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1
Acute and chronic traumatic encephalopathies: pathogenesis and biomarkers
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Acute and chronic traumatic encephalopathies: pathogenesis and biomarkers

Nature reviews. Neurology, 2013-04, Vol.9 (4), p.192-200 [Peer Reviewed Journal]

COPYRIGHT 2013 Nature Publishing Group ;COPYRIGHT 2013 Nature Publishing Group ;Copyright Nature Publishing Group Apr 2013 ;2013 Macmillan Publishers Limited. All rights reserved 2013 ;ISSN: 1759-4758 ;ISSN: 1759-4766 ;EISSN: 1759-4766 ;DOI: 10.1038/nrneurol.2013.36 ;PMID: 23558985

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2
Antibody against early driver of neurodegeneration cis P-tau blocks brain injury and tauopathy
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Antibody against early driver of neurodegeneration cis P-tau blocks brain injury and tauopathy

Nature (London), 2015-07, Vol.523 (7561), p.431-436 [Peer Reviewed Journal]

COPYRIGHT 2015 Nature Publishing Group ;COPYRIGHT 2015 Nature Publishing Group ;Copyright Nature Publishing Group Jul 23, 2015 ;ISSN: 0028-0836 ;EISSN: 1476-4687 ;DOI: 10.1038/nature14658 ;PMID: 26176913 ;CODEN: NATUAS

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3
Comparative Assessment of the Prognostic Value of Biomarkers in Traumatic Brain Injury Reveals an Independent Role for Serum Levels of Neurofilament Light
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Comparative Assessment of the Prognostic Value of Biomarkers in Traumatic Brain Injury Reveals an Independent Role for Serum Levels of Neurofilament Light

PloS one, 2015-07, Vol.10 (7), p.e0132177-e0132177 [Peer Reviewed Journal]

COPYRIGHT 2015 Public Library of Science ;COPYRIGHT 2015 Public Library of Science ;2015 Al Nimer et al. This is an open access article distributed under the terms of the Creative Commons Attribution License: http://creativecommons.org/licenses/by/4.0/ (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2015 Al Nimer et al 2015 Al Nimer et al ;ISSN: 1932-6203 ;EISSN: 1932-6203 ;DOI: 10.1371/journal.pone.0132177 ;PMID: 26136237

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4
The NLRP3 inflammasome in traumatic brain injury: potential as a biomarker and therapeutic target
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The NLRP3 inflammasome in traumatic brain injury: potential as a biomarker and therapeutic target

Journal of neuroinflammation, 2020-04, Vol.17 (1), p.104-104, Article 104 [Peer Reviewed Journal]

COPYRIGHT 2020 BioMed Central Ltd. ;2020. This work is licensed under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2020 ;ISSN: 1742-2094 ;EISSN: 1742-2094 ;DOI: 10.1186/s12974-020-01778-5 ;PMID: 32252777

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5
Apelin-13/APJ system attenuates early brain injury via suppression of endoplasmic reticulum stress-associated TXNIP/NLRP3 inflammasome activation and oxidative stress in a AMPK-dependent manner after subarachnoid hemorrhage in rats
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Apelin-13/APJ system attenuates early brain injury via suppression of endoplasmic reticulum stress-associated TXNIP/NLRP3 inflammasome activation and oxidative stress in a AMPK-dependent manner after subarachnoid hemorrhage in rats

Journal of neuroinflammation, 2019-12, Vol.16 (1), p.247-247, Article 247 [Peer Reviewed Journal]

COPYRIGHT 2019 BioMed Central Ltd. ;2019. This work is licensed under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s). 2019 ;ISSN: 1742-2094 ;EISSN: 1742-2094 ;DOI: 10.1186/s12974-019-1620-3 ;PMID: 31791369

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6
Comparison of therapeutic effects between pulsed and continuous wave 810-nm wavelength laser irradiation for traumatic brain injury in mice
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Comparison of therapeutic effects between pulsed and continuous wave 810-nm wavelength laser irradiation for traumatic brain injury in mice

PloS one, 2011-10, Vol.6 (10), p.e26212-e26212 [Peer Reviewed Journal]

COPYRIGHT 2011 Public Library of Science ;COPYRIGHT 2011 Public Library of Science ;2011 Ando et al. This is an open-access article distributed under the terms of the Creative Commons Attribution License: https://creativecommons.org/licenses/by/4.0/ (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;Ando et al. 2011 ;ISSN: 1932-6203 ;EISSN: 1932-6203 ;DOI: 10.1371/journal.pone.0026212 ;PMID: 22028832

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7
Activin receptors regulate the oligodendrocyte lineage in health and disease
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Activin receptors regulate the oligodendrocyte lineage in health and disease

Acta neuropathologica, 2018-06, Vol.135 (6), p.887-906 [Peer Reviewed Journal]

The Author(s) 2018 ;COPYRIGHT 2018 Springer ;Acta Neuropathologica is a copyright of Springer, (2018). All Rights Reserved. ;ISSN: 0001-6322 ;EISSN: 1432-0533 ;DOI: 10.1007/s00401-018-1813-3 ;PMID: 29397421

