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1
FGFR gene alterations in lung squamous cell carcinoma are potential targets for the multikinase inhibitor nintedanib
FGFR gene alterations in lung squamous cell carcinoma are potential targets for the multikinase inhibitor nintedanib
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FGFR gene alterations in lung squamous cell carcinoma are potential targets for the multikinase inhibitor nintedanib

Cancer science, 2016-11, Vol.107 (11), p.1667-1676 [Peer Reviewed Journal]

2016 The Authors. Cancer Science published by John Wiley & Sons Australia, Ltd on behalf of Japanese Cancer Association. ;2016. This work is published under http://creativecommons.org/licenses/by-nc-nd/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;ISSN: 1347-9032 ;EISSN: 1349-7006 ;DOI: 10.1111/cas.13071 ;PMID: 27581340

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2
Cancer‐associated fibroblasts that restrain cancer progression: Hypotheses and perspectives
Cancer‐associated fibroblasts that restrain cancer progression: Hypotheses and perspectives
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Cancer‐associated fibroblasts that restrain cancer progression: Hypotheses and perspectives

Cancer science, 2020-04, Vol.111 (4), p.1047-1057 [Peer Reviewed Journal]

2020 The Authors. published by John Wiley & Sons Australia, Ltd on behalf of Japanese Cancer Association. ;2020 The Authors. Cancer Science published by John Wiley & Sons Australia, Ltd on behalf of Japanese Cancer Association. ;2020. This work is published under http://creativecommons.org/licenses/by-nc/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;ISSN: 1347-9032 ;EISSN: 1349-7006 ;DOI: 10.1111/cas.14346 ;PMID: 32060987

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3
Fibroblast growth factor receptor‐1 protein expression is associated with prognosis in estrogen receptor‐positive/human epidermal growth factor receptor‐2‐negative primary breast cancer
Fibroblast growth factor receptor‐1 protein expression is associated with prognosis in estrogen receptor‐positive/human epidermal growth factor receptor‐2‐negative primary breast cancer
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Fibroblast growth factor receptor‐1 protein expression is associated with prognosis in estrogen receptor‐positive/human epidermal growth factor receptor‐2‐negative primary breast cancer

Cancer science, 2016-04, Vol.107 (4), p.491-498 [Peer Reviewed Journal]

2016 The Authors. Cancer Science published by John Wiley & Sons Australia, Ltd on behalf of Japanese Cancer Association. ;2016. This work is published under http://creativecommons.org/licenses/by-nc-nd/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;ISSN: 1347-9032 ;EISSN: 1349-7006 ;DOI: 10.1111/cas.12897 ;PMID: 26801869

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4
Heterogeneity of cancer‐associated fibroblasts: Opportunities for precision medicine
Heterogeneity of cancer‐associated fibroblasts: Opportunities for precision medicine
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Heterogeneity of cancer‐associated fibroblasts: Opportunities for precision medicine

Cancer science, 2020-08, Vol.111 (8), p.2708-2717 [Peer Reviewed Journal]

2020 The Authors. published by John Wiley & Sons Australia, Ltd on behalf of Japanese Cancer Association. ;2020 The Authors. Cancer Science published by John Wiley & Sons Australia, Ltd on behalf of Japanese Cancer Association. ;2020. This work is published under http://creativecommons.org/licenses/by-nc/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;ISSN: 1347-9032 ;ISSN: 1349-7006 ;EISSN: 1349-7006 ;DOI: 10.1111/cas.14537 ;PMID: 32573845

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5
Phenotypic heterogeneity, stability and plasticity in tumor‐promoting carcinoma‐associated fibroblasts
Phenotypic heterogeneity, stability and plasticity in tumor‐promoting carcinoma‐associated fibroblasts
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Phenotypic heterogeneity, stability and plasticity in tumor‐promoting carcinoma‐associated fibroblasts

The FEBS journal, 2022-05, Vol.289 (9), p.2429-2447 [Peer Reviewed Journal]

2021 Federation of European Biochemical Societies ;2021 Federation of European Biochemical Societies. ;Copyright © 2022 Federation of European Biochemical Societies ;ISSN: 1742-464X ;EISSN: 1742-4658 ;DOI: 10.1111/febs.15851 ;PMID: 33786982

