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1
Calcium handling in human induced pluripotent stem cell derived cardiomyocytes
Calcium handling in human induced pluripotent stem cell derived cardiomyocytes
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Calcium handling in human induced pluripotent stem cell derived cardiomyocytes

PloS one, 2011-04, Vol.6 (4), p.e18037-e18037 [Peer Reviewed Journal]

COPYRIGHT 2011 Public Library of Science ;COPYRIGHT 2011 Public Library of Science ;2011 Itzhaki et al. This is an open-access article distributed under the terms of the Creative Commons Attribution License: https://creativecommons.org/licenses/by/4.0/ (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;Itzhaki et al. 2011 ;ISSN: 1932-6203 ;EISSN: 1932-6203 ;DOI: 10.1371/journal.pone.0018037 ;PMID: 21483779

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2
MICU2, a paralog of MICU1, resides within the mitochondrial uniporter complex to regulate calcium handling
MICU2, a paralog of MICU1, resides within the mitochondrial uniporter complex to regulate calcium handling
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MICU2, a paralog of MICU1, resides within the mitochondrial uniporter complex to regulate calcium handling

PloS one, 2013-02, Vol.8 (2), p.e55785-e55785 [Peer Reviewed Journal]

COPYRIGHT 2013 Public Library of Science ;COPYRIGHT 2013 Public Library of Science ;2013 Plovanich et al. This is an open-access article distributed under the terms of the Creative Commons Attribution License: https://creativecommons.org/licenses/by/4.0/ (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2013 Plovanich et al 2013 Plovanich et al ;ISSN: 1932-6203 ;EISSN: 1932-6203 ;DOI: 10.1371/journal.pone.0055785 ;PMID: 23409044

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3
Quantification of the calcium signaling deficit in muscles devoid of triadin
Quantification of the calcium signaling deficit in muscles devoid of triadin
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Quantification of the calcium signaling deficit in muscles devoid of triadin

PloS one, 2022-02, Vol.17 (2), p.e0264146-e0264146 [Peer Reviewed Journal]

COPYRIGHT 2022 Public Library of Science ;2022 Manno et al. This is an open access article distributed under the terms of the Creative Commons Attribution License: http://creativecommons.org/licenses/by/4.0/ (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2022 Manno et al 2022 Manno et al ;ISSN: 1932-6203 ;EISSN: 1932-6203 ;DOI: 10.1371/journal.pone.0264146 ;PMID: 35213584

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4
Impact of Increasing Dietary Calcium Levels on Calcium Excretion and Vitamin D Metabolites in the Blood of Healthy Adult Cats
Impact of Increasing Dietary Calcium Levels on Calcium Excretion and Vitamin D Metabolites in the Blood of Healthy Adult Cats
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Impact of Increasing Dietary Calcium Levels on Calcium Excretion and Vitamin D Metabolites in the Blood of Healthy Adult Cats

PloS one, 2016-02, Vol.11 (2), p.e0149190-e0149190 [Peer Reviewed Journal]

COPYRIGHT 2016 Public Library of Science ;COPYRIGHT 2016 Public Library of Science ;2016 Paßlack et al. This is an open access article distributed under the terms of the Creative Commons Attribution License: http://creativecommons.org/licenses/by/4.0/ (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2016 Paßlack et al 2016 Paßlack et al ;ISSN: 1932-6203 ;EISSN: 1932-6203 ;DOI: 10.1371/journal.pone.0149190 ;PMID: 26870965

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5
Upregulation of SK3 and IK1 channels contributes to the enhanced endothelial calcium signaling and the preserved coronary relaxation in obese Zucker rats
Upregulation of SK3 and IK1 channels contributes to the enhanced endothelial calcium signaling and the preserved coronary relaxation in obese Zucker rats
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Upregulation of SK3 and IK1 channels contributes to the enhanced endothelial calcium signaling and the preserved coronary relaxation in obese Zucker rats

PloS one, 2014-10, Vol.9 (10), p.e109432-e109432 [Peer Reviewed Journal]

COPYRIGHT 2014 Public Library of Science ;COPYRIGHT 2014 Public Library of Science ;2014 Climent et al. This is an open-access article distributed under the terms of the Creative Commons Attribution License: http://creativecommons.org/licenses/by/4.0/ (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2014 Climent et al 2014 Climent et al ;ISSN: 1932-6203 ;EISSN: 1932-6203 ;DOI: 10.1371/journal.pone.0109432 ;PMID: 25302606

