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1
Mutation of A677 in histone methyltransferase EZH2 in human B-cell lymphoma promotes hypertrimethylation of histone H3 on lysine 27 (H3K27)
Mutation of A677 in histone methyltransferase EZH2 in human B-cell lymphoma promotes hypertrimethylation of histone H3 on lysine 27 (H3K27)
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Mutation of A677 in histone methyltransferase EZH2 in human B-cell lymphoma promotes hypertrimethylation of histone H3 on lysine 27 (H3K27)

Proceedings of the National Academy of Sciences - PNAS, 2012-02, Vol.109 (8), p.2989-2994 [Peer Reviewed Journal]

copyright © 1993-2008 National Acadamy of Sciences of the United States of America ;Copyright National Academy of Sciences Feb 21, 2012 ;ISSN: 0027-8424 ;EISSN: 1091-6490 ;DOI: 10.1073/pnas.1116418109 ;PMID: 22323599

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2
Autism-linked neuroligin-3 R451C mutation differentially alters hippocampal and cortical synaptic function
Autism-linked neuroligin-3 R451C mutation differentially alters hippocampal and cortical synaptic function
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Autism-linked neuroligin-3 R451C mutation differentially alters hippocampal and cortical synaptic function

Proceedings of the National Academy of Sciences - PNAS, 2011-08, Vol.108 (33), p.13764-13769 [Peer Reviewed Journal]

copyright © 1993–2008 National Academy of Sciences of the United States of America ;Copyright National Academy of Sciences Aug 16, 2011 ;ISSN: 0027-8424 ;EISSN: 1091-6490 ;DOI: 10.1073/pnas.1111093108 ;PMID: 21808020

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3
over-oxidized form of superoxide dismutase found in sporadic amyotrophic lateral sclerosis with bulbar onset shares a toxic mechanism with mutant SOD1
over-oxidized form of superoxide dismutase found in sporadic amyotrophic lateral sclerosis with bulbar onset shares a toxic mechanism with mutant SOD1
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over-oxidized form of superoxide dismutase found in sporadic amyotrophic lateral sclerosis with bulbar onset shares a toxic mechanism with mutant SOD1

Proceedings of the National Academy of Sciences - PNAS, 2012-03, Vol.109 (13), p.5074-5079 [Peer Reviewed Journal]

copyright © 1993-2008 National Academy of Sciences of the United States of America ;Copyright National Academy of Sciences Mar 27, 2012 ;ISSN: 0027-8424 ;EISSN: 1091-6490 ;DOI: 10.1073/pnas.1115402109 ;PMID: 22416121

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4
Essential Roles for Mycobacterium tuberculosis Rel beyond the Production of (p)ppGpp
Essential Roles for Mycobacterium tuberculosis Rel beyond the Production of (p)ppGpp
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Essential Roles for Mycobacterium tuberculosis Rel beyond the Production of (p)ppGpp

Journal of Bacteriology, 2013-12, Vol.195 (24), p.5629-5638 [Peer Reviewed Journal]

Copyright American Society for Microbiology Dec 2013 ;Copyright © 2013, American Society for Microbiology. All Rights Reserved. 2013 American Society for Microbiology ;ISSN: 0021-9193 ;EISSN: 1098-5530 ;EISSN: 1067-8832 ;DOI: 10.1128/JB.00759-13 ;PMID: 24123821 ;CODEN: JOBAAY

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5
Cancer-associated p53 tetramerization domain mutants: quantitative analysis reveals a low threshold for tumor suppressor inactivation
Cancer-associated p53 tetramerization domain mutants: quantitative analysis reveals a low threshold for tumor suppressor inactivation
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Cancer-associated p53 tetramerization domain mutants: quantitative analysis reveals a low threshold for tumor suppressor inactivation

The Journal of biological chemistry, 2011-01, Vol.286 (1), p.252-258 [Peer Reviewed Journal]