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6
A novel tdTomato transgenic mouse model to visualize FAP‐positive cancer‐associated fibroblasts
A novel tdTomato transgenic mouse model to visualize FAP‐positive cancer‐associated fibroblasts
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A novel tdTomato transgenic mouse model to visualize FAP‐positive cancer‐associated fibroblasts

The FEBS journal, 2023-05, Vol.290 (10), p.2604-2615 [Peer Reviewed Journal]

2022 Federation of European Biochemical Societies. ;Copyright © 2023 Federation of European Biochemical Societies ;ISSN: 1742-464X ;EISSN: 1742-4658 ;DOI: 10.1111/febs.16712 ;PMID: 36565059

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7
TL1A/TNFR2‐mediated mitochondrial dysfunction of fibroblast‐like synoviocytes increases inflammatory response in patients with rheumatoid arthritis via reactive oxygen species generation
TL1A/TNFR2‐mediated mitochondrial dysfunction of fibroblast‐like synoviocytes increases inflammatory response in patients with rheumatoid arthritis via reactive oxygen species generation
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TL1A/TNFR2‐mediated mitochondrial dysfunction of fibroblast‐like synoviocytes increases inflammatory response in patients with rheumatoid arthritis via reactive oxygen species generation

The FEBS journal, 2020-07, Vol.287 (14), p.3088-3104 [Peer Reviewed Journal]

2020 Federation of European Biochemical Societies ;2020 Federation of European Biochemical Societies. ;Copyright © 2020 Federation of European Biochemical Societies ;ISSN: 1742-464X ;EISSN: 1742-4658 ;DOI: 10.1111/febs.15181 ;PMID: 31953914

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8
Identification of a novel senolytic agent, navitoclax, targeting the Bcl‐2 family of anti‐apoptotic factors
Identification of a novel senolytic agent, navitoclax, targeting the Bcl‐2 family of anti‐apoptotic factors
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Identification of a novel senolytic agent, navitoclax, targeting the Bcl‐2 family of anti‐apoptotic factors

Aging cell, 2016-06, Vol.15 (3), p.428-435 [Peer Reviewed Journal]

2015 The Authors. published by the Anatomical Society and John Wiley & Sons Ltd. ;2015 The Authors. Aging Cell published by the Anatomical Society and John Wiley & Sons Ltd. ;Copyright © 2016 The Anatomical Society and John Wiley & Sons Ltd ;ISSN: 1474-9718 ;EISSN: 1474-9726 ;DOI: 10.1111/acel.12445 ;PMID: 26711051

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9
Functional diversity of cancer‐associated fibroblasts in modulating drug resistance
Functional diversity of cancer‐associated fibroblasts in modulating drug resistance
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Functional diversity of cancer‐associated fibroblasts in modulating drug resistance

Cancer science, 2020-10, Vol.111 (10), p.3468-3477 [Peer Reviewed Journal]

2020 The Authors. published by John Wiley & Sons Australia, Ltd on behalf of Japanese Cancer Association ;2020 The Authors. Cancer Science published by John Wiley & Sons Australia, Ltd on behalf of Japanese Cancer Association. ;2020. This work is published under http://creativecommons.org/licenses/by-nc/3.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;ISSN: 1347-9032 ;EISSN: 1349-7006 ;DOI: 10.1111/cas.14578 ;PMID: 33044028

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10
Rapamycin inhibits the secretory phenotype of senescent cells by a Nrf2‐independent mechanism
Rapamycin inhibits the secretory phenotype of senescent cells by a Nrf2‐independent mechanism
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Rapamycin inhibits the secretory phenotype of senescent cells by a Nrf2‐independent mechanism

Aging cell, 2017-06, Vol.16 (3), p.564-574 [Peer Reviewed Journal]

2017 The Authors. published by the Anatomical Society and John Wiley & Sons Ltd. ;2017 The Authors. Aging Cell published by the Anatomical Society and John Wiley & Sons Ltd. ;COPYRIGHT 2017 John Wiley & Sons, Inc. ;Copyright © 2017 The Anatomical Society and John Wiley & Sons Ltd. ;ISSN: 1474-9718 ;EISSN: 1474-9726 ;DOI: 10.1111/acel.12587 ;PMID: 28371119

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11
Reverse of non‐small cell lung cancer drug resistance induced by cancer‐associated fibroblasts via a paracrine pathway
Reverse of non‐small cell lung cancer drug resistance induced by cancer‐associated fibroblasts via a paracrine pathway
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Reverse of non‐small cell lung cancer drug resistance induced by cancer‐associated fibroblasts via a paracrine pathway