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8
Towards further progress in traumatic brain injury – Authors' reply
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Towards further progress in traumatic brain injury – Authors' reply

Lancet neurology, 2023-02, Vol.22 (2), p.110-110 [Peer Reviewed Journal]

2023 Elsevier Ltd ;2023. Elsevier Ltd ;ISSN: 1474-4422 ;EISSN: 1474-4465 ;DOI: 10.1016/S1474-4422(22)00516-6 ;PMID: 36681441

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9
Anti-inflammatory and immunomodulatory mechanisms of atorvastatin in a murine model of traumatic brain injury
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Anti-inflammatory and immunomodulatory mechanisms of atorvastatin in a murine model of traumatic brain injury

Journal of neuroinflammation, 2017-08, Vol.14 (1), p.167-167, Article 167 [Peer Reviewed Journal]

COPYRIGHT 2017 BioMed Central Ltd. ;The Author(s). 2017 ;ISSN: 1742-2094 ;EISSN: 1742-2094 ;DOI: 10.1186/s12974-017-0934-2 ;PMID: 28835272

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10
Aucubin alleviates oxidative stress and inflammation via Nrf2-mediated signaling activity in experimental traumatic brain injury
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Aucubin alleviates oxidative stress and inflammation via Nrf2-mediated signaling activity in experimental traumatic brain injury

Journal of neuroinflammation, 2020-06, Vol.17 (1), p.188-188, Article 188 [Peer Reviewed Journal]

COPYRIGHT 2020 BioMed Central Ltd. ;2020. This work is licensed under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2020 ;ISSN: 1742-2094 ;EISSN: 1742-2094 ;DOI: 10.1186/s12974-020-01863-9 ;PMID: 32539839

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11
Human bone marrow mesenchymal stem cell-derived extracellular vesicles attenuate neuroinflammation evoked by focal brain injury in rats
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Human bone marrow mesenchymal stem cell-derived extracellular vesicles attenuate neuroinflammation evoked by focal brain injury in rats

Journal of neuroinflammation, 2019-11, Vol.16 (1), p.216-216, Article 216 [Peer Reviewed Journal]

COPYRIGHT 2019 BioMed Central Ltd. ;The Author(s). 2019 ;ISSN: 1742-2094 ;EISSN: 1742-2094 ;DOI: 10.1186/s12974-019-1602-5 ;PMID: 31722731

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12
Intranasal Chromium Induces Acute Brain and Lung Injuries in Rats: Assessment of Different Potential Hazardous Effects of Environmental and Occupational Exposure to Chromium and Introduction of a Novel Pharmacological and Toxicological Animal Model
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Intranasal Chromium Induces Acute Brain and Lung Injuries in Rats: Assessment of Different Potential Hazardous Effects of Environmental and Occupational Exposure to Chromium and Introduction of a Novel Pharmacological and Toxicological Animal Model

PloS one, 2016-12, Vol.11 (12), p.e0168688 [Peer Reviewed Journal]

COPYRIGHT 2016 Public Library of Science ;COPYRIGHT 2016 Public Library of Science ;2016 Salama et al. This is an open access article distributed under the terms of the Creative Commons Attribution License: http://creativecommons.org/licenses/by/4.0/ (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2016 Salama et al 2016 Salama et al ;ISSN: 1932-6203 ;EISSN: 1932-6203 ;DOI: 10.1371/journal.pone.0168688 ;PMID: 27997619

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13
Uncovering precision phenotype-biomarker associations in traumatic brain injury using topological data analysis
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Uncovering precision phenotype-biomarker associations in traumatic brain injury using topological data analysis

PloS one, 2017-03, Vol.12 (3), p.e0169490-e0169490 [Peer Reviewed Journal]

COPYRIGHT 2017 Public Library of Science ;COPYRIGHT 2017 Public Library of Science ;2017 Nielson et al. This is an open access article distributed under the terms of the Creative Commons Attribution License: http://creativecommons.org/licenses/by/4.0/ (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2017 Nielson et al 2017 Nielson et al ;ISSN: 1932-6203 ;EISSN: 1932-6203 ;DOI: 10.1371/journal.pone.0169490 ;PMID: 28257413

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14
Preventing neuronal edema increases network excitability after traumatic brain injury
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Preventing neuronal edema increases network excitability after traumatic brain injury

The Journal of clinical investigation, 2020-11, Vol.130 (11), p.6005-6020 [Peer Reviewed Journal]

COPYRIGHT 2020 American Society for Clinical Investigation ;COPYRIGHT 2020 American Society for Clinical Investigation ;Copyright American Society for Clinical Investigation Nov 2020 ;2020 American Society for Clinical Investigation 2020 American Society for Clinical Investigation ;ISSN: 0021-9738 ;EISSN: 1558-8238 ;DOI: 10.1172/JCI134793 ;PMID: 33044227

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15
Mechanisms and therapeutic implications of RTA 408, an activator of Nrf2, in subarachnoid hemorrhage-induced delayed cerebral vasospasm and secondary brain injury
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Mechanisms and therapeutic implications of RTA 408, an activator of Nrf2, in subarachnoid hemorrhage-induced delayed cerebral vasospasm and secondary brain injury

