9173 ;EISSN: 1545-7885 ;DOI: 10.1371/journal.pbio.2003992 ;PMID: 29370167

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4
Identification of RNA-binding proteins in exosomes capable of interacting with different types of RNA: RBP-facilitated transport of RNAs into exosomes
Identification of RNA-binding proteins in exosomes capable of interacting with different types of RNA: RBP-facilitated transport of RNAs into exosomes
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Identification of RNA-binding proteins in exosomes capable of interacting with different types of RNA: RBP-facilitated transport of RNAs into exosomes

PloS one, 2018-04, Vol.13 (4), p.e0195969-e0195969 [Peer Reviewed Journal]

COPYRIGHT 2018 Public Library of Science ;COPYRIGHT 2018 Public Library of Science ;2018 Statello et al. This is an open access article distributed under the terms of the Creative Commons Attribution License: http://creativecommons.org/licenses/by/4.0/ (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2018 Statello et al 2018 Statello et al ;ISSN: 1932-6203 ;EISSN: 1932-6203 ;DOI: 10.1371/journal.pone.0195969 ;PMID: 29689087

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5
The SARS-CoV-2 Spike protein has a broad tropism for mammalian ACE2 proteins
The SARS-CoV-2 Spike protein has a broad tropism for mammalian ACE2 proteins
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The SARS-CoV-2 Spike protein has a broad tropism for mammalian ACE2 proteins

PLoS biology, 2020-12, Vol.18 (12), p.e3001016 [Peer Reviewed Journal]

2020 Conceicao et al. This is an open access article distributed under the terms of the Creative Commons Attribution License: http://creativecommons.org/licenses/by/4.0/ (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2020 Conceicao et al 2020 Conceicao et al ;ISSN: 1545-7885 ;ISSN: 1544-9173 ;EISSN: 1545-7885 ;DOI: 10.1371/journal.pbio.3001016 ;PMID: 33347434

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6
SARS-CoV-2 suppresses IFNβ production mediated by NSP1, 5, 6, 15, ORF6 and ORF7b but does not suppress the effects of added interferon
SARS-CoV-2 suppresses IFNβ production mediated by NSP1, 5, 6, 15, ORF6 and ORF7b but does not suppress the effects of added interferon
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SARS-CoV-2 suppresses IFNβ production mediated by NSP1, 5, 6, 15, ORF6 and ORF7b but does not suppress the effects of added interferon

PLoS pathogens, 2021-08, Vol.17 (8), p.e1009800-e1009800 [Peer Reviewed Journal]

2021 Shemesh et al 2021 Shemesh et al ;ISSN: 1553-7374 ;ISSN: 1553-7366 ;EISSN: 1553-7374 ;DOI: 10.1371/journal.ppat.1009800 ;PMID: 34437657

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7
SARS-CoV-2 variants with mutations at the S1/S2 cleavage site are generated in vitro during propagation in TMPRSS2-deficient cells
SARS-CoV-2 variants with mutations at the S1/S2 cleavage site are generated in vitro during propagation in TMPRSS2-deficient cells
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SARS-CoV-2 variants with mutations at the S1/S2 cleavage site are generated in vitro during propagation in TMPRSS2-deficient cells

PLoS pathogens, 2021-01, Vol.17 (1), p.e1009233-e1009233 [Peer Reviewed Journal]

COPYRIGHT 2021 Public Library of Science ;2021 Sasaki et al. This is an open access article distributed under the terms of the Creative Commons Attribution License: http://creativecommons.org/licenses/by/4.0/ (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2021 Sasaki et al 2021 Sasaki et al ;ISSN: 1553-7374 ;ISSN: 1553-7366 ;EISSN: 1553-7374 ;DOI: 10.1371/journal.ppat.1009233 ;PMID: 33476327

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8
Structure of mouse coronavirus spike protein complexed with receptor reveals mechanism for viral entry
Structure of mouse coronavirus spike protein complexed with receptor reveals mechanism for viral entry
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Structure of mouse coronavirus spike protein complexed with receptor reveals mechanism for viral entry

PLoS pathogens, 2020-03, Vol.16 (3), p.e1008392-e1008392 [Peer Reviewed Journal]