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6
Calmodulin Interacts with the Sodium/Calcium Exchanger NCX1 to Regulate Activity
Calmodulin Interacts with the Sodium/Calcium Exchanger NCX1 to Regulate Activity
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Calmodulin Interacts with the Sodium/Calcium Exchanger NCX1 to Regulate Activity

PloS one, 2015-09, Vol.10 (9), p.e0138856-e0138856 [Peer Reviewed Journal]

COPYRIGHT 2015 Public Library of Science ;COPYRIGHT 2015 Public Library of Science ;2015 Chou et al. This is an open access article distributed under the terms of the Creative Commons Attribution License: http://creativecommons.org/licenses/by/4.0/ (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2015 Chou et al 2015 Chou et al ;ISSN: 1932-6203 ;EISSN: 1932-6203 ;DOI: 10.1371/journal.pone.0138856 ;PMID: 26421717

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7
Characterisation of connexin expression and electrophysiological properties in stable clones of the HL-1 myocyte cell line
Characterisation of connexin expression and electrophysiological properties in stable clones of the HL-1 myocyte cell line
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Characterisation of connexin expression and electrophysiological properties in stable clones of the HL-1 myocyte cell line

PloS one, 2014-02, Vol.9 (2), p.e90266-e90266 [Peer Reviewed Journal]

COPYRIGHT 2014 Public Library of Science ;COPYRIGHT 2014 Public Library of Science ;2014 Dias et al. This is an open access article distributed under the terms of the Creative Commons Attribution License: http://creativecommons.org/licenses/by/4.0/ (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2014 Dias et al 2014 Dias et al ;ISSN: 1932-6203 ;EISSN: 1932-6203 ;DOI: 10.1371/journal.pone.0090266 ;PMID: 24587307

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8
Mitochondrial calcium uniporter MCU supports cytoplasmic Ca2+ oscillations, store-operated Ca2+ entry and Ca2+-dependent gene expression in response to receptor stimulation
Mitochondrial calcium uniporter MCU supports cytoplasmic Ca2+ oscillations, store-operated Ca2+ entry and Ca2+-dependent gene expression in response to receptor stimulation
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Mitochondrial calcium uniporter MCU supports cytoplasmic Ca2+ oscillations, store-operated Ca2+ entry and Ca2+-dependent gene expression in response to receptor stimulation

PloS one, 2014-07, Vol.9 (7), p.e101188-e101188 [Peer Reviewed Journal]

2014 Samanta et al. This is an open-access article distributed under the terms of the Creative Commons Attribution License: http://creativecommons.org/licenses/by/4.0/ (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2014 Samanta et al 2014 Samanta et al ;ISSN: 1932-6203 ;EISSN: 1932-6203 ;DOI: 10.1371/journal.pone.0101188 ;PMID: 25004162

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9
CaMKII activation participates in doxorubicin cardiotoxicity and is attenuated by moderate GRP78 overexpression
CaMKII activation participates in doxorubicin cardiotoxicity and is attenuated by moderate GRP78 overexpression
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CaMKII activation participates in doxorubicin cardiotoxicity and is attenuated by moderate GRP78 overexpression

PloS one, 2019-04, Vol.14 (4), p.e0215992 [Peer Reviewed Journal]

COPYRIGHT 2019 Public Library of Science ;COPYRIGHT 2019 Public Library of Science ;2019 Tscheschner et al. This is an open access article distributed under the terms of the Creative Commons Attribution License: http://creativecommons.org/licenses/by/4.0/ (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2019 Tscheschner et al 2019 Tscheschner et al ;ISSN: 1932-6203 ;EISSN: 1932-6203 ;DOI: 10.1371/journal.pone.0215992 ;PMID: 31034488

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10
Excessive signal transduction of gain-of-function variants of the calcium-sensing receptor (CaSR) are associated with increased ER to cytosol calcium gradient
Excessive signal transduction of gain-of-function variants of the calcium-sensing receptor (CaSR) are associated with increased ER to cytosol calcium gradient
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Excessive signal transduction of gain-of-function variants of the calcium-sensing receptor (CaSR) are associated with increased ER to cytosol calcium gradient

PloS one, 2013-11, Vol.8 (11), p.e79113 [Peer Reviewed Journal]

COPYRIGHT 2013 Public Library of Science ;COPYRIGHT 2013 Public Library of Science ;2013 Ranieri et al. This is an open-access article distributed under the terms of the Creative Commons Attribution License: http://creativecommons.org/licenses/by/3.0/ (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2013 Ranieri et al 2013 Ranieri et al ;ISSN: 1932-6203 ;EISSN: 1932-6203 ;DOI: 10.1371/journal.pone.0079113 ;PMID: 24244430