PloS one, 2020-10, Vol.15 (10), p.e0240122-e0240122 [Peer Reviewed Journal]

COPYRIGHT 2020 Public Library of Science ;2020 Tsai et al. This is an open access article distributed under the terms of the Creative Commons Attribution License: http://creativecommons.org/licenses/by/4.0/ (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2020 Tsai et al 2020 Tsai et al ;ISSN: 1932-6203 ;EISSN: 1932-6203 ;DOI: 10.1371/journal.pone.0240122 ;PMID: 33017422

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16
Trends in traumatic brain injury mortality in China, 2006-2013: A population-based longitudinal study
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Trends in traumatic brain injury mortality in China, 2006-2013: A population-based longitudinal study

PLoS medicine, 2017-07, Vol.14 (7), p.e1002332-e1002332 [Peer Reviewed Journal]

COPYRIGHT 2017 Public Library of Science ;COPYRIGHT 2017 Public Library of Science ;2017 Public Library of Science. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited: Cheng P, Yin P, Ning P, Wang L, Cheng X, Liu Y, et al. (2017) Trends in traumatic brain injury mortality in China, 2006-2013: A population-based longitudinal study. PLoS Med 14(7): e1002332. https://doi.org/10.1371/journal.pmed.1002332 ;2017 Cheng et al 2017 Cheng et al ;2017 Public Library of Science. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited: Cheng P, Yin P, Ning P, Wang L, Cheng X, Liu Y, et al. (2017) Trends in traumatic brain injury mortality in China, 2006-2013: A population-based longitudinal study. PLoS Med 14(7): e1002332. https://doi.org/10.1371/journal.pmed.1002332 ;ISSN: 1549-1676 ;ISSN: 1549-1277 ;EISSN: 1549-1676 ;DOI: 10.1371/journal.pmed.1002332 ;PMID: 28700591

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17
Astrocyte-derived exosomes enriched with miR-873a-5p inhibit neuroinflammation via microglia phenotype modulation after traumatic brain injury
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Astrocyte-derived exosomes enriched with miR-873a-5p inhibit neuroinflammation via microglia phenotype modulation after traumatic brain injury

Journal of neuroinflammation, 2020-03, Vol.17 (1), p.89-89, Article 89 [Peer Reviewed Journal]

COPYRIGHT 2020 BioMed Central Ltd. ;2020. This work is licensed under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2020 ;ISSN: 1742-2094 ;EISSN: 1742-2094 ;DOI: 10.1186/s12974-020-01761-0 ;PMID: 32192523

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18
The prolyl 4-hydroxylase inhibitor GSK360A decreases post-stroke brain injury and sensory, motor, and cognitive behavioral deficits
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The prolyl 4-hydroxylase inhibitor GSK360A decreases post-stroke brain injury and sensory, motor, and cognitive behavioral deficits

PloS one, 2017-09, Vol.12 (9), p.e0184049-e0184049 [Peer Reviewed Journal]

COPYRIGHT 2017 Public Library of Science ;COPYRIGHT 2017 Public Library of Science ;2017 Zhou et al. This is an open access article distributed under the terms of the Creative Commons Attribution License: http://creativecommons.org/licenses/by/4.0/ (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2017 Zhou et al 2017 Zhou et al ;ISSN: 1932-6203 ;EISSN: 1932-6203 ;DOI: 10.1371/journal.pone.0184049 ;PMID: 28880966

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19
Combination therapy of human umbilical cord blood cells and granulocyte colony stimulating factor reduces histopathological and motor impairments in an experimental model of chronic traumatic brain injury
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Combination therapy of human umbilical cord blood cells and granulocyte colony stimulating factor reduces histopathological and motor impairments in an experimental model of chronic traumatic brain injury

PloS one, 2014-03, Vol.9 (3), p.e90953 [Peer Reviewed Journal]

COPYRIGHT 2014 Public Library of Science ;COPYRIGHT 2014 Public Library of Science ;2014. This is an open-access article, free of all copyright, and may be freely reproduced, distributed, transmitted, modified, built upon, or otherwise used by anyone for any lawful purpose. The work is made available under the Creative Commons CC0 public domain dedication. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2014 ;ISSN: 1932-6203 ;EISSN: 1932-6203 ;DOI: 10.1371/journal.pone.0090953 ;PMID: 24621603

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20
Heat Shock Protein 70 (HSP70) Induction: Chaperonotherapy for Neuroprotection after Brain Injury
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Heat Shock Protein 70 (HSP70) Induction: Chaperonotherapy for Neuroprotection after Brain Injury

Cells (Basel, Switzerland), 2020-09, Vol.9 (9), p.2020 [Peer Reviewed Journal]

COPYRIGHT 2020 MDPI AG ;2020. This work is licensed under http://creativecommons.org/licenses/by/3.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2020 by the authors. 2020 ;ISSN: 2073-4409 ;EISSN: 2073-4409 ;DOI: 10.3390/cells9092020 ;PMID: 32887360

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