COPYRIGHT 2020 Public Library of Science ;COPYRIGHT 2020 Public Library of Science ;2020 Shang et al. This is an open access article distributed under the terms of the Creative Commons Attribution License: http://creativecommons.org/licenses/by/4.0/ (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2020 Shang et al 2020 Shang et al ;ISSN: 1553-7374 ;ISSN: 1553-7366 ;EISSN: 1553-7374 ;DOI: 10.1371/journal.ppat.1008392 ;PMID: 32150576

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9
Establishment of a well-characterized SARS-CoV-2 lentiviral pseudovirus neutralization assay using 293T cells with stable expression of ACE2 and TMPRSS2
Establishment of a well-characterized SARS-CoV-2 lentiviral pseudovirus neutralization assay using 293T cells with stable expression of ACE2 and TMPRSS2
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Establishment of a well-characterized SARS-CoV-2 lentiviral pseudovirus neutralization assay using 293T cells with stable expression of ACE2 and TMPRSS2

PloS one, 2021-03, Vol.16 (3), p.e0248348-e0248348 [Peer Reviewed Journal]

COPYRIGHT 2021 Public Library of Science ;COPYRIGHT 2021 Public Library of Science ;This is an open access article, free of all copyright, and may be freely reproduced, distributed, transmitted, modified, built upon, or otherwise used by anyone for any lawful purpose. The work is made available under the Creative Commons CC0 public domain dedication: https://creativecommons.org/publicdomain/zero/1.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;ISSN: 1932-6203 ;EISSN: 1932-6203 ;DOI: 10.1371/journal.pone.0248348 ;PMID: 33690649

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10
TDP-43 induces mitochondrial damage and activates the mitochondrial unfolded protein response
TDP-43 induces mitochondrial damage and activates the mitochondrial unfolded protein response
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TDP-43 induces mitochondrial damage and activates the mitochondrial unfolded protein response

PLoS genetics, 2019-05, Vol.15 (5), p.e1007947-e1007947 [Peer Reviewed Journal]

COPYRIGHT 2019 Public Library of Science ;COPYRIGHT 2019 Public Library of Science ;2019 Wang et al. This is an open access article distributed under the terms of the Creative Commons Attribution License: http://creativecommons.org/licenses/by/4.0/ (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2019 Wang et al 2019 Wang et al ;ISSN: 1553-7404 ;ISSN: 1553-7390 ;EISSN: 1553-7404 ;DOI: 10.1371/journal.pgen.1007947 ;PMID: 31100073

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11
An in-depth comparison of latent HIV-1 reactivation in multiple cell model systems and resting CD4+ T cells from aviremic patients
An in-depth comparison of latent HIV-1 reactivation in multiple cell model systems and resting CD4+ T cells from aviremic patients
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An in-depth comparison of latent HIV-1 reactivation in multiple cell model systems and resting CD4+ T cells from aviremic patients

PLoS pathogens, 2013-12, Vol.9 (12), p.e1003834-e1003834 [Peer Reviewed Journal]

COPYRIGHT 2013 Public Library of Science ;COPYRIGHT 2013 Public Library of Science ;2013 ;2013 Public Library of Science. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited: Citation: Spina CA, Anderson J, Archin NM, Bosque A, Chan J, et al. (2013) An In-Depth Comparison of Latent HIV-1 Reactivation in Multiple Cell Model Systems and Resting CD4+ T Cells from Aviremic Patients. PLoS Pathog 9(12): e1003834. doi:10.1371/journal.ppat.1003834 ;ISSN: 1553-7374 ;ISSN: 1553-7366 ;EISSN: 1553-7374 ;DOI: 10.1371/journal.ppat.1003834 ;PMID: 24385908

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12
Expression of the Long Non-Coding RNA HOTAIR Correlates with Disease Progression in Bladder Cancer and Is Contained in Bladder Cancer Patient Urinary Exosomes
Expression of the Long Non-Coding RNA HOTAIR Correlates with Disease Progression in Bladder Cancer and Is Contained in Bladder Cancer Patient Urinary Exosomes
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Expression of the Long Non-Coding RNA HOTAIR Correlates with Disease Progression in Bladder Cancer and Is Contained in Bladder Cancer Patient Urinary Exosomes

