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1
Fibroblasts in the Tumor Microenvironment: Shield or Spear?
Fibroblasts in the Tumor Microenvironment: Shield or Spear?
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Fibroblasts in the Tumor Microenvironment: Shield or Spear?

International journal of molecular sciences, 2018-05, Vol.19 (5), p.1532 [Peer Reviewed Journal]

Copyright MDPI AG 2018 ;2018 by the authors. 2018 ;ISSN: 1422-0067 ;ISSN: 1661-6596 ;EISSN: 1422-0067 ;DOI: 10.3390/ijms19051532 ;PMID: 29883428

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2
Scleral remodeling in early adulthood: the role of FGF-2
Scleral remodeling in early adulthood: the role of FGF-2
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Scleral remodeling in early adulthood: the role of FGF-2

Scientific reports, 2023-11, Vol.13 (1), p.20779-20779, Article 20779 [Peer Reviewed Journal]

2023. The Author(s). ;The Author(s) 2023. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2023 ;ISSN: 2045-2322 ;EISSN: 2045-2322 ;DOI: 10.1038/s41598-023-48264-5 ;PMID: 38012225

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3
Distinct fibroblast subsets drive inflammation and damage in arthritis
Distinct fibroblast subsets drive inflammation and damage in arthritis
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Distinct fibroblast subsets drive inflammation and damage in arthritis

Nature (London), 2019-06, Vol.570 (7760), p.246-251 [Peer Reviewed Journal]

COPYRIGHT 2019 Nature Publishing Group ;Copyright Nature Publishing Group Jun 13, 2019 ;ISSN: 0028-0836 ;EISSN: 1476-4687 ;DOI: 10.1038/s41586-019-1263-7 ;PMID: 31142839

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4
Biological heterogeneity and versatility of cancer-associated fibroblasts in the tumor microenvironment
Biological heterogeneity and versatility of cancer-associated fibroblasts in the tumor microenvironment
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Biological heterogeneity and versatility of cancer-associated fibroblasts in the tumor microenvironment

Oncogene, 2019-06, Vol.38 (25), p.4887-4901 [Peer Reviewed Journal]

2019© Springer Nature Limited 2019 ;ISSN: 0950-9232 ;EISSN: 1476-5594 ;DOI: 10.1038/s41388-019-0765-y ;PMID: 30816343

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5
Transition of Macrophages to Fibroblast-Like Cells in Healing Myocardial Infarction
Transition of Macrophages to Fibroblast-Like Cells in Healing Myocardial Infarction
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Transition of Macrophages to Fibroblast-Like Cells in Healing Myocardial Infarction

Journal of the American College of Cardiology, 2019-12, Vol.74 (25), p.3124-3135 [Peer Reviewed Journal]

2019 American College of Cardiology Foundation ;Copyright © 2019 American College of Cardiology Foundation. Published by Elsevier Inc. All rights reserved. ;2019. American College of Cardiology Foundation ;ISSN: 0735-1097 ;EISSN: 1558-3597 ;DOI: 10.1016/j.jacc.2019.10.036 ;PMID: 31856969

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6
CAF secreted miR-522 suppresses ferroptosis and promotes acquired chemo-resistance in gastric cancer
CAF secreted miR-522 suppresses ferroptosis and promotes acquired chemo-resistance in gastric cancer
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CAF secreted miR-522 suppresses ferroptosis and promotes acquired chemo-resistance in gastric cancer

Molecular cancer, 2020-02, Vol.19 (1), p.43-17, Article 43 [Peer Reviewed Journal]

COPYRIGHT 2020 BioMed Central Ltd. ;2020. This work is licensed under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2020 ;ISSN: 1476-4598 ;EISSN: 1476-4598 ;DOI: 10.1186/s12943-020-01168-8 ;PMID: 32106859

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7
A framework for advancing our understanding of cancer-associated fibroblasts
A framework for advancing our understanding of cancer-associated fibroblasts
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A framework for advancing our understanding of cancer-associated fibroblasts

Nature reviews. Cancer, 2020-03, Vol.20 (3), p.174-186 [Peer Reviewed Journal]

COPYRIGHT 2020 Nature Publishing Group ;This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2020. This work is published under https://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2020 ;ISSN: 1474-175X ;EISSN: 1474-1768 ;DOI: 10.1038/s41568-019-0238-1 ;PMID: 31980749

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8
Primary tumors release ITGBL1-rich extracellular vesicles to promote distal metastatic tumor growth through fibroblast-niche formation
Primary tumors release ITGBL1-rich extracellular vesicles to promote distal metastatic tumor growth through fibroblast-niche formation
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Primary tumors release ITGBL1-rich extracellular vesicles to promote distal metastatic tumor growth through fibroblast-niche formation

Nature communications, 2020-03, Vol.11 (1), p.1211-18, Article 1211 [Peer Reviewed Journal]

