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1
Blockade of IL-6 Trans signaling attenuates pulmonary fibrosis
Blockade of IL-6 Trans signaling attenuates pulmonary fibrosis
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Blockade of IL-6 Trans signaling attenuates pulmonary fibrosis

The Journal of immunology (1950), 2014-10, Vol.193 (7), p.3755-3768 [Peer Reviewed Journal]

Copyright © 2014 by The American Association of Immunologists, Inc. ;Copyright © 2014 by The American Association of Immunologists, Inc. 2014 Copyright © 2014 by The American Association of Immunologists, Inc. ;ISSN: 0022-1767 ;EISSN: 1550-6606 ;DOI: 10.4049/jimmunol.1302470 ;PMID: 25172494

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2
Emerging role of HMGB1 in fibrotic diseases
Emerging role of HMGB1 in fibrotic diseases
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Emerging role of HMGB1 in fibrotic diseases

Journal of cellular and molecular medicine, 2014-12, Vol.18 (12), p.2331-2339 [Peer Reviewed Journal]

2014 The Authors. Journal of Cellular and Molecular Medicine published by John Wiley & Sons Ltd and Foundation for Cellular and Molecular Medicine. ;Copyright © 2014 John Wiley & Sons Ltd and Foundation for Cellular and Molecular Medicine ;2014 The Authors. Journal of Cellular and Molecular Medicine published by John Wiley & Sons Ltd and Foundation for Cellular and Molecular Medicine. 2014 ;ISSN: 1582-1838 ;EISSN: 1582-4934 ;DOI: 10.1111/jcmm.12419 ;PMID: 25284457

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3
Type V collagen induced tolerance suppresses collagen deposition, TGF-β and associated transcripts in pulmonary fibrosis
Type V collagen induced tolerance suppresses collagen deposition, TGF-β and associated transcripts in pulmonary fibrosis
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Type V collagen induced tolerance suppresses collagen deposition, TGF-β and associated transcripts in pulmonary fibrosis

PloS one, 2013-10, Vol.8 (10), p.e76451 [Peer Reviewed Journal]

2013 Vittal et al. This is an open-access article distributed under the terms of the Creative Commons Attribution License: https://creativecommons.org/licenses/by/4.0/ (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2013 Vittal et al 2013 Vittal et al ;ISSN: 1932-6203 ;EISSN: 1932-6203 ;DOI: 10.1371/journal.pone.0076451 ;PMID: 24204629

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4
A functional CFTR assay using primary cystic fibrosis intestinal organoids
A functional CFTR assay using primary cystic fibrosis intestinal organoids
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A functional CFTR assay using primary cystic fibrosis intestinal organoids

Nature medicine, 2013-07, Vol.19 (7), p.939-945 [Peer Reviewed Journal]

COPYRIGHT 2013 Nature Publishing Group ;COPYRIGHT 2013 Nature Publishing Group ;Copyright Nature Publishing Group Jul 2013 ;ISSN: 1078-8956 ;EISSN: 1546-170X ;DOI: 10.1038/nm.3201 ;PMID: 23727931

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5
Pulmonary fibrosis: patterns and perpetrators
Pulmonary fibrosis: patterns and perpetrators
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Pulmonary fibrosis: patterns and perpetrators

The Journal of clinical investigation, 2012-08, Vol.122 (8), p.2756-2762 [Peer Reviewed Journal]

COPYRIGHT 2012 American Society for Clinical Investigation ;COPYRIGHT 2012 American Society for Clinical Investigation ;Copyright American Society for Clinical Investigation Aug 2012 ;Copyright © 2012, American Society for Clinical Investigation 2012 ;ISSN: 0021-9738 ;EISSN: 1558-8238 ;DOI: 10.1172/jci60323 ;PMID: 22850886

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6
Severe lung fibrosis requires an invasive fibroblast phenotype regulated by hyaluronan and CD44
Severe lung fibrosis requires an invasive fibroblast phenotype regulated by hyaluronan and CD44
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Severe lung fibrosis requires an invasive fibroblast phenotype regulated by hyaluronan and CD44