PloS one, 2016-01, Vol.11 (1), p.e0147236-e0147236 [Peer Reviewed Journal]

COPYRIGHT 2016 Public Library of Science ;COPYRIGHT 2016 Public Library of Science ;2016 Berrondo et al. This is an open access article distributed under the terms of the Creative Commons Attribution License: http://creativecommons.org/licenses/by/4.0/ (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2016 Berrondo et al 2016 Berrondo et al ;ISSN: 1932-6203 ;EISSN: 1932-6203 ;DOI: 10.1371/journal.pone.0147236 ;PMID: 26800519

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13
Oligodendrocytes support axonal transport and maintenance via exosome secretion
Oligodendrocytes support axonal transport and maintenance via exosome secretion
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Oligodendrocytes support axonal transport and maintenance via exosome secretion

PLoS biology, 2020-12, Vol.18 (12), p.e3000621-e3000621 [Peer Reviewed Journal]

2020 Frühbeis et al. This is an open access article distributed under the terms of the Creative Commons Attribution License: http://creativecommons.org/licenses/by/4.0/ (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2020 Frühbeis et al 2020 Frühbeis et al ;ISSN: 1545-7885 ;ISSN: 1544-9173 ;EISSN: 1545-7885 ;DOI: 10.1371/journal.pbio.3000621 ;PMID: 33351792

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14
β-Amyloid precursor protein does not possess ferroxidase activity but does stabilize the cell surface ferrous iron exporter ferroportin
β-Amyloid precursor protein does not possess ferroxidase activity but does stabilize the cell surface ferrous iron exporter ferroportin
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β-Amyloid precursor protein does not possess ferroxidase activity but does stabilize the cell surface ferrous iron exporter ferroportin

PloS one, 2014-12, Vol.9 (12), p.e114174-e114174 [Peer Reviewed Journal]

2014 Wong et al. This is an open-access article distributed under the terms of the Creative Commons Attribution License: http://creativecommons.org/licenses/by/4.0/ (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2014 Wong et al 2014 Wong et al ;ISSN: 1932-6203 ;EISSN: 1932-6203 ;DOI: 10.1371/journal.pone.0114174 ;PMID: 25464026

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15
DENV inhibits type I IFN production in infected cells by cleaving human STING
DENV inhibits type I IFN production in infected cells by cleaving human STING
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DENV inhibits type I IFN production in infected cells by cleaving human STING

PLoS pathogens, 2012-10, Vol.8 (10), p.e1002934-e1002934 [Peer Reviewed Journal]

COPYRIGHT 2012 Public Library of Science ;COPYRIGHT 2012 Public Library of Science ;Aguirre et al. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited: Aguirre S, Maestre AM, Pagni S, Patel JR, Savage T, et al. (2012) DENV Inhibits Type I IFN Production in Infected Cells by Cleaving Human STING. PLoS Pathog 8(10): e1002934. doi:10.1371/journal.ppat.1002934 ;2012 Aguirre et al 2012 Aguirre et al ;ISSN: 1553-7374 ;ISSN: 1553-7366 ;EISSN: 1553-7374 ;DOI: 10.1371/journal.ppat.1002934 ;PMID: 23055924

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16
Alu elements in ANRIL non-coding RNA at chromosome 9p21 modulate atherogenic cell functions through trans-regulation of gene networks
Alu elements in ANRIL non-coding RNA at chromosome 9p21 modulate atherogenic cell functions through trans-regulation of gene networks
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Alu elements in ANRIL non-coding RNA at chromosome 9p21 modulate atherogenic cell functions through trans-regulation of gene networks

PLoS genetics, 2013-07, Vol.9 (7), p.e1003588 [Peer Reviewed Journal]