This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2020 ;ISSN: 2041-1723 ;EISSN: 2041-1723 ;DOI: 10.1038/s41467-020-14869-x ;PMID: 32139701

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9
Preclinical Activity of ARQ 087, a Novel Inhibitor Targeting FGFR Dysregulation
Preclinical Activity of ARQ 087, a Novel Inhibitor Targeting FGFR Dysregulation
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Preclinical Activity of ARQ 087, a Novel Inhibitor Targeting FGFR Dysregulation

PloS one, 2016-09, Vol.11 (9), p.e0162594-e0162594 [Peer Reviewed Journal]

COPYRIGHT 2016 Public Library of Science ;COPYRIGHT 2016 Public Library of Science ;2016 Hall et al. This is an open access article distributed under the terms of the Creative Commons Attribution License: http://creativecommons.org/licenses/by/4.0/ (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2016 Hall et al 2016 Hall et al ;ISSN: 1932-6203 ;EISSN: 1932-6203 ;DOI: 10.1371/journal.pone.0162594 ;PMID: 27627808

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10
FGF-2 promotes angiogenesis through a SRSF1/SRSF3/SRPK1-dependent axis that controls VEGFR1 splicing in endothelial cells
FGF-2 promotes angiogenesis through a SRSF1/SRSF3/SRPK1-dependent axis that controls VEGFR1 splicing in endothelial cells
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FGF-2 promotes angiogenesis through a SRSF1/SRSF3/SRPK1-dependent axis that controls VEGFR1 splicing in endothelial cells

BMC biology, 2021-08, Vol.19 (1), p.173-173, Article 173 [Peer Reviewed Journal]

2021. The Author(s). ;2021. This work is licensed under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;Distributed under a Creative Commons Attribution 4.0 International License ;The Author(s) 2021 ;ISSN: 1741-7007 ;EISSN: 1741-7007 ;DOI: 10.1186/s12915-021-01103-3 ;PMID: 34433435

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11
Targeting FAPα-expressing hepatic stellate cells overcomes resistance to antiangiogenics in colorectal cancer liver metastasis models
Targeting FAPα-expressing hepatic stellate cells overcomes resistance to antiangiogenics in colorectal cancer liver metastasis models
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Targeting FAPα-expressing hepatic stellate cells overcomes resistance to antiangiogenics in colorectal cancer liver metastasis models

The Journal of clinical investigation, 2022-10, Vol.132 (19), p.1-18 [Peer Reviewed Journal]

Copyright American Society for Clinical Investigation Oct 2022 ;2022 Qi et al. 2022 Qi et al. ;ISSN: 1558-8238 ;ISSN: 0021-9738 ;EISSN: 1558-8238 ;DOI: 10.1172/JCI157399 ;PMID: 35951441

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12
Antibody-mediated activation of FGFR1 induces FGF23 production and hypophosphatemia
Antibody-mediated activation of FGFR1 induces FGF23 production and hypophosphatemia
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Antibody-mediated activation of FGFR1 induces FGF23 production and hypophosphatemia

PloS one, 2013-02, Vol.8 (2), p.e57322-e57322 [Peer Reviewed Journal]

COPYRIGHT 2013 Public Library of Science ;COPYRIGHT 2013 Public Library of Science ;2013 Wu et al. This is an open-access article distributed under the terms of the Creative Commons Attribution License: https://creativecommons.org/licenses/by/4.0/ (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2013 Wu et al 2013 Wu et al ;ISSN: 1932-6203 ;EISSN: 1932-6203 ;DOI: 10.1371/journal.pone.0057322 ;PMID: 23451204

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13
High-glucose inhibits human fibroblast cell migration in wound healing via repression of bFGF-regulating JNK phosphorylation
High-glucose inhibits human fibroblast cell migration in wound healing via repression of bFGF-regulating JNK phosphorylation
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High-glucose inhibits human fibroblast cell migration in wound healing via repression of bFGF-regulating JNK phosphorylation

PloS one, 2014-09, Vol.9 (9), p.e108182-e108182 [Peer Reviewed Journal]

COPYRIGHT 2014 Public Library of Science ;COPYRIGHT 2014 Public Library of Science ;2014 Xuan et al. This is an open-access article distributed under the terms of the Creative Commons Attribution License: http://creativecommons.org/licenses/by/4.0/ (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2014 Xuan et al 2014 Xuan et al ;ISSN: 1932-6203 ;EISSN: 1932-6203 ;DOI: 10.1371/journal.pone.0108182 ;PMID: 25244316

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14
FGFR gene alterations in lung squamous cell carcinoma are potential targets for the multikinase inhibitor nintedanib
FGFR gene alterations in lung squamous cell carcinoma are potential targets for the multikinase inhibitor nintedanib
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FGFR gene alterations in lung squamous cell carcinoma are potential targets for the multikinase inhibitor nintedanib