The Journal of experimental medicine, 2011-07, Vol.208 (7), p.1459-1471 [Peer Reviewed Journal]

2011 Li et al. 2011 ;ISSN: 0022-1007 ;EISSN: 1540-9538 ;DOI: 10.1084/jem.20102510 ;PMID: 21708929

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7
Origins of Cystic Fibrosis Lung Disease
Origins of Cystic Fibrosis Lung Disease
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Origins of Cystic Fibrosis Lung Disease

The New England journal of medicine, 2015-01, Vol.372 (4), p.351-362 [Peer Reviewed Journal]

Copyright © 2015 Massachusetts Medical Society. All rights reserved. ;ISSN: 0028-4793 ;EISSN: 1533-4406 ;DOI: 10.1056/NEJMra1300109 ;PMID: 25607428

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8
Capitalizing on the heterogeneous effects of CFTR nonsense and frameshift variants to inform therapeutic strategy for cystic fibrosis
Capitalizing on the heterogeneous effects of CFTR nonsense and frameshift variants to inform therapeutic strategy for cystic fibrosis
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Capitalizing on the heterogeneous effects of CFTR nonsense and frameshift variants to inform therapeutic strategy for cystic fibrosis

PLoS genetics, 2018-11, Vol.14 (11), p.e1007723-e1007723 [Peer Reviewed Journal]

COPYRIGHT 2018 Public Library of Science ;COPYRIGHT 2018 Public Library of Science ;2018 Sharma et al. This is an open access article distributed under the terms of the Creative Commons Attribution License: http://creativecommons.org/licenses/by/4.0/ (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;2018 Sharma et al 2018 Sharma et al ;ISSN: 1553-7404 ;ISSN: 1553-7390 ;EISSN: 1553-7404 ;DOI: 10.1371/journal.pgen.1007723 ;PMID: 30444886

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9
Matrix metalloproteinases as therapeutic targets for idiopathic pulmonary fibrosis
Matrix metalloproteinases as therapeutic targets for idiopathic pulmonary fibrosis
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Matrix metalloproteinases as therapeutic targets for idiopathic pulmonary fibrosis

American journal of respiratory cell and molecular biology, 2015-11, Vol.53 (5), p.585-600 [Peer Reviewed Journal]

Copyright American Thoracic Society Nov 2015 ;Copyright © 2015 by the American Thoracic Society 2015 ;ISSN: 1044-1549 ;EISSN: 1535-4989 ;DOI: 10.1165/rcmb.2015-0020tr ;PMID: 26121236

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10
Hyaluronan and TLR4 promote surfactant-protein-C-positive alveolar progenitor cell renewal and prevent severe pulmonary fibrosis in mice
Hyaluronan and TLR4 promote surfactant-protein-C-positive alveolar progenitor cell renewal and prevent severe pulmonary fibrosis in mice
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Hyaluronan and TLR4 promote surfactant-protein-C-positive alveolar progenitor cell renewal and prevent severe pulmonary fibrosis in mice

Nature medicine, 2016-11, Vol.22 (11), p.1285-1293 [Peer Reviewed Journal]

COPYRIGHT 2016 Nature Publishing Group ;COPYRIGHT 2016 Nature Publishing Group ;Copyright Nature Publishing Group Nov 2016 ;ISSN: 1078-8956 ;EISSN: 1546-170X ;DOI: 10.1038/nm.4192 ;PMID: 27694932

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11
Reduced airway surface pH impairs bacterial killing in the porcine cystic fibrosis lung
Reduced airway surface pH impairs bacterial killing in the porcine cystic fibrosis lung
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Reduced airway surface pH impairs bacterial killing in the porcine cystic fibrosis lung

Nature (London), 2012-07, Vol.487 (7405), p.109-113 [Peer Reviewed Journal]