2011 by The American Society for Biochemistry and Molecular Biology, Inc. ;ISSN: 0021-9258 ;EISSN: 1083-351X ;DOI: 10.1074/jbc.M110.174698 ;PMID: 20978130

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6
Abstract 1711 VRK1 Mutants and Implications for Kinase Stability and Activity in Neurodegenerative Disorders
Abstract 1711 VRK1 Mutants and Implications for Kinase Stability and Activity in Neurodegenerative Disorders
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Abstract 1711 VRK1 Mutants and Implications for Kinase Stability and Activity in Neurodegenerative Disorders

The Journal of biological chemistry, 2024-03, Vol.300 (3) [Peer Reviewed Journal]

2024 The American Society for Biochemistry and Molecular Biology, Inc. ;ISSN: 0021-9258 ;EISSN: 1083-351X ;DOI: 10.1016/j.jbc.2024.106959

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7
Ion-beam irradiation, gene identification, and marker-assisted breeding in the development of low-cadmium rice
Ion-beam irradiation, gene identification, and marker-assisted breeding in the development of low-cadmium rice
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Ion-beam irradiation, gene identification, and marker-assisted breeding in the development of low-cadmium rice

Proceedings of the National Academy of Sciences - PNAS, 2012-11, Vol.109 (47), p.19166-19171 [Peer Reviewed Journal]

copyright © 1993-2008 National Academy of Sciences of the United States of America ;Copyright National Academy of Sciences Nov 20, 2012 ;ISSN: 0027-8424 ;EISSN: 1091-6490 ;DOI: 10.1073/pnas.1211132109 ;PMID: 23132948

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8
Increased Wingless (Wnt) signaling in pituitary progenitor/stem cells gives rise to pituitary tumors in mice and humans
Increased Wingless (Wnt) signaling in pituitary progenitor/stem cells gives rise to pituitary tumors in mice and humans
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Increased Wingless (Wnt) signaling in pituitary progenitor/stem cells gives rise to pituitary tumors in mice and humans

Proceedings of the National Academy of Sciences - PNAS, 2011-07, Vol.108 (28), p.11482-11487 [Peer Reviewed Journal]

copyright © 1993–2008 National Academy of Sciences of the United States of America ;Copyright National Academy of Sciences Jul 12, 2011 ;ISSN: 0027-8424 ;EISSN: 1091-6490 ;DOI: 10.1073/pnas.1101553108 ;PMID: 21636786

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9
Mutations in mitochondrial histidyl tRNA synthetase HARS2 cause ovarian dysgenesis and sensorineural hearing loss of Perrault syndrome
Mutations in mitochondrial histidyl tRNA synthetase HARS2 cause ovarian dysgenesis and sensorineural hearing loss of Perrault syndrome
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Mutations in mitochondrial histidyl tRNA synthetase HARS2 cause ovarian dysgenesis and sensorineural hearing loss of Perrault syndrome

Proceedings of the National Academy of Sciences - PNAS, 2011-04, Vol.108 (16), p.6543-6548 [Peer Reviewed Journal]

Copyright © 1993-2008 National Academy of Sciences of the United States of America ;Copyright National Academy of Sciences Apr 19, 2011 ;ISSN: 0027-8424 ;EISSN: 1091-6490 ;DOI: 10.1073/pnas.1103471108 ;PMID: 21464306

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10
Picosecond Kinetics of Light Harvesting and Photoprotective Quenching in Wild-Type and Mutant Phycobilisomes Isolated from the Cyanobacterium Synechocystis PCC 6803
Picosecond Kinetics of Light Harvesting and Photoprotective Quenching in Wild-Type and Mutant Phycobilisomes Isolated from the Cyanobacterium Synechocystis PCC 6803
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Picosecond Kinetics of Light Harvesting and Photoprotective Quenching in Wild-Type and Mutant Phycobilisomes Isolated from the Cyanobacterium Synechocystis PCC 6803