Cancer science, 2016-11, Vol.107 (11), p.1667-1676 [Peer Reviewed Journal]

2016 The Authors. Cancer Science published by John Wiley & Sons Australia, Ltd on behalf of Japanese Cancer Association. ;2016. This work is published under http://creativecommons.org/licenses/by-nc-nd/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;ISSN: 1347-9032 ;EISSN: 1349-7006 ;DOI: 10.1111/cas.13071 ;PMID: 27581340

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15
Cancer‐associated fibroblasts that restrain cancer progression: Hypotheses and perspectives
Cancer‐associated fibroblasts that restrain cancer progression: Hypotheses and perspectives
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Cancer‐associated fibroblasts that restrain cancer progression: Hypotheses and perspectives

Cancer science, 2020-04, Vol.111 (4), p.1047-1057 [Peer Reviewed Journal]

2020 The Authors. published by John Wiley & Sons Australia, Ltd on behalf of Japanese Cancer Association. ;2020 The Authors. Cancer Science published by John Wiley & Sons Australia, Ltd on behalf of Japanese Cancer Association. ;2020. This work is published under http://creativecommons.org/licenses/by-nc/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;ISSN: 1347-9032 ;EISSN: 1349-7006 ;DOI: 10.1111/cas.14346 ;PMID: 32060987

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16
In vivo reprogramming of murine cardiac fibroblasts into induced cardiomyocytes
In vivo reprogramming of murine cardiac fibroblasts into induced cardiomyocytes
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In vivo reprogramming of murine cardiac fibroblasts into induced cardiomyocytes

Nature (London), 2012-05, Vol.485 (7400), p.593-598 [Peer Reviewed Journal]

2015 INIST-CNRS ;COPYRIGHT 2012 Nature Publishing Group ;COPYRIGHT 2012 Nature Publishing Group ;Copyright Nature Publishing Group May 31, 2012 ;ISSN: 0028-0836 ;EISSN: 1476-4687 ;DOI: 10.1038/nature11044 ;PMID: 22522929 ;CODEN: NATUAS

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17
Therapeutic effect of nanogel-based delivery of soluble FGFR2 with S252W mutation on craniosynostosis
Therapeutic effect of nanogel-based delivery of soluble FGFR2 with S252W mutation on craniosynostosis
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Therapeutic effect of nanogel-based delivery of soluble FGFR2 with S252W mutation on craniosynostosis

PloS one, 2014-07, Vol.9 (7), p.e101693 [Peer Reviewed Journal]

COPYRIGHT 2014 Public Library of Science ;COPYRIGHT 2014 Public Library of Science ;2014 Yokota et al. This is an open-access article distributed under the terms of the Creative Commons Attribution License: http://creativecommons.org/licenses/by/4.0/ (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2014 Yokota et al 2014 Yokota et al ;ISSN: 1932-6203 ;EISSN: 1932-6203 ;DOI: 10.1371/journal.pone.0101693 ;PMID: 25003957

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18
Chemokines at the crossroads of tumor-fibroblast interactions that promote malignancy
Chemokines at the crossroads of tumor-fibroblast interactions that promote malignancy
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Chemokines at the crossroads of tumor-fibroblast interactions that promote malignancy

Journal of leukocyte biology, 2011-01, Vol.89 (1), p.31-39 [Peer Reviewed Journal]

2011 Society for Leukocyte Biology ;ISSN: 0741-5400 ;EISSN: 1938-3673 ;DOI: 10.1189/jlb.0310182 ;PMID: 20628066

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19
Fibroblast heterogeneity: implications for human disease
Fibroblast heterogeneity: implications for human disease
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Fibroblast heterogeneity: implications for human disease

The Journal of clinical investigation, 2018-01, Vol.128 (1), p.26-35 [Peer Reviewed Journal]

COPYRIGHT 2018 American Society for Clinical Investigation ;Copyright American Society for Clinical Investigation Jan 2018 ;Copyright © 2018, American Society for Clinical Investigation 2018 American Society for Clinical Investigation ;ISSN: 0021-9738 ;EISSN: 1558-8238 ;DOI: 10.1172/JCI93555 ;PMID: 29293096

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20
Targeting interleukin-6 as a strategy to overcome stroma-induced resistance to chemotherapy in gastric cancer
Targeting interleukin-6 as a strategy to overcome stroma-induced resistance to chemotherapy in gastric cancer
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Targeting interleukin-6 as a strategy to overcome stroma-induced resistance to chemotherapy in gastric cancer

Molecular cancer, 2019-03, Vol.18 (1), p.68-68, Article 68 [Peer Reviewed Journal]

COPYRIGHT 2019 BioMed Central Ltd. ;2019. This work is licensed under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s). 2019 ;ISSN: 1476-4598 ;EISSN: 1476-4598 ;DOI: 10.1186/s12943-019-0972-8 ;PMID: 30927911

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