2015 INIST-CNRS ;COPYRIGHT 2012 Nature Publishing Group ;COPYRIGHT 2012 Nature Publishing Group ;Copyright Nature Publishing Group Jul 5, 2012 ;ISSN: 0028-0836 ;EISSN: 1476-4687 ;DOI: 10.1038/nature11130 ;PMID: 22763554 ;CODEN: NATUAS

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12
Blockade of CCL24 with a monoclonal antibody ameliorates experimental dermal and pulmonary fibrosis
Blockade of CCL24 with a monoclonal antibody ameliorates experimental dermal and pulmonary fibrosis
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Blockade of CCL24 with a monoclonal antibody ameliorates experimental dermal and pulmonary fibrosis

Annals of the rheumatic diseases, 2019-09, Vol.78 (9), p.1260-1268 [Peer Reviewed Journal]

Author(s) (or their employer(s)) 2019. Re-use permitted under CC BY-NC. No commercial re-use. See rights and permissions. Published by BMJ. ;2019 Author(s) (or their employer(s)) 2019. Re-use permitted under CC BY-NC. No commercial re-use. See rights and permissions. Published by BMJ. This is an open access article distributed in accordance with the Creative Commons Attribution Non Commercial (CC BY-NC 4.0) license, which permits others to distribute, remix, adapt, build upon this work non-commercially, and license their derivative works on different terms, provided the original work is properly cited, appropriate credit is given, any changes made indicated, and the use is non-commercial. See: http://creativecommons.org/licenses/by-nc/4.0/ . Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;Author(s) (or their employer(s)) 2019. Re-use permitted under CC BY-NC. No commercial re-use. See rights and permissions. Published by BMJ. 2019 ;ISSN: 0003-4967 ;EISSN: 1468-2060 ;DOI: 10.1136/annrheumdis-2019-215119 ;PMID: 31129606

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13
Inhalation of lung spheroid cell secretome and exosomes promotes lung repair in pulmonary fibrosis
Inhalation of lung spheroid cell secretome and exosomes promotes lung repair in pulmonary fibrosis
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Inhalation of lung spheroid cell secretome and exosomes promotes lung repair in pulmonary fibrosis

Nature communications, 2020-02, Vol.11 (1), p.1064-14, Article 1064 [Peer Reviewed Journal]

This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License. ;The Author(s) 2020 ;ISSN: 2041-1723 ;EISSN: 2041-1723 ;DOI: 10.1038/s41467-020-14344-7 ;PMID: 32111836

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14
Regulation of Transforming Growth Factor-β1-driven Lung Fibrosis by Galectin-3
Regulation of Transforming Growth Factor-β1-driven Lung Fibrosis by Galectin-3
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Regulation of Transforming Growth Factor-β1-driven Lung Fibrosis by Galectin-3

American journal of respiratory and critical care medicine, 2012-03, Vol.185 (5), p.537-546 [Peer Reviewed Journal]

2015 INIST-CNRS ;Copyright © 2012 by the American Thoracic Society 2012 ;ISSN: 1073-449X ;EISSN: 1535-4970 ;DOI: 10.1164/rccm.201106-0965oc ;PMID: 22095546

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15
miR-21 mediates fibrogenic activation of pulmonary fibroblasts and lung fibrosis
miR-21 mediates fibrogenic activation of pulmonary fibroblasts and lung fibrosis
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Article 		Add to My Research

miR-21 mediates fibrogenic activation of pulmonary fibroblasts and lung fibrosis

The Journal of experimental medicine, 2010-08, Vol.207 (8), p.1589-1597 [Peer Reviewed Journal]

2010 Liu et al. 2010 ;ISSN: 0022-1007 ;EISSN: 1540-9538 ;DOI: 10.1084/jem.20100035 ;PMID: 20643828

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16
Curcumin regulates pulmonary extracellular matrix remodeling and mitochondrial function to attenuate pulmonary fibrosis by regulating the miR-29a-3p/DNMT3A axis
Curcumin regulates pulmonary extracellular matrix remodeling and mitochondrial function to attenuate pulmonary fibrosis by regulating the miR-29a-3p/DNMT3A axis
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Curcumin regulates pulmonary extracellular matrix remodeling and mitochondrial function to attenuate pulmonary fibrosis by regulating the miR-29a-3p/DNMT3A axis

