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1
Cellular dynamics of regeneration reveals role of two distinct Pax7 stem cell populations in larval zebrafish muscle repair
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Cellular dynamics of regeneration reveals role of two distinct Pax7 stem cell populations in larval zebrafish muscle repair

Disease models & mechanisms, 2016-06, Vol.9 (6), p.671-684 [Peer Reviewed Journal]

2016. Published by The Company of Biologists Ltd. ;2016. This work is licensed under http://creativecommons.org/licenses/by/3.0 (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2016. Published by The Company of Biologists Ltd 2016 ;ISSN: 1754-8403 ;EISSN: 1754-8411 ;DOI: 10.1242/dmm.022251 ;PMID: 27149989

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2
Cellular and molecular mechanisms of muscle atrophy
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Cellular and molecular mechanisms of muscle atrophy

Disease models & mechanisms, 2013-01, Vol.6 (1), p.25-39 [Peer Reviewed Journal]

2013. This work is licensed under http://creativecommons.org/licenses/by-nc-sa/3.0 (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2012. Published by The Company of Biologists Ltd 2012 ;ISSN: 1754-8403 ;EISSN: 1754-8411 ;DOI: 10.1242/dmm.010389 ;PMID: 23268536

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3
Modeling autosomal recessive cutis laxa type 1C in mice reveals distinct functions for Ltbp-4 isoforms
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Modeling autosomal recessive cutis laxa type 1C in mice reveals distinct functions for Ltbp-4 isoforms

Disease models & mechanisms, 2015-04, Vol.8 (4), p.403-415 [Peer Reviewed Journal]

2015. Published by The Company of Biologists Ltd. ;2015. This work is licensed under http://creativecommons.org/licenses/by/3.0 (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2015. Published by The Company of Biologists Ltd 2015 ;ISSN: 1754-8403 ;EISSN: 1754-8411 ;DOI: 10.1242/dmm.018960 ;PMID: 25713297

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4
The role of mTOR signaling in the regulation of protein synthesis and muscle mass during immobilization in mice
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The role of mTOR signaling in the regulation of protein synthesis and muscle mass during immobilization in mice

Disease models & mechanisms, 2015-09, Vol.8 (9), p.1059-1069 [Peer Reviewed Journal]

2015. Published by The Company of Biologists Ltd. ;2015. This work is licensed under http://creativecommons.org/licenses/by/3.0 (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2015. Published by The Company of Biologists Ltd 2015 ;ISSN: 1754-8403 ;ISSN: 1754-8411 ;EISSN: 1754-8411 ;DOI: 10.1242/dmm.019414 ;PMID: 26092121

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5
A gene expression resource generated by genome-wide lacZ profiling in the mouse
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A gene expression resource generated by genome-wide lacZ profiling in the mouse

Disease models & mechanisms, 2015-11, Vol.8 (11), p.1467-1478 [Peer Reviewed Journal]

2015. Published by The Company of Biologists Ltd. ;2015. This work is licensed under http://creativecommons.org/licenses/by/3.0 (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2015. Published by The Company of Biologists Ltd 2015 ;ISSN: 1754-8403 ;ISSN: 1754-8411 ;EISSN: 1754-8411 ;DOI: 10.1242/dmm.021238 ;PMID: 26398943

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6
A novel rabbit model of Duchenne muscular dystrophy generated by CRISPR/Cas9
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A novel rabbit model of Duchenne muscular dystrophy generated by CRISPR/Cas9

Disease models & mechanisms, 2018-06, Vol.11 (6) [Peer Reviewed Journal]

2018. Published by The Company of Biologists Ltd. ;2018. This work is licensed under http://creativecommons.org/licenses/by/3.0 (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2018. Published by The Company of Biologists Ltd 2018 ;ISSN: 1754-8403 ;EISSN: 1754-8411 ;DOI: 10.1242/dmm.032201 ;PMID: 29871865

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7
In vivo cerebellar circuit function is disrupted in an mdx mouse model of Duchenne muscular dystrophy
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In vivo cerebellar circuit function is disrupted in an mdx mouse model of Duchenne muscular dystrophy

Disease models & mechanisms, 2019-12, Vol.13 (2) [Peer Reviewed Journal]