Cancer science, 2018-04, Vol.109 (4), p.944-955 [Peer Reviewed Journal]

2018 The Authors. published by John Wiley & Sons Australia, Ltd on behalf of Japanese Cancer Association. ;2018 The Authors. Cancer Science published by John Wiley & Sons Australia, Ltd on behalf of Japanese Cancer Association. ;2018. This work is published under http://creativecommons.org/licenses/by-nc/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;ISSN: 1347-9032 ;EISSN: 1349-7006 ;DOI: 10.1111/cas.13520 ;PMID: 29383798

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12
Heterogeneous cancer‐associated fibroblasts: A new perspective for understanding immunosuppression in pancreatic cancer
Heterogeneous cancer‐associated fibroblasts: A new perspective for understanding immunosuppression in pancreatic cancer
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Heterogeneous cancer‐associated fibroblasts: A new perspective for understanding immunosuppression in pancreatic cancer

Immunology, 2022-09, Vol.167 (1), p.1-14 [Peer Reviewed Journal]

2022 John Wiley & Sons Ltd. ;2022 John Wiley & Sons Ltd ;ISSN: 0019-2805 ;EISSN: 1365-2567 ;DOI: 10.1111/imm.13496 ;PMID: 35569095

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13
Cancer‐associated fibroblast‐derived interleukin‐1β activates protumor C‐C motif chemokine ligand 22 signaling in head and neck cancer
Cancer‐associated fibroblast‐derived interleukin‐1β activates protumor C‐C motif chemokine ligand 22 signaling in head and neck cancer
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Cancer‐associated fibroblast‐derived interleukin‐1β activates protumor C‐C motif chemokine ligand 22 signaling in head and neck cancer

Cancer science, 2019-09, Vol.110 (9), p.2783-2793 [Peer Reviewed Journal]

2019 The Authors. published by John Wiley & Sons Australia, Ltd on behalf of Japanese Cancer Association. ;2019 The Authors. Cancer Science published by John Wiley & Sons Australia, Ltd on behalf of Japanese Cancer Association. ;2019. This work is published under http://creativecommons.org/licenses/by-nc-nd/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;ISSN: 1347-9032 ;EISSN: 1349-7006 ;DOI: 10.1111/cas.14135 ;PMID: 31325403

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14
Involvement of local renin‐angiotensin system in immunosuppression of tumor microenvironment
Involvement of local renin‐angiotensin system in immunosuppression of tumor microenvironment
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Involvement of local renin‐angiotensin system in immunosuppression of tumor microenvironment

Cancer science, 2018-01, Vol.109 (1), p.54-64 [Peer Reviewed Journal]

2017 The Authors. published by John Wiley & Sons Australia, Ltd on behalf of Japanese Cancer Association. ;2017 The Authors. Cancer Science published by John Wiley & Sons Australia, Ltd on behalf of Japanese Cancer Association. ;2018. This work is published under http://creativecommons.org/licenses/by-nc-nd/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;ISSN: 1347-9032 ;EISSN: 1349-7006 ;DOI: 10.1111/cas.13423 ;PMID: 29034589

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15
Cancer‐associated fibroblasts contribute to cisplatin resistance by modulating ANXA3 in lung cancer cells
Cancer‐associated fibroblasts contribute to cisplatin resistance by modulating ANXA3 in lung cancer cells
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Cancer‐associated fibroblasts contribute to cisplatin resistance by modulating ANXA3 in lung cancer cells

Cancer science, 2019-05, Vol.110 (5), p.1609-1620 [Peer Reviewed Journal]

2019 The Authors. published by John Wiley & Sons Australia, Ltd on behalf of Japanese Cancer Association. ;2019 The Authors. Cancer Science published by John Wiley & Sons Australia, Ltd on behalf of Japanese Cancer Association. ;ISSN: 1347-9032 ;EISSN: 1349-7006 ;DOI: 10.1111/cas.13998 ;PMID: 30868675

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16
Tumor microenvironmental 15‐PGDH depletion promotes fibrotic tumor formation and angiogenesis in pancreatic cancer
Tumor microenvironmental 15‐PGDH depletion promotes fibrotic tumor formation and angiogenesis in pancreatic cancer
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Tumor microenvironmental 15‐PGDH depletion promotes fibrotic tumor formation and angiogenesis in pancreatic cancer

