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B-type natriuretic peptide trumps other prognostic markers in patients assessed for coronary disease
B-type natriuretic peptide trumps other prognostic markers in patients assessed for coronary disease
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B-type natriuretic peptide trumps other prognostic markers in patients assessed for coronary disease

BMC medicine, 2019-04, Vol.17 (1), p.72-72, Article 72 [Peer Reviewed Journal]

COPYRIGHT 2019 BioMed Central Ltd. ;COPYRIGHT 2019 BioMed Central Ltd. ;2019. This work is licensed under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;info:eu-repo/semantics/openAccess ;The Author(s). 2019 ;ISSN: 1741-7015 ;EISSN: 1741-7015 ;DOI: 10.1186/s12916-019-1306-9 ;PMID: 30943979

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2
Unsatisfactory risk factor control and high rate of new cardiovascular events in patients with myocardial infarction and prior coronary artery disease
Unsatisfactory risk factor control and high rate of new cardiovascular events in patients with myocardial infarction and prior coronary artery disease
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Unsatisfactory risk factor control and high rate of new cardiovascular events in patients with myocardial infarction and prior coronary artery disease

BMC cardiovascular disorders, 2019-03, Vol.19 (1), p.71-71, Article 71 [Peer Reviewed Journal]

COPYRIGHT 2019 BioMed Central Ltd. ;2019. This work is licensed under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;info:eu-repo/semantics/openAccess ;The Author(s). 2019 ;ISSN: 1471-2261 ;EISSN: 1471-2261 ;DOI: 10.1186/s12872-019-1062-y ;PMID: 30922234

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3
NBEAL1 controls SREBP2 processing and cholesterol metabolism and is a susceptibility locus for coronary artery disease
NBEAL1 controls SREBP2 processing and cholesterol metabolism and is a susceptibility locus for coronary artery disease
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NBEAL1 controls SREBP2 processing and cholesterol metabolism and is a susceptibility locus for coronary artery disease

Scientific reports, 2020-03, Vol.10 (1), p.4528-4528, Article 4528 [Peer Reviewed Journal]

This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;info:eu-repo/semantics/openAccess ;The Author(s) 2020 ;ISSN: 2045-2322 ;EISSN: 2045-2322 ;DOI: 10.1038/s41598-020-61352-0 ;PMID: 32161285

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4
Blocking phospholamban with VHH intrabodies enhances contractility and relaxation in heart failure
Blocking phospholamban with VHH intrabodies enhances contractility and relaxation in heart failure
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Blocking phospholamban with VHH intrabodies enhances contractility and relaxation in heart failure

Nature communications, 2022-05, Vol.13 (1), p.3018-3018, Article 3018 [Peer Reviewed Journal]

2022. The Author(s). ;The Author(s) 2022. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;info:eu-repo/semantics/openAccess ;The Author(s) 2022 ;ISSN: 2041-1723 ;EISSN: 2041-1723 ;DOI: 10.1038/s41467-022-29703-9 ;PMID: 35641497

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5
Associations between circulating microRNAs and lipid-rich coronary plaques measured with near-infrared spectroscopy
Associations between circulating microRNAs and lipid-rich coronary plaques measured with near-infrared spectroscopy
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Associations between circulating microRNAs and lipid-rich coronary plaques measured with near-infrared spectroscopy

Scientific reports, 2023-05, Vol.13 (1), p.7580-7580, Article 7580 [Peer Reviewed Journal]

2023. The Author(s). ;The Author(s) 2023. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;info:eu-repo/semantics/openAccess ;The Author(s) 2023 ;ISSN: 2045-2322 ;EISSN: 2045-2322 ;DOI: 10.1038/s41598-023-34642-6 ;PMID: 37165064

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6
The extracellular matrix glycoprotein ADAMTSL2 is increased in heart failure and inhibits TGFβ signalling in cardiac fibroblasts
The extracellular matrix glycoprotein ADAMTSL2 is increased in heart failure and inhibits TGFβ signalling in cardiac fibroblasts
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The extracellular matrix glycoprotein ADAMTSL2 is increased in heart failure and inhibits TGFβ signalling in cardiac fibroblasts

Scientific reports, 2021-10, Vol.11 (1), p.19757-19757, Article 19757 [Peer Reviewed Journal]

2021. The Author(s). ;The Author(s) 2021. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;info:eu-repo/semantics/openAccess ;The Author(s) 2021 ;ISSN: 2045-2322 ;EISSN: 2045-2322 ;DOI: 10.1038/s41598-021-99032-2 ;PMID: 34611183

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7
ADAMTSL3 knock-out mice develop cardiac dysfunction and dilatation with increased TGFβ signalling after pressure overload
ADAMTSL3 knock-out mice develop cardiac dysfunction and dilatation with increased TGFβ signalling after pressure overload
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ADAMTSL3 knock-out mice develop cardiac dysfunction and dilatation with increased TGFβ signalling after pressure overload

Communications biology, 2022-12, Vol.5 (1), p.1392-1392, Article 1392 [Peer Reviewed Journal]

2022. The Author(s). ;The Author(s) 2022. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;info:eu-repo/semantics/openAccess ;The Author(s) 2022 ;ISSN: 2399-3642 ;EISSN: 2399-3642 ;DOI: 10.1038/s42003-022-04361-1 ;PMID: 36539599

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