Biomedicine & pharmacotherapy, 2024-05, Vol.174, p.116572-116572, Article 116572 [Peer Reviewed Journal]

2024 The Authors ;Copyright © 2024 The Authors. Published by Elsevier Masson SAS.. All rights reserved. ;ISSN: 0753-3322 ;EISSN: 1950-6007 ;DOI: 10.1016/j.biopha.2024.116572 ;PMID: 38626519

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17
The porcine lung as a potential model for cystic fibrosis
The porcine lung as a potential model for cystic fibrosis
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The porcine lung as a potential model for cystic fibrosis

American journal of physiology. Lung cellular and molecular physiology, 2008-08, Vol.295 (2), p.L240-L263 [Peer Reviewed Journal]

Copyright © 2008, American Physiological Society ;ISSN: 1040-0605 ;EISSN: 1522-1504 ;DOI: 10.1152/ajplung.90203.2008 ;PMID: 18487356

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18
SLC15A3 plays a crucial role in pulmonary fibrosis by regulating macrophage oxidative stress
SLC15A3 plays a crucial role in pulmonary fibrosis by regulating macrophage oxidative stress
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SLC15A3 plays a crucial role in pulmonary fibrosis by regulating macrophage oxidative stress

Cell death and differentiation, 2024-04, Vol.31 (4), p.417-430 [Peer Reviewed Journal]

2024. The Author(s), under exclusive licence to ADMC Associazione Differenziamento e Morte Cellulare. ;The Author(s), under exclusive licence to ADMC Associazione Differenziamento e Morte Cellulare 2024. Springer Nature or its licensor (e.g. a society or other partner) holds exclusive rights to this article under a publishing agreement with the author(s) or other rightsholder(s); author self-archiving of the accepted manuscript version of this article is solely governed by the terms of such publishing agreement and applicable law. ;ISSN: 1350-9047 ;EISSN: 1476-5403 ;DOI: 10.1038/s41418-024-01266-w ;PMID: 38374230

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19
Human pluripotent stem cell-derived acinar/ductal organoids generate human pancreas upon orthotopic transplantation and allow disease modelling
Human pluripotent stem cell-derived acinar/ductal organoids generate human pancreas upon orthotopic transplantation and allow disease modelling
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Human pluripotent stem cell-derived acinar/ductal organoids generate human pancreas upon orthotopic transplantation and allow disease modelling

Gut, 2017-03, Vol.66 (3), p.473-486 [Peer Reviewed Journal]

Published by the BMJ Publishing Group Limited. For permission to use (where not already granted under a licence) please go to http://www.bmj.com/company/products-services/rights-and-licensing ;Published by the BMJ Publishing Group Limited. For permission to use (where not already granted under a licence) please go to http://www.bmj.com/company/products-services/rights-and-licensing/. ;Copyright: 2016 Published by the BMJ Publishing Group Limited. For permission to use (where not already granted under a licence) please go to http://www.bmj.com/company/products-services/rights-and-licensing ;Published by the BMJ Publishing Group Limited. For permission to use (where not already granted under a licence) please go to http://www.bmj.com/company/products-services/rights-and-licensing/ 2016 ;ISSN: 0017-5749 ;EISSN: 1468-3288 ;DOI: 10.1136/gutjnl-2016-312423 ;PMID: 27633923 ;CODEN: GUTTAK

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20
IL20Rb aggravates pulmonary fibrosis through enhancing bone marrow derived profibrotic macrophage activation
IL20Rb aggravates pulmonary fibrosis through enhancing bone marrow derived profibrotic macrophage activation
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IL20Rb aggravates pulmonary fibrosis through enhancing bone marrow derived profibrotic macrophage activation

Pharmacological research, 2024-05, Vol.203, p.107178 [Peer Reviewed Journal]

EISSN: 1096-1186 ;DOI: 10.1016/j.phrs.2024.107178

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