2019. Published by The Company of Biologists Ltd. ;2020. This work is licensed under https://creativecommons.org/licenses/by/4.0 (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2019. Published by The Company of Biologists Ltd 2019 ;ISSN: 1754-8403 ;EISSN: 1754-8411 ;DOI: 10.1242/dmm.040840 ;PMID: 31704708

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8
Diverse dystonin gene mutations cause distinct patterns of Dst isoform deficiency and phenotypic heterogeneity in Dystonia musculorum mice
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Diverse dystonin gene mutations cause distinct patterns of Dst isoform deficiency and phenotypic heterogeneity in Dystonia musculorum mice

Disease models & mechanisms, 2020-05, Vol.13 (5) [Peer Reviewed Journal]

2020. This work is licensed under https://creativecommons.org/licenses/by/4.0 (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2020. Published by The Company of Biologists Ltd 2020 ;ISSN: 1754-8403 ;EISSN: 1754-8411 ;DOI: 10.1242/dmm.041608 ;PMID: 32482619

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9
Digitization of indigenous knowledge on forest foods and medicines
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Digitization of indigenous knowledge on forest foods and medicines

IFLA journal, 2017-06, Vol.43 (2), p.187-197 [Peer Reviewed Journal]

The Author(s) 2016 ;ISSN: 0340-0352 ;EISSN: 1745-2651 ;DOI: 10.1177/0340035216681326

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10
Pumilio2-deficient mice show a predisposition for epilepsy
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Pumilio2-deficient mice show a predisposition for epilepsy

Disease models & mechanisms, 2017-11, Vol.10 (11), p.1333-1342 [Peer Reviewed Journal]

2017. Published by The Company of Biologists Ltd. ;2017. This work is licensed under http://creativecommons.org/licenses/by/3.0 (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2017. Published by The Company of Biologists Ltd 2017 ;ISSN: 1754-8403 ;ISSN: 1754-8411 ;EISSN: 1754-8411 ;DOI: 10.1242/dmm.029678 ;PMID: 29046322

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11
Notch signaling deficiency underlies age-dependent depletion of satellite cells in muscular dystrophy
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Notch signaling deficiency underlies age-dependent depletion of satellite cells in muscular dystrophy

Disease models & mechanisms, 2014-08, Vol.7 (8), p.997-1004 [Peer Reviewed Journal]

2014. Published by The Company of Biologists Ltd. ;2014. This work is licensed under http://creativecommons.org/licenses/by/3.0 (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2014. Published by The Company of Biologists Ltd 2014 ;ISSN: 1754-8403 ;EISSN: 1754-8411 ;DOI: 10.1242/dmm.015917 ;PMID: 24906372

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12
Inactivation of Zeb1 in GRHL2-deficient mouse embryos rescues mid-gestation viability and secondary palate closure
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Inactivation of Zeb1 in GRHL2-deficient mouse embryos rescues mid-gestation viability and secondary palate closure

Disease models & mechanisms, 2020, Vol.13 (3) [Peer Reviewed Journal]

2020. Published by The Company of Biologists Ltd. ;2020. This work is licensed under https://creativecommons.org/licenses/by/4.0 (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2020. Published by The Company of Biologists Ltd 2020 ;ISSN: 1754-8403 ;EISSN: 1754-8411 ;DOI: 10.1242/dmm.042218 ;PMID: 32005677

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13
Immune regulation of metastasis: mechanistic insights and therapeutic opportunities
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Article
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Immune regulation of metastasis: mechanistic insights and therapeutic opportunities

Disease models & mechanisms, 2018-10, Vol.11 (10) [Peer Reviewed Journal]

2018. Published by The Company of Biologists Ltd. ;2018. This work is licensed under http://creativecommons.org/licenses/by/3.0 (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2018. Published by The Company of Biologists Ltd 2018 ;ISSN: 1754-8403 ;EISSN: 1754-8411 ;DOI: 10.1242/dmm.036236 ;PMID: 30355585

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14
A high-sugar diet produces obesity and insulin resistance in wild-type Drosophila
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A high-sugar diet produces obesity and insulin resistance in wild-type Drosophila

Disease models & mechanisms, 2011-11, Vol.4 (6), p.842-849 [Peer Reviewed Journal]

2011. This work is licensed under http://creativecommons.org/licenses/by-nc-sa/3.0 (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2011. Published by The Company of Biologists Ltd 2011 ;ISSN: 1754-8403 ;EISSN: 1754-8411 ;DOI: 10.1242/dmm.007948 ;PMID: 21719444

