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First use of anatomical networks to study modularity and integration of heads, forelimbs and hindlimbs in abnormal anencephalic and cyclopic vs normal human development
First use of anatomical networks to study modularity and integration of heads, forelimbs and hindlimbs in abnormal anencephalic and cyclopic vs normal human development
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First use of anatomical networks to study modularity and integration of heads, forelimbs and hindlimbs in abnormal anencephalic and cyclopic vs normal human development

Scientific reports, 2019-05, Vol.9 (1), p.7821, Article 7821 [Peer Reviewed Journal]

The Author(s) 2019. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2019 ;ISSN: 2045-2322 ;EISSN: 2045-2322 ;DOI: 10.1038/s41598-019-44314-z ;PMID: 31127169

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Growth and cellular patterning during fetal human inner ear development studied by a correlative imaging approach
Growth and cellular patterning during fetal human inner ear development studied by a correlative imaging approach
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Growth and cellular patterning during fetal human inner ear development studied by a correlative imaging approach

BMC developmental biology, 2019-05, Vol.19 (1), p.11-11, Article 11 [Peer Reviewed Journal]

2019. This work is licensed under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s). 2019 ;ISSN: 1471-213X ;EISSN: 1471-213X ;DOI: 10.1186/s12861-019-0191-y ;PMID: 31109306

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3
Expression of epigenetic machinery genes is sensitive to maternal obesity and weight loss in relation to fetal growth in mice
Expression of epigenetic machinery genes is sensitive to maternal obesity and weight loss in relation to fetal growth in mice
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Expression of epigenetic machinery genes is sensitive to maternal obesity and weight loss in relation to fetal growth in mice

Clinical epigenetics, 2016-02, Vol.8 (22), p.22-22, Article 22 [Peer Reviewed Journal]

COPYRIGHT 2016 BioMed Central Ltd. ;Copyright BioMed Central 2016 ;Attribution ;Panchenko et al. 2016 ;ISSN: 1868-7075 ;ISSN: 1868-7083 ;EISSN: 1868-7083 ;DOI: 10.1186/s13148-016-0188-3 ;PMID: 26925174

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4
Two novel human NUMB isoforms provide a potential link between development and cancer
Two novel human NUMB isoforms provide a potential link between development and cancer
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Two novel human NUMB isoforms provide a potential link between development and cancer

Neural development, 2010-12, Vol.5 (1), p.31-31, Article 31 [Peer Reviewed Journal]

COPYRIGHT 2010 BioMed Central Ltd. ;2010 Karaczyn et al; licensee BioMed Central Ltd. This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/2.0), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited. ;Copyright ©2010 Karaczyn et al; licensee BioMed Central Ltd. 2010 Karaczyn et al; licensee BioMed Central Ltd. ;ISSN: 1749-8104 ;EISSN: 1749-8104 ;DOI: 10.1186/1749-8104-5-31 ;PMID: 21122105

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5
Delta-Notch signaling and lateral inhibition in zebrafish spinal cord development
Delta-Notch signaling and lateral inhibition in zebrafish spinal cord development
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Delta-Notch signaling and lateral inhibition in zebrafish spinal cord development

BMC developmental biology, 2001-07, Vol.1 (1), p.13-13, Article 13 [Peer Reviewed Journal]

Copyright © 2001 Appel et al; licensee BioMed Central Ltd. This is an Open Access article: verbatim copying and redistribution of this article are permitted in all media for any purpose, provided this notice is preserved along with the article's original URL. 2001 Appel et al; licensee BioMed Central Ltd. This is an Open Access article: verbatim copying and redistribution of this article are permitted in all media for any purpose, provided this notice is preserved along with the article's original URL. ;ISSN: 1471-213X ;EISSN: 1471-213X ;DOI: 10.1186/1471-213x-1-13 ;PMID: 11495630

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