Cancer science, 2022-10, Vol.113 (10), p.3579-3592 [Peer Reviewed Journal]

2022 The Authors. published by John Wiley & Sons Australia, Ltd on behalf of Japanese Cancer Association. ;2022 The Authors. Cancer Science published by John Wiley & Sons Australia, Ltd on behalf of Japanese Cancer Association. ;COPYRIGHT 2022 John Wiley & Sons, Inc. ;2022. This work is published under http://creativecommons.org/licenses/by-nc/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;ISSN: 1347-9032 ;EISSN: 1349-7006 ;DOI: 10.1111/cas.15495 ;PMID: 35848891

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17
IL‐35 ameliorates collagen‐induced arthritis by promoting TNF‐α‐induced apoptosis of synovial fibroblasts and stimulating M2 macrophages polarization
IL‐35 ameliorates collagen‐induced arthritis by promoting TNF‐α‐induced apoptosis of synovial fibroblasts and stimulating M2 macrophages polarization
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IL‐35 ameliorates collagen‐induced arthritis by promoting TNF‐α‐induced apoptosis of synovial fibroblasts and stimulating M2 macrophages polarization

The FEBS journal, 2019-05, Vol.286 (10), p.1972-1985 [Peer Reviewed Journal]

2019 Federation of European Biochemical Societies ;2019 Federation of European Biochemical Societies. ;Copyright © 2019 Federation of European Biochemical Societies ;ISSN: 1742-464X ;EISSN: 1742-4658 ;DOI: 10.1111/febs.14801 ;PMID: 30834683

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18
Inhibition of fibroblast growth factor receptor 2 attenuates proliferation and invasion of pancreatic cancer
Inhibition of fibroblast growth factor receptor 2 attenuates proliferation and invasion of pancreatic cancer
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Inhibition of fibroblast growth factor receptor 2 attenuates proliferation and invasion of pancreatic cancer

Cancer science, 2014-09, Vol.105 (9), p.1212-1219 [Peer Reviewed Journal]

2014 The Authors. Cancer Science published by Wiley Publishing Asia Pty Ltd on behalf of Japanese Cancer Association. ;2014. This work is published under http://creativecommons.org/licenses/by-nc-nd/3.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2014 The Authors. Cancer Science published by Wiley Publishing Asia Pty Ltd on behalf of Japanese Cancer Association. 2014 ;ISSN: 1347-9032 ;EISSN: 1349-7006 ;DOI: 10.1111/cas.12470 ;PMID: 24975163

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19
The heterogeneity of cancer‐associated fibroblast subpopulations: Their origins, biomarkers, and roles in the tumor microenvironment
The heterogeneity of cancer‐associated fibroblast subpopulations: Their origins, biomarkers, and roles in the tumor microenvironment
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The heterogeneity of cancer‐associated fibroblast subpopulations: Their origins, biomarkers, and roles in the tumor microenvironment

Cancer science, 2023-01, Vol.114 (1), p.16-24 [Peer Reviewed Journal]

2022 The Authors. published by John Wiley & Sons Australia, Ltd on behalf of Japanese Cancer Association. ;2022 The Authors. Cancer Science published by John Wiley & Sons Australia, Ltd on behalf of Japanese Cancer Association. ;COPYRIGHT 2023 John Wiley & Sons, Inc. ;2023. This work is published under http://creativecommons.org/licenses/by-nc/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;ISSN: 1347-9032 ;EISSN: 1349-7006 ;DOI: 10.1111/cas.15609 ;PMID: 36197901

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20
Tumor microenvironment in gastric cancers
Tumor microenvironment in gastric cancers
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Tumor microenvironment in gastric cancers

Cancer science, 2020-08, Vol.111 (8), p.2696-2707 [Peer Reviewed Journal]

2020 The Authors. published by John Wiley & Sons Australia, Ltd on behalf of Japanese Cancer Association. ;2020 The Authors. Cancer Science published by John Wiley & Sons Australia, Ltd on behalf of Japanese Cancer Association. ;2020. This work is published under http://creativecommons.org/licenses/by-nc/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;ISSN: 1347-9032 ;EISSN: 1349-7006 ;DOI: 10.1111/cas.14521 ;PMID: 32519436

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