Biophysical journal, 2012-04, Vol.102 (7), p.1692-1700 [Peer Reviewed Journal]

2012 Biophysical Society ;Copyright © 2012 Biophysical Society. Published by Elsevier Inc. All rights reserved. ;2012 by the Biophysical Society. 2012 Biophysical Society ;Wageningen University & Research ;ISSN: 0006-3495 ;EISSN: 1542-0086 ;DOI: 10.1016/j.bpj.2012.03.008 ;PMID: 22500770

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11
Lung cancers with acquired resistance to EGFR inhibitors occasionally harbor BRAF gene mutations but lack mutations in KRAS, NRAS, or MEK1
Lung cancers with acquired resistance to EGFR inhibitors occasionally harbor BRAF gene mutations but lack mutations in KRAS, NRAS, or MEK1
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Lung cancers with acquired resistance to EGFR inhibitors occasionally harbor BRAF gene mutations but lack mutations in KRAS, NRAS, or MEK1

Proceedings of the National Academy of Sciences - PNAS, 2012-07, Vol.109 (31), p.E2127-E2133 [Peer Reviewed Journal]

Copyright National Academy of Sciences Jul 31, 2012 ;ISSN: 0027-8424 ;EISSN: 1091-6490 ;DOI: 10.1073/pnas.1203530109 ;PMID: 22773810

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12
T790M mutation in EGFR kinase causes drug resistance by increasing the affinity for ATP
T790M mutation in EGFR kinase causes drug resistance by increasing the affinity for ATP
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T790M mutation in EGFR kinase causes drug resistance by increasing the affinity for ATP

Proceedings of the National Academy of Sciences - PNAS, 2008-02, Vol.105 (6), p.2070-2075 [Peer Reviewed Journal]

Copyright 2008 The National Academy of Sciences of the United States of America ;Copyright National Academy of Sciences Feb 12, 2008 ;2008 by The National Academy of Sciences of the USA ;ISSN: 0027-8424 ;EISSN: 1091-6490 ;DOI: 10.1073/pnas.0709662105 ;PMID: 18227510

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13
Mutant induced pluripotent stem cell lines recapitulate aspects of TDP-43 proteinopathies and reveal cell-specific vulnerability
Mutant induced pluripotent stem cell lines recapitulate aspects of TDP-43 proteinopathies and reveal cell-specific vulnerability
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Mutant induced pluripotent stem cell lines recapitulate aspects of TDP-43 proteinopathies and reveal cell-specific vulnerability

Proceedings of the National Academy of Sciences - PNAS, 2012-04, Vol.109 (15), p.5803-5808 [Peer Reviewed Journal]

copyright © 1993-2008 National Academy of Sciences of the United States of America ;Copyright National Academy of Sciences Apr 10, 2012 ;ISSN: 0027-8424 ;EISSN: 1091-6490 ;DOI: 10.1073/pnas.1202922109 ;PMID: 22451909

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14
Mouse model of Timothy syndrome recapitulates triad of autistic traits
Mouse model of Timothy syndrome recapitulates triad of autistic traits
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Mouse model of Timothy syndrome recapitulates triad of autistic traits

Proceedings of the National Academy of Sciences - PNAS, 2011-09, Vol.108 (37), p.15432-15437 [Peer Reviewed Journal]

Copyright National Academy of Sciences Sep 13, 2011 ;ISSN: 0027-8424 ;EISSN: 1091-6490 ;DOI: 10.1073/pnas.1112667108 ;PMID: 21878566

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15
The origins and genetic interactions of KRAS mutations are allele- and tissue-specific
The origins and genetic interactions of KRAS mutations are allele- and tissue-specific
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The origins and genetic interactions of KRAS mutations are allele- and tissue-specific

Nature communications, 2021-03, Vol.12 (1), p.1808-14, Article 1808 [Peer Reviewed Journal]