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15
The Menkes and Wilson disease genes counteract in copper toxicosis in Labrador retrievers: a new canine model for copper-metabolism disorders
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The Menkes and Wilson disease genes counteract in copper toxicosis in Labrador retrievers: a new canine model for copper-metabolism disorders

Disease models & mechanisms, 2016-01, Vol.9 (1), p.25-38 [Peer Reviewed Journal]

2016. Published by The Company of Biologists Ltd. ;2016. This work is licensed under http://creativecommons.org/licenses/by/3.0 (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2016. Published by The Company of Biologists Ltd 2016 ;ISSN: 1754-8403 ;ISSN: 1754-8411 ;EISSN: 1754-8411 ;DOI: 10.1242/dmm.020263 ;PMID: 26747866

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16
A comprehensive assessment of the SOD1G93A low-copy transgenic mouse, which models human amyotrophic lateral sclerosis
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A comprehensive assessment of the SOD1G93A low-copy transgenic mouse, which models human amyotrophic lateral sclerosis

Disease models & mechanisms, 2011-09, Vol.4 (5), p.686-700 [Peer Reviewed Journal]

2011. This work is licensed under http://creativecommons.org/licenses/by-nc-sa/3.0 (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2011. Published by The Company of Biologists Ltd 2011 ;ISSN: 1754-8403 ;EISSN: 1754-8411 ;DOI: 10.1242/dmm.007237 ;PMID: 21540242

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17
Subchondral drilling for articular cartilage repair: a systematic review of translational research
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Article
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Subchondral drilling for articular cartilage repair: a systematic review of translational research

Disease models & mechanisms, 2018-06, Vol.11 (6) [Peer Reviewed Journal]

2018. Published by The Company of Biologists Ltd. ;2018. This work is licensed under http://creativecommons.org/licenses/by/3.0 (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2018. Published by The Company of Biologists Ltd 2018 ;ISSN: 1754-8403 ;EISSN: 1754-8411 ;DOI: 10.1242/dmm.034280 ;PMID: 29728409

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18
High-resolution live imaging reveals axon-glia interactions during peripheral nerve injury and repair in zebrafish
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High-resolution live imaging reveals axon-glia interactions during peripheral nerve injury and repair in zebrafish

Disease models & mechanisms, 2015-06, Vol.8 (6), p.553-564 [Peer Reviewed Journal]

2015. Published by The Company of Biologists Ltd. ;2015. This work is licensed under http://creativecommons.org/licenses/by/3.0 (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;Distributed under a Creative Commons Attribution 4.0 International License ;2015. Published by The Company of Biologists Ltd 2015 ;ISSN: 1754-8403 ;ISSN: 1754-8411 ;EISSN: 1754-8411 ;DOI: 10.1242/dmm.018184 ;PMID: 26035865

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19
Loss of fibulin-5 binding to β1 integrins inhibits tumor growth by increasing the level of ROS
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Article
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Loss of fibulin-5 binding to β1 integrins inhibits tumor growth by increasing the level of ROS

Disease models & mechanisms, 2010-01, Vol.3 (5-6), p.333-342 [Peer Reviewed Journal]

2010. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the associated terms available at https://journals.biologists.com/dmm/pages/rights-permissions . ;2010 ;ISSN: 1754-8403 ;EISSN: 1754-8411 ;DOI: 10.1242/dmm.003707 ;PMID: 20197418

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20
The Meckel-Gruber syndrome protein TMEM67 controls basal body positioning and epithelial branching morphogenesis in mice via the non-canonical Wnt pathway
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The Meckel-Gruber syndrome protein TMEM67 controls basal body positioning and epithelial branching morphogenesis in mice via the non-canonical Wnt pathway

Disease models & mechanisms, 2015-06, Vol.8 (6), p.527-541 [Peer Reviewed Journal]

2015. Published by The Company of Biologists Ltd. ;2015. This work is licensed under http://creativecommons.org/licenses/by/3.0 (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2015. Published by The Company of Biologists Ltd 2015 ;ISSN: 1754-8403 ;ISSN: 1754-8411 ;EISSN: 1754-8411 ;DOI: 10.1242/dmm.019083 ;PMID: 26035863

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