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11
A 3D diffusional-compartmental model of the calcium dynamics in cytosol, sarcoplasmic reticulum and mitochondria of murine skeletal muscle fibers
A 3D diffusional-compartmental model of the calcium dynamics in cytosol, sarcoplasmic reticulum and mitochondria of murine skeletal muscle fibers
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A 3D diffusional-compartmental model of the calcium dynamics in cytosol, sarcoplasmic reticulum and mitochondria of murine skeletal muscle fibers

PloS one, 2018-07, Vol.13 (7), p.e0201050-e0201050 [Peer Reviewed Journal]

COPYRIGHT 2018 Public Library of Science ;COPYRIGHT 2018 Public Library of Science ;2018 Marcucci et al. This is an open access article distributed under the terms of the Creative Commons Attribution License: http://creativecommons.org/licenses/by/4.0/ (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2018 Marcucci et al 2018 Marcucci et al ;ISSN: 1932-6203 ;EISSN: 1932-6203 ;DOI: 10.1371/journal.pone.0201050 ;PMID: 30048500

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12
Characterization of intracellular calcium mobilization induced by remimazolam, a newly approved intravenous anesthetic
Characterization of intracellular calcium mobilization induced by remimazolam, a newly approved intravenous anesthetic
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Characterization of intracellular calcium mobilization induced by remimazolam, a newly approved intravenous anesthetic

PloS one, 2022-02, Vol.17 (2), p.e0263395-e0263395 [Peer Reviewed Journal]

COPYRIGHT 2022 Public Library of Science ;2022 Urabe et al. This is an open access article distributed under the terms of the Creative Commons Attribution License: http://creativecommons.org/licenses/by/4.0/ (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2022 Urabe et al 2022 Urabe et al ;ISSN: 1932-6203 ;EISSN: 1932-6203 ;DOI: 10.1371/journal.pone.0263395 ;PMID: 35104283

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13
Elevation of extracellular Ca2+ induces store-operated calcium entry via calcium-sensing receptors: a pathway contributes to the proliferation of osteoblasts
Elevation of extracellular Ca2+ induces store-operated calcium entry via calcium-sensing receptors: a pathway contributes to the proliferation of osteoblasts
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Elevation of extracellular Ca2+ induces store-operated calcium entry via calcium-sensing receptors: a pathway contributes to the proliferation of osteoblasts

PloS one, 2014-09, Vol.9 (9), p.e107217-e107217 [Peer Reviewed Journal]

2014 Hu et al. This is an open-access article distributed under the terms of the Creative Commons Attribution License: http://creativecommons.org/licenses/by/4.0/ (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2014 Hu et al 2014 Hu et al ;ISSN: 1932-6203 ;EISSN: 1932-6203 ;DOI: 10.1371/journal.pone.0107217 ;PMID: 25254954

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14
Protective effects of voltage-gated calcium channel antagonists against zinc toxicity in SN56 neuroblastoma cholinergic cells
Protective effects of voltage-gated calcium channel antagonists against zinc toxicity in SN56 neuroblastoma cholinergic cells
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Protective effects of voltage-gated calcium channel antagonists against zinc toxicity in SN56 neuroblastoma cholinergic cells

PloS one, 2018-12, Vol.13 (12), p.e0209363 [Peer Reviewed Journal]

COPYRIGHT 2018 Public Library of Science ;COPYRIGHT 2018 Public Library of Science ;2018 Zyśk et al. This is an open access article distributed under the terms of the Creative Commons Attribution License: http://creativecommons.org/licenses/by/4.0/ (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2018 Zyśk et al 2018 Zyśk et al ;ISSN: 1932-6203 ;EISSN: 1932-6203 ;DOI: 10.1371/journal.pone.0209363 ;PMID: 30571745

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15
RyR2 modulates a Ca2+-activated K+ current in mouse cardiac myocytes
RyR2 modulates a Ca2+-activated K+ current in mouse cardiac myocytes
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RyR2 modulates a Ca2+-activated K+ current in mouse cardiac myocytes

PloS one, 2014-04, Vol.9 (4), p.e94905-e94905 [Peer Reviewed Journal]

2014 Mu et al. This is an open-access article distributed under the terms of the Creative Commons Attribution License: http://creativecommons.org/licenses/by/4.0/ (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2014 Mu et al 2014 Mu et al ;ISSN: 1932-6203 ;EISSN: 1932-6203 ;DOI: 10.1371/journal.pone.0094905 ;PMID: 24747296