The Author(s) 2021. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2021 ;ISSN: 2041-1723 ;EISSN: 2041-1723 ;DOI: 10.1038/s41467-021-22125-z ;PMID: 33753749

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16
Functional analysis of receptor tyrosine kinase mutations in lung cancer identifies oncogenic extracellular domain mutations of ERBB2
Functional analysis of receptor tyrosine kinase mutations in lung cancer identifies oncogenic extracellular domain mutations of ERBB2
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Functional analysis of receptor tyrosine kinase mutations in lung cancer identifies oncogenic extracellular domain mutations of ERBB2

Proceedings of the National Academy of Sciences - PNAS, 2012-09, Vol.109 (36), p.14476-14481 [Peer Reviewed Journal]

copyright © 1993-2008 National Academy of Sciences of the United States of America ;Copyright National Academy of Sciences Sep 4, 2012 ;ISSN: 0027-8424 ;EISSN: 1091-6490 ;DOI: 10.1073/pnas.1203201109 ;PMID: 22908275

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17
KCC2 activity is critical in limiting the onset and severity of status epilepticus
KCC2 activity is critical in limiting the onset and severity of status epilepticus
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KCC2 activity is critical in limiting the onset and severity of status epilepticus

Proceedings of the National Academy of Sciences - PNAS, 2015-03, Vol.112 (11), p.3523-3528 [Peer Reviewed Journal]

Volumes 1–89 and 106–112, copyright as a collective work only; author(s) retains copyright to individual articles ;Copyright National Academy of Sciences Mar 17, 2015 ;ISSN: 0027-8424 ;EISSN: 1091-6490 ;DOI: 10.1073/pnas.1415126112 ;PMID: 25733865

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18
Oncogenic mutations mimic and enhance dynamic events in the natural activation of phosphoinositide 3-kinase p110α (PIK3CA)
Oncogenic mutations mimic and enhance dynamic events in the natural activation of phosphoinositide 3-kinase p110α (PIK3CA)
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Oncogenic mutations mimic and enhance dynamic events in the natural activation of phosphoinositide 3-kinase p110α (PIK3CA)

Proceedings of the National Academy of Sciences - PNAS, 2012-09, Vol.109 (38), p.15259-15264 [Peer Reviewed Journal]

copyright © 1993-2008 National Academy of Sciences of the United States of America ;ISSN: 0027-8424 ;EISSN: 1091-6490 ;DOI: 10.1073/pnas.1205508109 ;PMID: 22949682

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19
In vivo, Pikfyve generates PI(3,5)P₂, which serves as both a signaling lipid and the major precursor for PI5P
In vivo, Pikfyve generates PI(3,5)P₂, which serves as both a signaling lipid and the major precursor for PI5P
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In vivo, Pikfyve generates PI(3,5)P₂, which serves as both a signaling lipid and the major precursor for PI5P

Proceedings of the National Academy of Sciences - PNAS, 2012-10, Vol.109 (43), p.17472-17477 [Peer Reviewed Journal]

copyright © 1993-2008 National Academy of Sciences of the United States of America ;Copyright National Academy of Sciences Oct 23, 2012 ;ISSN: 0027-8424 ;EISSN: 1091-6490 ;DOI: 10.1073/pnas.1203106109 ;PMID: 23047693

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20
Disruption of PH–kinase domain interactions leads to oncogenic activation of AKT in human cancers
Disruption of PH–kinase domain interactions leads to oncogenic activation of AKT in human cancers
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Disruption of PH–kinase domain interactions leads to oncogenic activation of AKT in human cancers

Proceedings of the National Academy of Sciences - PNAS, 2012-11, Vol.109 (47), p.19368-19373 [Peer Reviewed Journal]

copyright © 1993-2008 National Academy of Sciences of the United States of America ;Copyright National Academy of Sciences Nov 20, 2012 ;ISSN: 0027-8424 ;EISSN: 1091-6490 ;DOI: 10.1073/pnas.1204384109 ;PMID: 23134728

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