COPYRIGHT 2013 Public Library of Science ;COPYRIGHT 2013 Public Library of Science ;2013 Holdt et al 2013 Holdt et al ;2013 Holdt et al. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited: Holdt LM, Hoffmann S, Sass K, Langenberger D, Scholz M, et al. (2013) Alu Elements in ANRIL Non-Coding RNA at Chromosome 9p21 Modulate Atherogenic Cell Functions through Trans-Regulation of Gene Networks. PLoS Genet 9(7): e1003588. doi:10.1371/journal.pgen.1003588 ;ISSN: 1553-7404 ;ISSN: 1553-7390 ;EISSN: 1553-7404 ;DOI: 10.1371/journal.pgen.1003588 ;PMID: 23861667

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17
Functional and Evolutionary Analyses Identify Proteolysis as a General Mechanism for NLRP1 Inflammasome Activation
Functional and Evolutionary Analyses Identify Proteolysis as a General Mechanism for NLRP1 Inflammasome Activation
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Functional and Evolutionary Analyses Identify Proteolysis as a General Mechanism for NLRP1 Inflammasome Activation

PLoS pathogens, 2016-12, Vol.12 (12), p.e1006052-e1006052 [Peer Reviewed Journal]

COPYRIGHT 2016 Public Library of Science ;COPYRIGHT 2016 Public Library of Science ;2016 Chavarría-Smith et al 2016 Chavarría-Smith et al ;ISSN: 1553-7374 ;ISSN: 1553-7366 ;EISSN: 1553-7374 ;DOI: 10.1371/journal.ppat.1006052 ;PMID: 27926929

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18
TRIM25 Enhances the Antiviral Action of Zinc-Finger Antiviral Protein (ZAP)
TRIM25 Enhances the Antiviral Action of Zinc-Finger Antiviral Protein (ZAP)
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TRIM25 Enhances the Antiviral Action of Zinc-Finger Antiviral Protein (ZAP)

PLoS pathogens, 2017-01, Vol.13 (1), p.e1006145-e1006145 [Peer Reviewed Journal]

COPYRIGHT 2017 Public Library of Science ;COPYRIGHT 2017 Public Library of Science ;2017 Public Library of Science. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited: Li MMH, Lau Z, Cheung P, Aguilar EG, Schneider WM, Bozzacco L, et al. (2017) TRIM25 Enhances the Antiviral Action of Zinc-Finger Antiviral Protein (ZAP). PLoS Pathog 13(1): e1006145. doi:10.1371/journal.ppat.1006145 ;2017 Public Library of Science. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited: Li MMH, Lau Z, Cheung P, Aguilar EG, Schneider WM, Bozzacco L, et al. (2017) TRIM25 Enhances the Antiviral Action of Zinc-Finger Antiviral Protein (ZAP). PLoS Pathog 13(1): e1006145. doi:10.1371/journal.ppat.1006145 ;ISSN: 1553-7374 ;ISSN: 1553-7366 ;EISSN: 1553-7374 ;DOI: 10.1371/journal.ppat.1006145 ;PMID: 28060952

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19
In vitro glycoengineering of IgG1 and its effect on Fc receptor binding and ADCC activity
In vitro glycoengineering of IgG1 and its effect on Fc receptor binding and ADCC activity
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In vitro glycoengineering of IgG1 and its effect on Fc receptor binding and ADCC activity

PloS one, 2015-08, Vol.10 (8), p.e0134949-e0134949 [Peer Reviewed Journal]

2015 Thomann et al. This is an open access article distributed under the terms of the Creative Commons Attribution License: http://creativecommons.org/licenses/by/4.0/ (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2015 Thomann et al 2015 Thomann et al ;ISSN: 1932-6203 ;EISSN: 1932-6203 ;DOI: 10.1371/journal.pone.0134949 ;PMID: 26266936

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20
Real-time resolution studies of the regulation of lactate production by hexokinases binding to mitochondria in single cells
Real-time resolution studies of the regulation of lactate production by hexokinases binding to mitochondria in single cells
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Real-time resolution studies of the regulation of lactate production by hexokinases binding to mitochondria in single cells

PloS one, 2024-03, Vol.19 (3), p.e0300150-e0300150 [Peer Reviewed Journal]

Copyright: © 2024 John et al. This is an open access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. ;COPYRIGHT 2024 Public Library of Science ;ISSN: 1932-6203 ;EISSN: 1932-6203 ;DOI: 10.1371/journal.pone.0300150 ;PMID: 38457438

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