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16
A vesicular Na+/Ca2+ exchanger in coral calcifying cells
A vesicular Na+/Ca2+ exchanger in coral calcifying cells
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A vesicular Na+/Ca2+ exchanger in coral calcifying cells

PloS one, 2018-10, Vol.13 (10), p.e0205367-e0205367 [Peer Reviewed Journal]

2018 Barron et al. This is an open access article distributed under the terms of the Creative Commons Attribution License: http://creativecommons.org/licenses/by/4.0/ (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2018 Barron et al 2018 Barron et al ;ISSN: 1932-6203 ;EISSN: 1932-6203 ;DOI: 10.1371/journal.pone.0205367 ;PMID: 30379874

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17
Regulatory Action of Calcium Ion on Cyclic AMP-Enhanced Expression of Implantation-Related Factors in Human Endometrial Cells
Regulatory Action of Calcium Ion on Cyclic AMP-Enhanced Expression of Implantation-Related Factors in Human Endometrial Cells
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Regulatory Action of Calcium Ion on Cyclic AMP-Enhanced Expression of Implantation-Related Factors in Human Endometrial Cells

PloS one, 2015-07, Vol.10 (7), p.e0132017-e0132017 [Peer Reviewed Journal]

COPYRIGHT 2015 Public Library of Science ;COPYRIGHT 2015 Public Library of Science ;2015 Kusama et al. This is an open access article distributed under the terms of the Creative Commons Attribution License: http://creativecommons.org/licenses/by/4.0/ (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2015 Kusama et al 2015 Kusama et al ;ISSN: 1932-6203 ;EISSN: 1932-6203 ;DOI: 10.1371/journal.pone.0132017 ;PMID: 26161798

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18
Modulation of intracellular calcium waves and triggered activities by mitochondrial ca flux in mouse cardiomyocytes
Modulation of intracellular calcium waves and triggered activities by mitochondrial ca flux in mouse cardiomyocytes
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Modulation of intracellular calcium waves and triggered activities by mitochondrial ca flux in mouse cardiomyocytes

PloS one, 2013-11, Vol.8 (11), p.e80574 [Peer Reviewed Journal]

COPYRIGHT 2013 Public Library of Science ;COPYRIGHT 2013 Public Library of Science ;2013 Zhao et al. This is an open-access article distributed under the terms of the Creative Commons Attribution License: https://creativecommons.org/licenses/by/4.0/ (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2013 Zhao et al 2013 Zhao et al ;ISSN: 1932-6203 ;EISSN: 1932-6203 ;DOI: 10.1371/journal.pone.0080574 ;PMID: 24348912

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19
Serotonin 5-HT3 receptor-mediated vomiting occurs via the activation of Ca2+/CaMKII-dependent ERK1/2 signaling in the least shrew (Cryptotis parva)
Serotonin 5-HT3 receptor-mediated vomiting occurs via the activation of Ca2+/CaMKII-dependent ERK1/2 signaling in the least shrew (Cryptotis parva)
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Serotonin 5-HT3 receptor-mediated vomiting occurs via the activation of Ca2+/CaMKII-dependent ERK1/2 signaling in the least shrew (Cryptotis parva)

PloS one, 2014-08, Vol.9 (8), p.e104718-e104718 [Peer Reviewed Journal]

2014 Zhong et al. This is an open-access article distributed under the terms of the Creative Commons Attribution License: http://creativecommons.org/licenses/by/4.0/ (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2014 Zhong et al 2014 Zhong et al ;ISSN: 1932-6203 ;EISSN: 1932-6203 ;DOI: 10.1371/journal.pone.0104718 ;PMID: 25121483

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20
Kokumi substances, enhancers of basic tastes, induce responses in calcium-sensing receptor expressing taste cells
Kokumi substances, enhancers of basic tastes, induce responses in calcium-sensing receptor expressing taste cells
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Kokumi substances, enhancers of basic tastes, induce responses in calcium-sensing receptor expressing taste cells

PloS one, 2012-04, Vol.7 (4), p.e34489-e34489 [Peer Reviewed Journal]

COPYRIGHT 2012 Public Library of Science ;COPYRIGHT 2012 Public Library of Science ;2012 Maruyama et al. This is an open-access article distributed under the terms of the Creative Commons Attribution License: https://creativecommons.org/licenses/by/4.0/ (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;Maruyama et al. 2012 ;ISSN: 1932-6203 ;EISSN: 1932-6203 ;DOI: 10.1371/journal.pone.0034489 ;PMID: 22511946

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